200 research outputs found
A statistical method for region-based meta-analysis of genome-wide association studies in genetically diverse populations
- Author
- A Torkamani
- AG Clark
- BF Voight
- Chen Suo
- Chiea-Chuen Khor
- CM Lindgren
- Daniel Peng-Keat Ng
- DR Nyholt
- DY Lin
- E Zeggini
- E Zeggini
- E-Shyong Tai
- EN Smith
- F Takeuchi
- H Lango Allen
- Haiyan Xu
- HK Perera
- I Mukhopadhyay
- J Li
- J Marchini
- Jianjun Liu
- JM Cheverud
- KA Frazer
- Kee-Seng Chia
- LJ Scott
- M Jallow
- M Kanehisa
- M Kanehisa
- M Kanehisa
- M Stephens
- MC Campbell
- MC Wu
- MI McCarthy
- NA Rosenberg
- P Donnelly
- R Saxena
- R Sladek
- Rick Twee-Hee Ong
- RL Hanson
- S Kathiresan
- SR Browning
- TA Manolio
- Tien-Yin Wong
- Tin Aung
- V Moskvina
- W Pan
- Wan-Ting Poh
- Wan-Ting Tay
- Wellcome Trust Case Control Consortium
- X Han
- X Sim
- Xu Wang
- Xuanyao Liu
- Xueling Sim
- Y Li
- Y Wang
- Yik-Ying Teo
- YY Teo
- YY Teo
- YY Teo
- Publication venue
- Nature Publishing Group
- Publication date
- 01/04/2012
- Field of study
Genome-wide association studies (GWAS) have become the preferred experimental design in exploring the genetic etiology of complex human traits and diseases. Standard SNP-based meta-analytic approaches have been utilized to integrate the results from multiple experiments. This fundamentally assumes that the patterns of linkage disequilibrium (LD) between the underlying causal variants and the directly genotyped SNPs are similar across the populations for the same SNPs to emerge with surrogate evidence of disease association. We introduce a novel strategy for assessing regional evidence of phenotypic association that explicitly incorporates the extent of LD in the region. This provides a natural framework for combining evidence from multi-ethnic studies of both dichotomous and quantitative traits that (i) accommodates different patterns of LD, (ii) integrates different genotyping platforms and (iii) allows for the presence of allelic heterogeneity between the populations. Our method can also be generalized to perform gene-based or pathway-based analyses. Applying this method on real GWAS data in type 2 diabetes (T2D) boosted the association evidence in regions well-established for T2D etiology in three diverse South-East Asian populations, as well as identified two novel gene regions and a biologically convincing pathway that are subsequently validated with data from the Wellcome Trust Case Control Consortium
A study assessing the association of glycated hemoglobin a1C (HbA1C) associated variants with HbA1C, chronic kidney disease and diabetic retinopathy in populations of asian ancestry
- Author
- AL Price
- AM Simonis-Bik
- AS Levey
- AW Foong
- B Devlin
- B Howie
- BF Voight
- C Li
- C Sabanayagam
- C Sabanayagam
- CE Tan
- Chen Suo
- Chiea-Chuen Khor
- Ching-Yu Cheng
- CJ Willer
- CS Franklin
- DM Nathan
- E Selvin
- E Zeggini
- E-Shyong Tai
- EE Nang
- Eranga Vithana
- F Takeuchi
- G Paré
- Haiyan Xu
- J Marchini
- J Ryu
- Jianjun Liu
- JL Grimsby
- JT Tan
- KA Frazer
- Kee-Seng Chia
- L Sobrin
- LJ Scott
- Mingguang He
- Mohammad Ali
- N Soranzo
- Peng Chen
- R Saxena
- Rick Twee-Hee Ong
- RT Ong
- Terri L. Young
- Tien-Yin Wong
- Tin Aung
- TY Wong
- TY Wong
- TY Wong
- V TanhÀuserovå
- VM Monnier
- Wan-Ting Tay
- Wei-Yen Lim
- Xueling Sim
- Y Zheng
- YC Klimentidis
- Yik-Ying Teo
- YY Teo
- YY Teo
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 07/11/2013
- Field of study
10.1371/journal.pone.0079767PLoS ONE811-POLN
Genome-Wide Meta-Analysis of Five Asian Cohorts Identifies PDGFRA as a Susceptibility Locus for Corneal Astigmatism
- Author
- A Cagigrigoriu
- A Kim
- AL Price
- AM Grjibovski
- AM Solouki
- AW Foong
- B Peng
- Belinda Cornes
- C Lange
- CC Khor
- CE Tan
- Chen Suo
- Chiea-Chuen Khor
- Ching-Yu Cheng
- CJ Hammond
- CJ Hammond
- CS Kee
- DJ Schaid
- DO Mutti
- DQ Li
- E Ciner
- E.-Shyong Tai
- EM Harvey
- EM Harvey
- EN Vithana
- Eranga Vithana
- F Carbonaro
- G Heidary
- GR Kazeem
- H Hashemi
- H Kaur
- J Cui
- J Cutter
- J Kawagishi
- Janey Wiggs
- Jianjun Liu
- JM Ip
- K Attebo
- K Hughes
- K Hughes
- K Zadnik
