9 research outputs found
An innate defense peptide BPIFA1/SPLUNC1 restricts influenza A virus infection
- Author
- A Garcia-Caballero
- A Kipar
- A Kipar
- A Portela
- A Schorlemmer
- B Ghafouri
- B Meng
- B Zhang
- C D Bingle
- C Ehre
- CD Bingle
- CD Bingle
- CD Bingle
- CD Bingle
- CD Bingle
- CJ Britto
- EE LeClair
- EE LeClair
- EE Leclair
- F Krammer
- F Ning
- G H Leeming
- G McGillivary
- G Radicioni
- GH Leeming
- H Zaraket
- HD Zhou
- HW Chu
- HW Chu
- J P Stewart
- JA Whitsett
- JJ Skehel
- JP Stewart
- JV Thaikoottathil
- K M Akram
- L Bingle
- L Bingle
- L Gakhar
- L Huang
- LM Teran
- M Kesimer
- M Matrosovich
- M Musa
- N A Moyo
- NM Bouvier
- P Digard
- R A Tripp
- R V Shohet
- S Boulo
- S Hussain
- S Jasim
- SA Wharton
- V Alva
- WM Weston
- Y He
- Y Liu
- Y You
- Y You
- YP Di
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2017
- Field of study
The airway epithelium secretes proteins that function in innate defense against
infection. BPI fold-containing family member A1 (BPIFA1) is secreted into airways
and has a protective role during bacterial infections, but it is not known whether it
also has an antiviral role. To determine a role in host defense against influenza A
virus (IAV) infection and to find the underlying defense mechanism we developed
transgenic mouse models that are deficient in BPIFA1 and used these, in
combination with in vitro 3D mouse tracheal epithelial cell (mTEC) cultures, to
investigate its antiviral properties. We show that BPIFA1 has a significant role in
mucosal defense against IAV infection. BPIFA1 secretion was highly modulated after
IAV infection. Mice deficient in BPIFA1 lost more weight after infection, supported a
higher viral load and virus reached the peripheral lung earlier, indicative of a defect
in the control of infection. Further analysis using mTEC cultures showed that
BPIFA1-deficient cells bound more virus particles, displayed increased nuclear
import of IAV ribonucleoprotein complexes and supported higher levels of viral
replication. Our results identify a critical role for BPIFA1 in the initial phase of
infection by inhibiting the binding and entry of IAV into airway epithelial cells
Expression of CCSP and SPLUNC1, Putative Anti-inflammatory Proteins, Following Murine Respiratory Viral Infection
- Publication venue
- Publication date
- 01/01/2012
- Field of study
Introducing forensic entomology in cases of suspect animal neglect
- Publication venue
- 'BMJ'
- Publication date
- 20/12/2017
- Field of study
The role of M1 protein in the acute infection of wood mice with MHV-68 (Murid herpesvirus 4)
- Publication venue
- Publication date
- 01/01/2009
- Field of study
Transgenic plants are an alternative to oily fish for providing omega-3 polyunsaturated fatty acids in the human diet: A summary of the findings of a BBSRC funded project
- Author
- Publication venue
- Publication date
- 01/01/2011
- Field of study
The n-3 polyunsaturated fatty acids (PUFA) present primarily in oily fish, namely eicosapentaenoic acid (EPA) and docosahexaenoic acid (DHA), are important components of cell membranes and are needed for normal development and cell function. Humans have very limited capacity for EPA and DHA synthesis from α-linolenic acid and so they must be obtained preformed from the diet. However, perceived unpalatability of oily fish and fish oil, concerns about contamination with environmental pollutants, dietary choices that exclude fish and animal products, and price limit the effectiveness of recommendations