27 research outputs found
Bisphosphonates as antimyeloma drugs
- Author
- A Aviles
- A Corso
- A Corso
- A Guenther
- AJ Roelofs
- AU Ural
- C Baulch-Brown
- C Scavelli
- CM Shipman
- CM Shipman
- D Chauhan
- D Ribatti
- D Ribatti
- D Santini
- EV McCloskey
- EV McCloskey
- F Fiore
- GJ Morgan
- H Fleisch
- H Monkkonen
- HG Drexler
- J Raikkonen
- JR Berenson
- JR Berenson
- K Sato
- KL Kavanagh
- M Coscia
- M Kellinsalmi
- M Moschetta
- MA Lawson
- ME Puhaindran
- MS Raab
- MT Drake
- N Giuliani
- P Clezardin
- P Musto
- P Tassone
- PI Croucher
- R Casetti
- R Schmidmaier
- RG Russell
- S Kumar
- SP Luckman
- SP Luckman
- T Hideshima
- T Yuasa
- V Kunzmann
- WM Kuehl
- Y Abe
- Publication venue
- Nature Publishing Group
- Publication date
- 01/01/2011
- Field of study
In patients with symptomatic multiple myeloma (MM), bisphosphonate (BP) treatment has been widely used to prevent bone loss and preserve skeletal health because of its proven effects on inhibiting osteoclast-mediated bone resorption. In addition to their effects on osteoclasts, it is becoming increasingly evident that BPs may have additional effects on the bone microenvironment and cells other than osteoclasts that may potentially inhibit the development and progression of MM. This review focuses on the pathophysiology of MM with an emphasis on the events that drive MM progression within the bone and the mechanisms by which BPs may inhibit specific processes. The underlying molecular mechanisms that drive the modulation of cellular fate and function and consequent physiological outcomes are described. Direct effects on myeloma cell growth and survival and the interactions between myeloma cells and the bone microenvironment are discussed. Clinical evidence of the antimyeloma effects of BPs is emerging and is also reviewed
Effect of Ketoprofen on acute phase protein concentrations in goats undergoing castration
- Author
- A Dowling
- A Pfeffer
- AD Fisher
- AD Fisher
- B Earley
- C Cray
- C Ganheim
- D Werling
- DB Faulkner
- F Ceciliani
- FHD Gonzalez
- FHD González
- Gamze Sevri Ekren Asici
- GM Friton
- GR Duncanson
- H Murata
- H Murata
- HA Ravin
- Hasan Erdogan
- Ibrahim Akin
- J Kaba
- KA Mathews
- Kerem Ural
- LJ Dawson
- M Hashemnia
- MM Benjamin
- MM Rahman
- MS Bergh
- N Katoh
- N Katoh
- NU Horadagodo
- PA Ulutas
- PD Eckersall
- PD Eckersall
- PD Eckersall
- PK Saini
- PMH Heegaard
- S Nazifi
- SL Curry
- ST Ting
- STL Ting
- Umit Karademir
- V Molony
- WM El-Deeb
- WY Pang
- WY Pang
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Effects of alirocumab on types of myocardial infarction: insights from the ODYSSEY OUTCOMES trial
- Author
- Abbas J
- Abbate A
- Abdullakutty J
- Abhyanakar AD
- Aboyans V
- Abrantes JAM
- Abu-Fadel M
- Acevedo M
- Adamkova V
- Adhyapak S
- Agarwal H
- Aggarwal R
- Aggarwal RK
- Aguirre A
- Ahmed H
- Ahremark U
- Ahuad Guerrero RA
- Aidar Ayoub JC
- Airaksinen JK
- Aitken D
- Akatova E
- Akhter F
- Ako J
- Akright L
- Akyea-Djamson A
- Al Joundi T
- Al-Windy NYY
- Alan D
- Alcocer Gamba MA
- Alexander JH
- Alexander K
- Alexandru TM
- Alings M
- Alonso Martin JJ
- Alvarisqueta AF
- Alves De Lima AE
- Amarasekera S
- Amarasena N
- Amir O
- Amlani M
- Amosova E
- Amuchastegui M
- Andersen K
- Andersen R
- Anderson J
- Ando K
- Angel Hominal M
- Annam S
- Anscombe R
- Apostolovic SR
- Appel K-F
- Arandjelovic A
- Araneda G
- Arango Benecke JL
- Arbel Y
- Arif I
- Armaganijan L
- Arroyo JAC
- Arstall MA
- Asanin MR
- Atar S
- Athyros V
- Auguadro C
- Ayala CAC
- Aylward PE
- Azizad MM
- Bagai A
- Bagriy A
- Bahit MC
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- Balaguer Recena J
- Baldari D
- Balinovac J
- Ball E
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- Banuru S
- Barakovic F
- Baranov E
- Baranova E
- Barbarash OL
- Barbato E
- Barna OM
- Barone-Rochette G
- Barr C
- Barrucco R
- Bartels GL
- Bashir R
- Basson MMDV
- Bastos JM
- Bata I
- Batushkin V
- Bayat J
- Bayram Llamas EA
- Bayron C
- Beauloye C
- Bednarski J
- Behrens S
- Belhassane A
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- Berger R
