109 research outputs found
Active liquid crystal tuning of metallic nanoantenna enhanced light emission from colloidal quantum dots
- Author
- Abass A.
- Aimi Abass
- Anger P.
- Beeckman J.
- Belacel C.
- Bharadwaj P.
- Bjorn Maes
- Christ A.
- Chu K. C.
- Coenen T.
- Curto A. G.
- Dickson W.
- Dridi M.
- Dries Van Thourhout
- Evans P. R.
- Flamee S.
- GarcĂa de Abajo F. J.
- Hancu I. M.
- Hicks E. M.
- Hu C.
- Jaime GĂłmez Rivas
- Jeroen Beeckman
- Jin P.
- Jun Y. C.
- Khatua S.
- Kossyrev P. A.
- KĂŒhn S.
- Li H.
- Li J.
- Li J.
- Li J.
- Li J. J.
- Livneh N.
- Lozano G.
- Lukâyanchuk B.
- Lumdee C.
- Marc Verschuuren
- Mertens H.
- Ming T.
- Miroshnichenko A. E.
- Muskens O. L.
- MĂŒller J.
- Olcum S.
- Pellegrini G.
- Ringler M.
- Rodriguez S. R. K.
- Rodriguez S. R. K.
- Said Rahimzadeh-Kalaleh Rodriguez
- Schwartz T.
- Shadrivov I. V.
- Tangi Aubert
- Teperik T. V.
- Thomas Ako
- Vecchi G.
- Wokaun A.
- Xiao S.
- Zeger Hens
- Zhou W.
- Zou S.
- Publication venue
- 'American Chemical Society (ACS)'
- Publication date
- 01/01/2014
- Field of study
A system comprising an aluminum nanoantenna array on top of a luminescent colloidal quantum dot waveguide and covered by a thermotropic liquid crystal (LC) is introduced. By heating the LC above its critical temperature, we demonstrate that the concomitant refractive index change modifies the hybrid plasmonic-photonic resonances in the system. This enables active control of the spectrum and directionality of the narrow-band (similar to 6 nm) enhancement of quantum dot photoluminescence by the metallic nanoantennas
LRR Conservation Mapping to Predict Functional Sites within Protein Leucine-Rich Repeat Domains
- Author
- A Bateman
- A Di Matteo
- A Forsyth
- A Mercer
- A Sali
- AC Papageorgiou
- Andrew Bent
- B Hao
- B Kobe
- B Kobe
- B Park
- B Schwessinger
- BR Herrin
- C Morlot
- C Schmitz-Linneweber
- C Sigrist
- C Zipfel
- C Zipfel
- C Zipfel
- CA Innis
- CA Velikovsky
- D Chinchilla
- D Chinchilla
- D Zeidler
- E Myers
- F Leckie
- F Sicilia
- FM Dunning
- G Felix
- G Kunze
- H Ashkenazy
- H Binz
- H HĂ€weker
- J Bella
- J Dolan
- J FernĂĄndez-Recio
- J Kang
- J Wan
- JD Jones
- Juan FernĂĄndez-Recio
- K Dickson
- K Kishimoto
- K Takeda
- K Tamura
- KU Torii
- KV Krasileva
- L Adams-Phillips
- L Federici
- L GĂłmez-GĂłmez
- L GĂłmez-GĂłmez
- L Liu
- L Zhou
- Laura Helft
- LB Clark
- LK Mosavi
- Luca Federici
- M Albert
- M Casasoli
- M Padmanabhan
- M Parniske
- M Proell
- M Rafiqi
- M Shaw
- Miguel A. Blazquez
- MS Jin
- N Dharmasiri
- N Dharmasiri
- P Emsley
- PN Dodds
- Q Fan
- R Celikel
- R D'ovidio
- R Finn
- R Napier
- RA Laskowski
- RA van der Hoorn
- Rishabh Gupta
- S Kepinski
- S Lacombe
- S Tasumi
- SA Morillo
- SH Shiu
- T Boller
- T Kajander
- T Nakagawa
- T Van Loy
- T Walsh
- Teresa Koller
- TK Eitas
- TL Couvreur
- V Nicaise
- V Shanmugam
- Vignyan Reddy
- W Wilbur
- X Tan
- Xiyang Chen
- Y Suzuki
- Z Yang
- Z Ye
- Publication venue
- Public Library of Science
- Publication date
- 18/07/2011
- Field of study
Computational prediction of protein functional sites can be a critical first step for analysis of large or complex proteins. Contemporary methods often require several homologous sequences and/or a known protein structure, but these resources are not available for many proteins. Leucine-rich repeats (LRRs) are ligand interaction domains found in numerous proteins across all taxonomic kingdoms, including immune system receptors in plants and animals. We devised Repeat Conservation Mapping (RCM), a computational method that predicts functional sites of LRR domains. RCM utilizes two or more homologous sequences and a generic representation of the LRR structure to identify conserved or diversified patches of amino acids on the predicted surface of the LRR. RCM was validated using solved LRR+ligand structures