65 research outputs found

    Partonic flow and ϕ\phi-meson production in Au+Au collisions at sNN\sqrt{s_{NN}} = 200 GeV

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    We present first measurements of the ϕ\phi-meson elliptic flow (v2(pT)v_{2}(p_{T})) and high statistics pTp_{T} distributions for different centralities from sNN\sqrt{s_{NN}} = 200 GeV Au+Au collisions at RHIC. In minimum bias collisions the v2v_{2} of the ϕ\phi meson is consistent with the trend observed for mesons. The ratio of the yields of the Ω\Omega to those of the ϕ\phi as a function of transverse momentum is consistent with a model based on the recombination of thermal ss quarks up to pT4p_{T}\sim 4 GeV/cc, but disagrees at higher momenta. The nuclear modification factor (RCPR_{CP}) of ϕ\phi follows the trend observed in the KS0K^{0}_{S} mesons rather than in Λ\Lambda baryons, supporting baryon-meson scaling. Since ϕ\phi-mesons are made via coalescence of seemingly thermalized ss quarks in central Au+Au collisions, the observations imply hot and dense matter with partonic collectivity has been formed at RHIC.Comment: 6 pages, 4 figures, submit to PR

    Measurement of Transverse Single-Spin Asymmetries for Di-Jet Production in Proton-Proton Collisions at s=200\sqrt{s} = 200 GeV

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    We report the first measurement of the opening angle distribution between pairs of jets produced in high-energy collisions of transversely polarized protons. The measurement probes (Sivers) correlations between the transverse spin orientation of a proton and the transverse momentum directions of its partons. With both beams polarized, the wide pseudorapidity (1η+2-1 \leq \eta \leq +2) coverage for jets permits separation of Sivers functions for the valence and sea regions. The resulting asymmetries are all consistent with zero and considerably smaller than Sivers effects observed in semi-inclusive deep inelastic scattering (SIDIS). We discuss theoretical attempts to reconcile the new results with the sizable transverse spin effects seen in SIDIS and forward hadron production in pp collisions.Comment: 6 pages total, 1 Latex file, 3 PS files with figure

    Energy and system size dependence of \phi meson production in Cu+Cu and Au+Au collisions

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    We study the beam-energy and system-size dependence of \phi meson production (using the hadronic decay mode \phi -- K+K-) by comparing the new results from Cu+Cu collisions and previously reported Au+Au collisions at \sqrt{s_NN} = 62.4 and 200 GeV measured in the STAR experiment at RHIC. Data presented are from mid-rapidity (|y|<0.5) for 0.4 < pT < 5 GeV/c. At a given beam energy, the transverse momentum distributions for \phi mesons are observed to be similar in yield and shape for Cu+Cu and Au+Au colliding systems with similar average numbers of participating nucleons. The \phi meson yields in nucleus-nucleus collisions, normalised by the average number of participating nucleons, are found to be enhanced relative to those from p+p collisions with a different trend compared to strange baryons. The enhancement for \phi mesons is observed to be higher at \sqrt{s_NN} = 200 GeV compared to 62.4 GeV. These observations for the produced \phi(s\bar{s}) mesons clearly suggest that, at these collision energies, the source of enhancement of strange hadrons is related to the formation of a dense partonic medium in high energy nucleus-nucleus collisions and cannot be alone due to canonical suppression of their production in smaller systems.Comment: 20 pages and 5 figure

    System size dependence of associated yields in hadron-triggered jets

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    We present results on the system size dependence of high transverse momentum di-hadron correlations at sNN\sqrt{s_{NN}} = 200 GeV as measured by STAR at RHIC. Measurements in d+Au, Cu+Cu and Au+Au collisions reveal similar jet-like correlation yields at small angular separation (Δϕ0\Delta\phi\sim0, Δη0\Delta\eta\sim0) for all systems and centralities. Previous measurements have shown that the away-side yield is suppressed in heavy-ion collisions. We present measurements of the away-side suppression as a function of transverse momentum and centrality in Cu+Cu and Au+Au collisions. The suppression is found to be similar in Cu+Cu and Au+Au collisions at a similar number of participants. The results are compared to theoretical calculations based on the parton quenching model and the modified fragmentation model. The observed differences between data and theory indicate that the correlated yields presented here will provide important constraints on medium density profile and energy loss model parameters.Comment: 12 pages, 5 figure

