Estudio del sistemas de regulación de ventilación en un edificios como parte de la auditoria energética de sistemas HVAC

Este artículo presenta los resultados de un estudio experimental del sistema de ventilación de un edifico de tipo comercial. Se presenta el resultado de mediciones efectuadas durante un proceso de auditoría energética típico en sistemas de ventilación. Se analizan y presentan las principales alternativas de economía de la regulación tanto en el circuito de suministro como el de extracción y renovación del aire

Modelamiento simplificado de un edificio de una zona mediante una red R-C para aplicaciones HVAC.

Este artículo presenta los resultados del estudio realizado para desarrollar un modelo térmico computacional de un edificio. El modelo considera el intercambio de calor y humedad entre un espacio climatizado (mono-zona) y una red capacitiva R-C de las paredes y ventanas. Este modelo pude ser usado en el marco de una pre-auditoria energética del edificio, considerando que no se dispone usualmente de una identificación experimental del consumo de calefacción o enfriamiento (por lo general no se instalan contadores independientes de estos sistemas). Sin embargo el cálculo de la demanda energética es requerida en la búsqueda de oportunidades de reducción del consumo de energía global del edificio

Estudio del sistemas de regulación de ventilación en un edificios como parte de la auditoria energética de sistemas HVAC

Este artículo presenta los resultados de un estudio experimental del sistema de ventilación de un edifico de tipo comercial. Se presenta el resultado de mediciones efectuadas durante un proceso de auditoría energética típico en sistemas de ventilación. Se analizan y presentan las principales alternativas de economía de la regulación tanto en el circuito de suministro como el de extracción y renovación del aire

Identification and data-driven model reduction of state-space representations of lossless and dissipative systems from noise-free data

We illustrate procedures to identify a state-space representation of a lossless- or dissipative system from a given noise-free trajectory; important special cases are passive- and bounded-real systems. Computing a rank-revealing factorization of a Gramian-like matrix constructed from the data, a state sequence can be obtained; state-space equations are then computed solving a system of linear equations. This idea is also applied to perform model reduction by obtaining a balanced realization directly from data and truncating it to obtain a reduced-order mode

GOLVEN peptide signalling through RGI receptors and MPK6 restricts asymmetric cell division during lateral root initiation

During lateral root initiation, lateral root founder cells undergo asymmetric cell divisions that generate daughter cells with different sizes and fates, a prerequisite for correct primordium organogenesis. An excess of the GLV6/RGF8 peptide disrupts these initial asymmetric cell divisions, resulting in more symmetric divisions and the failure to achieve lateral root organogenesis. Here, we show that loss-of-function GLV6 and its homologue GLV10 increase asymmetric cell divisions during lateral root initiation, and we identified three members of the RGF1 INSENSITIVE/RGF1 receptor subfamily as likely GLV receptors in this process. Through a suppressor screen, we found that MITOGEN-ACTIVATED PROTEIN KINASE6 is a downstream regulator of the GLV pathway. Our data indicate that GLV6 and GLV10 act as inhibitors of asymmetric cell divisions and signal through RGF1 INSENSITIVE receptors and MITOGEN-ACTIVATED PROTEIN KINASE6 to restrict the number of initial asymmetric cell divisions that take place during lateral root initiation. The authors demonstrate the negative role of GOLVEN peptides during lateral root initiation in Arabidopsis, at the very early stage of the first asymmetric cell division of lateral root founder cells, and identify the receptors for these peptides

Auxin-regulated reversible inhibition of TMK1 signaling by MAKR2 modulates the dynamics of root gravitropism

Plants are able to orient their growth according to gravity, which ultimately controls both shoot and root architecture.1 Gravitropism is a dynamic process whereby gravistimulation induces the asymmetric distribution of the plant hormone auxin, leading to asymmetric growth, organ bending, and subsequent reset of auxin distribution back to the original pre-gravistimulation situation.1, 2, 3 Differential auxin accumulation during the gravitropic response depends on the activity of polarly localized PIN-FORMED (PIN) auxin-efflux carriers.1, 2, 3, 4 In particular, the timing of this dynamic response is regulated by PIN2,5,6 but the underlying molecular mechanisms are poorly understood. Here, we show that MEMBRANE ASSOCIATED KINASE REGULATOR2 (MAKR2) controls the pace of the root gravitropic response. We found that MAKR2 is required for the PIN2 asymmetry during gravitropism by acting as a negative regulator of the cell-surface signaling mediated by the receptor-like kinase TRANSMEMBRANE KINASE1 (TMK1).2,7, 8, 9, 10 Furthermore, we show that the MAKR2 inhibitory effect on TMK1 signaling is antagonized by auxin itself, which triggers rapid MAKR2 membrane dissociation in a TMK1-dependent manner. Our findings suggest that the timing of the root gravitropic response is orchestrated by the reversible inhibition of the TMK1 signaling pathway at the cell surface