245 research outputs found
A mouse model reproducing the pathophysiology of neonatal group B streptococcal infection
- Author
- A Kinsner
- A Mottahedin
- A Six
- A Tazi
- AC Kreitzer
- AJ Carey
- B Korin
- CC Portugal
- CJ Kim
- D Fernandez-Lopez
- DM Norden
- E Alves
- E Boldenow
- EB Andrade
- FY Lin
- G Leisman
- G Mancuso
- GW Heyne
- H Hagberg
- H Meziane
- H Patterson
- HB Worp van der
- I Miyairi
- IO Okike
- IO Okike
- J Vornhagen
- JM Garre
- JR Verani
- K Doare Le
- K Grimwood
- KA Patras
- KS Doran
- KS Doran
- KS Kim
- L Mohle
- LA Meyn
- LC Ku
- LM Biase De
- M Coureuil
- M Ernst
- M Kohli-Lynch
- M Lalancette-Hebert
- M Maurer
- M Nedergaard
- M Rosa-Fraile
- M Stalhammar-Carlemalm
- MC Thigpen
- MJ Allard
- MP Forquin
- MR Hammerschlag
- MS Badri
- MS Edwards
- MV Sofroniew
- N Jones
- O Baud
- O Cauli
- P Calabresi
- P Ferrieri
- PT Heath
- R Libster
- RW Guillery
- S Perrin
- SE Hickman
- SJ Schrag
- SL Leib
- SM Hansen
- T Barichello
- T Barichello
- TD Stoner
- TE Wiswell
- TL Lamagni
- TM Randis
- TR Kollmann
- UK Hanisch
- YD Bifrare
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2018
- Field of study
Group B streptococcal (GBS) meningitis remains a devastating disease. The absence of an animal model reproducing the natural infectious process has limited our understanding of the disease and, consequently, delayed the development of effective treatments. We describe here a mouse model in which bacteria are transmitted to the offspring from vaginally colonised pregnant females, the natural route of infection. We show that GBS strain BM110, belonging to the CC17 clonal complex, is more virulent in this vertical transmission model than the isogenic mutant BM110∆cylE, which is deprived of hemolysin/cytolysin. Pups exposed to the more virulent strain exhibit higher mortality rates and lung inflammation than those exposed to the attenuated strain. Moreover, pups that survive to BM110 infection present neurological developmental disability, revealed by impaired learning performance and memory in adulthood. The use of this new mouse model, that reproduces key steps of GBS infection in newborns, will promote a better understanding of the physiopathology of GBS-induced meningitis.The authors gratefully acknowledge the help of Encarnaca̧ ̃o Ribeiro for excellent technical assistance, Joana Tavares for assisting with IVIS Lumina LT, Susana Roque for the
luminex instrument experiments, the Molecular Microbiology group at i3S for microscope use, and the Portuguese architect and artist Gil Ferreira da Silva for the artworks
included in the last figure. This work was supported by funds from Foundation for
Science and Technology (FCT), European Regional Development Fund (FEDER) and
Compete under project POCI-01-0145-FEDER-016607 (PTDC/IMI-MIC/1049/2014)
and from the project NORTE-01-0145-FEDER-000012, supported by Norte Portugal
Regional Operational Programme (NORTE 2020), under the PORTUGAL 2020 Partnership Agreement, through the European Regional Development Fund (ERDF). T.S.
