474 research outputs found
Confining Domains Lead to Reaction Bursts: Reaction Kinetics in the Plasma Membrane
- Author
- A Chaudhuri
- A Eldar
- A Kusumi
- A Kusumi
- A Mocciaro
- Akihiro Kusumi
- B Treanor
- D Fange
- D Goswami
- D Lingwood
- DV Nicolau
- E Melo
- G Wang
- H Grecco
- I Chung
- I Schoen
- IR Nabi
- JG Powles
- JJ Tyson
- JK Klarlund
- K Murase
- KG Suzuki
- KG Suzuki
- KM Lerea
- L Cai
- LD Landau
- M Mears
- M Riley
- MJ Saxton
- MN Costa
- MN Costa
- N Morone
- PD Calvert
- R Grima
- R Grima
- R Grima
- R Heinrich
- Roeland M. H. Merks
- RS Kasai
- S Krishnamurthy
- S Schnell
- SJ Singer
- T Fujiwara
- T Ryan
- Takahiro K. Fujiwara
- VM Kenkre
- Z Kalay
- Ziya Kalay
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2011
- Field of study
Confinement of molecules in specific small volumes and areas within a cell is likely to be a general strategy that is developed during evolution for regulating the interactions and functions of biomolecules. The cellular plasma membrane, which is the outermost membrane that surrounds the entire cell, was considered to be a continuous two-dimensional liquid, but it is becoming clear that it consists of numerous nano-meso-scale domains with various lifetimes, such as raft domains and cytoskeleton-induced compartments, and membrane molecules are dynamically trapped in these domains. In this article, we give a theoretical account on the effects of molecular confinement on reversible bimolecular reactions in a partitioned surface such as the plasma membrane. By performing simulations based on a lattice-based model of diffusion and reaction, we found that in the presence of membrane partitioning, bimolecular reactions that occur in each compartment proceed in bursts during which the reaction rate is sharply and briefly increased even though the asymptotic reaction rate remains the same. We characterized the time between reaction bursts and the burst amplitude as a function of the model parameters, and discussed the biological significance of the reaction bursts in the presence of strong inhibitor activity
Measurement of CP-violation asymmetries in D0 to Ks pi+ pi-
- Author
- Aaltonen T
- Ahmed S
- Alam MS
- Albert J
- Amerio S
- Amidei D
- Anastassov A
- Annovi A
- Antos J
- Apollinari G
- Appel JA
- Arisawa T
- Artikov A
- Asaadi J
- Ashmanskas W
- Auerbach B
- Aurisano A
- Azfar F
- Azzolini V
- Badgett W
- Bae T
- Band HR
- Banerjee S
- Barbaro-Galtieri A
- Barnes VE
- Barnett BA
- Barria P
- Bartos P
- Bauce M
- Bedeschi F
- Behari S
- Bellettini G
- Bellinger J
- Benjamin D
- Beretvas A
- Bhatti A
- Bhuyan B
- Bianchi F
- Bisello D
- Bizjak I
- Bland KR
- Blumenfeld B
- Bocci A
- Bodek A
- Bomben M
- Bortoletto D
- Bosisio L
- Boudreau J
- Boveia A
- Brigliadori L
- Bromberg C
- Brucken E
- Budagov J
- Budd HS
- Bula R
- Burchat PR
- Burkett K
- Busetto G
- Bussey P
- Buzatu A
- Calamba A
- Calancha C
- Camarda S
- Campanelli M
- Campbell M
- Canelli F
- Carls B
- Carlsmith D
- Carosi R
- Carrillo S
- Carron S
- Cartaro C
- Casal B
- Casarsa M
- Castro A
- Catastini P
- Cauz D
- Cavaliere V
- Cavalli-Sforza M
- Cerri A
- Cerrito L
- Chen X
- Chen YC
- Chertok M
