556 research outputs found
Far-infrared vibrational properties of high-pressure-high-temperature C60 polymers and the C60 dimer
We report high-resolution far-infrared transmission measurements of the 2 + 2 cycloaddition C-60 dimer and two-dimensional rhombohedral and one-dimensional orthorhombic high-pressure high-temperature C60 polymers. In the spectral region investigated(20-650 cm(-1)), we see no low-energy interball modes, but symmetry breaking of the linked C-60 balls is evident in the complex spectrum of intramolecular modes. Experimental features suggest large splittings or frequency shifts of some IhC60-derived modes that are activated by symmetry reduction, implying that the balls are strongly distorted in these structures. We have calculated the vibrations of all three systems by first-principles quantum molecular dynamics and use them to assign the predominant IhC60 symmetries of observed modes. Pur calculations show unprecedentedly large downshifts of T-1u(2)-derived modes and extremely large splittings of other modes, both of which are consistent with the experimental spectra. For the rhombohedral and orthorhombic polymers, the T-1u(2)-derived mode that is polarized along the bonding direction is calculated to downshift below any T-1u(1)-derived modes. We also identify a previously unassigned feature near 610 cm(-1) in all three systems as a widely split or shifted mode derived from various silent IhC60 vibrations, confirming a strong perturbation model for these linked fullerene structures
Partial anomalous pulmonary venous connection with intact atrial septum in a child with ventricular septal defect: a case report
Partial anomalous pulmonary vein connection (PAPVC) is a rare congenital abnormal cardiac defect involving the pulmonary veins draining into the right atrium (RA) directly or indirectly by venous connection. Ninety percent of PAPVCs are accompanied by atrial septal defect (ASD). To our knowledge, there is no previous report of PAPVC with ventricular septal defect (VSD) without ASD in Korea, and in this paper, we report the first such case. A 2-day-old girl was admitted into the Chonnam National University Hospital for evaluation of a cardiac murmur. An echocardiogram revealed perimembranous VSD without ASD. She underwent patch closure of the VSD at 5 months of age. Although the VSD was completely closed, she had persistent cardiomegaly with right ventricular volume overload, as revealed by echocardiography. Three years later, cardiac catheterization and chest computed tomography revealed a PAPVC, with the right upper pulmonary vein draining into the right SVC. Therefore, correction of the PAPVC was surgically performed at 3 years of age. We conclude that it is important to suspect PAPVC in patients with right ventricular volume overload, but without ASD
Low Temperature Shear Modulus Changes in Solid 4-He and Connection to Supersolidity
Superfluidity, liquid flow without friction, is familiar in helium. The first
evidence for "supersolidity", its analogue in quantum solids, came from recent
torsional oscillator (TO) measurements involving 4-He. At temperatures below
200 mK, TO frequencies increased, suggesting that some of the solid decoupled
from the oscillator. This behavior has been replicated by several groups but
solid 4-He does not respond to pressure differences and persistent currents and
other signatures of superflow have not been seen. Both experiments and theory
indicate that defects are involved. These should also affect the solid's
mechanical behavior and so we have measured the shear modulus of solid 4-He at
low frequencies and strains. We observe large increases below 200 mK, with the
same dependence on measurement amplitude, 3-He impurity concentration and
annealing as the decoupling seen in TO experiments. This unusual elastic
behavior is explained in terms of a dislocation network which is pinned by 3-He
at the lowest temperatures but becomes mobile above 100 mK. The frequency
changes in TO experiments appear to be related to the motion of these
dislocations, perhaps by disrupting a possible supersolid state.Comment: 18 pages, 4 figues, Supplementary Informatio
Different mechanism of two-proton emission from proton-rich nuclei Al and Mg
Two-proton relative momentum () and opening angle ()
distributions from the three-body decay of two excited proton-rich nuclei,
namely Al p + p + Na and Mg p
+ p + Ne, have been measured with the projectile fragment separator
(RIPS) at the RIKEN RI Beam Factory. An evident peak at MeV/c as
well as a peak in around 30 are seen in the two-proton
break-up channel from a highly-excited Mg. In contrast, such peaks are
absent for the Al case. It is concluded that the two-proton emission
mechanism of excited Mg is quite different from the Al case, with
the former having a favorable diproton emission component at a highly excited
state and the latter dominated by the sequential decay process
Probing oscillations into sterile neutrinos with cosmology, astrophysics and experiments
We perform a thorough analysis of oscillation signals generated by one extra
sterile neutrino, extending previous analyses done in simple limiting cases and
including the effects of established oscillations among active neutrinos. We
consider the following probes: solar, atmospheric, reactor and beam neutrinos,
Big-Bang Nucleosynthesis (helium-4, deuterium), Cosmic Microwave Background,
Large Scale Structure, supernovae, neutrinos from other astrophysical sources.