from multiple taxa, identifying ligand interaction sites. RCM was then used for de novo dissection of two plant microbe-associated molecular pattern (MAMP) receptors, EF-TU RECEPTOR (EFR) and FLAGELLIN-SENSING 2 (FLS2). In vivo testing of Arabidopsis thaliana EFR and FLS2 receptors mutagenized at sites identified by RCM demonstrated previously unknown functional sites. The RCM predictions for EFR, FLS2 and a third plant LRR protein, PGIP, compared favorably to predictions from ODA (optimal docking area), Consurf, and PAML (positive selection) analyses, but RCM also made valid functional site predictions not available from these other bioinformatic approaches. RCM analyses can be conducted with any LRR-containing proteins at www.plantpath.wisc.edu/RCM, and the approach should be modifiable for use with other types of repeat protein domains
Comparative analysis of Neph gene expression in mouse and chicken development
- Author
- A Fornoni
- A Sugie
- AM Goffinet
- Andreas Kispert
- Beate Brand-Saberi
- Bernhard Schermer
- BJ Dickson
- DB Donoviel
- E Mizuhara
- E Neumann-Haefelin
- EH Chen
- Faisal Yusuf
- G Guo
- G Kuo
- G Liu
- G Piluso
- G-M Barletta
- GS Sellick
- H Putaala
- H Yajima
- Heiko Schweizer
- HM Young
- I Konstantinova
- J Funahashi
- J Najm
- K Bhalla
- K Nishida
- K Shen
- K-F Fischbach
- L Sellin
- Linus A. Völker
- LM Wilson
- M Frotscher
- M Hornberg
- M Hoshino
- M Kestila
- M Manto
- M Pascual
- M Ristola
- M Ruiz-GĂłmez
- M Strunkelnberg
- MA Cooper
- Marianne Petry
- Martin Höhne
- Mohammad Abdelsabour-Khalaf
- N Hoveyda
- Oliver Kretz
- P Gerke
- P Gerke
- P Gerke
- P Ihalmo
- PF Maness
- PS Tarpey
- RG Ramos
- RS Tuan
- S Bao
- S Serizawa
- SA Newman
- SD Menon
- T Komori
- T Schneider
- TB Huber
- Thomas Benzing
- Tilman Busch
- TR Clandinin
- V Duboc
- Y Minaki
- Y Morikawa
- Y Yue
- Publication venue
- Springer-Verlag
- Publication date
- 01/01/2011
- Field of study
Neph proteins are evolutionarily conserved members of the immunoglobulin superfamily of adhesion proteins and regulate morphogenesis and patterning of different tissues. They share a common protein structure consisting of extracellular immunoglobulin-like domains, a transmembrane region, and a carboxyl terminal cytoplasmic tail required for signaling. Neph orthologs have been widely characterized in invertebrates where they mediate such diverse processes as neural development, synaptogenesis, or myoblast fusion. Vertebrate Neph proteins have been described first at the glomerular filtration barrier of the kidney. Recently, there has been accumulating evidence suggesting a function of Neph proteins also outside the kidney. Here we demonstrate that Neph1, Neph2, and Neph3 are expressed differentially in various tissues during ontogenesis in mouse and chicken. Neph1 and Neph2 were found to be amply expressed in the central nervous system while Neph3 expression remained localized to the cerebellum anlage and the spinal cord. Outside the nervous system, Neph mRNAs were also differentially expressed in branchial arches, somites, heart, lung bud, and apical ectodermal ridge. Our findings support the concept that vertebrate Neph proteins, similarly to their Drosophila and C. elegans orthologs, provide guidance cues for cell recognition and tissue patterning in various organs which may open interesting perspectives for future research on Neph1-3 controlled morphogenesis
A systematic review of progranulin concentrations in biofluids in over 7,000 peopleâassessing the pathogenicity of GRN mutations and other influencing factors
- Author
- Abel Emily
- AgĂŒero Pablo
- Alcolea Daniel
- Almeida Maria Rosario
- Andres-Cerezo Lucie
- Antonell Anna
- Arcaro Marina
- Arighi Andrea
- Arndt Philipp
- Bagnoli Silvia
- Baker Matt
- Barandiaran Myriam
- Benussi Luisa
- Binetti Giuliano
- Block Matthew S.