and A.M. were supported by Investigador FCT (IF/00875/2012 and IF/00753/2014),
POPH and Fundo Social Europeu. E.B.A. and C.C.P. hold postdoctoral fellowships from
FCT (PTDC/IMI-MIC/1049/2014 and SFRH/BPD/91962/2012). Ar.F. and P.T.C. were
supported by Laboratoire d’Excellence (LABEX) Integrative Biology of Emerging Infectious Diseases (grant ANR-10-LABX-62-IBEID).info:eu-repo/semantics/publishedVersio
Embryonic Stem Cell-Derived L1 Overexpressing Neural Aggregates Enhance Recovery after Spinal Cord Injury in Mice
- Author
- A Bessis
- A De Biase
- A Irintchev
- AA Lavdas
- C Bernreuther
- CD Barnes
- Christian Bernreuther
- CP Hofstetter
- D Li
- DA Kerr
- DD Pearse
- DM Basso
- F Appel
- FG Rathjen
- G Loers
- GB Miles
- Gunnar Hargus
- H Kimura
- H Nishimune
- H Okano
- HJ Lee
- HS Keirstead
- I Apostolova
- I Jakovcevski
- I Kalus
- Igor Jakovcevski
- J Chen
- J Chen
- J Dekkers
- J Hellstrom
- J Lindner
- Jin-Chong Xu
- JM Harper
- K Fouad
- K Sadoul
- KK Jain
- L Dong
- M Celio
- M Dihne
- M Dihne
- M Gozariu
- M Joshi
- M Moos
- Mark Mattson
- Melitta Schachner
- MJ Howard
- MR Celio
- MS Davidoff
- O Kiehn
- O Nesic
- P Hulley
- R Curtis
- RS Nandoe Tewarie
- S Di Giovanni
- S Shapiro
- S Silletti
- S Thuret
- SA Busch
- SH Lee
- U Schuch
- V Belegu
- V Menet
- Y Ogawa
- YF Cui
- Yi-Fang Cui
- Publication venue
- Public Library of Science
- Publication date
- 18/03/2011
- Field of study
An obstacle to early stem cell transplantation into the acutely injured spinal cord is poor survival of transplanted cells. Transplantation of embryonic stem cells as substrate adherent embryonic stem cell-derived neural aggregates (SENAs) consisting mainly of neurons and radial glial cells has been shown to enhance survival of grafted cells in the injured mouse brain. In the attempt to promote the beneficial function of these SENAs, murine embryonic stem cells constitutively overexpressing the neural cell adhesion molecule L1 which favors axonal growth and survival of grafted and imperiled cells in the inhibitory environment of the adult mammalian central nervous system were differentiated into SENAs and transplanted into the spinal cord three days after compression lesion. Mice transplanted with L1 overexpressing SENAs showed improved locomotor function when compared to mice injected with wild-type SENAs. L1 overexpressing SENAs showed an increased number of surviving cells, enhanced neuronal differentiation and reduced glial differentiation after transplantation when compared to SENAs not engineered to overexpress L1. Furthermore, L1 overexpressing SENAs rescued imperiled host motoneurons and parvalbumin-positive interneurons and increased numbers of catecholaminergic nerve fibers distal to the lesion. In addition to encouraging the use of embryonic stem cells for early therapy after spinal cord injury L1 overexpression in the microenvironment of the lesioned spinal cord is a novel finding in its functions that would make it more attractive for pre-clinical studies in spinal cord regeneration and most likely other diseases of the nervous system
Network analysis of the transcriptional pattern of young and old cells of Escherichia coli during lag phase
- Author
- A Clauset
- A De Las Penas
- A Fredriksson
- AL Barabasi
- BK Cho
- C Luna-Chavez
- C Pin
- C Pin
- C Stathopoulos
- CA Ouzounis
- Carmen Pin
- CL Winder
- D De Biase
- D Koch
- DA Skibinski
- DH Weichart
- E Klauck
- EJ Stewart
- ER Stadtman
- EV Patridge
- F Govantes
- G Balazsi
- GA Gaitanaris
- H Jeong
- H Jung
- HT Richard
- IL Jung
- J Bascompte
- J Brejning
- J Brejning
- J Lin
- J Mecsas
- Jay CD Hinton
- JE Purvis
- JW Campbell
- József Baranyi
- KA Hopkin
- KP Stim-Herndon
- L Brondsted
- L Nachin
- LN Csonka
- LT Benov
- M Arifuzzaman
- M D'Arrigo
- M Ling
- M Radonjic
- Marina Muñoz-Cuevas
- Matthew D Rolfe
- MD Island
- MF Anjum
- Michael W Peck
- MS Cetin
- MW Covert
- N Peekhaus
- Nicholas J Walton
- O Francetic
- R Guimera
- RW Corbin
- S Eriksson
- S Gama-Castro
- S Saavedra
- SE Finkel
- SM Moorhead
- T Yura
- WS Yew
- Y Li
- Y Wei
- YL Ganduri
- ZN Oltvai
- Publication venue
- BioMed Central
- Publication date
- 16/11/2009
- Field of study
Background: The aging process of bacteria in stationary phase is halted if cells are subcultured and enter lag phase and it is then followed by cellular division. Network science has been applied to analyse the transcriptional response, during lag phase, of bacterial cells starved previously in stationary phase for 1 day (young cells) and 16 days (old cells).