for EPA and DHA intakes. Moreover, marine sources of EPA and DHA are diminishing in the face of increasing demands. Therefore, an alternative source of EPA and DHA is needed that is broadly acceptable, can be upscaled and is sustainable. This review discusses these challenges and, using findings from recent nutritional trials, explains how they may be overcome by seed oils from transgenic plants engineered to produce EPA and DHA. Trials in healthy men and women assessed the acute uptake and appearance in blood over 8 hours of EPA and DHA from transgenic Camelina sativa compared to fish oil, and the incorporation of these PUFA into blood lipids after dietary supplementation. The findings showed that postprandial EPA and DHA incorporation into blood lipids and accumulation in plasma lipids after dietary supplementation was as good as that achieved with fish oil. The oil derived from this transgenic plant was well tolerated. This review also discusses the implications for human nutrition, marine ecology and agriculture
Appropriate antibiotic therapy improves Ureaplasma sepsis outcome in the neonatal mouse
- Author
- A Berger
- A Matlow
- AD Heggie
- Angela H. Leeming
- B Katz
- DC Kasper
- F Brus
- F Kong
- GH Cassell
- GH Cassell
- GS Pinna
- HO Ballard
- HO Ballard
- I Morioka
- J Yi
- KB Waites
- KB Waites
- KB Waites
- KB Waites
- KB Waites
- Leonard E. Weisman
- Lingkun Kong
- LT Fonseca
- M Kim
- MA Martínez
- ML Beeton
- MR Bayraktar
- P Zarogoulidis
- PT Rudd
- R Ozdemir
- RJ Baier
- RL Goldenberg
- RM Viscardi
- S Sethi
- SA Walls
- ST Tay
- V Clifford
- X Zheng
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Relationships between Religion and Two Forms of Homonegativity in Europe—A Multilevel Analysis of Effects of Believing, Belonging and Religious Practice
- Author
- A Adamczyk
- A Agadjanian
- A Crocket
- AL Whitehead
- AL Whitehead
- AM Burdette
- B Altemeyer
- B Griffiths
- B Laythe
- B Rye
- BC Hayes
- BC Hayes
- BC Hayes
- C Bader
- C Plugge-Foust
- D Hervieu-Leger
- D Pollack
- D Voas
- DA Clairmont
- DA Leeming
- DS Ferguson
- E Durkheim
- E Pace
- F Asbrock
- FR Ammaturo
- G Davie
- G Mavrogordatos
- GH Weinberg
- GM Fitzmaurice
- GM Herek
- GM Herek
- GM Herek
- GW Allport
- H Goldstein
- H Goldstein
- H Tajfel
- H van den Akker
- I Iveković
- I Szalma
- J Cesari
- J Chen
- J Gerhards
- J Loftus
- J Ohlander
- J Puar
- J Puar
- J Ritchie
- J Symons
- J Takács
- J Takács
- J Van Assche
- JP Schwartz
- JPD Cecco
- JR Seul
- JRJ Fontaine
- K Beatty
- K Rios
- L Quillian
- L Wylie
- LA Kirkpatrick
- M Taqi-ud-Din Al-Hilali
- MA Eisenstein
- Martin Voracek
- MD Regnerus
- MO Emerson
- MR Lee
- MS Rye
- O’Neil James
- P Froese
- P Froese
- P Nannestad
- P Norris
- P Norris
- P Scheepers
- P Scheepers
- R Andersen
- R Inglehart
- R Inglehart
- R Inglehart
- R Mole
- R Sona
- R Stark
- R Stark
- R Stark
- R Traunmüller
- R Wilkinson
- RD Putnam
- RD Woodberry
- RG Wilkinson
- RJ Duck
- SA Stouffer
- SE Bogardus
- SP Huntington
- Stefanie Doebler
- T Glendinning
- T Marsh
- TF Pettigrew
- VN Parrillo
- W Mayfield
- WJ Serdahely
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- Field of study
Dermal Pathology of Superficial Fungus Infections
- Author
- AA Strachan
- AC Allen
- AG Coiscou
- AG Franks
- AG Franks
- AGE Pearse
- AH Foresman
- AH Mehregan
- AH Mehregan
- AH Slepyan
- AL Weiner
- AM Kligman
- AM Kligman
- AM Kligman
- AM Kligman
- AN Chakraborty
- AR Birt
- BD Davis
- BJ Reid
- BK Fisher
- BM Partridge
- BM Partridge
- CF Burgoon Jr.