- Boeve Bradley
- Borroni Barbara
- Bruni Amalia
- BrÄthen Geir
- Cantoni Valentina
- Carlson Aaron M.
- Clarimon Jordi
- Clot Fabienne
- Coppola Giovanni
- Dickson Dennis W.
- DurĂŁes JoĂŁo
- Feneberg Emily
- Fenoglio Chiara
- Finch Ni Cole
- Fortea Juan
- Gabilondo Alazne
- Galimberti Daniela
- Gazzina Stefano
- Ghidoni Roberta
- Gohda Tomohito
- Graff Caroline
- Graff-Radford Neill R.
- Greither Thomas
- GrĂžntvedt GĂžril Rolfseng
- GĂłmez-NĂșñez Alba
- GĂłmez-Tortosa Estrella
- Heller Carolin
- Heslegrave Amanda
- Hiemisch Andreas
- IllĂĄn-Gala Ignacio
- Jian Jinlong
- Jiskoot Lize C.
- Jurkutat Anne
- Kamei Nozomu
- Kiess Wieland
- Kittel-Schneider Sarah
- Lamari Foudil
- Lauridsen Camilla
- LeBer Isabelle
- Lemos JoĂŁo
- Lima Marisa
- Liu Chuanju
- LleĂł Alberto
- Lohmann Ebba
- Meeter Lieke H.H.
- Mengel David
- Moreno FermĂn
- Murakoshi Maki
- Otto Markus
- Petersen Ronald C.
- Puey Roger
- Quante Mirja
- Rademakers Rosa
- Reif Andreas
- Rivolta Jasmine
- Rohrer Jonathan D.
- Rossi Giacomina
- Rotondo Enmanuela
- Sainz M. José
- Sando Sigrid Botne
- Santana Isabel
- Schreiber Stefanie
- Seelaar Harro
- Sellami Leila
- Serpente Maria
- Serrero Ginette
- Sogorb-Esteve Aitana
- Steinacker Petra
- Stubert Johannes
- Swift Imogen J.
- Synofzik Matthis
- SĂĄnchez-Valle Raquel
- Takada Leonel T.
- Theil Gerit
- TĂĄbuas-Pereira Miguel
- Van Damme Philip
- van der Ende Emma L.
- van Swieten John C.
- Vielhaber Stefan
- Weigl Johannes
- Wilke Carlo
- Zetterberg Henrik
- Publication venue
- Publication date
- 28/03/2024
- Field of study
Background: Pathogenic heterozygous mutations in the progranulin gene (GRN) are a key cause of frontotemporal dementia (FTD), leading to significantly reduced biofluid concentrations of the progranulin protein (PGRN). This has led to a number of ongoing therapeutic trials aiming to treat this form of FTD by increasing PGRN levels in mutation carriers. However, we currently lack a complete understanding of factors that affect PGRN levels and potential variation in measurement methods. Here, we aimed to address this gap in knowledge by systematically reviewing published literature on biofluid PGRN concentrations. Methods: Published data including biofluid PGRN concentration, age, sex, diagnosis and GRN mutation were collected for 7071 individuals from 75 publications. The majority of analyses (72%) had focused on plasma PGRN concentrations, with many of these (56%) measured with a single assay type (Adipogen) and so the influence of mutation type, age at onset, sex, and diagnosis were investigated in this subset of the data. Results: We established a plasma PGRN concentration cut-off between pathogenic mutation carriers and non-carriers of 74.8 ng/mL using the Adipogen assay based on 3301 individuals, with a CSF concentration cut-off of 3.43 ng/mL. Plasma PGRN concentration varied by GRN mutation type as well as by clinical diagnosis in those without a GRN mutation. Plasma PGRN concentration was significantly higher in women than men in GRN mutation carriers (p = 0.007) with a trend in non-carriers (p = 0.062), and there was a significant but weak positive correlation with age in both GRN mutation carriers and non-carriers. No significant association was seen with weight or with TMEM106B rs1990622 genotype. However, higher