- Berglund S
- Berk M
- Berland J
- Berli MA
- Berlowitz M
- Berman B
- Bernan A
- Berti S
- Bhagwat A
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- Bhatt DL
- Binder R
- Birchem J
- Bittner VA
- Blicharski T
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- Botelho A
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- Botia DCL
- Bottcher M
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- Breedveld RW
- Brennan JM
- Brodison A
- Bronzwaer PNA
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- Budaj A
- Bugan V
- Busmane A
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- Camsari A
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- Carrillo Calvillo J
- Carroll PA
- Cartasegna LR
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- Chandna H
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- Chen D
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- Chen Y
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- Commerford PJ
- Concha YMR
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- Constantine G
- Constantinescu MCA
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- Davis W
- De Berrazueta Fernandez JR
- De Cesare NB
- De Leo A
- De Luca G
- De Mora Martin M
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- Edelberg JM
- Edes I
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- Eisenberg S
- Ekanayaka R
- El Shahawy M
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- Elias M
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- Faes D
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- Fairlamb J
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- Falukozy J
- Fang C-Y
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- Fanghanel Salmon G
- Farhat N
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- Fausto Ovando SR
- Fazekas F
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- Feitosa GS
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- Fernandes Manenti ERF
- Fernandez Portales J
- Fernandez AM
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- Ferrolino A
- Filardi PP
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- Fishberg R
- Fisher D
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- Florenzano F
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- French WJ
- Friedman D
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- Fulop P
- Fulwani MC
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- Garcia Duran RO
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- Gardner G
- Garrahy PJ
- Garrido M
- Garza Ruiz JA
- Gavish D
- Ge J
- Gelormini J
- Gerber J
- Gerber R
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- Giesler G
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- Gilbert JM
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- Giordano JA
- Giorgeto FE
- Gislason G
- Giuliano ME
- Gniot J
- Goch A
- Goloborodko B
- Gomez Vilamajo OA
- Gonsorcik J
- Gonzales RJV
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- Gonzalez Salas LG
- Gonzalez-Juanatey C
- Gonzalez-Juanatey JR
- Goodman SG
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- Gordeev I
- Gordienko A
- Gorny J
- Gosselin G
- Gosselink ATM
- Gotcheva NN
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- Groenemeijer BE
- Groutars RGEJ
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- Hansen O
- Haraguchi T
- Harding S
- Harkut P
- Harrington RA
- Harris B
- Harris B
- Hart H
- Hata Y
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- Hedman A
- Heidenreich L
- Heinc P
- Heitzer T
- Hemetsberger R
- Henderson D
- Henkin Y
- Herath JI
- Herazo AS
- Hermans K
- Hermans WRM
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- Herrman JPR
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- Hess CN
- Higashikata T
- Higuera JD
- Hii CLS
- Hirayama A
- Hirooka K
- Hlatky MA
- Hoag G
- Hodes Z
- Hoffer E
- Hoffman D
- Hong T
- Hoogslag PAM
- Hoppe U
- Horak D
- Horowitz J
- Horvath I
- Horwitz P
- Hove JD
- Hrabar AD
- Hranai M
- Hsieh I-C
- Huang L
- Huang S
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- Huang W