- Kee-Seng Chia
- KW Wright
- L Cowen
- L Llorente
- L O'Donoghue
- L Tong
- Liang-Kee Goh
- LK Yeh
- M Clementi
- M Dirani
- M Dirani
- M Fruttiger
- M He
- M KhabazKhoob
- Mark Seielstad
- MC Heinrich
- Mohammad Kamran Ikram
- N Vij
- P Raju
- PG Hysi
- PI de Bakker
- PR Keller
- Qiao Fan
- R Lavanya
- Rick Twee-Hee Ong
- S Han
- S Vitale
- SA Read
- SB Thom
- SC Huynh
- Seang-Mei Saw
- SG Robbins
- SM Saw
- T Grosvenor
- Terri L. Young
- Tien-Yin Wong
- Tin Aung
- TL Young
- TY Wong
- V Dobson
- VP Hoppenreijs
- Wan-Ting Tay
- WJ Kim
- WR Baldwin
- Xin Zhou
- Xueling Sim
- Y Wang
- Yi-Ju Li
- Yik-Ying Teo
- YJ Li
- YY Teo
- Publication venue
- Public Library of Science
- Publication date
- 01/12/2011
- Field of study
Corneal astigmatism refers to refractive abnormalities and irregularities in the curvature of the cornea, and this interferes with light being accurately focused at a single point in the eye. This ametropic condition is highly prevalent, influences visual acuity, and is a highly heritable trait. There is currently a paucity of research in the genetic etiology of corneal astigmatism. Here we report the results from five genome-wide association studies of corneal astigmatism across three Asian populations, with an initial discovery set of 4,254 Chinese and Malay individuals consisting of 2,249 cases and 2,005 controls. Replication was obtained from three surveys comprising of 2,139 Indians, an additional 929 Chinese children, and an independent 397 Chinese family trios. Variants in PDGFRA on chromosome 4q12 (lead SNP: rs7677751, allelic odds ratioâ=â1.26 (95% CI: 1.16â1.36), Pmetaâ=â7.87Ă10â9) were identified to be significantly associated with corneal astigmatism, exhibiting consistent effect sizes across all five cohorts. This highlights the potential role of variants in PDGFRA in the genetic etiology of corneal astigmatism across diverse Asian populations
Transferability of Type 2 Diabetes Implicated Loci in Multi-Ethnic Cohorts from Southeast Asia
- Author
- A Ramachandran
- AL Gloyn
- AL Gloyn
- AL Price
- AW Foong
- BF Voight
- BN Howie
- C Herder
- C Hu
- C Hu
- CE Tan
- Chen Suo
- CJ Groves
- D Altshuler
- D Bodhini
- D Schleinitz
- Daniel Peng-Keat Ng
- DK Sanghera
- E Zeggini
- E Zeggini
- E-Shyong Tai
- F Takeuchi
- FJ Tsai
- Greg Gibson
- H Unoki
- J Cutter
- J Marchini
- J Wen
- JB Zhou
- JC Chambers
- JC Chan
- Jianjun Liu
- JK Hertel
- JT Tan
- JT Tan
- JV van Vliet-Ostaptchouk
- K Hughes
- K Hughes
- K Yasuda
- KA Frazer
- Kee-Seng Chia
- LJ Scott
- M Horikoshi
- Mark Seielstad
- MC Ng
- Michael Boehnke
- MS Sandhu
- N Grarup
- PW Franks
- Q Qi
- R Lavanya
- R Saxena
- R Sladek
- Rick Twee-Hee Ong
- RT Ong
- S Omori
- S Omori
- SE Humphries
- SF Grant
- Tien-Yin Wong
- V Lyssenko
- W Yang
- Wan-Ting Tay
- X Han
- Xueling Sim
- Y Furukawa
- Y Horikawa
- Y Lin
- Y Liu
- Y Liu
- Y Tabara
- Y Wu
- YH Lee
- Yik-Ying Teo
- YY Teo
- YY Teo
- Publication venue
- Public Library of Science
- Publication date
- 01/04/2011
- Field of study
Recent large genome-wide association studies (GWAS) have identified multiple loci
which harbor genetic variants associated with type 2 diabetes mellitus (T2D),
many of which encode proteins not previously suspected to be involved in the
pathogenesis of T2D. Most GWAS for T2D have focused on populations of European
descent, and GWAS conducted in other populations with different ancestry offer a
unique opportunity to study the genetic architecture of T2D. We performed
genome-wide association scans for T2D in 3,955 Chinese (2,010 cases, 1,945
controls), 2,034 Malays (794 cases, 1,240 controls), and 2,146 Asian Indians
(977 cases, 1,169 controls). In addition to the search for novel variants
implicated in T2D, these multi-ethnic cohorts serve to assess the
transferability and relevance of the previous findings from European descent
populations in the three major ethnic populations of Asia, comprising half of
the world's population. Of the SNPs associated with T2D in previous GWAS,
only variants at CDKAL1 and
HHEX/IDE/KIF11 showed the strongest
association with T2D in the meta-analysis including all three ethnic groups.