plasma PGRN levels were seen in those with the GRN rs5848 CC genotype in both GRN mutation carriers and non-carriers. Conclusions: These results further support the usefulness of PGRN concentration for the identification of the large majority of pathogenic mutations in the GRN gene. Furthermore, these results highlight the importance of considering additional factors, such as mutation type, sex and age when interpreting PGRN concentrations. This will be particularly important as we enter the era of trials for progranulin-associated FTD.</p
Gene therapy for carcinoma of the breast: Pro-apoptotic gene therapy
- Author
- A Ashkenazi
- A Gross
- A Haimovitz-Friedman
- A Neubauer
- A Phelan
- A Piche
- A Sierra
- A Sierra
- A Sierra
- A Strasser
- A Vinyals
- AA Danen-Van Oorschot
- AC Lazaris
- AJ Brenner
- AM Pietersen
- AN Rakkar
- B Zhivotovsky
- BE Rogers
- BM Putzer
- BM Putzer
- C Heise
- CA Schmitt
- CM Perou
- D Wallach
- DE Fisher
- DJ Allan
- DR Ciocca
- DR Ciocca
- DR Green
- E White
- EC LaCasse
- EM Berns
- F Li
- F Paillard
- FF Liu
- G Ambrosini
- G Ambrosini
- G Elliott
- G Evan
- G Gasparini
- G Kroemer
- GI Perez
- GJ Zhang
- GJ Zhang
- GN Hortobagyi
- GT Williams
- H Gahery-Segard
- H Joensuu
- H Li
- H Li
- HJ van Slooten
- HK Boxhorn
- I Marzo
- I Tamm
- J Hurlimann
- J Wu
- J Xiang
- J Xiang
- JC Reed
- JC Reed
- JesĂșs GĂłmez-Navarro
- JF Kerr
- JF Kerr
- Jialing Xiang
- JL Cook
- JL Gooch
- JM Gee
- JM Ruppert
- JT Douglas
- JY Chang
- K Becker
- K Binley
- KB Elkon
- KL Molnar-Kimber
- L Buckbinder
- LA Lesoon-Wood
- LD Stratford-Perricaudet
- LL Nielsen
- LL Nielsen
- LL Nielsen
- LL Nielsen
- LM Anderson
- LM Vargas-Roig
- LM Vargas-Roig
- LR Livingstone
- M Bouvet
- M Favrot
- M Jaattela
- M Jaattela
- M Marcelli
- M Muschen
- M Mustonen
- M Nacht
- M Vakkala
- M Wright
- MA Sheard
- MA Shibata
- MF Clarke
- MM Keane
- MS Dilber
- MS Sheikh
- MW Ealovega
- N Shao
- NA Thornberry
- NA Thornberry
- OI Olopade
- P Holmqvist
- P Li
- P Rochaix
- P Seth
- P Seth
- PM Vrancken
- PN Reynolds
- Q Zhan
- QL Deveraux
- QR Chen
- R Kumar
- R Silvestrini
- RA Walker
- RB Dickson
- RC Bargou
- RD Leek
- RJ Debs
- RJ Muschel
- RM Elledge
- S Eissa
- S Fawell
- S Krajewski
- S Veronese
- SL Brody
- SM Frisch
- SM Frisch
- SR Schwarze
- SS Gambhir
- T Kafri
- VN Sumantran
- Waleed Arafat
- XF Le
- XM Yin
- Y Takamiya
- Y Wei
- YA Hannun
- YT Tai
- ZM Shao
- ZN Oltvai
- ZZ Su
- Publication venue
- Publication date
- 17/12/1999
- Field of study
The dysregulation of apoptosis contributes in a variety of ways to the malignant phenotype. It is increasingly recognized that the alteration of pro-apoptotic and anti-apoptotic molecules determines not only escape from mechanisms that control cell cycle and DNA damage, but also endows the cancer cells with the capacity to survive in the presence of a metabolically adverse milieu, to resist the attack of the immune system, to locally invade and survive despite a lack of tissue anchorage, and to evade the otherwise lethal insults induced by drugs and radiotherapy. A multitude of apoptosis mediators has been identified in the past decade, and the roles of several of them in breast cancer have been delineated by studying the clinical correlates of pathologically documented abnormalities. Using