- Huber K
- Huidobro L
- Huikuri H
- Hunter J
- Hurtig U
- Hussein O
- Huynh T
- Hwang J-J
- II TC
- III DG
- III KG
- Ilic S
- Ilieva-Pandeva KA
- Ince C
- Iniguez A
- Investigators ODYSSEYOUTCOMES
- Iqbal SMR
- Irimpen A
- Istratoaie O
- Ito K
- Ivanov IG
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- Ivanovic N
- Iyengar SS
- Izzo M
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- Jafar Z
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- Jaramillo N
- Jatin FGM
- Jaworska K
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- Jeppesen J
- Jeserich M
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- Jintapakorn W
- Johansen PB
- Jonas M
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- Jortveit J
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- Kadr H
- Kaewsuwanna P
- Kahali D
- Kaikkonen K
- Kaiser S
- Kalashetti S
- Kalil R
- Kamensky G
- Kamiya H
- Kandasamy B
- Kapin T
- Kaplan R
- Kapp IE
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- Katsuda Y
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- Kedev S
- Keen W
- Kehasukcharoen W
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- Knickelbine T
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- Kosinski E
- Kosmachova E
- Kostarska-Srokosz E
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- Kristensen KS
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- Zuniga JDH
- Zurakowski A
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- Zweiker R
- Publication venue
- 'Oxford University Press (OUP)'
- Publication date
- 01/01/2019
- Field of study
Aims The third Universal Definition of Myocardial Infarction (MI) Task Force classified MIs into five types: Type 1, spontaneous; Type 2, related to oxygen supply/demand imbalance; Type 3, fatal without ascertainment of cardiac biomarkers; Type 4, related to percutaneous coronary intervention; and Type 5, related to coronary artery bypass surgery. Low-density lipoprotein cholesterol (LDL-C) reduction with statins and proprotein convertase subtilisin–kexin Type 9 (PCSK9) inhibitors reduces risk of MI, but less is known about effects on types of MI. ODYSSEY OUTCOMES compared the PCSK9 inhibitor alirocumab with placebo in 18 924 patients with recent acute coronary syndrome (ACS) and elevated LDL-C (≥1.8 mmol/L) despite intensive statin therapy. In a pre-specified analysis, we assessed the effects of alirocumab on types of MI. Methods and results Median follow-up was 2.8 years. Myocardial infarction types were prospectively adjudicated and classified. Of 1860 total MIs, 1223 (65.8%) were adjudicated as Type 1, 386 (20.8%) as Type 2, and 244 (13.1%) as Type 4. Few events were Type 3 (n = 2) or Type 5 (n = 5). Alirocumab reduced first MIs [hazard ratio (HR) 0.85, 95% confidence interval (CI) 0.77–0.95; P = 0.003], with reductions in both Type 1 (HR 0.87, 95% CI 0.77–0.99; P = 0.032) and Type 2 (0.77, 0.61–0.97; P = 0.025), but not Type 4 MI. Conclusion After ACS, alirocumab added to intensive statin therapy favourably impacted on Type 1 and 2 MIs. The data indicate for the first time that a lipid-lowering therapy can attenuate the risk of Type 2 MI. Low-density lipoprotein cholesterol reduction below levels achievable with statins is an effective preventive strategy for both MI types.For complete list of authors see http://dx.doi.org/10.1093/eurheartj/ehz299</p
International Consensus Statement on Rhinology and Allergy: Rhinosinusitis
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- Passali D
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- Patel NA
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- Peng Y
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- Perloff JR
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- Ramakrishnan V
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- Rotenberg BW
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- Schwarzbach HL
- Schwitzguébel AJ‐P
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- Sedaghat AR
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- Seiberling K