- Chiarelli G
- Chlachidze G
- Chlebana F
- Cho K
- Chokheli D
- Chung WH
- Chung YS
- Ciocci MA
- Clark A
- Clarke C
- Compostella G
- Convery ME
- Conway J
- Corbo M
- Cordelli M
- Cossutti F
- Cox CA
- Cox DJ
- Crescioli F
- Cuevas J
- Culbertson R
- d'Ascenzo N
- d'Errico M
- D'Onofrio M
- da Costa JG
- Dagenhart D
- Dasu S
- Datta M
- de Barbaro P
- Dell'Orso M
- Della Ricca G
- Demortier L
- Deninno M
- Devoto F
- Di Canto A
- Di Ruzza B
- Dittmann JR
- Donati S
- Dong P
- Dorigo M
- Dorigo T
- Ebina K
- Eckmann R
- Edwards AJ
- Elagin A
- Eppig A
- Erbacher R
- Ernst JA
- Errede S
- Ershaidat N
- Eusebi R
- Farrington S
- Feindt M
- Fernandez JP
- Fernandez PM
- Field R
- Flanagan G
- Flood KT
- Forrest R
- Frank MJ
- Franklin M
- Freeman JC
- Funakoshi Y
- Furic I
- Gallinaro M
- Gallo F
- Gamba D
- Garcia JE
- Garfinkel AF
- Garosi P
- Gerberich H
- Gerchtein E
- Giagu S
- Giakoumopoulou V
- Giannetti P
- Gibson K
- Ginsburg CM
- Giokaris N
- Giromini P
- Giurgiu G
- Glagolev V
- Glenzinski D
- Gold M
- Goldin D
- Goldschmidt N
- Golossanov A
- Gomez G
- Gomez-Ceballos G
- Goncharov M
- Gonzalez BA
- Gonzalez O
- Gorelov I
- Goshaw AT
- Goulianos K
- Grinstein S
- Griso SP
- Grosso-Pilcher C
- Group RC
- Hahn SR
- Halkiadakis E
- Hamaguchi A
- Hamano K
- Han JY
- Happacher F
- Hara K
- Hare D
- Hare M
- Harr RF
- Harrison PF
- Hatakeyama K
- Hays C
- Heck M
- Heinrich J
- Herndon M
- Hewamanage S
- Hocker A
- Hollar JJ
- Hopkins W
- Horn D
- Hou S
- Hughes RE
- Hurwitz M
- Husemann U
- Hussain N
- Hussein M
- Huston J
- Ilic J
- Introzzi G
- Iori M
- Ivanov A
- Izen JM
- Jain V
- James E
- Jang D
- Jayatilaka B
- Jeon EJ
- Jindariani S
- Jones M
- Joo KK
- Jun SY
- Junk TR
- Kamon T
- Karchin PE
- Kasmi A
- Kato Y
- Ketchum W
- Keung J
- Khotilovich V
- Kilminster B
- Kim DH
- Kim HS
- Kim JE
- Kim MJ
- Kim P
- Kim SB
- Kim SH
- Kim YJ
- Kim YK
- Kimura N
- Kirby M
- Klimenko S
- Knoepfel K
- Kocian ML
- Kondo K
- Kong DJ
- Konigsberg J
- Kotwal AV
- Kowalewski R
- Kreps M
- Krishnamurthy M
- Kroll J
- Krop D
- Kruse M
- Krutelyov V
- Kuhr T
- Kurata M
- Kutter PE
- Kwang S
- Laasanen AT
- Lami S
- Lammel S
- Lancaster M
- Lanceri L
- Lander RL
- Lannon K
- Lath A
- Latham TE
- Latino G
- LeCompte T
- Lee E
- Lee HS
- Lee JS
- Lee SW
- Leith DWGS
- Leo S
- Leone S
- Lewis JD
- Li S
- Limosani A
- Lin CJ
- Lindgren M
- Lipeles E
- Lister A
- Litvintsev DO
- Liu C
- Liu H
- Liu Q
- Liu T
- Lockwitz S
- Loginov A
- Lopez-March N
- Lou XC
- Lucchesi D
- Lueck J
- Luitz S
- Lujan P
- Lukens P
- Lungu G
- Luth V
- Lynch HL
- Lys J
- Lysak R
- MacFarlane DB
- Madrak R
- Maeshima K
- Maestro P
- Majewski SA
- Malik S
- Manca G
- Manousakis-Katsikakis A
- Margaroli F
- Marino C
- Marsiske H
- Martinez M
- Martinez-Vidal F
- Mastrandrea P
- Matera K
- Mattson ME
- Mazzacane A
- Mazzanti P
- McFarland KS
- McIntyre P
- McNulty R
- Mehta A
- Mehtala P
- Mesropian C
- Messner R
- Miao T
- Mietlicki D
- Milanes DA
- Mitra A
- Miyake H
- Miyashita TS
- Moed S
- Moggi N
- Mohanty GB
- Mondragon MN
- Moon CS
- Moore R
- Morello MJ
- Morlock J
- Mukherjee A
- Muller DR
- Muller T
- Murat P
- Mussini M
- Nachtman J