Results: A genome scale network was constructed for E. coli K-12 by connecting genes with operons, transcription and sigma factors, metabolic pathways and cell functional categories. Most of the transcriptional changes were detected immediately upon entering lag phase and were maintained throughout this period. The lag period was longer for older cells and the analysis of the transcriptome revealed different intracellular activity in young and old cells. The number of genes differentially expressed was smaller in old cells (186) than in young cells (467). Relatively, few genes (62) were up- or down-regulated in both cultures. Transcription of genes related to osmotolerance, acid resistance, oxidative stress and adaptation to other stresses was down-regulated in both young and old cells. Regarding carbohydrate metabolism, genes related to the citrate cycle were up-regulated in young cells while old cells up-regulated the Entner Doudoroff and gluconate pathways and down-regulated the pentose phosphate pathway. In both old and young cells, anaerobic respiration and fermentation pathways were down-regulated, but only young cells up-regulated aerobic respiration while there was no evidence of aerobic respiration in old cells.Numerous genes related to DNA maintenance and replication, translation, ribosomal biosynthesis and RNA processing as well as biosynthesis of the cell envelope and flagellum and several components of the chemotaxis signal transduction complex were up-regulated only in young cells. The genes for several transport proteins for iron compounds were up-regulated in both young and old cells. Numerous genes encoding transporters for carbohydrates and organic alcohols and acids were down-regulated in old cells only.
Conclusion: Network analysis revealed very different transcriptional activities during the lag period in old and young cells. Rejuvenation seems to take place during exponential growth by replicative dilution of old cellular components
2017 HRS/EHRA/ECAS/APHRS/SOLAECE expert consensus statement on catheter and surgical ablation of atrial fibrillation: executive summary.
- Author
- Hugh Calkins
- Gerhard Hindricks
- Riccardo Cappato
- Young-Hoon Kim
- Eduardo B. Saad
- Luis Aguinaga
- Joseph G. Akar
- Vinay Badhwar
- Josep Brugada
- John Camm
- Peng-Sheng Chen
- Shih-Ann Chen
- Mina K. Chung
- Jens Cosedis Nielsen
- Anne B. Curtis
- D. Wyn Davies
- John D. Day
- André d’Avila
- N.M.S. de Groot
- Luigi Di Biase
- Mattias Duytschaever
- James R. Edgerton
- Kenneth A. Ellenbogen
- Patrick T. Ellinor
