Syariah Governance dan Maqashid Syariah di Perbankan Syariah di Indonesia

Latar Belakang: Pesatnya perkembangan perbankan syariah di Indonesia, baik itu dalam hal peningkatan total aset yang di miliki dan jumlah bank yang mengalami kenaikan membuat bank syariah harus selalu melakukan evaluasi terkait kinerja operasional perbankan syariah.Tujuan: Penelitian ini bertujuan untuk mengkaji pengaruh mekanisme syariah governance terhadap kinerja maqashid syariah menggunakan pengukuran Integrated Maqashid Al-Sharīʿah Based Performance Measure (IMSPM).Metode Penelitian: Pada penelitian ini menggunakan data sekunder yang bersifat kuantitatif yang berasal dari laporan keuangan dan laporan good corporate governance yang telah diterbitkan oleh masing-masing Bank Umum Syariah di Indonesia selama tahun 2015 hingga tahun 2020. Teknik pengambilan sampel pada penelitian ini menggunakan metode purposive sampling. Alat analisis penelitian ini menggunakan software SPSS versi 22. Alat analisis yang digunakan adalah analisis regresi berganda.Hasil Penelitian: Hasil penelitian bahwa jumlah dewan komisaris, jumlah komite audit, jumlah dewan direksi, jumlah dewan pengawas syariah, kepemilikan institusional dan umur perusahaan syariah tidak berpengaruh terhadap kinerja maqashid syariah.Keaslian/Kebaruan Penelitian: Penelitian mengenai tata kelola dan kinerja perbankan syariah dengan menggunakan tolok ukur maqasid syariah index telah banyak dilakukan oleh peneliti terdahulu. Pengukuran kinerja menggunakan maqasid syariah index dianggap kurang efektif karena hanya mencakup aspek keagamaan saja tanpa memperhitungkan apek keuangan. Oleh sebab itu, penelitian ini menggunakan pengukuran terbaru yang lebih luas untuk mengukur penerapan syariah governance terhadap kinerja maqasid syariah menggunakan konsep Integrated Maqashid Al-Shari’ah Performance Measure (IMSPM)

On the Mechanism of Time--Delayed Feedback Control

The Pyragas method for controlling chaos is investigated in detail from the experimental as well as theoretical point of view. We show by an analytical stability analysis that the revolution around an unstable periodic orbit governs the success of the control scheme. Our predictions concerning the transient behaviour of the control signal are confirmed by numerical simulations and an electronic circuit experiment.Comment: 4 pages, REVTeX, 4 eps-figures included Phys. Rev. Lett., in press also available at http://athene.fkp.physik.th-darmstadt.de/public/wolfram.htm

Generation of Large-Scale Vorticity in a Homogeneous Turbulence with a Mean Velocity Shear

An effect of a mean velocity shear on a turbulence and on the effective force which is determined by the gradient of Reynolds stresses is studied. Generation of a mean vorticity in a homogeneous incompressible turbulent flow with an imposed mean velocity shear due to an excitation of a large-scale instability is found. The instability is caused by a combined effect of the large-scale shear motions (''skew-induced" deflection of equilibrium mean vorticity) and ''Reynolds stress-induced" generation of perturbations of mean vorticity. Spatial characteristics, such as the minimum size of the growing perturbations and the size of perturbations with the maximum growth rate, are determined. This instability and the dynamics of the mean vorticity are associated with the Prandtl's turbulent secondary flows. This instability is similar to the mean-field magnetic dynamo instability. Astrophysical applications of the obtained results are discussed.Comment: 8 pages, 3 figures, REVTEX4, submitted to Phys. Rev.

Southern Infrared Proper Motion Survey II: A sample of low mass stars with \mu > 0.1''/yr

We present details of the second part of the Southern Infrared Proper Motion Survey (SIPS). Here accurate relative astrometry allows us to reduce the minimum proper motion to 0.1 arcseconds per year. This yields 6904 objects with proper motions between our minum cut and half an arcsecond a year. A small overspill sample with proper motions greater than this is also included. We examine our sample to identify interesting individual objects such as common proper motion binaries, potential L dwarfs and candidate nearby stars. Finally we show our survey is incomplete due to many factors, factors which we will take into account when simulating these survey results in the next paper in this series.Comment: Submitted to A&A, Tables 7 and 8 available electronically at http://www.astro.ru.nl/~ndeacon/SIPSII.htm

Cellular mechanisms of somatostatin action in the gut

We have used isolated canine parietal cells to examine the receptor and postreceptor events mediating the inhibitory effects of somatostatin on acid secretion. Somatostatin-14 (S14) and somatostatin-28 (S28) dose dependently inhibited parietal cells stimulated by secretagogues that activate both the adenylate cyclase/cyclic adenosine monophosphate and the inositol phospholipid/protein kinase C cascades. The inhibitory action was mediated via a specific cell surface receptor that consists of a single subunit protein (molecular weight 99,000 d). This receptor recognized S14 and S28 equally well. Somatostatin inhibited parietal cell activity via mechanisms that are both dependent on and independent of a pertussis toxin-sensitive inhibitory guanine nucleotide binding protein.Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/28402/1/0000177.pd

Biochemistry and physiology of gastrointestinal somatostatin

Somatostatin, a tetradecapeptide initially isolated from the ovine hypothalamus, is widely distributed throughout the gastrointestinal tract where it may act as a hormone, local chemical messenger, or neurotransmitter to elicit many physiological actions. Release of somatostatin from D cells in the gut is regulated by mechanisms that are both dependent on and independent of cAMP. In most cases somatostatin acts to inhibit the function of its target cells. It performs this action in part via pertussis-toxin-sensitive inhibitory guanine nucleotide-binding proteins that regulate adenylate cyclase activity. Other mechanisms may involve sites of action distal to intracellular second messenger systems .Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/44411/1/10620_2005_Article_BF01536041.pd