- Nagai Y
- Naganoma J
- Nakano I
- Napier A
- Neal H
- Nett J
- Neu C
- Neubauer MS
- Nielsen J
- Nodulman L
- Noh SY
- Norniella O
- Nugent IM
- O'Grady CP
- Oakes L
- Ofte I
- Oh SH
- Oh YD
- Oksuzian I
- Okusawa T
- Orava R
- Ortolan L
- Oyanguren A
- Pagliarone C
- Palencia E
- Pan B
- Pan Y
- Papadimitriou V
- Paramonov AA
- Patrick J
- Pauletta G
- Paulini M
- Paus C
- Pellett DE
- Pelliccioni M
- Penzo A
- Perazzo A
- Perl M
- Petersen BA
- Phillips TJ
- Piacentino G
- Pianori E
- Pierini M
- Pilot J
- Pitts K
- Plager C
- Pondrom L
- Poprocki S
- Potamianos K
- Pranko A
- Prepost R
- Prokoshin F
- Ptohos F
- Pulliam T
- Punzi G
- Rahaman A
- Ramakrishnan V
- Ranjan N
- Ratcliff BN
- Redondo I
- Renton P
- Rescigno M
- Riddick T
- Rimondi F
- Ristori L
- Ritchie JL
- Robson A
- Rodrigo T
- Rodriguez T
- Rogers E
- Rolli S
- Roney JM
- Roodman A
- Roser R
- Ruffini F
- Ruiz A
- Ruland AM
- Russ J
- Rusu V
- Saeed MA
- Safonov A
- Sakumoto WK
- Sakurai Y
- Salnikov AA
- Santi L
- Sato K
- Saveliev V
- Savoy-Navarro A
- Schilling CJ
- Schindler RH
- Schlabach P
- Schmidt A
- Schmidt EE
- Schwarz T
- Schwiening J
- Schwitters RF
- Scodellaro L
- Scribano A
- Scuri F
- Seidel S
- Seiya Y
- Semenov A
- Sforza F
- Shalhout SZ
- Shears T
- Shepard PF
- Shimojima M
- Shochet M
- Shreyber-Tecker I
- Simonenko A
- Sinervo P
- Sliwa K
- Smith JR
- Snider FD
- Snyder A
- Sobie RJ
- Soha A
- Song H
- Sorin V
- Spanier SM
- Squillacioti P
- St Denis R
- Stancari M
- Stelzer B
- Stelzer-Chilton O
- Stentz D
- Strologas J
- Strycker GL
- Su D
- Sudo Y
- Sukhanov A
- Sullivan MK
- Suslov I
- Suzuki K
- Swain SK
- Takemasa K
- Takeuchi Y
- Tang J
- Tecchio M
- Teng PK
- Thom J
- Thome J
- Thompson GA
- Thompson JM
- Thomson E
- Toback D
- Tokar S
- Tollefson K
- Tomura T
- Tonelli D
- Torre S
- Torretta D
- Totaro P
- Trovato M
- Ukegawa F
- Uozumi S
- Va'vra J
- Varganov A
- Vazquez F
- Velev G
- Vellidis C
- Vidal M
- Vila I
- Vilar R
- Vitale L
- Vizan J
- Vogel M
- Volpi G
- Wagner AP
- Wagner P
- Wagner RL
- Wakisaka T
- Wallny R
- Wang SM
- Wappler FR
- Warburton A
- Waters D
- Weaver M
- Wester WC
- Whiteson D
- Wick F
- Wicklund AB
- Wicklund E
- Wilbur S
- Wilden L
- Williams HH
- Wilson JS
- Wilson P
- Winer BL
- Wisniewski WJ
- Wittgen M
- Wittich P
- Wolbers S
- Wolfe H
- Wright DH
- Wright T
- Wu SL
- Wu X
- Wu Z
- Yamamoto K
- Yamato D
- Yang T
- Yang UK
- Yang YC
- Yao WM
- Yarritu AK
- Ye S
- Yeh GP
- Yi K
- Yi K
- Yoh J
- Yorita K
- Yoshida T
- Young CC
- Yu GB
- Yu I
- Yu SS
- Yun JC
- Zain SB
- Zanetti A
- Zeng Y
- Zhou C
- Ziegler V
- Zucchelli S
- Publication venue
- 'American Physical Society (APS)'
- Publication date
- 01/01/2012
- Field of study
We report a measurement of time-integrated CP-violation asymmetries in the
resonant substructure of the three-body decay D0 to Ks pi+ pi- using CDF II
data corresponding to 6.0 invfb of integrated luminosity from Tevatron ppbar
collisions at sqrt(s) = 1.96 TeV. The charm mesons used in this analysis come
from D*+(2010) to D0 pi+ and D*-(2010) to D0bar pi-, where the production
flavor of the charm meson is determined by the charge of the accompanying pion.