- Sabine Ernst
- Guilherme Fenelon
- Edward P. Gerstenfeld
- David E. Haines
- Michel Haissaguerre
- Robert H. Helm
- Elaine Hylek
- Warren M. Jackman
- Jose Jalife
- Jonathan M. Kalman
- Josef Kautzner
- Hans Kottkamp
- Karl Heinz Kuck
- Koichiro Kumagai
- Richard Lee
- Thorsten Lewalter
- Bruce D. Lindsay
- Laurent Macle
- Moussa Mansour
- Francis E. Marchlinski
- Gregory F. Michaud
- Hiroshi Nakagawa
- Andrea Natale
- Stanley Nattel
- Ken Okumura
- Douglas Packer
- Evgeny Pokushalov
- Matthew R. Reynolds
- Prashanthan Sanders
- Mauricio Scanavacca
- Richard Schilling
- Claudio Tondo
- Hsuan-Ming Tsao
- Atul Verma
- David J. Wilber
- Teiichi Yamane
- H Calkins
- H Calkins
- AK Jacobs
- JL Anderson
- CT January
- P Kirchhof
- P Jais
- H Calkins
- DL Packer
- KH Kuck
- SR Dukkipati
- VY Reddy
- A Natale
- DJ Wilber
- H Sohara
- C Pappone
- G Stabile
- GB Forleo
- A Verma
- D Scherr
- D Tamborero
- J Hummel
- M Bassiouny
- R Krittayaphong
- H Oral
- L Mont
- N Calvo
- F Furlanello
- OM Wazni
- J Cosedis Nielsen
- CA Morillo
- A Hakalahti
- M Hocini
- YW Chen
- K Inada
- MS Chen
- PJ Gentlesk
- MN Khan
- MR MacDonald
- RJ Hunter
- C Tondo
- BA Lutomsky
- AD Choi
- T Potter De
- YM Cha
- DG Jones
- T Machino-Ohtsuka
- S Halabi Al
- TJ Bunch
- TJ Lobo
- LH Ling
- LF Hsu
- DD Spragg
- F Kusumoto
- TJ Bunch
- P Santangeli
- K Nademanee
- TJ Bunch
- I Metzner
- TJ Bunch
- I Olivotto
- R Providencia
- P Leong-Sit
- KR Chun
- P Koopman
- GB Forleo
- L Wu
- S Mohanty
- V Badhwar
- CA Abreu Filho
- G Doukas
- C Blomstrom-Lundqvist
- P Chevalier
- DC Cheng
- P Budera
- K Phan
- AM Gillinov
- JS Rankin
- Y Louagie
- B Chiappini
- SD Barnett
- JR Edgerton
- JR Edgerton
- D Lockwood
- SC Malaisrie
- A Cherniavsky
- JS Yoo
- AH Driessen
- LV Boersma
- MC Henn
- SP Krul
- JL Cox
- E Rodriguez
- RK Wolf
- JR Edgerton
- JR Edgerton
- E Beyer
- K Kearney
- N Ad
- AH Driessen
- K Khargi
- OM Wazni
- AR Magnano
- HT McElderry
- PM McCarthy
- Y Zeng
- R Lee
- KH Kuck
- A Verma
- L Macle
- A Cheema
- K Rajappan
- D Bansch
- K Nakamura
- XH Wang
- WH Sauer
- Y Ninomiya
- T Yamane
- A Kobori
- C Pratola
- RH Jiang
- T Arentz
- M Tritto
- T Datino
- PD Dallaglio
- S Kapa
- JG Andrade
- C Eitel
- D Steven
- JG Andrade
- D Steven
- B Schaeffer
- EP Gerstenfeld
- P Vijayaraman
- JE Ip
- P Spector
- S Chen
- JY Kim
- FJ Perez
- NJ Patel
- O Wazni
- A Natale
- C Pappone
- N Sawhney
- S Chae
- F Ouyang
- S Matsuo
- S Tzeis
- SA Chen
- M Haissaguerre
- SH Lee
- MH Hsieh
- D Shah
- D Lin
- L Biase Di
- P Santangeli
- WS Lin
- RJ Lee
- Y Zhao
- S Dixit
