463 research outputs found
Scaling Relations for Logarithmic Corrections
Multiplicative logarithmic corrections to scaling are frequently encountered
in the critical behavior of certain statistical-mechanical systems. Here, a
Lee-Yang zero approach is used to systematically analyse the exponents of such
logarithms and to propose scaling relations between them. These proposed
relations are then confronted with a variety of results from the literature.Comment: 4 page
Fisher Renormalization for Logarithmic Corrections
For continuous phase transitions characterized by power-law divergences,
Fisher renormalization prescribes how to obtain the critical exponents for a
system under constraint from their ideal counterparts. In statistical
mechanics, such ideal behaviour at phase transitions is frequently modified by
multiplicative logarithmic corrections. Here, Fisher renormalization for the
exponents of these logarithms is developed in a general manner. As for the
leading exponents, Fisher renormalization at the logarithmic level is seen to
be involutory and the renormalized exponents obey the same scaling relations as
their ideal analogs. The scheme is tested in lattice animals and the Yang-Lee
problem at their upper critical dimensions, where predictions for logarithmic
corrections are made.Comment: 10 pages, no figures. Version 2 has added reference
Scaling behaviour of lattice animals at the upper critical dimension
We perform numerical simulations of the lattice-animal problem at the upper
critical dimension d=8 on hypercubic lattices in order to investigate
logarithmic corrections to scaling there. Our stochastic sampling method is
based on the pruned-enriched Rosenbluth method (PERM), appropriate to linear
polymers, and yields high statistics with animals comprised of up to 8000
sites. We estimate both the partition sums (number of different animals) and
the radii of gyration. We re-verify the Parisi-Sourlas prediction for the
leading exponents and compare the logarithmic-correction exponents to two
partially differing sets of predictions from the literature. Finally, we
propose, and test, a new Parisi-Sourlas-type scaling relation appropriate for
the logarithmic-correction exponents.Comment: 10 pages, 5 figure
Does the XY Model have an integrable continuum limit?
The quantum field theory describing the massive O(2) nonlinear sigma-model is
investigated through two non-perturbative constructions: The form factor
bootstrap based on integrability and the lattice formulation as the XY model.
The S-matrix, the spin and current two-point functions, as well as the 4-point
coupling are computed and critically compared in both constructions. On the
bootstrap side a new parafermionic super selection sector is found; in the
lattice theory a recent prediction for the (logarithmic) decay of lattice
artifacts is probed.Comment: 69 pages, 18 figures. Equation (3.20) correcte
Density of states, Potts zeros, and Fisher zeros of the Q-state Potts model for continuous Q
The Q-state Potts model can be extended to noninteger and even complex Q in
the FK representation. In the FK representation the partition function,Z(Q,a),
is a polynomial in Q and v=a-1(a=e^-T) and the coefficients of this
polynomial,Phi(b,c), are the number of graphs on the lattice consisting of b
bonds and c connected clusters. We introduce the random-cluster transfer matrix
to compute Phi exactly on finite square lattices. Given the FK representation
of the partition function we begin by studying the critical Potts model
Z_{CP}=Z(Q,a_c), where a_c=1+sqrt{Q}. We find a set of zeros in the complex
w=sqrt{Q} plane that map to the Beraha numbers for real positive Q. We also
identify tilde{Q}_c(L), the value of Q for a lattice of width L above which the
locus of zeros in the complex p=v/sqrt{Q} plane lies on the unit circle. We
find that 1/tilde{Q}_c->0 as 1/L->0. We then study zeros of the AF Potts model
in the complex Q plane and determine Q_c(a), the largest value of Q for a fixed
value of a below which there is AF order. We find excellent agreement with
Q_c=(1-a)(a+3). We also investigate the locus of zeros of the FM Potts model in
the complex Q plane and confirm that Q_c=(a-1)^2. We show that the edge
singularity in the complex Q plane approaches Q_c as Q_c(L)~Q_c+AL^-y_q, and
determine the scaling exponent y_q. Finally, by finite size scaling of the
Fisher zeros near the AF critical point we determine the thermal exponent y_t
as a function of Q in the range 2<Q<3. We find that y_t is a smooth function of
Q and is well fit by y_t=(1+Au+Bu^2)/(C+Du) where u=u(Q). For Q=3 we find
y_t~0.6; however if we include lattices up to L=12 we find y_t~0.50.Comment: to appear in Physical Review
Inflammation-driven bone formation in a mouse model of ankylosing spondylitis: sequential not parallel processes
Background\ud
\ud
Ankylosing spondylitis (AS) is an immune-mediated arthritis particularly targeting the spine and pelvis and is characterised by inflammation, osteoproliferation and frequently ankylosis. Current treatments that predominately target inflammatory pathways have disappointing efficacy in slowing disease progression. Thus, a better understanding of the causal association and pathological progression from inflammation to bone formation, particularly whether inflammation directly initiates osteoproliferation, is required.\ud
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Methods\ud
\ud
The proteoglycan-induced spondylitis (PGISp) mouse model of AS was used to histopathologically map the progressive axial disease events, assess molecular changes during disease progression and define disease progression using unbiased clustering of semi-quantitative histology. PGISp mice were followed over a 24-week time course. Spinal disease was assessed using a novel semi-quantitative histological scoring system that independently evaluated the breadth of pathological features associated with PGISp axial disease, including inflammation, joint destruction and excessive tissue formation (osteoproliferation). Matrix components were identified using immunohistochemistry.\ud