However, consistent direction of effect was observed for many of the other SNPs
in our study and in those carried out in European populations. Close examination
of the associations at both the CDKAL1 and
HHEX/IDE/KIF11 loci provided some evidence of locus and
allelic heterogeneity in relation to the associations with T2D. We also detected
variation in linkage disequilibrium between populations for most of these loci
that have been previously identified. These factors, combined with limited
statistical power, may contribute to the failure to detect associations across
populations of diverse ethnicity. These findings highlight the value of
surveying across diverse racial/ethnic groups towards the fine-mapping efforts
for the casual variants and also of the search for variants, which may be
population-specific
Genetic Variants on Chromosome 1q41 Influence Ocular Axial Length and High Myopia
- Author
- AJ Fischer
- Akira Meguro
- AL Price
- AM Solouki
- AP Klein
- AP Klein
- AP Klein
- AW Foong
- C Liu
- CA Anderson
- Candice E. H. Ho
- CC Khor
- CC Khor
- Chiea-Chuen Khor
- Ching-Yu Cheng
- CJ Hammond
- CM Lindgren
- CW Pan
- Daniel Chua
- DS Fan
- E.-Shyong Tai
- EN Vithana
- Eranga Vithana
- F Schaeffel
- Felicia Hawthorne
- Fumihiko Matsuda
- G Biino
- G Zhu
- GR Steinberg
- H Gao
- H Hayashi
- H Jensen
- H Nakanishi
- Hidetoshi Inoko
- I D'Errico
- IM Heid
- Isao Nakata
- J Liang
- Janey L. Wiggs
- JH Laity
- JM Ip
- Keitaro Matsuo
- Kenji Yamashiro
- KW Leung
- Kyoko Ohno-Matsui
- L Cordain
- L Cordain
- L Poliseno
- L Salmena
- Liang-Kee Goh
- LS Lim
- M Bitzer
- M Dirani
- M Dirani
- M Seve
- M Ugarte
- Mark Seielstad
- MI McCarthy
- N Brink
- N Lyhne
- NA McBrien
- Nagahisa Yoshimura
- NJ Schork
- Nobuhisa Mizuki
- P Simon
- PG Hysi
- PG Sanfilippo
- Q Fan
- Q Fan
- Qiao Fan
- R Lavanya
- R Plomin
- R Schippert
- R van Leeuwen
- R Wu
- Roger W. Beuerman
- S Purcell
- S Rozen
- Seang-Mei Saw
- SM Saw
- SM Saw
- SM Saw
- SM Saw
- SM Saw
- T Tokoro
- Terri L. Young
- Tien-Yin Wong
- Tin Aung
- TP Grosvenor
- TY Wong
- TY Wong
- V Baron
- VA Barathi
- Veluchamy A. Barathi
- Wan'e Lim
- X Huibi
- X Sim
- Xin Zhou
- Y Shi
- Y Shi
- Y Zha
- Yi-Ju Li
- Yik-Ying Teo
- Yingfeng Zheng
- YJ Li
- YY Teo
- Z Li
- Publication venue
- Public Library of Science
- Publication date
- 01/06/2012
- Field of study
As one of the leading causes of visual impairment and blindness, myopia poses a significant public health burden in Asia. The primary determinant of myopia is an elongated ocular axial length (AL). Here we report a meta-analysis of three genome-wide association studies on AL conducted in 1,860 Chinese adults, 929 Chinese children, and 2,155 Malay adults. We identified a genetic locus on chromosome 1q41 harboring the zinc-finger 11B pseudogene ZC3H11B showing genome-wide significant association with AL variation (rs4373767, ÎČâ=ââ0.16 mm per minor allele, Pmetaâ=â2.69Ă10â10). The minor C allele of rs4373767 was also observed to significantly associate with decreased susceptibility to high myopia (per-allele odds ratio (OR)â=â0.75, 95% CI: 0.68â0.84, Pmetaâ=â4.38Ă10â7) in 1,118 highly myopic cases and 5,433 controls. ZC3H11B and two neighboring genes SLC30A10 and LYPLAL1 were expressed in the human neural retina, retinal pigment epithelium, and sclera. In an experimental myopia mouse model, we observed significant alterations to gene and protein expression in the retina and sclera of the unilateral induced myopic eyes for the murine genes ZC3H11A, SLC30A10, and LYPLAL1. This supports the likely role of genetic variants at chromosome 1q41 in influencing AL variation and high myopia
Prognostic model to predict postoperative acute kidney injury in patients undergoing major gastrointestinal surgery based on a national prospective observational cohort study.