- P Neuzil
- K Yokoyama
- A Ikeda
- H Nakagawa
- H Nakagawa
- S Kumar
- S Kumar
- VY Reddy
- S Haldar
- F Perna
- M Kimura
- C Sohns
- M Martinek
- E Marijon
- E Sigmund
- W Ullah
- R Wakili
- K Kumagai
- Y Yamaguchi
- K Kumagai
- JS Kim
- X He
- L Biase Di
- L Biase Di
- CA Morillo
- A Harada
- RA Gray
- O Berenfeld
- M Mansour
- S Lazar
- F Atienza
- F Atienza
- J Vogler
- M Haissaguerre
- K Nademanee
- MD O'Neill
- LW Lo
- Z Zhang
- TH Kim
- GJ Wynn
- H Kottkamp
- H Kottkamp
- S Rolf
- R Bai
- MJ Cutler
- G Yang
- A Verma
- S Kapa
- RS Oakes
- C McGann
- N Dagres
- K Nademanee
- M Haissaguerre
- M Haissaguerre
- Y Takahashi
- SM Singh
- SM Narayan
- A Verma
- JG Quintanilla
- BJ Hansen
- PS Cuculich
- M Haissaguerre
- SM Narayan
- YJ Lin
- Y-J Lin
- JM Miller
- YJ Lin
- SM Narayan
- WJ Rappel
- C Gianni
- SM Narayan
- P Sommer
- E Buch
- P Benharash
- C Ramanathan
- HS Lim
- S Yamashita
- MS Guillem
- MS Guillem
- M Rodrigo
- JA Armour
- SS Po
- E Patterson
- EK Choi
- DG Katritsis
- H Nakagawa
- M Bettoni
- DH Pauza
- BJ Scherlag
- E Patterson
- K Lemola
- K Nishida
- K Nishida
- S Stavrakis
- E Pokushalov
- E Pokushalov
- E Pokushalov
- E Pokushalov
- DM Lloyd-Jones
- R Mahajan
- A Wokhlu
- S Dublin
- AS Gami
- UB Tedrow
- RR Huxley
- TM Munger
- HS Abed
- B Richter
- K Jongnarangsin
- AN Shah
- SL Chang
- KP Letsas
- RB Tang
- H Jin Hwang
- D Patel
- D Patel
- D Patel
- TF Chao
- RA Winkle
- CX Wong
- JH Kang
- S Mohanty
- K Ejima
- KP Letsas
- CX Wong
- A Alonso
- RK Pathak
- T Bitter
- EC Fletcher
- H Kraiczi
- M Ghias
- D Linz
- D Linz
- D Linz
- YK Iwasaki
- YK Iwasaki
- H Dimitri
- IH Stevenson
- F Holmqvist
- Y Kwon
- R Kanagala
- AS Fein
- M Matiello
- Y Naruse
- TG Neilan
- L Li
- MH Raitt
- N Chalfoun
- M Igarashi
- L Rivard
- S Mohanty
- IM Robbins
- S Ernst
- M Mansour
- DR Holmes Jr
- EA Fender
- L Biase Di
- LR Prieto
- DL Packer
- GW Taylor
- T Arentz
- HF Tse
- L Kasper
- J Dong
- RH Hoyt
- EB Saad
- S Hilbert
- S Hilbert
- EA Fender
- TJ Potter De
- KR Kanter
- NS Patel
- A Bharat
- SP Ponamgi
- L Biase Di
- R Cappato
- C Pappone
- M Martinek
- SM Singh
- TJ Bunch
- R Cappato
- C Tan
- S Mohanty
- S Mohanty
- R Cappato
- AM Gillinov
- FW Mohr
- N Doll
- B Sonmez
- MI Scanavacca
- B Borchert
- KK Ghia
- GW Gilcrease
- F Stockigt
- O Yousuf
- PG Jackson
- G Nolker
- JT Medeiros De Vasconcelos
- A Rillig
- P Chavez
- JC Mateos
- HB Shim
- E Black-Maier
- P Santangeli
- L Biase Di
- OM Wazni
- M Schmidt
- A Hakalahti
- L Biase Di
- SH Hohnloser
- H Calkins
- R Cappato
- L Biase Di
- M Bassiouny
- AA Bin Abdulhak