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Results\ud
\ud
Disease initiated with inflammation at the periphery of the intervertebral disc (IVD) adjacent to the longitudinal ligament, reminiscent of enthesitis, and was associated with upregulated tumor necrosis factor and metalloproteinases. After a lag phase, established inflammation was temporospatially associated with destruction of IVDs, cartilage and bone. At later time points, advanced disease was characterised by substantially reduced inflammation, excessive tissue formation and ectopic chondrocyte expansion. These distinct features differentiated affected mice into early, intermediate and advanced disease stages. Excessive tissue formation was observed in vertebral joints only if the IVD was destroyed as a consequence of the early inflammation. Ectopic excessive tissue was predominantly chondroidal with chondrocyte-like cells embedded within collagen type II- and X-rich matrix. This corresponded with upregulation of mRNA for cartilage markers Col2a1, sox9 and Comp. Osteophytes, though infrequent, were more prevalent in later disease.\ud
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Conclusions\ud
\ud
The inflammation-driven IVD destruction was shown to be a prerequisite for axial disease progression to osteoproliferation in the PGISp mouse. Osteoproliferation led to vertebral body deformity and fusion but was never seen concurrent with persistent inflammation, suggesting a sequential process. The findings support that early intervention with anti-inflammatory therapies will be needed to limit destructive processes and consequently prevent progression of AS
Chebyshev type lattice path weight polynomials by a constant term method
We prove a constant term theorem which is useful for finding weight
polynomials for Ballot/Motzkin paths in a strip with a fixed number of
arbitrary `decorated' weights as well as an arbitrary `background' weight. Our
CT theorem, like Viennot's lattice path theorem from which it is derived
primarily by a change of variable lemma, is expressed in terms of orthogonal
polynomials which in our applications of interest often turn out to be
non-classical. Hence we also present an efficient method for finding explicit
closed form polynomial expressions for these non-classical orthogonal
polynomials. Our method for finding the closed form polynomial expressions
relies on simple combinatorial manipulations of Viennot's diagrammatic
representation for orthogonal polynomials. In the course of the paper we also
provide a new proof of Viennot's original orthogonal polynomial lattice path
theorem. The new proof is of interest because it uses diagonalization of the
transfer matrix, but gets around difficulties that have arisen in past attempts
to use this approach. In particular we show how to sum over a set of implicitly
defined zeros of a given orthogonal polynomial, either by using properties of
residues or by using partial fractions. We conclude by applying the method to
two lattice path problems important in the study of polymer physics as models
of steric stabilization and sensitized flocculation.Comment: 27 pages, 14 figure
Phase-transitions induced by easy-plane anisotropy in the classical Heisenberg antiferromagnet on a triangular lattice: a Monte Carlo simulation
We present the results of Monte Carlo simulations for the antiferromagnetic
classical XXZ model with easy-plane exchange anisotropy on the triangular
lattice, which causes frustration of the spin alignment. The behaviour of this
system is similar to that of the antiferromagnetic XY model on the same
lattice, showing the signature of a Berezinskii-Kosterlitz-Thouless transition,
associated to vortex-antivortex unbinding, and of an Ising-like one due to the
chirality, the latter occurring at a slightly higher temperature. Data for
internal energy, specific heat, magnetic susceptibility, correlation length,
and some properties associated with the chirality are reported in a broad
temperature range, for lattice sizes ranging from 24x24 to 120x120; four values
of the easy-plane anisotropy are considered. Moving from the strongest towards
the weakest anisotropy (1%) the thermodynamic quantities tend to the isotropic
model behaviour, and the two transition temperatures decrease by about 25% and
22%, respectively.Comment: 11 pages, 13 figures (embedded by psfig), 3 table
Fisher zeros of the Q-state Potts model in the complex temperature plane for nonzero external magnetic field
The microcanonical transfer matrix is used to study the distribution of the
Fisher zeros of the Potts models in the complex temperature plane with
nonzero external magnetic field . Unlike the Ising model for
which has only a non-physical critical point (the Fisher edge singularity), the
Potts models have physical critical points for as well as the
Fisher edge singularities for . For the cross-over of the Fisher
zeros of the -state Potts model into those of the ()-state Potts model
is discussed, and the critical line of the three-state Potts ferromagnet is
determined. For we investigate the edge singularity for finite lattices
and compare our results with high-field, low-temperature series expansion of
Enting. For we find that the specific heat, magnetization,
susceptibility, and the density of zeros diverge at the Fisher edge singularity
with exponents , , and which satisfy the scaling
law .Comment: 24 pages, 7 figures, RevTeX, submitted to Physical Review
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