We apply a Dalitz-amplitude analysis for the description of the dynamic decay
structure and use two complementary approaches, namely a full Dalitz-plot fit
employing the isobar model for the contributing resonances and a
model-independent bin-by-bin comparison of the D0 and D0bar Dalitz plots. We
find no CP-violation effects and measure an asymmetry of ACP = (-0.05 +- 0.57
(stat) +- 0.54 (syst))% for the overall integrated CP-violation asymmetry,
consistent with the standard model prediction.Comment: 15 page
Two Coregulated Efflux Transporters Modulate Intracellular Heme and Protoporphyrin IX Availability in Streptococcus agalactiae
- Author
- AD Baughn
- AH Klein
- AK Sijpesteijn
- Alexandra Gruss
- Annabelle Fernandez
- AP Kaur
- AS Seoane
- Aurélie Derré-Bobillot
- B Epe
- B Koebmann
- C Poyart
- CE Allen
- D Lim
- DB Friedman
- Delphine Lechardeur
- DL Stauff
- DL Stauff
- DW Ellison
- E Maguin
- EH Lee
- Elisabeth Couvé
- G Fiorentino
- G Lindahl
- GW Kaatz
- GW Kaatz
- I Biswas
- J Goris
- J Law
- J Sambrook
- JA Leigh
- JG Quigley
- K Poole
- KS Doran
- L Frankenberg
- L Rezaiki
- L Shi
- L Winstedt
- LA Bibb
- LM Blank
- M Brochet
- M el Karoui
- M Shepherd
- MA Gilles-Gonzalez
- MA Providenti
- MB Pedersen
- MC Lamy
- Michael R. Wessels
- MM Huycke
- MN Alekshun
- O Gaillot
- P Chomczynski
- P Duwat
- P Gaudu
- P Krishnamurthy
- P Krishnamurthy
- Philippe Gaudu
- PN Ward
- R Tatsumi
- RA Proctor
- RJ Brooijmans
- RJ Brooijmans
- S Cescau
- S Endley
- S Kumar
- S Letoffe
- SB Keel
- SC Andrews
- SM Mense
- SP Wilkinson
- SP Wilkinson
- SS Pao
- SS Way
- TW Ritchey
- VJ Torres
- Y Yamamoto
- Y Yamamoto
- YH Han
- Z Malik
- Z Qi
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2010
- Field of study
Streptococcus agalactiae is a major neonatal pathogen whose infectious route involves septicemia. This pathogen does not synthesize heme, but scavenges it from blood to activate a respiration metabolism, which increases bacterial cell density and is required for full virulence. Factors that regulate heme pools in S. agalactiae are unknown. Here we report that one main strategy of heme and protoporphyrin IX (PPIX) homeostasis in S. agalactiae is based on a regulated system of efflux using two newly characterized operons, gbs1753 gbs1752 (called pefA pefB), and gbs1402 gbs1401 gbs1400 (called pefR pefC pefD), where pef stands for ‘porphyrin-regulated efflux’. In vitro and in vivo data show that PefR, a MarR-superfamily protein, is a repressor of both operons. Heme or PPIX both alleviate PefR-mediated repression. We show that bacteria inactivated for both Pef efflux systems display accrued sensitivity to these porphyrins, and give evidence that they accumulate intracellularly. The ΔpefR mutant, in which both pef operons are up-regulated, is defective for heme-dependent respiration, and attenuated for virulence. We conclude that this new efflux regulon controls intracellular heme and PPIX availability in S. agalactiae, and is needed for its capacity to undergo respiration metabolism, and to infect the host
Modelling survival : exposure pattern, species sensitivity and uncertainty
- Author
- A Gergs
- A Gergs
- A-M Nyman
- A-M Nyman
- B Mihaylova
- BA Carnes
- C Albert
- C Ceconi
- CI Bliss
- CL Fordham
- D Kulkarni
- DB Buchwalter
- DR Fox
- EP Panel
- F Gabsi
- G Guo
- H Lu
- IK Wittmer
- J Baas
- J Berkson
- J De Bruijn
- J Stadnicka-Michalak
- JM Box-Steffensmeier
- KA Garrett
- KJ Groh
- KonKam King
- KR Solomon
- L Maltby
- LA Gavrilov
- M Moerbeek
- M Rubach
- MC Newman
- MD Poteat
- MN Rubach
- PPR EFSA
- R Ashauer
- R Ashauer
- R Ashauer
- R Ashauer
- R Ashauer
- R Ashauer
- R Beaudouin
- R Laskowski
- RD Chew
- S Raimondo
- SALM Kooijman
- SALM Kooijman
- SK Au
- SV Krishnamurthy
- T Jager
- T Jager
- T Jager
- T Jager
- T Jager
- T Jager
- VE Forbes
- WQ Meeker
- Y Zhao
- Z Ahmad