- Author
- Aamir A
- Abbas J
- Abbas N
- Abbott TEF
- Abdalla M
- Abdikadir H
- Abuhussein N
- Acquaah F
- Adamson R
- Adeleye O
- Adeogun A
- Adlan A
- Aftab R
- Afzal Z
- Agarwal M
- Aglan H
- Agnew CJF
- Agrawal R
- Ahern D
- Ahluwalia J
- Ahluwalia V
- Ahmad A
- Ahmad K
- Ahmed H
- Ahmed I
- Ahmed L
- Ahmed N
- Ahmed P
- Ainger E
- Ainsworth P
- Aishwarya G
- Ajibola-Taylor O
- Akhbari M
- Akhtar A
- Akhtar R
- Akpenyi O
- Al-Ausi M
- Al-Huneidi R
- Al-Khudairi R
- Al-Masri S
- Al-Mousawi A
- Al-Saadi N
- Alagappan A
- Alberts J
- Alfa-Wali M
- Ali A
- Ali B
- Ali I
- Ali M
- Ali Q
- Ali S
- Alturki N
- Alwandi A
- Amani L
- Amarnath T
- Amer M
- Amin H
- Amin MN
- Amoah R
- Amoah-Arko A
- Anandarajah C
- Ananthavarathan P
- Andah EJE
- Andrew K
- Andrews H
- Ang A
- Ang HL
- Ang JJ
- Ani C
- Anilkumar A
- Anto N
- Anwar S
- Apperley H
- Appleton S
- Aquilina T
- Archer M
- Arif T
- Arneill M
- Arnell S
- Arnold TJ
- Arshad A
- Arthur J
- Arulkumaran N
- Arya J
- Asad M
- Asemota N
- Ashaye T
- Ashdown T
- Ashour R
- Ashraf MR
- Ashwood J
- Asif U
- Atkin G
- Atkins B
- Attalla M
- Aubrey-Jones D
- Auckburally S
- Auld F
- Auluck I
- Azam S
- Aziz R
- Azizi A
- Azmi F
- Babatunde F
- Babu VS
- Bachi A
- Bagga R
- Bains H
- Bajwa DS
- Baker A
- Baker C
- Balai E
- Balian V
- Ball M
- Bamford M
- Bana R
- Banfield DA
- Bannard-Smith J
- Barai I
- Barclay J
- Barfi L
- Barker C
- Barker M
- Barmayehvar B
- Barnfield J
- Barnor-Ahiaku E
- Baron C
- Barr CJ
- Barraclough J
- Baryan HK
- Basra M
- Bassam N
- Bath MF
- Battle J
- Beasley W
- Beattie G
- Beattie S
- Beatty M
- Beghal G
- Begum F
- Behranwala K
- Belchos J
- Belgaid DR
- Belgaumkar A
- Bell S
- Ben-Gal O
- Benchetrit S
- Bennett M
- Benons N
- Bernal TL
- Bertelli M
- Berthaume-Hawkins SD
- Best R
- Betts A
- Bevan K
- Bews-Hair M
- Bhambra R
- Bhamra N
- Bhangu A
- Bhatte S
- Bhatti A
- Bhide I
- Bhome R
- Bhudia R
- Bhullar S
- Bienias A
- Billyard C
- Bird C
- Birkinshaw F
- Black J
- Blake E
- Blazeby JM
- Bleakley A
- Bloom I
- Bogle R
- Bolton W
- Borakati A
- Boraluwe-Rallage H
- Borg CM
- Botha A
- Boualbanat Y
- Boulton APR
- Bountra K
- Bowley D
- Boyd G
- Boyles L
- Bradley P
- Brannick S
- Brayley J
- Brecher J
- Breen H
- Bristow S
- Britton N
- Broad J
- Brobbey P
- Brock E
- Brooke M
- Brown A
- Brown B
- Brown F
- Brown FS
- Brown H
- Brown K
- Brown LR
- Brown M
- Brown N
- Brown R
- Brown S
- Brown T
- Bruce C
- Bruce P
- Budd E
- Bull K
- Burgin A
- Burke D
- Burke J
- Burnage S
- Burns N
- Burrows A
- Busti S
- Butler A
- Cabdi NMO
- Cadden F
- Cairns C
- Calabria C
- Calciu A
- Campbell A
- Campbell B
- Campbell G
- Campbell HS
- Cannon SP
- Cant M
- Capella S
- Cardus C
- Cardwell A
- Cargill Z
- Caroll P
- Carr G
- Carr R
- Carr W
- Carse S
- Carter N
- Carter R
- Castle A
- Cato L
- Cave D
- Cecil E
- Chadwick H
- Chadwick M
- Chan F
- Chan L
- Chan M
- Chan SSN
- Chan-lam N
- Chandler E
- Chandler S
- Chandramoorthy L
- Chandramoorthy S
- Chang A
- Chang LH
- Chang M
- Chappelow I
- Chapple K
- Charnley R
- Chaudhry A
- Chaudhry S
- Chauhan P
- Chavan A
- Chean CS
- Chen L
- Chen Y
- Chenciner L
- Cheng J
- Chesner R
- Cheung W
- Chin YR
- China Z
- Chitsabesan P
- Chohan K
- Choi JE
- Chong A
- Chong P
- Choo S
- Chopada A
- Chouari T
- Choudhary Y
- Choudhry M
- Choy CH
- Christie S
- Christopher E
- Chudek D
- Chui J
- Church M
- Church R
- Cipparrone M
- Claireaux HA
- Clark K
- Clarke B
- Clement KD
- Clements B
- Clements SE
- Cobley H
- Coe P
- Cohen J
- Coldicutt O
- Cole A
- Coleman M
- Collaborative S
- Collins J
- Collishaw A
- Common M
- Concannon K
- Conchie H
- Connelly A
- Connor M
- Cookson P
- Cope EA
- Cope T
- Copeland M
- Copley HC
- Coppel J
- Corbridge O
- Cotter M
- Courquin GR
- Court J
- Cox L
- Craig ARJ
- Craig N
- Crane E
- Cremen S
- Cresswell B
- Crewe A
- Crilly C
- Crilly L
- Croghan N
- Croitoru C
- Cromwell D
- Cronbach P
- Crook H
- Crozier L
- Cruddas L
- Cullum R
- Cumber E
- Cumming S
- Cummings D
- Cunliffe L
- Cunningham L
- Curtis A
- Curtis J
- D'Auria M
- D'Souza F
- Dada J
- Dalal F
- Dalrymple J
- Daly D
- Daniels J
- Das E
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- Datta U