- R Providencia
- RA Winkle
- HL Armbruster
- JF Ren
- S Saksena
- J Baran
- JF Ren
- E Anter
- CS Sriram
- K Maleki
- OM Wazni
- D Shah
- JF Ren
- CJ Bruce
- S Asbach
- DF Briceno
- K Chilukuri
- K Thygesen
- SC Helps
- GS Krivonyak
- B Cauchemez
- T Kuwahara
- OW Franzen
- KH Ryu
- F Gaita
- C Herrera Siklody
- A Verma
- Y Greef De
- T Deneke
- FM Merchant
- L Lickfett
- F Gaita
- JW Schrickel
- T Deneke
- LD Sauren
- M Wieczorek
- M Bendszus
- RW Kruis
- C Medi
- H Ichiki
- E Nagy-Balo
- SE Vermeer
- K Neven
- KL Ripley
- H Ahmed
- R Kawasaki
- HW Lim
- K Yokoyama
- SM Singh
- T Kuwahara
- FM Contreras-Valdes
- LR Leite
- CM Tschabrunn
- T Deneke
- BJ Carroll
- P Muller
- T Tsuchiya
- MS Arruda
- T Kuwahara
- T Kuwahara
- A Chugh
- JS Koruth
- S Zellerhoff
- S Zellerhoff
- M Khan
- PJ Kahrilas
- NJ Shaheen
- U Halm
- H Knopp
- N Dagres
- C Eitel
- W Hazell
- A Cazavet
- E Podgaetz
- L Queneherve
- MF Gunes
- MA Qadeer
- CR Ellis
- SR Markar
- JG Andrade
- A Deshmukh
- R Cappato
- OJ Eick
- JD Fisher
- LF Hsu
- TJ Bunch
- LF Hsu
- Y Michowitz
- TS Tsang
- MD O'Neill
- Y Takahashi
- A Chugh
- H Makimoto
- T Kitamura
- S Ouali
- A Mykytsey
- C Sticherling
- KC Wong
- G Fayad
- JP Piccini
- D Lakkireddy
- JM Dumonceau
- TJ Bunch
- T Kuwahara
- S Miyazaki
- N Chopra
- TW Schwartz
- W Ajaj
- CF Pisani
- T Kanaeda
- LW Lo
- J Janssens
- MP Jones
- R Tavernier
- R Grove
- D Lakkireddy
- HM Zeljko
- CV Desimone
- O Gurbuz
- M Mansour
- RC Wu
- S Deftereos
- S Deftereos
- G Stabile
- M Luckie
- T Lambert
- S Torihashi
- DR Kim
- M Kesek
- KH Kuck
- H Sohara
- RF Evonich
- KR Chun
- H Hoyt
- EN Guhl
- MJ Earley
- AV Sarabanda
- A Guiot
- A Metzner
- Y Okumura
- M Arruda
- S Miyazaki
- E Wissner
- CS Elayi
- H Liu
- S Yong Ji
- F Franceschi
- S Miyazaki
- B Mondesert
- F Sacher
- JG Andrade
- C Pappone
- SR Dukkipati
- ES Katz
- D Caponi
- R Proietti
- JD Ferguson
- H Calkins
- BD Lindsay
- P Kovoor
- L Macle
- L Lickfett
- J Ector
- J Chen
- Y Khaykin
- G Stabile
- R Ponti De
- T Kleemann
- D Steven
- JP Weiss
- O Dragusin
- VY Reddy
- A Bulava
- H Cochet
- DN Gibson
- MB Shoemaker
- TD Welch
- C Witt
- L Pilote
- J Kosiuk
- H Ghanbari
- D Scherr
- Y Liu
- PA Noseworthy
- D Patel
- S Kochhauser
- J Kosiuk
- F Abhishek
- B Aldhoon
- G Mugnai
- Y Murakawa
- C Palaniswamy
- P Peichl
- J Waigand
- D Lakkireddy
- CC Tanaka-Esposito
- A Errahmouni
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2017
- Field of study
S
MicroRNA Dysregulation in the Spinal Cord following Traumatic Injury
- Author
- A Cimmino
- A De Biase
