Aspects of radiative K^+_e3 decays

We re-investigate the radiative charged kaon decay K+- --> pi0 e+- nu_e gamma in chiral perturbation theory, merging the chiral expansion with Low's theorem. We thoroughly analyze the precision of the predicted branching ratio relative to the non-radiative decay channel. Structure dependent terms and their impact on differential decay distributions are investigated in detail, and the possibility to see effects of the chiral anomaly in this decay channel is emphasized.Comment: 15 pages, 6 figure

Scalar form factors of light mesons

The scalar radius of the pion plays an important role in CHPT, because it is related to one of the basic effective coupling constants, viz. the one which controls the quark mass dependence of F_pi at one loop. In a recent paper, Yndurain derives a {\it robust lower bound} for this radius, which disagrees with earlier determinations. We show that such a bound does not exist: the "derivation" relies on an incorrect claim. Moreover, we discuss the physics of the form factors associated with the operators \ubar u, \dbar d and \sbar s and show that their structure in the vicinity of the K \Kbar threshold is quite different. Finally, we draw attention to the fact that the new data on the slope of the scalar K_l3 form factor confirm a recent, remarkably sharp theoretical prediction.Comment: 13 pages, 2 figures. v2: minor changes - version to appear on Phys. Lett. B. v3: Published version. Values of the measured K-pi radii correcte

Experimental study of direct photon emission in K- --> pi- pi0 gamma decay using ISTRA+ detector

The branching ratio in the charged-pion kinetic energy region of 55 to 90 MeV for the direct photon emission in the K- --> pi- pi0 gamma decay has been measured using in-flight decays detected with the ISTRA+ setup operating in the 25 GeV/c negative secondary beam of the U-70 PS. The value Br(DE)=[0.37+-0.39(stat)+-0.10(syst)]*10^(-5) obtained from the analysis of 930 completely reconstructed events is consistent with the average value of two stopped-kaon experiments, but it differs by 2.5 standard deviations from the average value of three in-flight-kaon experiments. The result is also compared with recent theoretical predictions.Comment: 13 pages, 8 figure

Gold-plated Mode of CP-Violation in Decays of B_c Meson from QCD Sum Rules

The model-independent method based on the triangle ideology is implemented to extract the CKM-matrix angle gamma in the decays of doubly heavy long-lived meson B_c. We analyze a color structure of diagrams and conditions to reconstruct two reference-triangles by tagging the flavor and CP eigenstates of D^0 meson in the fixed exclusive channels. The characteristic branching ratios are evaluated in the framework of QCD sum rules.Comment: 11 pages, RevTeX4 file, 4 eps-figure

Photoswitchable diacylglycerols enable optical control of protein kinase C.

Increased levels of the second messenger lipid diacylglycerol (DAG) induce downstream signaling events including the translocation of C1-domain-containing proteins toward the plasma membrane. Here, we introduce three light-sensitive DAGs, termed PhoDAGs, which feature a photoswitchable acyl chain. The PhoDAGs are inactive in the dark and promote the translocation of proteins that feature C1 domains toward the plasma membrane upon a flash of UV-A light. This effect is quickly reversed after the termination of photostimulation or by irradiation with blue light, permitting the generation of oscillation patterns. Both protein kinase C and Munc13 can thus be put under optical control. PhoDAGs control vesicle release in excitable cells, such as mouse pancreatic islets and hippocampal neurons, and modulate synaptic transmission in Caenorhabditis elegans. As such, the PhoDAGs afford an unprecedented degree of spatiotemporal control and are broadly applicable tools to study DAG signaling

Observation of the radiative kaon decay K- --> mu-pi0gammanu

Using data collected with the ISTRA+ spectrometer during the 2001 run of the U-70 proton synchrotron in Protvino, we report the first observation of the radiative kaon decay K- -->mu-pi0gammanu. We find Br(Kmu3g)/Br(Kmu3) for two regions 5<Eg<30 and 30<Eg<60 MeV consistent with theoretical predictions. The measured angular distribution asymmetry for the first region, 0.093+/-0.141, is two standard deviations away from the theoretical prediction of 0.354. The measured asymmetry in the T-odd variable is -0.03+/-0.13.Comment: 15 pages, 8 figurs, submitted to Yadernaya Fizik

Determination of the b quark mass at the M_Z scale with the DELPHI detector at LEP

An experimental study of the normalized three-jet rate of b quark events with respect to light quarks events (light= \ell \equiv u,d,s) has been performed using the CAMBRIDGE and DURHAM jet algorithms. The data used were collected by the DELPHI experiment at LEP on the Z peak from 1994 to 2000. The results are found to agree with theoretical predictions treating mass corrections at next-to-leading order. Measurements of the b quark mass have also been performed for both the b pole mass: M_b and the b running mass: m_b(M_Z). Data are found to be better described when using the running mass. The measurement yields: m_b(M_Z) = 2.85 +/- 0.18 (stat) +/- 0.13 (exp) +/- 0.19 (had) +/- 0.12 (theo) GeV/c^2 for the CAMBRIDGE algorithm. This result is the most precise measurement of the b mass derived from a high energy process. When compared to other b mass determinations by experiments at lower energy scales, this value agrees with the prediction of Quantum Chromodynamics for the energy evolution of the running mass. The mass measurement is equivalent to a test of the flavour independence of the strong coupling constant with an accuracy of 7 permil.Comment: 24 pages, 10 figures, Accepted by Eur. Phys. J.

Implications of MMP9 for Blood Brain Barrier Disruption and Hemorrhagic Transformation Following Ischemic Stroke.

Numerous studies have documented increases in matrix metalloproteinases (MMPs), specifically MMP-9 levels following stroke, with such perturbations associated with disruption of the blood brain barrier (BBB), increased risk of hemorrhagic complications, and worsened outcome. Despite this, controversy remains as to which cells release MMP-9 at the normal and pathological BBB, with even less clarity in the context of stroke. This may be further complicated by the influence of tissue plasminogen activator (tPA) treatment. The aim of the present review is to examine the relationship between neutrophils, MMP-9 and tPA following ischemic stroke to elucidate which cells are responsible for the increases in MMP-9 and resultant barrier changes and hemorrhage observed following stroke