Crowdfunding: the moderating role of the funding goal on factors influencing project success

Purpose: The factors determining the success of crowdfunding projects is one of the central aspects for crowdfunding researchers. Most quantitative approaches recognize the amount of funds targeted as an important control. However, little is known about the impact of the funding goal on other factors that impact crowdfunding success. We hypothesize that the effect of crowdfunding success factors might vary dependent on funding goal level. Design/methodology/approach: A dataset of 338 crowdfunding projects on the German crowdfunding platform StartNext, with a vast majority of projects founded in Germany and a few projects from international European founders, in the years 2015 to 2016 is analysed by conducting regression analyses controlling for varying funding goal sizes. We use the dependent variables success, the degree of success, number of project supporters and the average contribution per supporter and control whether the effect of independent variables such as comments, updates and social media depend on different funding goals. Findings: Our study indicates that the impact of the investigated success factors in fact strongly depends on the goal sizes of crowdfunding projects. By grouping projects into clusters of varying funding goal sizes, we find that the impact of individual success factors changes and that the funding goal plays a moderating role for factors impacting project success. Research/practical implications: These results help both researchers and future entrepreneurs to better understand supporter behaviour. First, we suggest researchers to include the projects’ funding goals as moderators in most cases especially when assessing success factors for crowdfunding projects. Second, future entrepreneurs should be aware that factors influencing the success of a crowdfunding project strongly depend on the set funding goal. Depending on funding goal, some factors become less relevant whilst other factors’ importance is increasing. Originality/value: The funding goal of a crowdfunding project determines whether instruments used by project founders have an impact on their project’s success. Although the funding goal is a central issue in crowdfunding research, it is often used as independent variable, in contrast we suggest incorporating it as a moderator for other success factors

Citizen concerns and acceptance for novel energy technologies

The authors would like to thank the Helmholtz project “Energy System 2050” for the financial support. A special thanks belongs to the whole group of the research topic 4 which has supported the work in several ways. For the development of the survey we would like to thank Prof. Dr. Michael Braun from GESIS and for hosting the survey the platform SoSci-survey (www.soscisurvey.de).The introduction of new energy technologies in the context of the transformation of energy systems has repeatedly led to acceptance problems. These can have a decisive impact on the success of individual projects or the introduction of entire technologies. Therefore, three new energy technologies have been selected for an acceptance examination of the population: hydrogen filling stations, stationary battery storage systems, and production facilities for biofuels, which will contribute to the decarbonisation of the energy sector in the future. Based on interviews with experts, a survey was developed to: analyse the acceptance based on the citizen concerns for the selected technologies, the attitude towards financial support, the perceived influence on the implementation process, and the current state of knowledge regarding the respective technology. The first analysis points to a certain acceptance of the selected energy technologies among the population in Germany. The work provides a first cornerstone for a more detailed explanation of the acceptance genesis for new energy technologies.Michael Brauninfo:eu-repo/semantics/publishedVersio

Three Personality Trait Combinations for Agile Employees: The Relationship Between the Big Five and Agile Mindset

Agile IT projects need employees who not only follow agile structures but have a specific attitude called the agile mindset. While the relevance of the agile mindset is clear, findings on when it can be developed, are very limited. Stable personality traits, like the big five, influence attitude. Providing how these traits interact with the agile mindset gives orientation regarding in which cases an agile mindset is more trainable than in other cases. To investigate these relationships, we conducted an online survey with 327 students of a project management lecture. As a result of our SEM and QCA analysis, we found three combinations of personality traits that influence the agile mindset including different extents of conscientiousness, openness, agreeableness and neuroticism. We deepen and extend the theory around the agile mindset and enable practitioners to choose data-driven cases for development activities. Limitations and future research based on these results are given

Assessing the social acceptance of key technologies for the German energy transition