this information, attempts are being made to correct the fundamental anomalies at the genetic level. Fundamental to this end are the design of more efficient and selective gene transfer systems, and the employment of complex interventions that are tailored to breast cancer and that are aimed concomitantly towards different components of the redundant regulatory pathways. The combination of such genetic modifications is most likely to be effective when combined with conventional treatments, thus robustly activating several pro-apoptotic pathways
Erratum to: 36th International Symposium on Intensive Care and Emergency Medicine
- Author
- A Boing
- A Gaspari
- A KĂŒbler
- A Marudi
- A Matheson
- A Tridente
- A. A. Amelia
- A. Abbas
- A. Adams
- A. Agnoli
- A. Al Khulaifi
- A. Al Khulaifi
- A. Aldawood
- A. AlHussain
- A. Ali
- A. Almenyar
- A. Alrashid
- A. Alsheikhly
- A. Alves
- A. Ankuli
- A. Arbouti
- A. Armaganidis
- A. Artigas
- A. Asimakos
- A. B. Belli
- A. B. Cayci Sivri
- A. Bandyopadhyay
- A. Basu
- A. Bedova
- A. Belli
- A. Ben Souissi
- A. Ben Souissi
- A. Ben Souissi
- A. Ben Souissi
- A. Bond
- A. Borgman
- A. Borowska
- A. Boutin
- A. Boutin
- A. Boyce
- A. Braschi
- A. Budnyuk
- A. C. Rodrigues
- A. Cariou
- A. Casazza
- A. Chakraborty
- A. Cirodde
- A. Crizaldo Toledo
- A. Cudia
- A. Cuoghi
- A. D. Dubrawski
- A. Dardashti
- A. Dean
- A. Dean
- A. Delfosse
- A. Dhaliwal
- A. Dhaliwal
- A. Dobru
- A. Docherty
- A. Doronzio
- A. Douvdevani
- A. Dukoff-Gordon
- A. Durward
- A. E Van den Berg
- A. Eden
- A. Eissa
- A. Enguix-Armada
- A. Escalante
- A. Escalante
- A. Ezzamouri
- A. F. Francke
- A. F. Turgeon
- A. F. Turgeon
- A. F. Van Rootselaar
- A. F. Van Rootselaar
- A. Fayed
- A. Fernandez
- A. Fischer
- A. Fogagnolo
- A. Fuller
- A. Fuller
- A. G. Murchison
- A. G. Proudfoot
- A. Garcia
- A. Garcia-Alcantara
- A. Garcia-De la Torre
- A. Garland
- A. Garland
- A. Gaspari
- A. Ghuysen
- A. Giannini
- A. Gobatto
- A. Goloubev
- A. Grubb
- A. Gruber
- A. Gultekin
- A. H. Zwinderman
- A. Hagiwara
- A. Hana
- A. Hana
- A. Harts
- A. Hejazi
- A. HernĂĄndez-Lozano
- A. HernĂĄndez-Lozano
- A. Hovingh
- A. Huber
- A. Itani
- A. J. Groeneveld
- A. J. Hoogendijk
- A. K. Kink
- A. K. Kuzovlev
- A. K. Tashima
- A. Kapuagasi
- A. Kar
- A. Kaskovagheorgescu
- A. Kaushal
- A. Khaghani
- A. Kindawi
- A. Kleinsasser
- A. Kodaira
- A. Kutz
- A. Kuzovlev
- A. Körner
- A. L Luciani
- A. L. Lange
- A. Lepape
- A. Lesmes
- A. Lima
- A. Lima
- A. Linder
- A. Loza
- A. LĂłpez Lago
- A. M. Magnuson
- A. M. Tuip-de Boer
- A. Maaoui
- A. Machado
- A. Mahrous
- A. Malamas
- A. Marinho
- A. Marinho
- A. Marinho
- A. Marinho
- A. Marinho
- A. Marinho
- A. Marinho
- A. Martins
- A. Marudi
- A. Marudi
- A. Matroud
- A. Mebazaa
- A. Mignon
- A. Min
- A. Molokhia
- A. Molokhia
- A. Molokhia
- A. Molokhia
- A. Molokhia
- A. Morelli
- A. Morris
- A. Motos
- A. N. Ahmad
- A. Nadeem
- A. Nau
- A. Neill
- A. Nyman
- A. NĂșñez
- A. Oglinda
- A. Omar
- A. Omar
- A. Omar
- A. Omar
- A. Orlando
- A. Osipov
- A. Ozcan
- A. OâLoughlin
- A. OâSullivan
- A. OâSullivan
- A. OâSullivan
- A. P. Pagano
- A. P. Torelly
- A. Pagano
- A. Patarchi
- A. Pautova
- A. Pavli
- A. Pesenti
- A. Prekates
- A. Psirides
- A. Puerto-Morlan
- A. Puxty
- A. PĂ©rez-Calatayud
- A. R. Robles Caballero