- Davies E
- Davies G
- Davies J
- Davies P
- Davis H
- Davison C
- Dawe V
- Dawood S
- Day L
- De Cates C
- De Freitas M
- Dean S
- Debnath D
- Deekonda P
- Deepak S
- Deery L
- Delvi A
- Denley S
- Dennahy IS
- Dennis R
- Dennis Y
- Desai H
- Desai K
- Devalia S
- Dhaliwal R
- Dhillon AK
- Dhillon P
- Dhir T
- Dhuna S
- Diamond J
- Dib NP
- Dickson G
- Dietrich A
- Dindyal S
- Dixon O
- Do V
- Dobrzynska M
- Doherty C
- Donaldson G
- Donohoe C
- Doshi A
- Doull C
- Downes C
- Doyle C
- Drake TM
- Draper A
- Dudley B
- Duff F
- Duffield J
- Duggal A
- Dumann E
- Dunleavy K
- Dunne E
- Dupaguntla YS
- Durand C
- Durbacz M
- Durham-Hall A
- Durno J
- Durrigan T
- Duthie F
- Dutta A
- Dyal A
- Dynes K
- Easby S
- Easdon S
- Eastwood M
- Ebhogiaye O
- Eccles L
- Ecclestone T
- Eddama M
- Edgerton K
- Edozie F
- Edwin C
- Egan E
- Eiben I
- Eiben P
- Eilon T
- El Matary R
- El Tokhy O
- El-Sawy D
- El-Taji O
- Elangovan S
- Elbuzidi M
- Elder P
- Elias J
- Ellerby N
- Elliot J
- Elliott J
- Elsaddig M
- Elseedawy E
- Emberton J
- En BNW
- Engledow A
- English W
- Epanomeritakis E
- Episkopos C
- Epstein J
- Esmail R
- Evans D
- Evans E
- Evans L
- Evans R
- Ezzat A
- Fafemi O
- Falamarzi A
- Fam M
- Faraj A
- Farhan-Alanie MMH
- Farmer N
- Farooq M
- Farooqi M
- Farrar E
- Farwana M
- Faulkner S
- Fawole A
- Fearnhead N
- Feldman M
- Fenton K
- Ferris B
- Filipescu T
- Finch E
- Fitzgerald I
- Fitzgerald JE
- Fitzpatrick H
- Flack V
- Flamini T
- Fletcher D
- Foley MP
- Fong J
- Fowler AJ
- Fox H
- Fozard T
- France B
- Francis N
- Francis S
- Francois V
- Francombe J
- Freeman SK
- Frere M
- Frew K
- Friend C
- Froggatt P
- Frost A
- Frost J
- Fung H
- Gabriel R
- Gaddah M
- Gadsby C
- Gaffing S
- Galea M
- Gallogly P
- Galloway F
- Gammeri E
- Gani A
- Ganyani R
- Gao C
- Gardner I
- Gardner S
- Gardner T
- Garner C
- Garsaa T
- Gaukroger A
- Ge Y
- Gentry R
- George A
- George D
- Gerard L
- Ghaffar A
- Ghag S
- Ghailan N
- Gharatya A
- Gibbons D
- Gibson W
- Gidwani A
- Gil H
- Gilbert A
- Gill A
- Gill K
- Gillespie H
- Gimson A
- Girdlestone T
- Given A
- Glace S
- Glasbey J
- Glasbey JC
- Glen P
- Glover M
- Gnanappiragasam D
- Goaman A
- Goergen N
- Goh C
- Goh ET
- Gohil J
- Gokani S
- Golding D
- Gomaa A
- Gonzalez-Ciscar A
- Goodson R
- Gopfert A
- Gordon J
- Gorman D
- Gower T
- Grace R
- Graham CJ
- Graham S
- Grant A
- Grant M
- Gravell R
- Green B
- Green S
- Greenan E
- Greenhalgh S
- Gregoriou K
- Gregory C
- Gresly J
- Grey A
- Gribbon C
- Griffith J
- Griffiths B
- Griffiths H
- Griffiths N
- Gruhn A
- Guevel B
- Guillotte C
- Gulzar I
- Gundogan B
- Gunwa T
- Gupta A
- Gupta R
- Gurjar S
- Gurung E
- Guy C
- Gwilym B
- Gwozdz AM
- Hack J
- Hackney E
- Haddad S
- Hague M
- Hair J
- Hall M
- Hall N
- Hallan R
- Hamdan K
- Hamid H
- Hammad A
- Hanson M
- Haq T
- Haque A
- Haray P
- Hare L
- Harinath G
- Harlinska A
- Harris A
- Harris C
- Harris L
- Harris R
- Harris S
- Harrison E
- Harrison EM
- Harrison P
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- STARSurg Collaborative Writing Committee, Data analysis, Steering committee, Advisory group, Regional leads, Collaborators and validators
- STARSurg Collaborative Writing Committee, Data analysis, Steering committee, Advisory group, Regional leads, Collaborators and validators, Nepogodiev, D
- Stechman M
- Stewart D
- Stewart E
- Stewart H
- Stewart R
- Stickler E
- Stoddart MT
- Stokes E
- Stott G
- Strang S
- Stringer H
- Stringfellow TD
- Stubbing-Moore A
- Sturrock S
- Subar D
- Subratty SM
- Sukirthan N
- Sukumar S
- Sulaiman SK
- Sultan S
- Sun L
- Sundaram K
- Suresh R
- Suresh S
- Sureshkumar A
- Suri A
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- Swan A
- Swan I
- Swaray A
- Swartbol T
- Symons L
- Szczap A
- Szuman A
- Tailor S
- Tam J
- Tam JP
- Tan CY
- Tan KL
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- Tan TSE
- Tang A
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- Tariq A
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- Tayeh S
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- Taylor O
- Taylor R
- Taylor Z
- Telfer R
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- Tenters F
- Teo R
- Thachettu A