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2016
- Field of study
The General Unified Threshold model for Survival (GUTS) integrates previously published toxicokinetic-toxicodynamic models and estimates survival with explicitly defined assumptions. Importantly, GUTS accounts for time-variable exposure to the stressor. We performed three studies to test the ability of GUTS to predict survival of aquatic organisms across different pesticide exposure patterns, time scales and species. Firstly, using synthetic data, we identified experimental data requirements which allow for the estimation of all parameters of the GUTS proper model. Secondly, we assessed how well GUTS, calibrated with short-term survival data of Gammarus pulex exposed to four pesticides, can forecast effects of longer-term pulsed exposures. Thirdly, we tested the ability of GUTS to estimate 14-day median effect concentrations of malathion for a range of species and use these estimates to build species sensitivity distributions for different exposure patterns. We find that GUTS adequately predicts survival across exposure patterns that vary over time. When toxicity is assessed for time-variable concentrations species may differ in their responses depending on the exposure profile. This can result in different species sensitivity rankings and safe levels. The interplay of exposure pattern and species sensitivity deserves systematic investigation in order to better understand how organisms respond to stress, including humans
The FGGY carbohydrate kinase family : insights into the evolution of functional specificities
- Author
- A Osterman
- A Vendeville
- Adam Godzik
- AE Todd
- AE Todd
- AM Schnoes
- Andrei Osterman
- B Reva
- BE Engelhardt
- BG Magor
- CA Bonner
- CA Orengo
- Christos A. Ouzounis
- CM Seibert
- D Grueninger
- D Wu
- DA Lee
- DA Rodionov
- E Di Luccio
- G Casari
- GE Crooks
- HM Berman
- I Letunic
- Irina Rodionova
- JA Capra
- JA Capra
- JA Gerlt
- JH Hurley
- JH Hurley
- JI Yeh
- K Sjolander
- K Ye
- KB Xavier
- LA David
- M Ormo
- M Pachkov
- ME Glasner
- MN Price
- MV Omelchenko
- N Krishnamurthy
- Olga Zagnitko
- OV Kalinina
- P Shannon
- R Overbeek
- RC Edgar
- RC Edgar
- RD Finn
- RK Aziz
- S Cheek
- SS Hannenhalli
- TA Tatusova
- TT Nguyen
- W-D Fessner
- Y Zhang
- Ying Zhang
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 01/12/2011
- Field of study
© The Author(s), 2011. This article is distributed under the terms of the Creative Commons Attribution License. The definitive version was published in PLoS Computational Biology 7 (2011): e1002318, doi:10.1371/journal.pcbi.1002318.Function diversification in large protein families is a major mechanism driving expansion of cellular networks, providing organisms with new metabolic capabilities and thus adding to their evolutionary success. However, our understanding of the evolutionary mechanisms of functional diversity in such families is very limited, which, among many other reasons, is due to the lack of functionally well-characterized sets of proteins. Here, using the FGGY carbohydrate kinase family as an example, we built a confidently annotated reference set (CARS) of proteins by propagating experimentally verified functional assignments to a limited number of homologous proteins that are supported by their genomic and functional contexts. Then, we analyzed, on both the phylogenetic and the molecular levels, the evolution of different functional specificities in this family. The results show that the different functions (substrate specificities) encoded by FGGY kinases have emerged only once in the evolutionary history following an apparently simple divergent evolutionary model. At the same time, on the molecular level, one isofunctional group (L-ribulokinase, AraB) evolved at least two independent solutions that employed distinct specificity-determining residues for the recognition of a same substrate (L-ribulose). Our analysis provides a detailed model of the evolution of the FGGY kinase family. It also shows that only combined molecular and phylogenetic approaches can help reconstruct a full picture of functional diversifications in such diverse families.This study was funded by NIH and DOE grants