- A. Radford
- A. Raithatha
- A. Raithatha
- A. Raithatha
- A. Reiter
- A. Rhodes
- A. Rhodes
- A. Rhodes
- A. Riahi
- A. Riahi
- A. Riahi
- A. Riahi
- A. Riviere
- A. Riviere
- A. Riviere
- A. Robles
- A. Rogers
- A. Rousseau
- A. Roze des Ordons
- A. Ruijter
- A. Rutten
- A. S. Alsaawi
- A. S. Stieglitz
- A. Salgueiro
- A. Salgueiro
- A. Salgueiro
- A. Sarmento
- A. Sarmento
- A. Savitsky
- A. Scheirer
- A. Schiavi
- A. Seldon
- A. Sell
- A. Sen
- A. Sergeev
- A. Shabanov
- A. Shafie
- A. Shafquat
- A. Shiraishi
- A. Silva-Pinto
- A. Silva-Pinto
- A. Spassov
- A. Stella
- A. Steuber
- A. Stieglitz
- A. Stilwell
- A. SĂĄnchez-LĂłpez
- A. SĂĄnchez-LĂłpez
- A. Taha
- A. Taha
- A. Taskin
- A. Temple
- A. Tinmouth
- A. Tinmouth
- A. Torres
- A. Torres
- A. Torres
- A. Touborg Lassen
- A. Tridente
- A. Tridente
- A. Tridente
- A. Tridente
- A. Trifi
- A. Trifi
- A. Tsimogianni
- A. Turgeon
- A. Turgeon
- A. Vakalos
- A. Vakalos
- A. Vakalos
- A. Vakalos
- A. Van Assche
- A. Van Assche
- A. Varma
- A. Vassi
- A. Vercueil
- A. Vieillard-Baron
- A. Violaki
- A. Violaki
- A. Vuylsteke
- A. W. Waldmann
- A. Wald
- A. Waldmann
- A. Waldmann
- A. Webber
- A. Wheeler
- A. Yalcin
- A. Zepeda-Mendoza
- A. Zerman
- AF Donneau
- AK GĂŒler
- AT Tesnieres
- B Adamik
- B Avard
- B. Al Raiy
- B. AlGhamdi
- B. Alhamadi
- B. Alotaibi
- B. Avard
- B. Avard
- B. Balicco
- B. BasarĂœk Aydogan
- B. Besen
- B. Bilgili
- B. Bilgili
- B. Borgatta
- B. Bouhemad
- B. C. Civantos
- B. Carr
- B. Civantos
- B. De Cagny
- B. De Cagny
- B. De Cagny
- B. De Cagny
- B. De Laat
- B. Delwarde
- B. Eilers
- B. François
- B. François
- B. Fyntanidou
- B. Fyntanidou
- B. Fyntanidou
- B. Goncalves
- B. Guidet
- B. J. Hunt
- B. Jeribi
- B. Jones
- B. K. Lopansri
- B. Khwannimit
- B. Khwannimit
- B. Köves
- B. Laribi
- B. Laribi
- B. Lord
- B. Loureiro
- B. LĂłpez Matamala
- B. Miles
- B. Mueller
- B. Mueller
- B. Mueller
- B. Nyamaizi
- B. O. Alotabi
- B. Oliveira
- B. Oliveira
- B. Oliveira
- B. P. Scicluna
- B. Pasquet
- B. Peroni
- B. Saugel
- B. Sauneuf
- B. Shenkman
- B. Sloan
- B. Stessel
- B. Stessel
- B. Söderquist
- B. Tudor
- B. Vidal
- B. Waldum-Grevbo
- B. Wanta
- B. Xavier
- C. A. Volta
- C. Aldecoa
- C. Aldecoa
- C. Arrowsmith
- C. Ashton
- C. Battle
- C. Battle
- C. Battle
- C. Bell
- C. Bor
- C. Bor
- C. Brunborg
- C. Carvalho
- C. Chaddock
- C. Chiurazzi
- C. Choong
- C. Cobilinschi
- C. Cobilinschi
- C. Cobilinschi
- C. Cobilinschi
- C. Cohen
- C. Dalrymple
- C. De Deyne
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- Y. Cohen
- Y. Dupertuis
- Y. Haraguchi
- Y. Himmel
- Y. Hoydonckx
- Y. Izutani
- Y. Izutani
- Y. Izutani
- Y. Kawano
- Y. Koide
- Y. Kokhanovsky
- Y. Masson
- Y. Nakamura
- Y. Nassar
- Y. Podoksenov
- Y. Que
- Y. S. Chiew
- Y. S. Chiew
- Y. Sakr
- Y. Sarshor
- Y. Schriel
- Y. Shouman
- Y. Su
- Y. Su
- Y. Svirko
- Y. Tekdos
- Y. Wong
- Z. Aidoni
- Z. Ali
- Z. Douira
- Z. Ghiorghiu
- Z. Ghiorghiu
- Z. Ghiorghiu
- Z. Ghiorghiu
- Z. GĂŒllĂŒ
- Z. GĂŒllĂŒ
- Z. IsĂœkdogan
- Z. MolnĂĄr
- Z. Ramsheva
- Z. Stanga
- Z. Stathi
- Z. Uzundurukan
- Z. Ălger
- Ă. MartĂnez GonzĂĄlez
- Ă. Ăzdedeoglu
- Ă. GaygĂœsĂœz
- Ă. Kara
- Publication venue
- Springer Nature
- Publication date
- 01/01/2016
- Field of study
[This corrects the article DOI: 10.1186/s13054-016-1208-6.]