- CF Burgoon Jr.
- CF Merwin
- CG Lane
- CJ White
- CT Bishop
- CT Bishop
- CT Bishop
- CT Bishop
- CT Bishop
- CW Emmons
- D Gruby
- D Majocchi
- D Majocchi
- D Munro-Ashman
- D Taplin
- DE Temple
- DI Williams
- DL McCaffree
- DWR Mackenzie
- EB Ritchie
- EH Mercer
- F Blank
- F Blank
- F Blank
- F Blank
- F Blank
- F Blank
- F Blank
- F Blank
- F Blank
- F Blank
- F Blank
- F Blank
- F Blank
- F Keddie
- F Reiss
- F Sagher
- FF Hellier
- FV Hauser
- G Bauslaugh
- G Cremer
- G Daniels
- G Hildick-Smith
- GC Ainsworth
- GH Percival
- GM Lewis
- GM Lewis
- GM Lewis
- GM Lewis
- H Blank
- H Blank
- H Blank
- H Blank
- H Blank
- H Götz
- H Götz
- H Götz
- H Montgomery
- H Neves
- H VanVelsor
- HA Luscombe
- HG Adamson
- HL Saferstein
- HL Saferstein
- HL Saferstein
- HS Yaffee
- I Sarkany
- I Sarkany
- I Sarkany
- I Sarkany
- I Sarkany
- J Shanon
- JA Johnson
- JA Neves
- JA Neves
- JA Tolmach
- JAL Leeming
- JC Patterson
- JC Wirth
- JC Wirth
- JC Wirth
- JF Daly
- JG Smith Jr.
- JH Graham
- JH Graham
- JH Graham
- JH Harris
- JH Sequeira
- JH Swartz
- JL Callaway
- JL Cooper
- JL Pipkin
- JM Brock
- JM Hitch
- JM Hitch
- JP Pickett
- JW Rippon
- JW Rippon
- JW Rippon
- JW Rippon
- JW Wilson
- JW Wilson
- JW Wilson
- KH Burdick
- KM Halprin
- KW Thompson
- KW Thompson
- L Ajello
- L Kapica
- L Kapica
- LF Montes
- LF Montes
- LK Georg
- LK Georg
- LM Nelson
- M Langeron
- M Moore
- M Waisman
- MA Gordon
- MF Gridley
- MJ Marples
- MJ Scott
- MM Schreiber
- MP English
- MRJ Salton
- NF Conant
- O Canizares
- O Fischman
- O Gans
- O Male
- O Male
- OF Jillson
- P Telner
- PE Weary
- PE Weary
- PE Weary
- PH Manson-Bahr
- PJ Imperato
- PJ Koblenzer
- R Michalowski
- RCS Audette
- RD Baker
- RD Lillie
- RD Lillie
- RDG Simons
- RG Grocott
- RJ Myers
- RJ Yu
- RJA Sabouraud
- RL Baer
- RNB MacKenna
- RVP Hutter
- RW Riddell
- S Ayres Jr.