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- Sella GCP
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- Senior BA
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- Senturk M
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- Snidvongs K
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- Zuskin E
- Čulig J
- Publication venue
- 'Wiley'
- Publication date
- 01/01/2021
- Field of study
Background: The 5 years since the publication of the first International Consensus Statement on Allergy and Rhinology: Rhinosinusitis (ICAR‐RS) has witnessed foundational progress in our understanding and treatment of rhinologic disease. These advances are reflected within the more than 40 new topics covered within the ICAR‐RS‐2021 as well as updates to the original 140 topics. This executive summary consolidates the evidence‐based findings of the document. Methods: ICAR‐RS presents over 180 topics in the forms of evidence‐based reviews with recommendations (EBRRs), evidence‐based reviews, and literature reviews. The highest grade structured recommendations of the EBRR sections are summarized in this executive summary. Results: ICAR‐RS‐2021 covers 22 topics regarding the medical management of RS, which are grade A/B and are presented in the executive summary. Additionally, 4 topics regarding the surgical management of RS are grade A/B and are presented in the executive summary. Finally, a comprehensive evidence‐based management algorithm is provided. Conclusion: This ICAR‐RS‐2021 executive summary provides a compilation of the evidence‐based recommendations for medical and surgical treatment of the most common forms of RS
Effect of alirocumab on mortality after acute coronary syndromes. An analysis of the ODYSSEY OUTCOMES randomized clinical trial
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- Yakushin S
- Yan AT-K
- Yan BPY
- Yanez M
- Yang E
- Yang X
- Yang X
- Yao Z
- Yazdani S
- Yerra SK
- Yin W-H
- Yong H
- Yoon JH
- Younis L
- Yu Y
- Yuan Z
- Yusoff K
- Yusoff K
- Zahger D
- Zaidman CJ
- Zainea M
- Zaman A
- Zapata G
- Zdravko B
- Zea Nunez CA
- Zeiher AM
- Zeiher AM
- Zhang R
- Zhang W
- Zhang X
- Zhang Z
- Zhao X
- Zheleznyy V
- Zheng Y
- Zheng Z
- Zhou Y
- Zhu C
- Zhu H
- Zhu J
- Zirlik A
- Zizka J
- Zobouyan I
- Zong W
- Zrazhevsky K
- Zurakowski A
- Zuran I
- Zweiker R
- Publication venue
- 'Ovid Technologies (Wolters Kluwer Health)'
- Publication date
- 04/01/2019
- Field of study
Background: Previous trials of PCSK9 (proprotein convertase subtilisin-kexin type 9) inhibitors demonstrated reductions in major adverse cardiovascular events, but not death. We assessed the effects of alirocumab on death after index acute coronary syndrome. Methods: ODYSSEY OUTCOMES (Evaluation of Cardiovascular Outcomes After an Acute Coronary Syndrome During Treatment With Alirocumab) was a double-blind, randomized comparison of alirocumab or placebo in 18 924 patients who had an ACS 1 to 12 months previously and elevated atherogenic lipoproteins despite intensive statin therapy. Alirocumab dose was blindly titrated to target achieved low-density lipoprotein cholesterol (LDL-C) between 25 and 50 mg/dL. We examined the effects of treatment on all-cause death and its components, cardiovascular and noncardiovascular death, with log-rank testing. Joint semiparametric models tested associations between nonfatal cardiovascular events and cardiovascular or noncardiovascular death. Results: Median follow-up was 2.8 years. Death occurred in 334 (3.5%) and 392 (4.1%) patients, respectively, in the alirocumab and placebo groups (hazard ratio [HR], 0.85; 95% CI, 0.73 to 0.98; P=0.03, nominal P value). This resulted from nonsignificantly fewer cardiovascular (240 [2.5%] vs 271 [2.9%]; HR, 0.88; 95% CI, 0.74 to 1.05; P=0.15) and noncardiovascular (94 [1.0%] vs 121 [1.3%]; HR, 0.77; 95% CI, 0.59 to 1.01; P=0.06) deaths with alirocumab. In a prespecified analysis of 8242 patients eligible for ≥3 years follow-up, alirocumab reduced death (HR, 0.78; 95% CI, 0.65 to 0.94; P=0.01). Patients with nonfatal cardiovascular events were at increased risk for