Post Genome-Wide Association Studies of Novel Genes Associated with Type 2 Diabetes Show Gene-Gene Interaction and High Predictive Value
- Author
- A Helgason
- AC Janssens
- AH Mokdad
- Anne-Claire Vergnaud
- Benjamin Glaser
- Beverley Balkau
- Christine Proença
- D Meyre
- David Meyre
- Don Husereau
- E Rampersaud
- E Zeggini
- Emmanuelle Durand
- F Chimienti
- F Chimienti
- Franck De Graeve
- Frederic Allegaert
- Guillaume Charpentier
- H Esterbauer
- H Oberkofler
- Hélène Choquet
- Ilana Blech
- J Krishnamurthy
- JC Florez
- Jon Wasson
- Jérôme Delplanque
- LJ Scott
- M Horikoshi
- Marshall Alan Permutt
- MB Schulze
- MG Hayes
- Michel Marre
- MN Weedon
- Mohamed Chikri
- N Grarup
- Philippe Froguel
- Q Yang
- Q Yang
- R Saxena
- R Sladek
- RL Hanson
- Robert Sladek
- S Cauchi
- S Hadjadj
- S Hercberg
- S Hercberg
- S Omori
- Samy Hadjadj
- Stefan Gaget
- Stéphane Cauchi
- Sébastien Czernichow
- T Koblas
- T Sing
- TM Frayling
- V Steinthorsdottir
- Wolfgang Patsch
- Y Furukawa
- Publication venue
- Public Library of Science
- Publication date
- 07/05/2008
- Field of study
Recently, several Genome Wide Association (GWA) studies in populations of European descent have identified and validated novel single nucleotide polymorphisms (SNPs), highly associated with type 2 diabetes (T2D). Our aims were to validate these markers in other European and non-European populations, then to assess their combined effect in a large French study comparing T2D and normal glucose tolerant (NGT) individuals. rs7903146 SNP, were combined (8.68-fold for the 14% of French individuals carrying 18 to 30 risk alleles with an allelic OR of 1.24). With an area under the ROC curve of 0.86, only 15 novel loci were necessary to discriminate French individuals susceptible to develop T2D. strongly associate with T2D in French individuals, and mostly in populations of Central European descent but not in Moroccan subjects. Genes expressed in the pancreas interact together and their combined effect dramatically increases the risk for T2D, opening avenues for the development of genetic prediction tests
The Human Serum Metabolome
- Author
- A Jiye
- A Wu
- A Zlatkis
- AA Spector
- AM Weljie
- An Chi Guo
- B Bicalho
- B Tan
- BF Cravatt
- Bijaya Gautam
- BL Ackermann
- Bruce McManus
- C Burtis
- C Lentner
- CA Burtis
- CA Daykin
- CA Smith
- CJ DeLong
- CN Serhan
- CR Pace-Asciak
- Craig Knox
- D Cheng
- D Pratico
- Darren Flower
- David D. Hau
- David S. Wishart
- DJ Merkler
- DL Wheeler
- DL Wheeler
- DM Lambert
- DS Wishart
- DS Wishart
- DS Wishart
- DS Wishart
- DS Wishart
- E Fahy
- E Zelena
- Edison Dong
- EE Carlson
- F Borrelli
- F Martini
- F Massa
- F Rossi
- FF Hsu
- FF Hsu
- Fiona Bamforth
- G Corso
- GC Shearer
- GC Shearer
- GH Grant
- GP Mueller
- H Martin
- HB Bradshaw
- HE Solberg
- I Beheshti
- Igor Sinelnikov
- J Folch
- JC Lindon
- Jianguo Xia
- JM Buscher
- JM Walker
- John W. Newman
- Jun Peng
- K Apel
- K Cottingham
- K Degtyarenko
- K Ekroos
- K Guo
- K Guo
- K Hollywood
- K Kuriki
- K Sandra
- K Yang
- KA Lawton
- KL Woo
- L Akoto
- L Bjerkenstedt
- L Hagenfeldt
- L Viinikka
- M Bou Khalil
- M Kanehisa
- M Koivusalo
- ML Smith
- MN Hill
- MP Gonthier
- MR Ninonuevo
- MR Wenk
- N Chiang
- Nelson Young
- Nikolaos Psychogios
- O Quehenberger
- P Caraceni
- P Chen
- P Kohli
- Paul Huang
- PJ Foxall
- PT Ivanova
- Q Cui
- Q Zhang
- R Grasbeck
- R Taguchi
- RA de Graaf
- RA Kekwick
- Ramanarayan Krishnamurthy
- Roman Eisner
- Rupasri Mandal
- Russ Greiner
