36 research outputs found

    The effects of non-universal extra dimensions on the radiative lepton flavor decays \mu\to e\gamma and \tau\to \mu\gamma in the two Higgs doublet model

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    We study the effect of non-universal extra dimensions on the branching ratios of the lepton flavor violating processes \mu\to e\gamma and \tau\to \mu\gamma in the general two Higgs doublet model. We observe that these effects are small for a single extra dimension, however, in the case of two extra dimensions there is a considerable enhancement in the additional contributions.Comment: 16 Pages, 9 Figure

    Static quantities of the W boson in the SU_L(3) X U_X(1) model with right-handed neutrinos

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    The static electromagnetic properties of the WW boson, Δκ\Delta \kappa and ΔQ\Delta Q, are calculated in the SU_L(3)} \times U_X(1) model with right-handed neutrinos. The new contributions from this model arise from the gauge and scalar sectors. In the gauge sector there is a new contribution from a complex neutral gauge boson Y0Y^0 and a singly-charged gauge boson Y±Y^\pm. The mass of these gauge bosons, called bileptons, is expected to be in the range of a few hundreds of GeV according to the current bounds from experimental data. If the bilepton masses are of the order of 200 GeV, the size of their contribution is similar to that obtained in other weakly coupled theories. However the contributions to both ΔQ\Delta Q and Δκ\Delta \kappa are negligible for very heavy or degenerate bileptons. As for the scalar sector, an scenario is examined in which the contribution to the WW form factors is identical to that of a two-Higgs-doublet model. It is found that this sector would not give large corrections to Δκ\Delta \kappa and ΔQ\Delta Q.Comment: New material included. Final version to apppear in Physical Review

    FCNC Top Quark Decays in Extra Dimensions

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    The flavor changing neutral top quark decay t -> c X is computed, where X is a neutral standard model particle, in a extended model with a single extra dimension. The cases for the photon, X= \gamma,andaStandardModelHiggsboson,X=H,areanalyzedindetailinanonlinear, and a Standard Model Higgs boson, X = H, are analyzed in detail in a non-linearR_\xi gauge. We find that the branching ratios can be enhanced by the dynamics originated in the extra dimension. In the limit where 1/R >> ->, we have found Br(t -> c \gamma) \simeq 10^{-10} for 1/R = 0.5 TeV. For the decay t -> c H, we have found Br(t -> cH) \simeq 10^{-10} for a low Higgs mass value. The branching ratios go to zero when 1/R -> \infty.Comment: Accepted to be published in the Europ. Phys. Jour. C; 16 pages, 2 figure

    Top-Quark Decay Via the Anomalous Coupling tˉcγ\bar t c \gamma at Hadron Colliders

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    We determine the constraints on the anomalous top-quark coupling associated with the flavor-changing neutral current vertex tˉcγ\bar t c \gamma from the limits on the bb-quark rare decay bsγb\rightarrow s \gamma and non-standard top-quark decays. Based on these constraints, we discuss the experimental observability of the rare decay mode tcγt \rightarrow c \gamma, both at the Fermilab Tevatron with a luminosity-upgrade and at the LHC.Comment: 19 pages, 8 figures, RevTeX, postscript files available via anonymous ftp: ftp://ucdhep.ucdavis.edu/han/top/ (get paper_top.ps and fig*.ps

    Dynamic protein methylation in chromatin biology

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    Post-translational modification of chromatin is emerging as an increasingly important regulator of chromosomal processes. In particular, histone lysine and arginine methylation play important roles in regulating transcription, maintaining genomic integrity, and contributing to epigenetic memory. Recently, the use of new approaches to analyse histone methylation, the generation of genetic model systems, and the ability to interrogate genome wide histone modification profiles has aided in defining how histone methylation contributes to these processes. Here we focus on the recent advances in our understanding of the histone methylation system and examine how dynamic histone methylation contributes to normal cellular function in mammals

    Catching Element Formation In The Act ; The Case for a New MeV Gamma-Ray Mission: Radionuclide Astronomy in the 2020s

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    High Energy Astrophysic

    The spectrum of type III hyperlipoproteinemia

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    Item does not contain fulltextBACKGROUND: Type III hyperlipoproteinemia is a highly atherogenic dyslipoproteinemia characterized by hypercholesterolemia and hypertriglyceridemia due to markedly increased numbers of cholesterol-enriched chylomicron and very-low-density lipoprotein (VLDL) remnant lipoprotein particles. Type III can be distinguished from mixed hyperlipidemia based on a simple diagnostic algorithm, which involves total cholesterol, triglycerides, and apolipoprotein B (apoB). However, apoB is not measured routinely. OBJECTIVE: The objective of the present study was to determine if patients with type III could be distinguished from mixed hyperlipidemia based on lipoprotein lipids. METHODS: Classification was based first on total cholesterol and triglyceride and then on the apoB diagnostic algorithm using apoB plus total cholesterol plus triglycerides, and validated by sequential ultracentrifugation. Four hundred and forty normals, 637 patients with hypertriglyceridemia, and 714 with hypertriglyceridemia and hypercholesterolemia were studied. Plasma lipoproteins were separated by sequential ultracentrifugation and heparin-manganese precipitation. Cholesterol, triglyceride, and apoB were measured in plasma and isolated lipoprotein fractions. RESULTS: Of the 1351 patients with hypertriglyceridemia, 49 had type III hyperlipoproteinemia, as diagnosed by the apoB algorithm and validated by ultracentrifugation. Plasma triglycerides were higher in the type III subjects: 4.16 mmol/L (3.35-6.08, 25th-75th percentile), but there was considerable overlap with the hypertriglyceridemic subjects 2.65 mmol/L (1.91-4.20, 25th-75th percentile) and the combined hyperlipidemic subjects 3.02 mmol/L (2.07-5.32, 25th-75th percentile). Similarly, total cholesterol was 4.79 mmol/L (4.31-5.58, 25th-75th percentile) for type III vs 5.5 mmol/L (4.64-5.78, 25th-75th percentile) and 7.02 mmol/L (6.39-7.96, 25th-75th percentile), respectively. By contrast, as identified by the apoB algorithm, the VLDL-C/TG, VLDL-C/VLDL-TG, VLDL-C/VLDL apoB, and VLDL apoB/LDL apoB ratios were all higher in type III than in the other hypertriglyceridemic dyslipoproteinemias with the exception of type V as diagnosed by the apoB algorithm. CONCLUSION: Cholesterol and triglycerides cannot reliably distinguish type III hyperlipoproteinemia from mixed hyperlipidemia. Adding apoB and applying the apoB algorithm makes reliable diagnosis possible and easy. However, unless apoB is introduced into routine clinical care, type III hyperlipoproteinemia will often not be recognized. Given the cardiovascular risk associated with type III and its responsiveness to treatment, this should not be acceptable
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