- S Hanusova
- S Hanusova
- S Hanusova
- S Rothman
- S Rothman
- SC Desai
- SC Desai
- SF Grappel
- SM Peck
- T Butterworth
- TH Sternberg
- TP Kugelman
- VD Newcomer
- VD Newcomer
- W Kaplan
- W Kaplan
- WB Shelley
- WC Johnson
- WE Weber
- WF Lever
- WJ Miles
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/1971
- Field of study
Mapping the human genetic architecture of COVID-19
- Author
- Abdelrazik M
- Abdullah T
- Abe R
- Abe S
- Abecasis GR
- Abel L
- Abernathy C
- Abraheem A
- Abul-Husn NS
- Acosta-Herrera M
- Acquilini D
- Acquilini D
- Adachi T
- Adachi Y
- Adams C
- Adams K
- Adanini O
- Adeleye O
- Adeniji K
- Adra D
- Afifi N
- Afilalo J
- Afilalo M
- Afolabi D
- Afrasiabi Z
- Afset JE
- Agasou A
- Aghemo A
- Agranoff D
- Agrawal S
- Aguirre LA
- Agwuh K
- Ahmad HF
- Ahmad N
- Ahmed A
- Ahmed C
- Ahmed KA
- Ai M
- Ail D
- Akeroyd L
- Akhtar MN
- Akhtar S
- Akinkugbe O
- Aksentijevich A
- Al Thani A
- Al-Muftah W
- Al-Sarraj Y
- Alarcon C
- Alarcon-Riquelme ME
- Alasdair F
- Alaverdian D
- Alavere H
- Albertos R
- Albillos A
- Albrecht W
- Albrich W
- Albrich WC
- Aldera EL
- Aldridge J
- Alegria A
- Alex B
- Alexander P
- Alfonso J
- Algera AG
- Ali A
- Ali IMA
- Ali S
- Aliberti S
- Allan A
- Allan E
- Allan J
- Alldis Z
- Allen L
- Allen S
- Allibone S
- Allison KS
- Allos R
- Ally SM
- Almaden-Boyle C
- Altabaibeh A
- Altmueller J
- Alvaro A
- Amar D
- Amin V
- Amitrano S
- Amiya S
- Ampuero J
- Anan R
- Anania S
- Anastasescu E
- Anderson F
- Anderson J
- Anderson M
- Anderson P
- Anderson S
- Anderson S
- Anderson T
- Ando A
- Andolfo I
- Andrade V
- Andretta F
- Andreucci E
- Andreucci E
- Andrew A
- Andrew B
- Andrew G
- Andrews E
- Andrews SJ
- Andrews SJ
- Andrews SJ
- Andrews SJ
- Andrikopoulos P
- Anedda F
- Aneli S
- Anemoli V
- Angelini C
- Angus B
- Annis A
- Antcliffe D
- Antinori A
- Antoni MD
- Anumakonda V
- Anwar M
- Anzai T
- Apetri E
- Arai D
- Arana E
- Arbane G
- Archer K
- Arciero E
- Arden T
- Arias SS
- Arif S
- Arimura K
- Armstrong J
- Armstrong JA
- Armstrong L
- Armstrong R
- Arning N
- Arnold S
- Arora J
- Artuso R
- Arumugam P
- Asakura T
- Asano K
- Aschard H
- Ascolillo S
- Ashish A
- Ashley E
- Ashraf S
- Ashraf S
- Ashton L
- Ashworth M
- Asiimwe IG
- Aslibekyan S
- Asselta R
- Atanasovska B
- Atkinson D
- Atkinson EG
- Attwood B
- Aucella F
- Augustin M
- Auld D
- Auld F
- Austin K
- Austin P
- Auton A
- Auton A
- Avendano-Ortiz J
- Awano N
- Ayers A
- Azarzar S
- Aziz F
- Azuma M
- Azuure C
- Azzolini E
- Baba R
- Baba T
- Bach B
- Bacher P
- Bachetti T
- Badalamenti S
- Badia JR
- Badji RM
- Bahmer T
- Baikady RR
- Baillie JK
- Baillie JK
- Baillie JK
- Baillie JK
- Baines B
- Baines D
- Baines K
- Baird T
- Baird Y
- Bakshi S
- Bakthavatsalam D
- Balaconis MK
- Baldassarri M
- Baldini M
- Ball CA
- Bals R
- Baltzell AH
- Bamford A
- Bamford P
- Banach D
- Banagan J
- Banales JM
- Bancroft H
- Band G
- Bandera A
- Bandini M
- Bandla N
- Bano S
- Baptista D
- Barada O
- Baras A
- Baratti S
- Barber R
- Barbieri C
- Barbieri C
- Barbour A
- Barclay L
- Barclay WS
- Barda B
- Bargagli E
- Bari S
- Barker J
- Barlow G
- Barlow SL
- Barnass S
- Barnes KC
- Baroni S
- Baroni S
- Barreira A
- Barrett F
- Barrett KMS
- Barrett N
- Barron A
- Barry P
- Bartels M
- Bartley S
- Baruah R
- Baselli G
- Bashyal A
- Basikolo C
- Bassetti M
- Bassford C
- Basso MEF
- Bastion V
- Basude S
- Bates H
- Bates M
- Battle C
- Bauchmuller K
- Bax D
- Baxter D
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- Baya N
- Bayo L-A
- Beadle J
- Beadsworth M
- Beale R
- Bean S
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- Beekes M
- Beesley K
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- Beith CM
- Belagodu Z
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- Belfield H
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- Beligni G