- S Fischer
- S O'Hagan
- S Perwaiz
- S Pillarisetti
- S Tiziani
- S Watkins
- SI Fox
- SM Huang
- SN Whitehead
- Souhaila Bouatra
- Steven R. Smith
- T Sugiura
- Theodore Goodfriend
- Theresa L. Pedersen
- TP Carr
- VV Bezuglov
- W Wang
- X Han
- X Ye
- YI Shurubor
- Zsuzsanna Hollander
- Publication venue
- Public Library of Science
- Publication date
- 01/02/2011
- Field of study
Continuing improvements in analytical technology along with an increased interest in performing comprehensive, quantitative metabolic profiling, is leading to increased interest pressures within the metabolomics community to develop centralized metabolite reference resources for certain clinically important biofluids, such as cerebrospinal fluid, urine and blood. As part of an ongoing effort to systematically characterize the human metabolome through the Human Metabolome Project, we have undertaken the task of characterizing the human serum metabolome. In doing so, we have combined targeted and non-targeted NMR, GC-MS and LC-MS methods with computer-aided literature mining to identify and quantify a comprehensive, if not absolutely complete, set of metabolites commonly detected and quantified (with today's technology) in the human serum metabolome. Our use of multiple metabolomics platforms and technologies allowed us to substantially enhance the level of metabolome coverage while critically assessing the relative strengths and weaknesses of these platforms or technologies. Tables containing the complete set of 4229 confirmed and highly probable human serum compounds, their concentrations, related literature references and links to their known disease associations are freely available at http://www.serummetabolome.ca
Combination of searches for heavy spin-1 resonances using 139 fb−1 of proton-proton collision data at s = 13 TeV with the ATLAS detector
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- Zhemchugov A
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- Zimine NI
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- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- Springer Nature
- Publication date
- 19/04/2024
- Field of study
A combination of searches for new heavy spin-1 resonances decaying into different pairings of W, Z, or Higgs bosons, as well as directly into leptons or quarks, is presented. The data sample used corresponds to 139 fb−1 of proton-proton collisions at
= 13 TeV collected during 2015–2018 with the ATLAS detector at the CERN Large Hadron Collider. Analyses selecting quark pairs (qq, bb,
, and tb) or third-generation leptons (τν and ττ) are included in this kind of combination for the first time. A simplified model predicting a spin-1 heavy vector-boson triplet is used. Cross-section limits are set at the 95% confidence level and are compared with predictions for the benchmark model. These limits are also expressed in terms of constraints on couplings of the heavy vector-boson triplet to quarks, leptons, and the Higgs boson. The complementarity of the various analyses increases the sensitivity to new physics, and the resulting constraints are stronger than those from any individual analysis considered. The data exclude a heavy vector-boson triplet with mass below 5.8 TeV in a weakly coupled scenario, below 4.4 TeV in a strongly coupled scenario, and up to 1.5 TeV in the case of production via vector-boson fusion
Measurement of the tt¯tt¯ production cross section in pp collisions at √s=13 TeV with the ATLAS detector
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- Zeißner SV
- Zemaityte G
- Zeng JC
- Zenin O
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- Zerwas D
- Zgubič M
- Zhang B
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- Zhang G
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- Zhao P
- Zhao Y
- Zhao Z
- Zhemchugov A
- Zheng Z
- Zhong D