New insights into the genetic etiology of Alzheimer's disease and related dementias
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- Aaltonen L.
- Aaltonen V.
- Aarsland Dag
- Aavikko M.
- Abalos M.S.
- Abdelnour Carla
- Abner E.
- Abraham R.
- Adams H.
- Adams P.M.
- Adarmes-GĂłmez A.
- Adarmes-GĂłmez A.D.
- Aguilera N.
- Aguilera N.
- Aguirre A.
- Ahmad S.
- Akinyemi R.O.
- Al-Chalabi A.
- AlarcĂłn-MartĂn Emilio
- Albert M.S.
- Albin R.L.
- Alcolea Daniel
- Alegret Montserrat
- Ali M.
- Allen M.
- Allende I.R.
- Alonso M.D.
- Alonso M.D.
- Alvarez I.
- Alvarez L.
- Amer-Ferrer G.
- Amin N.
- Amouyel Philippe
- Anastasiou A.
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- Andrade V.
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- Antikainen R.
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- Publication venue
- Publication date
- 01/01/2022
- Field of study
Characterization of the genetic landscape of Alzheimer's disease (AD) and related dementias (ADD) provides a unique opportunity for a better understanding of the associated pathophysiological processes. We performed a two-stage genome-wide association study totaling 111,326 clinically diagnosed/'proxy' AD cases and 677,663 controls. We found 75 risk loci, of which 42 were new at the time of analysis. Pathway enrichment analyses confirmed the involvement of amyloid/tau pathways and highlighted microglia implication. Gene prioritization in the new loci identified 31 genes that were suggestive of new genetically associated processes, including the tumor necrosis factor alpha pathway through the linear ubiquitin chain assembly complex. We also built a new genetic risk score associated with the risk of future AD/dementia or progression from mild cognitive impairment to AD/dementia. The improvement in prediction led to a 1.6- to 1.9-fold increase in AD risk from the lowest to the highest decile, in addition to effects of age and the APOE Δ4 allele
Estudos de avaliação do conhecimento nutricional de adultos: uma revisão sistemåtica
- Author
- Al Riyami A
- Alcides ECA
- Aldrich L
- Axelson ML
- Barros DC
- Carrillo E
- Castro NMG
- Chapman KM
- Dattilo M
- De Vriendt T
- Després JP
- Dickson-Spillmann M
- Diez-Garcia RW
- Duncan BB
- Freire P
- Freitas ECB
- Galindo GĂłmez C
- Glanz K
- Gomes MA
- GĂĄmbaro A
- Harnack L
- Henderson Sabry J
- Hendrie GA
- Holdsworth M
- Kolodinsky J
- KresiÄ G
- Lin W
- Lin W
- Lourani Oliveira dos Santos Correia
- LĂdia Bezerra Barbosa
- Mataratzis PSR
- McLeod ER
- Mendes KG
- Mirmiran P
- Montero Bravo A
- Motta DG
- Nicastro H
- Nuss H
- Obayashi S
- Oliveira FL
- OâBrien G
- Parmenter K
- Parmenter K
- Parmenter K
- Pon LW
- Raphaela Costa Ferreira
- Rodrigues LPF
- Sandra Mary Lima Vasconcelos
- Sapp SG
- Sarno F
- Scagliusi FB
- Schaller C
- Sharma SV
- Silveira DS
- Spronk I
- Triches RM
- Verbeek A
- Verner D
- Von Elm E
- Vuori I
- Wardle J
- Worsley A
- Zawila LG
- Publication venue
- 'FapUNIFESP (SciELO)'
- Publication date
- Field of study
Evaluation of appendicitis risk prediction models in adults with suspected appendicitis
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- Abbas Sh
- Abbas Sh
- Abbassi Oa
- Abbott T
- Abdelgadir Am
- Abdelrahman A
- Abdelrahman M
- Abdelrahman M
- Abdelwahed A
- Abellan M
- AbellĂĄn Am
- AbellĂĄn Am
- Abulafi M
- Acharya A
- Acosta A
- Adam Me
- Adams Re
- Adegbola So
- Adegbola So
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- Afshar S
- Agresta F
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- Carvalho Lc
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- Cavallo D
- Cavero A
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- CerdĂĄn-Santacruz C
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- CerdĂĄn-Santacruz C
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- Charalabopoulos A
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- Chatzikonstantinou M
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- Chauhan P
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- Cheah Wl
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- Chessa A
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- Chetta G
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- Chillitupa Cz
- Chimetto A
- Chin My
- Chishti Ia
- Chitsabesan P
- Chng S
- Choi S
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- Chong B
- Choolani-Bhojwani E
- Choong Jh
- Choong Jh
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- Chowdhary M
- Chowdhury F
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- Chrastek D
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- Cidoncha A
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- Clements Ja
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- Clements Js
- Clerici F
- Clingan R
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- Clough E
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- Coe Po
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- Colangelo E
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- Collier-Wakefield O
- Colombo F
- Colombo F
- Colvin Da
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- Connor Kl
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- Conti D