We find no evidence for a sterile neutrino in present data, identify the still
allowed regions, and study which future experiments can best probe them:
sub-MeV solar experiments, more precise studies of CMB or BBN, future supernova
explosions, etc. We discuss how the LSND hint is strongly disfavoured by the
constraints of (standard) cosmology.Comment: 50 pages, many (14) figures. The text is divided into "results" and
"technical details" sections. Final updated versio
Hepatic oxidative DNA damage is associated with increased risk for hepatocellular carcinoma in chronic hepatitis C
Although the oxidative stress frequently occurs in patients with chronic hepatitis C, its role in future hepatocellular carcinoma (HCC) development is unknown. Hepatic 8-hydroxydeoxyguanosine (8-OHdG) was quantified using liver biopsy samples from 118 naĂŻve patients who underwent liver biopsy from 1995 to 2001. The predictability of 8-OHdG for future HCC development and its relations to epidemiologic, biochemical and histological baseline characteristics were evaluated. During the follow-up period (mean was 6.7±3.3 years), HCC was identified in 36 patients (30.5%). Univariate analysis revealed that 16 variables, including 8-OHdG counts (65.2±20.2 vs 40.0±23.5 cells per 105âÎŒm2, P<0.0001), were significantly different between patients with and without HCC. Cox proportional hazard analysis showed that the hepatic 8-OHdG (P=0.0058) and fibrosis (P=0.0181) were independent predicting factors of HCC. Remarkably, 8-OHdG levels were positively correlated with body and hepatic iron storage markers (vs ferritin, P<0.0001 vs hepatic iron score, P<0.0001). This study showed that oxidative DNA damage is associated with increased risk for HCC and hepatic 8-OHdG levels are useful as markers to identify the extreme high-risk subgroup. The strong correlation between hepatic DNA damage and iron overload suggests that the iron content may be a strong mediator of oxidative stress and iron reduction may reduce HCC incidence in patients with chronic hepatitis C
Hindered proton collectivity in the proton-rich nucleus ^<28>S : Possible magic number Z = 16
NUCLEAR STRUCTURE AND DYNAMICS 20129â13 July 201
Clinical practice: Protein-losing enteropathy in children
Protein-losing enteropathy (PLE) is a rare complication of a variety of intestinal disorders characterized by an excessive loss of proteins into the gastrointestinal tract due to impaired integrity of the mucosa. The clinical presentation of patients with PLE is highly variable, depending upon the underlying cause, but mainly consists of edema due to hypoproteinemia. While considering PLE, other causes of hypoproteinemia such as malnutrition, impaired synthesis, or protein loss through other organs like the kidney, liver, or skin, have to be excluded. The disorders causing PLE can be divided into those due to protein loss from intestinal lymphatics, like primary intestinal lymphangiectasia or congenital heart disease and those with protein loss due to an inflamed or abnormal mucosal surface. The diagnosis is confirmed by increased fecal concentrations of alpha-1-antitrypsin. After PLE is diagnosed, the underlying cause should be identified by stool cultures, serologic evaluation, cardiac screening, or radiographic imaging. Treatment of PLE consists of nutrition state maintenance by using a high protein diet with supplement of fat-soluble vitamins. In patients with lymphangiectasia, a low fat with medium chain triglycerides (MCT) diet should be prescribed. Besides dietary adjustments, appropriate treatment for the underlying etiology is necessary and supportive care to avoid complications of edema. PLE is a rare complication of various diseases, mostly gastrointestinal or cardiac conditions that result into loss of proteins in the gastrointestinal tract. Prognosis depends upon the severity and treatment options of the underlying disease
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