cardiovascular and noncardiovascular deaths (P<0.0001 for the associations). Alirocumab reduced total nonfatal cardiovascular events (P<0.001) and thereby may have attenuated the number of cardiovascular and noncardiovascular deaths. A post hoc analysis found that, compared to patients with lower LDL-C, patients with baseline LDL-C ≥100 mg/dL (2.59 mmol/L) had a greater absolute risk of death and a larger mortality benefit from alirocumab (HR, 0.71; 95% CI, 0.56 to 0.90; Pinteraction=0.007). In the alirocumab group, all-cause death declined wit h achieved LDL-C at 4 months of treatment, to a level of approximately 30 mg/dL (adjusted P=0.017 for linear trend). Conclusions: Alirocumab added to intensive statin therapy has the potential to reduce death after acute coronary syndrome, particularly if treatment is maintained for ≥3 years, if baseline LDL-C is ≥100 mg/dL, or if achieved LDL-C is low. Clinical Trial Registration: URL: https://www.clinicaltrials.gov. Unique identifier: NCT01663402
Calcium orthophosphate-based biocomposites and hybrid biomaterials
- Author
- A Baji
- A Balamurugan
- A Bigi
- A Bigi
- A Bigi
- A Bigi
- A Bigi
- A Bigi
- A Borzacchiello
- A Cavalcanti
- A Chiba
- A Dorner-Reisel
- A Dunn
- A Geçer
- A John
- A Korematsu
- A Korematsu
- A Krout
- A Martino Di
- A Mathijsen
- A Matsuda
- A Nakahira
- A Oyane
- A Oyane
- A Pandey
- A Plachokova
- A Rapacz-Kmita
- A Rapacz-Kmita
- A Rapacz-Kmita
- A Rovira
- A Scabbia
- A Sinha
- A Sinha
- A Slosarcyk
- A Sogal
- A Sugawara
- A Svensson
- A Tampieri
- A Wittkampf
- A Yokoyama
- A Yoshida
- AA Ignatius
- AA Ignatius
- AA Ignatius
- AA White
- AC Lawson
- AC Tas
- ACA Wan
- ACA Wan
- AG Coombes
- AG Pinheiro
- AJ Ruys
- AK Lynn
- AK Mohanty
- AL Boskey
- AL Daniel-da-Silva
- AL Oliveira
- AM Moursi
- AMP Dupraz
- AMP Dupraz
- AP Marques
- AP Marques
- AP Unwin
- AR Boccaccini
- AR Boyd
- AS Greenwald
- AU Daniels
- B Boyan
- B Chen
- B Rai
- B Rai
- B Rai
- B Sreedhar
- B Szaraniec
- B Zhao
- BH Yoon
- BJ Meenan
- BL Seal
- BR Kumar
- BT Lee
- BY Chou
- C Chenglim
- C Chu
- C Chu
- C Chu
- C Deng
- C Deng
- C Doyle
- C Du
- C Du
- C Du
- C Durucan
- C Durucan
- C Durucan
- C Göbel
- C Hellmich
- C Kealley
- C Kealley
- C Lin
- C Mansur
- C Ohtsuki
- C Piconi
- C Schiller
- C Schiller
- C Sender
- C Trojani
- C You
- C Zhang
- C Zou
- CC Silva
- CCPM Verheyen
- CCPM Verheyen
- CCPM Verheyen
- CE Holy
- CE Wen
- CG Finkemeier
- CH Yao
- CI Vallo
- CJ Damien
- CJ Damien
- CK Chan
- CM Agrawal
- CM Agrawal
- CM Serre
- CM Vaz
- CM Vaz
- CQ Ning
- CQ Ning
- CR Kothapalli
- CS Reed
- CT Laurencin
- CT Laurencin
- CT Laurencin
- CV Ragel
- CVM Rodrigues
- D Bakoš
- D Choi
- D Enlow
- D Green
- D Hakimimehr
- D Lickorish
- D Lickorish
- D Nihouannen Le
- D Nihouannen Le
- D Nihouannen Le
- D Nihouannen Le
- D Rautaray
- D Scharnweber
- D Skrtic
- D Skrtic
- D Skrtic
- D Skrtic
- D Stojanovic
- D Tadic
- D Tadic
- D Walsh
- D Zahn
- D Zhang
- DA Shimko
- DA Wahl
- DB Burr
- DC Tancred
- DF Williams
- DG Guo
- DG Shchukin
- DK Pattanayak
- DM Yunos
- DN Misra
- DR Mehlisch
- DS Zhou
- DW Hutmacher
- DW Hutmacher
- DZ Chen
- E Adolfsson
- E Adolfsson
- E Behravesh
- E Bertoni
- E Boanini
- E Boanini
- E Boanini
- E Bonucci
- E Dawes
- E Fidancevska
- E Khor
- E Milella
- E Piskin
- E Sachlos
- E Salernitano
- E Ural
- ED Boland
- EE Keller
- EJ Harper
- EJ Harper
- EM Christenson
- EM Younger
- ES Ahn
- ES Ahn
- F Causa
- F Heilmann
- F Jegoux
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- F Witte
- F Xu
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- F Xu
- F Zhang
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- F Zhao
- FH Lin
- FJ Guild
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- FL Matthews
- FN Oktar
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- FRAJ Rose
- FY Hsu
- FZ Cui
- G Carotenuto
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- G Fortunato
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- G Wei
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- H Dong
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- H Ikeda