- Thakrar D
- Thatcher A
- Theodoropoulou K
- Thomas A
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- Wright OW
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- Wynell-Mayow W
- Xu Y
- Yahaya AA
- Yalamarthi S
- Yang DD
- Yang N
- Yap J
- Yardimci E
- Yarham E
- Yasin IH
- Yasin T
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- Yeo A
- Yeoh T
- Yeung J
- Yin ZD
- Yip J
- Yoganayagam N
- Yogeswara T
- York J
- Yousaf A
- Youssef H
- Yu T
- Yule A
- Zaat S
- Zanichelli D
- Zaver V
- Zeng R
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- Zheleniakova T
- Zhu Y
- Zornoza A
- Zubikarai G
- Zulkifli A
- Zuzarte L
- Publication venue
- 'Wiley'
- Publication date
- 18/05/2018
- Field of study
Background: Acute illness, existing co-morbidities and surgical stress response can all contribute to postoperative acute kidney injury (AKI) in patients undergoing major gastrointestinal surgery. The aim of this study was prospectively to develop a pragmatic prognostic model to stratify patients according to risk of developing AKI after major gastrointestinal surgery. Methods: This prospective multicentre cohort study included consecutive adults undergoing elective or emergency gastrointestinal resection, liver resection or stoma reversal in 2-week blocks over a continuous 3-month period. The primary outcome was the rate of AKI within 7 days of surgery. Bootstrap stability was used to select clinically plausible risk factors into the model. Internal model validation was carried out by bootstrap validation. Results: A total of 4544 patients were included across 173 centres in the UK and Ireland. The overall rate of AKI was 14·2 per cent (646 of 4544) and the 30-day mortality rate was 1·8 per cent (84 of 4544). Stage 1 AKI was significantly associated with 30-day mortality (unadjusted odds ratio 7·61, 95 per cent c.i. 4·49 to 12·90; P < 0·001), with increasing odds of death with each AKI stage. Six variables were selected for inclusion in the prognostic model: age, sex, ASA grade, preoperative estimated glomerular filtration rate, planned open surgery and preoperative use of either an angiotensin-converting enzyme inhibitor or an angiotensin receptor blocker. Internal validation demonstrated good model discrimination (c-statistic 0·65). Discussion: Following major gastrointestinal surgery, AKI occurred in one in seven patients. This preoperative prognostic model identified patients at high risk of postoperative AKI. Validation in an independent data set is required to ensure generalizability
The global burden of cancer attributable to risk factors, 2010-19 : a systematic analysis for the Global Burden of Disease Study 2019
- Author
- Aali Amirali
- Abbas Qamar
- Abbasi Behzad
- Abbasi-Kangevari Mohsen
- Abbasi-Kangevari Zeinab
- Abbastabar Hedayat
- Abd-Elsalam Sherief
- Abdelmasseh Michael
- Abdelwahab Ahmed Abdelwahab
- Abdoli Gholamreza
- Abdulkader Rizwan Suliankatchi
- Abdulkadir Hanan Abdulkadir
- Abedi Aidin
- Abegaz Kedir Hussein
- Abidi Hassan
- Aboagye Richard Gyan
- Abolhassani Hassan
- Absalan Abdorrahim
- Abtew Yonas Derso
- Abu-Gharbieh Eman
- Achappa Basavaprabhu
- Acheson Alistair R
- Acuna Juan Manuel
- Addison Daniel
- Addo Isaac Yeboah
- Adegboye Oyelola A
- Adesina Miracle Ayomikun
- Adnan Mohammad
- Adnani Qorinah Estiningtyas Sakilah
- Advani Shailesh M
- Afrin Sumia
- Afzal Muhammad Sohail
- Aggarwal Manik
- Ahinkorah Bright Opoku
- Ahmad Araz Ramazan
- Ahmad Rizwan
- Ahmad Sajjad
- Ahmad Sohail
- Ahmadi Sepideh
- Ahmed Haroon
- Ahmed Luai A
- Ahmed Muktar Beshir
- Aiman Wajeeha
- Ajami Marjan
- Akalu Gizachew Taddesse
- Akbarzadeh-Khiavi Mostafa
- Aklilu Addis
- Akonde Maxwell
- Akunna Chisom Joyqueenet
- Al-Hanawi Mohammed Khaled
- Al-Maweri Sadeq Ali Ali
- Al-Raddadi Rajaa M
- Al-Rifai Rami H Hani
- Al-Sabah Salman Khalifah
- Al-Tammemi Ala'a B
- Alahdab Fares
- Alanezi Fahad Mashhour
- Alanzi Turki M
- Alessy Saleh Ali
- Algammal Abdelazeem M
- Alhassan Robert Kaba
- Ali Adel Hajj
- Ali Beriwan Abdulqadir
- Ali Hiwa Abubaker
- Ali Liaqat
- Ali Syed Shujait
- Alimohamadi Yousef
- Alipour Vahid
- Aljunid Syed Mohamed
- Alkhayyat Motasem
- Almustanyir Sami
- Alonso Nivaldo
- Alqalyoobi Shehabaldin