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- Zhu H
- Zhu HL
- Zhu J
- Zhu Y
- Zhuang X
- Zhukov K
- Zhulanov V
- Zieminska D
- Zimine NI
- Zimmermann S
- Ziolkowski M
- Zoccoli A
- Zoch K
- Zorbas TG
- Zormpa O
- Zou W
- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 16/11/2021
- Field of study
A measurement of four-top-quark production using proton-proton collision data at a centre-of-mass energy of 13 TeV collected by the ATLAS detector at the Large Hadron Collider corresponding to an integrated luminosity of 139 fb−1 is presented. Events are selected if they contain a single lepton (electron or muon) or an opposite-sign lepton pair, in association with multiple jets. The events are categorised according to the number of jets and how likely these are to contain b-hadrons. A multivariate technique is then used to discriminate between signal and background events. The measured four-top-quark production cross section is found to be 26+17−15 fb, with a corresponding observed (expected) significance of 1.9 (1.0) standard deviations over the background-only hypothesis. The result is combined with the previous measurement performed by the ATLAS Collaboration in the multilepton final state. The combined four-top-quark production cross section is measured to be 24+7−6 fb, with a corresponding observed (expected) signal significance of 4.7 (2.6) standard deviations over the background-only predictions. It is consistent within 2.0 standard deviations with the Standard Model expectation of 12.0 ± 2.4 fb
Measurements of Higgs bosons decaying to bottom quarks from vector boson fusion production with the ATLAS experiment at √=13TeV
- Author
- Aad G
- Abbott B
- Abbott DC
- Abeling K
- Abhayasinghe DK
- Abidi SH
- AbouZeid OS
- Abraham NL
- Abramowicz H
- Abreu H
- Abud AA
- Abulaiti Y
- Acharya BS
- Achkar B
- Adam L
- Adamczyk L
- Adamek L
- Adelman J
- Adiguzel A
- Adorni S
- Adye T
- Affolder AA
- Afik Y
- Agapopoulou C
- Agaras MN
- Aggarwal A
- Agheorghiesei C
- Aguilar-Saavedra JA
- Agullo PM
- Ahmad A
- Ahmadov F
- Ahmed WS
- Ahnen JV
- Ai X
- Aielli G
- Akatsuka S
- Akbiyik M
- Akilli E
- Akimov AV
- Al Khoury K
- Alberghi GL
- Albert J
- Alderweireldt S
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexopoulos T
- Alfonsi A
- Alfonsi F
- Alhroob M
- Ali B
- Ali S
- Aliev M
- Alimonti G
- Allaire C
- Allbrooke BMM
- Allport PP
- Aloisio A
- Alonso F
- Alpigiani C
- Alves FLL
- Alviggi MG
- Ambler A
- Ambroz L
- Amelung C
- Amidei D
- Amoroso S
- Amrouche CS
- Anastopoulos C
- Andari N
- Andeen T
- Anders JK
- Andrean SY
- Andreazza A
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- Butterworth JM
- Buttinger W
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- Chevalérias TJA
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- Chiu I
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- Ciocio A
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- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 23/06/2021
- Field of study
The paper presents a measurement of the Standard Model Higgs Boson decaying to b-quark pairs in the vector boson fusion (VBF) production mode. A sample corresponding to 126 fb−1 of s√=13TeV proton–proton collision data, collected with the ATLAS experiment at the Large Hadron Collider, is analyzed utilizing an adversarial neural network for event classification. The signal strength, defined as the ratio of the measured signal yield to that predicted by the Standard Model for VBF Higgs production, is measured to be 0.95+0.38−0.36 , corresponding to an observed (expected) significance of 2.6 (2.8) standard deviations from the background only hypothesis. The results are additionally combined with an analysis of Higgs bosons decaying to b-quarks, produced via VBF in association with a photon
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