1,930 research outputs found

    The completion of the General Magnetic Survey of Australia by the Carnegie Institution of Washington

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    The plan of the General Magnetic Survey of Australia by the Department of Terrestrial Magnetism of the Carnegie Institution of Washington was explained in a paper read by the author before the Australasian Association at Melbourne in 1913. As there stated, the object was to secure approximately one station for every 10,000 square miles of territory, or about 300 stations in all, with a uniform distribution over the Continent. Lack of facilities for travelling over large areas of the interior, of course, prevented the execution of this plan in its entirety, but the number and distribution of the stations established by the close of the survey in November, 1914, may be considered very satisfactory under the circumstances. The finally accepted results are given in the appended table, which is self explanatory. Some have already been published in the volumes of the Department of Terrestrial Magnetism, where descriptions of stations will also be found, but some have not yet appeared in print

    A Station-Based Southern Annular Mode Index from 1884 to 2005

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    Atmospheric pressure observations from the Southern Hemisphere are used to estimate monthly and annually averaged indexes of the southern annular mode (SAM) back to 1884. This analysis groups all relevant observations in the following four regions: one for Antarctica and three in the subtropical zone. Continuous surface pressure observations are available at a number of locations in the subtropical regions since the end of the nineteenth century. However, year-round observations in the subpolar region near the Antarctic continent began only during the 1940-60 period. The shorter Antarctic records seriously compromise the length of a traditionally estimated SAM index. To improve the situation "proxy'' estimates of Antarctic sea level pressure anomalies are provided based on the concept of atmospheric mass conservation poleward of 208S. This allows deriving a longer SAM index back to 1884. Several aspects of the new record, its statistical properties, seasonal trends, and the regional pressure anomaly correlations, are presented

    Quaker Pacifism during the Irish Revolution

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    Finite automata and composite realisations.

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    SIGLEAvailable from British Library Document Supply Centre- DSC:D34350/81 / BLDSC - British Library Document Supply CentreGBUnited Kingdo

    The Evolution of Clef Signatures

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    A Review of Obstetric Practice: With Deductions

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    "Bessy Bell" and a Welsh Air

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    Phenotypic and molecular assessment of seven patients with 6p25 deletion syndrome: Relevance to ocular dysgenesis and hearing impairment

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    BACKGROUND: Thirty-nine patients have been described with deletions involving chromosome 6p25. However, relatively few of these deletions have had molecular characterization. Common phenotypes of 6p25 deletion syndrome patients include hydrocephalus, hearing loss, and ocular, craniofacial, skeletal, cardiac, and renal malformations. Molecular characterization of deletions can identify genes that are responsible for these phenotypes. METHODS: We report the clinical phenotype of seven patients with terminal deletions of chromosome 6p25 and compare them to previously reported patients. Molecular characterization of the deletions was performed using polymorphic marker analysis to determine the extents of the deletions in these seven 6p25 deletion syndrome patients. RESULTS: Our results, and previous data, show that ocular dysgenesis and hearing impairment are the two most highly penetrant phenotypes of the 6p25 deletion syndrome. While deletion of the forkhead box C1 gene (FOXC1) probably underlies the ocular dysgenesis, no gene in this region is known to be involved in hearing impairment. CONCLUSIONS: Ocular dysgenesis and hearing impairment are the two most common phenotypes of 6p25 deletion syndrome. We conclude that a locus for dominant hearing loss is present at 6p25 and that this locus is restricted to a region distal to D6S1617. Molecular characterization of more 6p25 deletion patients will aid in refinement of this locus and the identification of a gene involved in dominant hearing loss

    INTERACTIONS OF HORMONAL STEROIDS WITH NUCLEIC ACIDS, I. A SPECIFIC REQUIREMENT FOR GUANINE

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