- Altawalah Haya
- Alvis-Guzman Nelson
- Amare Firehiwot
- Ameyaw Edward Kwabena
- Amirzade-Iranaq Mohammad Hosein
- Amu Hubert
- Amusa Ganiyu Adeniyi
- Ancuceanu Robert
- Anderson Jason A
- Animut Yaregal Animut
- Anoushiravani Amir
- Anoushirvani Ali Arash
- Ansari-Moghaddam Alireza
- Ansha Mustafa Geleto
- Antony Benny
- Antwi Maxwell Hubert
- Anwar Sumadi Lukman
- Anwer Razique
- Anyasodor Anayochukwu Edward
- Arab-Zozani Morteza
- Arabloo Jalal
- Aremu Olatunde
- Argaw Ayele Mamo
- Ariffin Hany
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- Villeneuve Paul J
- Violante Francesco S
- Vladimirov Sergey Konstantinovitch
- Vlassov Vasily
- Vo Bay
- Vos Theo
- Vu Linh Gia
- Wadood Abdul Wadood
- Waheed Yasir
- Walde Mandaras Tariku
- Wamai Richard G
- Wang Cong
- Wang Fang
- Wang Ning
- Wang Yu
- Ward Paul
- Waris Abdul
- Westerman Ronny
- Wickramasinghe Nuwan Darshana
- Woldemariam Melat
- Woldu Berhanu
- Xiao Hong
- Xu Rixing
- Xu Suowen
- Xu Xiaoyue
- Yadav Lalit
- Yang Lin
- Yazdanpanah Fereshteh
- Yeshaw Yigizie
- Yismaw Yazachew
- Yonemoto Naohiro
- Younis Mustafa Z
- Yousefi Zabihollah
- Yousefian Fatemeh
- Yu Chuanhua
- Yu Yong
- Yunusa Ismaeel
- Zahir Mazyar
- Zaki Nazar
- Zaman Burhan Abdullah
- Zangiabadian Moein
- Zare Fariba
- Zare Iman
- Zareshahrabadi Zahra
- Zarrintan Armin
- Zastrozhin Mikhail Sergeevich
- Zeineddine Mohammad A
- Zhang Dongyu
- Zhang Jianrong
- Zhang Yunquan
- Zhang Zhi-Jiang
- Zhou Linghui
- Zodpey Sanjay
- Zoladl Mohammad
- Ć ekerija Mario
- Publication venue
- Publication date
- 01/01/2022
- Field of study
Background Understanding the magnitude of cancer burden attributable to potentially modifiable risk factors is crucial for development of effective prevention and mitigation strategies. We analysed results from the Global Burden of Diseases, Injuries, and Risk Factors Study (GBD) 2019 to inform cancer control planning efforts globally. Methods The GBD 2019 comparative risk assessment framework was used to estimate cancer burden attributable to behavioural, environmental and occupational, and metabolic risk factors. A total of 82 risk-outcome pairs were included on the basis of the World Cancer Research Fund criteria. Estimated cancer deaths and disability-adjusted life-years (DALYs) in 2019 and change in these measures between 2010 and 2019 are presented. Findings Globally, in 2019, the risk factors included in this analysis accounted for 4.45 million (95% uncertainty interval 4.01-4.94) deaths and 105 million (95.0-116) DALYs for both sexes combined, representing 44.4% (41.3-48.4) of all cancer deaths and 42.0% (39.1-45.6) of all DALYs. There were 2.88 million (2.60-3.18) risk-attributable cancer deaths in males (50.6% [47.8-54.1] of all male cancer deaths) and 1.58 million (1.36-1.84) risk-attributable cancer deaths in females (36.3% [32.5-41.3] of all female cancer deaths). The leading risk factors at the most detailed level globally for risk-attributable cancer deaths and DALYs in 2019 for both sexes combined were smoking, followed by alcohol use and high BMI. Risk-attributable cancer burden varied by world region and Socio-demographic Index (SDI), with smoking, unsafe sex, and alcohol use being the three leading risk factors for risk-attributable cancer DALYs in low SDI locations in 2019, whereas DALYs in high SDI locations mirrored the top three global risk factor rankings. From 2010 to 2019, global risk-attributable cancer deaths increased by 20.4% (12.6-28.4) and DALYs by 16.8% (8.8-25.0), with the greatest percentage increase in metabolic risks (34.7% [27.9-42.8] and 33.3% [25.8-42.0]). Interpretation The leading risk factors contributing to global cancer burden in 2019 were behavioural, whereas metabolic risk factors saw the largest increases between 2010 and 2019. Reducing exposure to these modifiable risk factors would decrease cancer mortality and DALY rates worldwide, and policies should be tailored appropriately to local cancer risk factor burden. Copyright (C) 2022 The Author(s). Published by Elsevier Ltd. This is an Open Access article under the CC BY 4.0 license.Peer reviewe
Measurement and interpretation of same-sign W boson pair production in association with two jets in pp collisions at s = 13 TeV with the ATLAS detector
