37 research outputs found
The role of atmospheric deposition in the biogeochemistry of the Mediterranean Sea
Estimates of atmospheric inputs to the Mediterranean (MED) and some coastal areas are reviewed, and uncertainities in these estimates considered. Both the magnitude and the mineralogical composition of atmospheric dust inputs indicate that eolian deposition is an important (50%) or prevailing (>80%) contribution to sediments in the offshore waters of the entire Mediterranean (MED) basin. Model data for trace metals and nutrients indicate that the atmosphere delivers more than half the lead and nitrogen, one-third of total phosphorus, and 10% of the zinc entering the entire basin. Measured data in sub-basins, such as the north-western MED and northern Adriatic indicate an even greater proportion of atmospheric versus riverine inputs. When dissolved fluxes are compared (the form most likely to impinge on surface water biogeochemical cycles), the atmosphere is found to be 5 to 50 times more important than rivers for dissolved Zn and 15 to 30 times more important for Pb fluxes. Neglecting co-limitation by other nutrients, new production supported by atmospheric nitrogen deposition ranges from 2-4 g C m-2 yr-1, whereas atmospheric phosphorus deposition appears to support less than 1 g C m-2 yr-1. In spite of the apparently small contribution of atmospheric deposition to overall production in the basin it has been suggested that certain episodes of phytoplankton blooms are triggered by atmospheric deposition of N, P or Fe. Future studies are needed to clarify the extent and causal links between these episodic blooms and atmospheric/oceanographic forcing functions. A scientific program aimed at elucidating the possible biogeochemical effects of Saharan outbreaks in the MED through direct sampling of the ocean and atmosphere before and after such events is therefore highly recommended
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A terminal selector prevents a Hox transcriptional switch to safeguard motor neuron identity throughout life
To become and remain functional, individual neuron types must select during development and maintain throughout life their distinct terminal identity features, such as expression of specific neurotransmitter receptors, ion channels and neuropeptides. Here, we report a molecular mechanism that enables cholinergic motor neurons (MNs) in the C. elegans ventral nerve cord to select and maintain their unique terminal identity. This mechanism relies on the dual function of the conserved terminal selector UNC-3 (Collier/Ebf). UNC-3 synergizes with LIN-39 (Scr/ Dfd/Hox4-5) to directly co-activate multiple terminal identity traits specific to cholinergic MNs, but also antagonizes LIN-39’s ability to activate terminal features of alternative neuronal identities. Loss of unc-3 causes a switch in the transcriptional targets of LIN-39, thereby alternative, not cholinergic MN-specific, terminal features become activated and locomotion defects occur. The strategy of a terminal selector preventing a transcriptional switch may constitute a general principle for safeguarding neuronal identity throughout life