- Contine Ac
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- Cook V
- Cooke F
- Coppola M
- Coratti F
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- Cordoba E
- Cordoba E
- Cormenzana Ob
- Correia D
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- Cortés Gv
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- Cossu F
- Costa Sd
- Costamagna D
- Cotton Ae
- Couch Dg
- Cousins L
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- Cox M
- Coyle D
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- Cruickshank N
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- Cynthia G
- CĂĄnovas-Moreno G
- D'Ambra L
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- Danquah-Boateng D
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- De Chaves-RodrĂguez Pg
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- Del-Valle-Ruiz Sr
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- Delimpalta C
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- FernĂĄndez-Candela A
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- Zhang Ay
- Zhou S
- Zorrilla L
- Zuin M
- Zurleni Tz
- Publication venue
- 'Wiley'
- Publication date
- 01/01/2019
- Field of study
Background
Appendicitis is the most common general surgical emergency worldwide, but its diagnosis remains challenging. The aim of this study was to determine whether existing risk prediction models can reliably identify patients presenting to hospital in the UK with acute right iliac fossa (RIF) pain who are at low risk of appendicitis.
Methods
A systematic search was completed to identify all existing appendicitis risk prediction models. Models were validated using UK data from an international prospective cohort study that captured consecutive patients aged 16â45âyears presenting to hospital with acute RIF in March to June 2017. The main outcome was best achievable model specificity (proportion of patients who did not have appendicitis correctly classified as low risk) whilst maintaining a failure rate below 5 per cent (proportion of patients identified as low risk who actually had appendicitis).
Results
Some 5345 patients across 154 UK hospitals were identified, of which twoâthirds (3613 of 5345, 67·6 per cent) were women. Women were more than twice as likely to undergo surgery with removal of a histologically normal appendix (272 of 964, 28·2 per cent) than men (120 of 993, 12·1 per cent) (relative risk 2·33, 95 per cent c.i. 1·92 to 2·84; Pâ<â0·001). Of 15 validated risk prediction models, the Adult Appendicitis Score performed best (cutâoff score 8 or less, specificity 63·1 per cent, failure rate 3·7 per cent). The Appendicitis Inflammatory Response Score performed best for men (cutâoff score 2 or less, specificity 24·7 per cent, failure rate 2·4 per cent).
Conclusion
Women in the UK had a disproportionate risk of admission without surgical intervention and had high rates of normal appendicectomy. Risk prediction models to support shared decisionâmaking by identifying adults in the UK at low risk of appendicitis were identified
Reducing the environmental impact of surgery on a global scale: systematic review and co-prioritization with healthcare workers in 132 countries
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- ĆœateckĂœ J.
- Publication venue
- Oxford University Press (OUP)
- Publication date
- 01/07/2023
- Field of study
Background
Healthcare cannot achieve net-zero carbon without addressing operating theatres. The aim of this study was to prioritize feasible interventions to reduce the environmental impact of operating theatres.
Methods
This study adopted a four-phase Delphi consensus co-prioritization methodology. In phase 1, a systematic review of published interventions and global consultation of perioperative healthcare professionals were used to longlist interventions. In phase 2, iterative thematic analysis consolidated comparable interventions into a shortlist. In phase 3, the shortlist was co-prioritized based on patient and clinician views on acceptability, feasibility, and safety. In phase 4, ranked lists of interventions were presented by their relevance to high-income countries and lowâmiddle-income countries.
Results
In phase 1, 43 interventions were identified, which had low uptake in practice according to 3042 professionals globally. In phase 2, a shortlist of 15 intervention domains was generated. In phase 3, interventions were deemed acceptable for more than 90 per cent of patients except for reducing general anaesthesia (84 per cent) and re-sterilization of âsingle-useâ consumables (86 per cent). In phase 4, the top three shortlisted interventions for high-income countries were: introducing recycling; reducing use of anaesthetic gases; and appropriate clinical waste processing. In phase 4, the top three shortlisted interventions for lowâmiddle-income countries were: introducing reusable surgical devices; reducing use of consumables; and reducing the use of general anaesthesia.
Conclusion
This is a step toward environmentally sustainable operating environments with actionable interventions applicable to both highâ and lowâmiddleâincome countries
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