Background The widespread use of sustainable energy technologies is a key element in the transformation of the energy system from fossil-based to zero-carbon. In line with this, technology acceptance is of great importance as resistance from the public can slow down or hinder the construction of energy technology projects. The current study assesses the social acceptance of three energy technologies relevant for the German energy transition: stationary battery storage, biofuel production plants and hydrogen fuel station. Methods An online survey was conducted to examine the public’s general and local acceptance of energy technologies. Explored factors included general and local acceptance, public concerns, trust in relevant stakeholders and attitudes towards financial support. Results The results indicate that general acceptance for all technologies is slightly higher than local acceptance. In addition, we discuss which public concerns exist with regard to the respective technologies and how they are more strongly associated with local than general acceptance. Further, we show that trust in stakeholders and attitudes towards financial support is relatively high across the technologies discussed. Conclusions Taken together, the study provides evidence for the existence of a “general–local” gap, despite measuring general and local acceptance at the same level of specificity using a public sample. In addition, the collected data can provide stakeholders with an overview of worries that might need to be addressed when planning to implement a certain energy project

Comparative patent analysis for the identification of global research trends for the case of battery storage, hydrogen and bioenergy

Patent documents provide knowledge about which countries are investing in certain technologies and make it possible to identify potential innovation trends. The aim of this article is to analyze trends in patenting that might result in innovations for three energy technologies: thermochemical conversion of biomass (Bioenergy), lithium-ion battery storage, and hydrogen production by alkaline water electrolysis. Based on different patent indicators, the most active countries are compared to provide insights into the global market position of a country, particularly Germany which is used as a reference here. In line with this, a freely available patent analysis software tool was developed directly using the European Patent Office database through their Open Patent Services. The results for named technologies show that patenting activity of Germany is low in comparison to other countries such as Japan, China, and the US. Whereas the position of Germany for batteries and hydrogen is comparable, bioenergy shows different results regarding the identified countries and the number of patents found. However, a broader context beyond patenting is suggested for consideration to make robust statements about particular technology trends. The presented tool and methodology in this study can serve as a blueprint for explorative assessments in any technological domain

An unexpected twist to the activation of IKKβ:TAK1 primes IKKβ for activation by autophosphorylation

IKKβ {IκB [inhibitor of NF-κB (nuclear factor κB)] kinase β} is required to activate the transcription factor NF-κB, but how IKKβ itself is activated in vivo is still unclear. It was found to require phosphorylation by one or more ‘upstream’ protein kinases in some reports, but by autophosphorylation in others. In the present study, we resolve this contro-versy by demonstrating that the activation of IKKβ induced by IL-1 (interleukin-1) or TNF (tumour necrosis factor) in embryonic fibroblasts, or by ligands that activate Toll-like receptors in macrophages, requires two distinct phosphorylation events: first, the TAK1 [TGFβ (transforming growth factor β)-activated kinase-1]-catalysed phosphorylation of Ser(177) and, secondly, the IKKβ-catalysed autophosphorylation of Ser(181). The phosphorylation of Ser(177) by TAK1 is a priming event required for the subsequent autophosphorylation of Ser(181), which enables IKKβ to phosphorylate exogenous substrates. We also provide genetic evidence which indicates that the IL-1-stimulated, LUBAC (linear ubiquitin chain assembly complex)-catalysed formation of linear ubiquitin chains and their interaction with the NEMO (NF-κB essential modulator) component of the canonical IKK complex permits the TAK1-catalysed priming phosphorylation of IKKβ at Ser(177) and IKKα at Ser(176). These findings may be of general significance for the activation of other protein kinases

Lys63/Met1-hybrid ubiquitin chains are commonly formed during the activation of innate immune signalling.

AbstractWe have reported previously that activation of the MyD88-signaling network rapidly induces the formation of hybrid ubiquitin chains containing both Lys63-linked and Met1-linked ubiquitin (Ub) oligomers, some of which are attached covalently to Interleukin Receptor Associated kinase 1. Here we show that Lys63/Met1-Ub hybrids are also formed rapidly when the TNFR1/TRADD, TLR3/TRIF- and NOD1/RIP2-signaling networks are activated, some of which are attached covalently to Receptor-Interacting Protein 1 (TNFR1 pathway) or Receptor-Interacting Protein 2 (NOD1 pathway). These observations suggest that the formation of Lys63/Met1-Ub hybrids are of general significance for the regulation of innate immune signaling systems, and their potential roles in vivo are discussed. We also report that TNFα induces the attachment of Met1-linked Ub chains directly to TNF receptor 1, which do not seem to be attached covalently to Lys63-linked or other types of ubiquitin chain