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- H Kweon
- H Li
- H Li
- H Li
- H Matsuno
- H Mckellop
- H Monma
- H Tlatlik
- H Wang
- H Zhang
- HA Lowenstam
- HA Marouf
- HB Wen
- HH Fu
- HHK Xu
- HHK Xu
- HHK Xu
- HHK Xu
- HHK Xu
- HJ Lee
- HJ Lee
- HJ Qiu
- HL Dornhoffer
- HR Lin
- HR Ramay
- HS Wang
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- HY Juang
- I Balac
- I Kangasniemi
- I Resiak
- I Tcacencu
- I Thompson
- I Yamaguchi
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- YM Kong
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- YM Sung
- YP Lu
- Yun-Mo Sung
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- YW Li
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- YX Pang
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- Z Ajdukovic
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- Z Shen
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- Z Yi
- ZAC Schnepp
- ZB Luklinska
- ZE Erkmen
- ZK Hong
- Α Rovira
- Α Tampieri
- ΝH Ladizesky
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Are patients with lichen planus really prone to urolithiasis? Lichen planus and urolithiasis
- Author
- Publication venue
- 'FapUNIFESP (SciELO)'
- Publication date
- 01/01/2016
- Field of study
WOS: 000380066200025PubMed: 27286123Purpose: to investigate whether patients with lichen planus (LP) are really prone to urolithiasis or not. Patients and Methods: We performed a prospective analysis of 40 patients diagnosed with lichen planus (LP) (group I), and 40 volunteers did not have LP before (group II). Participants were all checked for urolithiasis by radiological investigations. Blood samples were analyzed for biochemistry parameters including calcium and uric acid. 24-h urine samples were analyzed to investigate oxalate, citrate calcium, uric acid, magnesium, sodium and creatinine. Results: Men/women ratio and mean age were similar between group I and II (p> 0.05). A presence or history of urolithiasis was detected in 8 (20%) and 2 (% 5) patients in group I and II, respectively (p< 0.05). Hypocitraturia was the most common anomaly with 35% (n: 14) in group I. The rate of hypocitraturia in group II was 12.5% (n: 5) and the difference was statistically significantly different (p= 0.036). In group I, hyperuricosuria and hyperoxaluria followed with rates of 27.5% (n: 11) and 25% (n: 10), respectively. The rate of hyperuricosuria and hyperoxaluria were both 5% (n: 2) in group II and the differences were significant (p< 0.05). Hyperuricemia was another important finding in the patients with LP. It was detected in 13 (32.5%) patients in group I and in 1 (2.5%) participant in group II (p= 0.001). Conclusion: According to our results, metabolic disorders of urolithiasis were highly detected in the patients with LP. However, similar to the etiology of LP, the exact reasons for these metabolic abnormalities in LP remain a mystery
Caspase 1, Caspase 3, TNF-alpha, p53, and Hif1-alpha gene expression status of the brain tissues and hippocampal neuron loss in short-term dichlorvos exposed rats
- Author
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Shear bond, wettability and AFM evaluations on CO2 laser-irradiated CAD/CAM ceramic surfaces
- Author
- A Della Bona
- A Della Bona
- A Gamal El
- A Piwowarczyk
- ACC Saracoglu
- AH Suliman
- Ahmed El Gamal
- AM Filho
- B Ersu
- B Gökçe
- C Ural
- C Ural
- Carlo Fornaini
- Etienne Medioni
- G Sjögren
- H Akin
- Jean Paul Rocca
- JH Chen
- JH Chen
- JL Román-Rodríguez
- JP Rocca
- JW Thurmond
- KA Kupiec
- M Hayashi
- M He
- M Ozcan
- M Ozcan
- MA Bergman
- MB Blatz
- ME Jensen
- MN Aboushelib
- MP Paranhos
- MS Akyil
- MS Akyıl
- N Krämer
- Nathalie Brulat-Bouchard
- Omid H. Muhammad
- P Shiu
- PH Corazza
- R Li
- RC Oyagu De
- RG Craig
- RR Braga
- S Kasraei
- S Naichuan
- S Stubinger
- TM Chen
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Behavioural and respiratory dysfunction in the freshwater fish, Labeo rohita (Hamilton) under malathion intoxication
- Author
- Publication venue
- 'Walter de Gruyter GmbH'
- Publication date
- Field of study