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- Bellinghausen C
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- Below JE
- Bemand T
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- Bercades G
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- Bewley J
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- Board S
- Bocciardi R
- Bocciolone M
- Bochud M
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- Bociek A
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- Bogaard HJ
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- Boillat-Blanco N
- Bokhari M
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- Butte MJ
- Butterworth AS
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- Buxbaum JD
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- Chassot J
- Chaudhary K
- Chavan S
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- Chernitsov A
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- Cheyne C
- Chiba S
- Chihara Y
- Child J
- Childs D
- Chittoor G
- Chiu A
- Cho JH
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- Choe K-W
- Choi S
- Chouchane O
- Choudhury Y
- Chowdhury M
- Christle J
- Chubachi S
- Chueca N
- Chukkambotla S
- Chukkambotla S
- Chung R
- Chung W
- Churchhouse C
- Chwialkowska K
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- Ciccarelli M
- Ciesek S
- Cirulli ET
- Claassen S
- Claire W
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- Clapham M
- Claringbould A
- Clarinval M
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- Clement I
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- Coetzee S
- Coghlan P
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- Conyngham J-A
- Cooke GS
- Cooper J
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- Corcoran E
- Cordero AIH
- Cordioli M
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- Cordioli M
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- Wyllie DH
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- Zhen J
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- Zoller H
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- zu Bentrup FM
- Zucchi P
- Zwinderman AHK
- Zyndorf M
- Publication venue
- Publication date
- 01/01/2021
- Field of study
The genetic make-up of an individual contributes to the susceptibility and response to viral infection. Although environmental, clinical and social factors have a role in the chance of exposure to SARS-CoV-2 and the severity of COVID-191,2, host genetics may also be important. Identifying host-specific genetic factors may reveal biological mechanisms of therapeutic relevance and clarify causal relationships of modifiable environmental risk factors for SARS-CoV-2 infection and outcomes. We formed a global network of researchers to investigate the role of human genetics in SARS-CoV-2 infection and COVID-19 severity. Here we describe the results of three genome-wide association meta-analyses that consist of up to 49,562 patients with COVID-19 from 46 studies across 19 countries. We report 13 genome-wide significant loci that are associated with SARS-CoV-2 infection or severe manifestations of COVID-19. Several of these loci correspond to previously documented associations to lung or autoimmune and inflammatory diseases3,4,5,6,7. They also represent potentially actionable mechanisms in response to infection. Mendelian randomization analyses support a causal role for smoking and body-mass index for severe COVID-19 although not for type II diabetes. The identification of novel host genetic factors associated with COVID-19 was made possible by the community of human genetics researchers coming together to prioritize the sharing of data, results, resources and analytical frameworks. This working model of international collaboration underscores what is possible for future genetic discoveries in emerging pandemics, or indeed for any complex human disease