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- Zhemchugov A
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- Zimine NI
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- Ă kesson TPA
- Ăncel ĂO
- ĆœeniĆĄ T
- ĆœivkoviÄ L
- Publication venue
- Springer Nature
- Publication date
- 01/04/2024
- Field of study
This paper presents the measurement of fducial and diferential cross sections for both the inclusive and electroweak production of a same-sign W-boson pair in association with two jets (W±W±jj) using 139 fbâ1 of proton-proton collision data recorded at a centre-of-mass energy of âs = 13 TeV by the ATLAS detector at the Large Hadron Collider. The analysis is performed by selecting two same-charge leptons, electron or muon, and at least two jets with large invariant mass and a large rapidity diference. The measured fducial cross sections for electroweak and inclusive W±W±jj production are 2.92 ± 0.22 (stat.) ± 0.19 (syst.)fb and 3.38±0.22 (stat.)±0.19 (syst.)fb, respectively, in agreement with Standard Model predictions. The measurements are used to constrain anomalous quartic gauge couplings by extracting 95% confdence level intervals on dimension-8 operators. A search for doubly charged Higgs bosons H±± that are produced in vector-boson fusion processes and decay into a same-sign W boson pair is performed. The largest deviation from the Standard Model occurs for an H±± mass near 450 GeV, with a global signifcance of 2.5 standard deviations
Combination of searches for heavy spin-1 resonances using 139 fbâ1 of proton-proton collision data at s = 13 TeV with the ATLAS detector
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- Zheng K
- Zheng X
- Zheng Z
- Zhong D
- Zhou B
- Zhou H
- Zhou N
- Zhou Y
- Zhou Y
- Zhu CG
- Zhu J
- Zhu Y
- Zhu Y
- Zhuang X
- Zhukov K
- Zimine NI
- Zinsser J
- Ziolkowski M
- Zoccoli A
- Zoch K
- Zorbas TG
- Zormpa O
- Zou W
- Zwalinski L
- Ă kesson TPA
- Ăncel ĂO
- ĆœeniĆĄ T
- ĆœivkoviÄ L
- Publication venue
- Springer Nature
- Publication date
- 19/04/2024
- Field of study
A combination of searches for new heavy spin-1 resonances decaying into different pairings of W, Z, or Higgs bosons, as well as directly into leptons or quarks, is presented. The data sample used corresponds to 139 fbâ1 of proton-proton collisions at
= 13 TeV collected during 2015â2018 with the ATLAS detector at the CERN Large Hadron Collider. Analyses selecting quark pairs (qq, bb,
, and tb) or third-generation leptons (ÏÎœ and ÏÏ) are included in this kind of combination for the first time. A simplified model predicting a spin-1 heavy vector-boson triplet is used. Cross-section limits are set at the 95% confidence level and are compared with predictions for the benchmark model. These limits are also expressed in terms of constraints on couplings of the heavy vector-boson triplet to quarks, leptons, and the Higgs boson. The complementarity of the various analyses increases the sensitivity to new physics, and the resulting constraints are stronger than those from any individual analysis considered. The data exclude a heavy vector-boson triplet with mass below 5.8 TeV in a weakly coupled scenario, below 4.4 TeV in a strongly coupled scenario, and up to 1.5 TeV in the case of production via vector-boson fusion
Detection of dengue viruses using reverse transcription-loop-mediated isothermal amplification
- Author
- A Igarashi
- A Postel
- A Wilder-Smith
- AK Falconar
- BL Innis
- Boon-Teong Teoh
- BT Teoh
- CL Seah
- DJ Gubler
- DJ Gubler
- EA Henchal
- EC Holmes
- GP Kouri
- HY Chee
- I Kurane
- J Mongkolsapaya
- JD Thompson
- JE Levi
- Jefree Johari
- K Lapphra
- Kim-Kee Tan
- LJ Markoff
- M Parida
- MG Guzman
- MG Guzman
- Mohammed Bashar Danlami
- N Kobayashi
- N Tomita
- P Koraka
- PK Russell
- Poh-Sim Hooi
- PR Young
- PY Shu
- PY Shu
- R Lima Mda
- R Saito
- Rafi Md-Esa
- RB Tesh
- RM Ratcliff
- RS Lanciotti
- S AbuBakar
- S Alcon
- S Li
- Sazaly AbuBakar
- SB Halstead
- SB Halstead
- Sing-Sin Sam
- SS Sam
- T Laue
- T Notomi
- T Pang
- VT Hang
- X Lu
- Y Mori
- YY Kong
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
- âŠ