Roles of the TRAF6 and Pellino E3 ligases in MyD88 and RANKL signaling

It is widely accepted that the essential role of TRAF6 in vivo is to generate the Lys63-linked ubiquitin (K63-Ub) chains needed to activate the "master" protein kinase TAK1. Here, we report that TRAF6 E3 ligase activity contributes to but is not essential for the IL-1-dependent formation of K63-Ub chains, TAK1 activation, or IL-8 production in human cells, because Pellino1 and Pellino2 generate the K63-Ub chains required for signaling in cells expressing E3 ligase-inactive TRAF6 mutants. The IL-1-induced formation of K63-Ub chains and ubiquitylation of IRAK1, IRAK4, and MyD88 was abolished in TRAF6/Pellino1/Pellino2 triple-knockout (KO) cells, but not in TRAF6 KO or Pellino1/2 double-KO cells. The reexpression of E3 ligase-inactive TRAF6 mutants partially restored IL-1 signaling in TRAF6 KO cells, but not in TRAF6/Pellino1/Pellino2 triple-KO cells. Pellino1-generated K63-Ub chains activated the TAK1 complex in vitro with similar efficiently to TRAF6-generated K63-Ub chains. The early phase of TLR signaling and the TLR-dependent secretion of IL-10 (controlled by IRAKs 1 and 2) was only reduced modestly in primary macrophages from knockin mice expressing the E3 ligase-inactive TRAF6[L74H] mutant, but the late-phase production of IL-6, IL-12, and TNFα (controlled only by the pseudokinase IRAK2) was abolished. RANKL-induced signaling in macrophages and the differentiation of bone marrow to osteoclasts was similar in TRAF6[L74H] and wild-type cells, explaining why the bone structure and teeth of the TRAF6[L74H] mice was normal, unlike TRAF6 KO mice. We identify two essential roles of TRAF6 that are independent of its E3 ligase activity

Identification of TBK1 complexes required for the phosphorylation of IRF3 and the production of interferon β

The double-stranded RNA mimetic poly(I:C) and lipopolysaccharide (LPS) activate Toll-like receptors 3 (TLR3) and TLR4, respectively, triggering the activation of TANK (TRAF family member-associated NF-κB activator)-binding kinase 1 (TBK1) complexes, the phosphorylation of interferon regulatory factor 3 (IRF3) and transcription of the interferon β (IFNβ) gene. Here, we demonstrate that the TANK–TBK1 and optineurin (OPTN)–TBK1 complexes control this pathway. The poly(I:C)- or LPS-stimulated phosphorylation of IRF3 at Ser396 and production of IFNβ were greatly reduced in bone marrow-derived macrophages (BMDMs) from TANK knockout (KO) mice crossed to knockin mice expressing the ubiquitin-binding-defective OPTN[D477N] mutant. In contrast, IRF3 phosphorylation and IFNβ production were not reduced significantly in BMDM from OPTN[D477N] knockin mice and only reduced partially in TANK KO BMDM. The TLR3/TLR4-dependent phosphorylation of IRF3 and IFNβ gene transcription were not decreased in macrophages from OPTN[D477N] crossed to mice deficient in IκB kinase ε, a TANK-binding kinase related to TBK1. In contrast with the OPTN–TBK1 complex, TBK1 associated with OPTN[D477N] did not undergo phosphorylation at Ser172 in response to poly(I:C) or LPS, indicating that the interaction of ubiquitin chains with OPTN is required to activate OPTN–TBK1 in BMDM. The phosphorylation of IRF3 and IFNβ production induced by Sendai virus infection were unimpaired in BMDM from TANK KO × OPTN[D477N] mice, suggesting that other/additional TBK1 complexes control the RIG-I-like receptor-dependent production of IFNβ. Finally, we present evidence that, in human HACAT cells, the poly(I:C)-dependent phosphorylation of TBK1 at Ser172 involves a novel TBK1-activating kinase(s)