    Energy dependence of charged pion, proton and anti-proton transverse momentum spectra for Au+Au collisions at \sqrt{s_NN} = 62.4 and 200 GeV

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    We study the energy dependence of the transverse momentum (pT) spectra for charged pions, protons and anti-protons for Au+Au collisions at \sqrt{s_NN} = 62.4 and 200 GeV. Data are presented at mid-rapidity (|y| < 0.5) for 0.2 < pT < 12 GeV/c. In the intermediate pT region (2 < pT < 6 GeV/c), the nuclear modification factor is higher at 62.4 GeV than at 200 GeV, while at higher pT (pT >7 GeV/c) the modification is similar for both energies. The p/pi+ and pbar/pi- ratios for central collisions at \sqrt{s_NN} = 62.4 GeV peak at pT ~ 2 GeV/c. In the pT range where recombination is expected to dominate, the p/pi+ ratios at 62.4 GeV are larger than at 200 GeV, while the pbar/pi- ratios are smaller. For pT > 2 GeV/c, the pbar/pi- ratios at the two beam energies are independent of pT and centrality indicating that the dependence of the pbar/pi- ratio on pT does not change between 62.4 and 200 GeV. These findings challenge various models incorporating jet quenching and/or constituent quark coalescence.Comment: 19 pages and 6 figure

    Observation of a new boson at a mass of 125 GeV with the CMS experiment at the LHC

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    Overview of the JET results in support to ITER

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    Vascular control of kidney epithelial transporters.

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    A major pathway in hypertension pathogenesis involves direct activation of ANG II type 1 (AT(1)) receptors in the kidney, stimulating Na(+) reabsorption. AT(1) receptors in tubular epithelia control expression and stimulation of Na(+) transporters and channels. Recently, we found reduced blood pressure and enhanced natriuresis in mice with cell-specific deletion of AT(1) receptors in smooth muscle (SMKO mice). Although impaired vasoconstriction and preserved renal blood flow might contribute to exaggerated urinary Na(+) excretion in SMKO mice, we considered whether alterations in Na(+) transporter expression might also play a role; therefore, we carried out proteomic analysis of key Na(+) transporters and associated proteins. Here, we show that levels of Na(+)-K(+)-2Cl(-) cotransporter isoform 2 (NKCC2) and Na(+)/H(+) exchanger isoform 3 (NHE3) are reduced at baseline in SMKO mice, accompanied by attenuated natriuretic and diuretic responses to furosemide. During ANG II hypertension, we found widespread remodeling of transporter expression in wild-type mice with significant increases in the levels of total NaCl cotransporter, phosphorylated NaCl cotransporter (Ser(71)), and phosphorylated NKCC2, along with the cleaved, activated forms of the α- and γ-epithelial Na(+) channel. However, the increases in α- and γ-epithelial Na(+) channel with ANG II were substantially attenuated in SMKO mice. This was accompanied by a reduced natriuretic response to amiloride. Thus, enhanced urinary Na(+) excretion observed after cell-specific deletion of AT(1) receptors from smooth muscle cells is associated with altered Na(+) transporter abundance across epithelia in multiple nephron segments. These findings suggest a system of vascular-epithelial in the kidney, modulating the expression of Na(+) transporters and contributing to the regulation of pressure natriuresis.NEW & NOTEWORTHY The use of drugs to block the renin-angiotensin system to reduce blood pressure is common. However, the precise mechanism for how these medications control blood pressure is incompletely understood. Here, we show that mice lacking angiotensin receptors specifically in smooth muscle cells lead to alternation in tubular transporter amount and function. Thus, demonstrating the importance of vascular-tubular cross talk in the control of blood pressure
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