- A Dharap
- A Duran
- A Izzotti
- A Sharma
- AI Pogue
- AI Saeed
- AI Saeed
- AM Cheng
- AM Krichevsky
- B Ason
- B Izumi
- B Kefas
- BA Citron
- C Cheng
- C Lindenblatt
- C Luna
- C Profyris
- C Santoscoy
- C Xiao
- C Xu
- CS Rau
- D Bhaumik
- D Iliopoulos
- D Iliopoulos
- D Ovcharenko
- D Shin
- D. Wolfgang Pita-Thomas
- David Reigada
- DM Basso
- DM McTigue
- DP Bartel
- E Elyakim
- E Mor
- E Sonkoly
- E Tili
- E Tsitsiou
- E Wienholds
- ED Ponomarev
- ER Hutchison
- ER Strickland
- F Bazzoni
- FB Gao
- FJ Sheedy
- FM Bareyre
- FM Bareyre
- Francisco J. Esteban
- G Gabriely
- G Li
- G Martello
- G Shafi
- G Sui
- GK Scott
- GK Smyth
- GT Bommer
- H Hohjoh
- H Matsubara
- H Su
- H Wu
- HF Xia
- IK Guttilla
- J Ho
- J Li
- J Li
- J Li
- J Lu
- J Qiu
- J Tao
- J Wang
- J Yao
- JA Saugstad
- JB Aimone
- JC Dugas
- JL Mott
- JR Bethea
- JR Neilson
- JW Fawcett
- JW Rowland
- K Nakanishi
- K Zheng
- KD Rasmussen
- KD Taganov
- L He
- L Schnell
- L Smirnova
- L Xia
- LB Frankel
- LP Weng
- M Bak
- M Crawford
- M Deo
- M Sachdeva
- M Wang
- M Yamakuchi
- MA Lindsay
- Manuel Nieto-Díaz
- Marcos Caballero-López
- MJ Crowe
- MR Young
- MS Kumar
- MS Ostenfeld
- Mónica Yunta
- N Mellios
- N Saito
- N Schonrock
- NH Foley
- NK Liu
- ON Hausmann
- P Mestdagh
- R Breitling
- R Chen
- R Fiore
- R Spizzo
- R Zhou
- Rodrigo M. Maza
- Rosa Navarro-Ruíz
- S Casha
- S Di Giovanni
- S Dudoit
- S Moncini
- S Shimizu
- S Thomas
- S Wang
- SD Grossman
- SE Linsen
- Simone Di Giovanni
- SK Fineberg
- SM Allan
- SM Eacker
- SS Hebert
- SS Hebert
- SW Scheff
- SY Park
- T Andersson
- T Li
- T Sugawara
- T Tjoa
- Teresa Muñoz-Galdeano
- V Ambros
- V Sahni
- VB Sampson
- W Huang da
- W Huber
- W Konopka
- WP Tsang
- WR Tyor
- X Ma
- X Pan
- X Tang
- X Zhang
- X Zhao
- X Zhao
- XY Yu
- XZ Liu
- Y Benjamini
- Y Gusev
- Y Gusev
- Y Suzuki
- Y Tang
- Y Ye
- Y Yuan
- YJ Hua
- YM Yu
- Z Wang
- ZX Shan
- Ángela del Águila
- Publication venue
- Public Library of Science
- Publication date
- 12/04/2012
- Field of study
Spinal cord injury (SCI) triggers a multitude of pathophysiological events that are tightly regulated by the expression levels of specific genes. Recent studies suggest that changes in gene expression following neural injury can result from the dysregulation of microRNAs, short non-coding RNA molecules that repress the translation of target mRNA. To understand the mechanisms underlying gene alterations following SCI, we analyzed the microRNA expression patterns at different time points following rat spinal cord injury
Determinants of SARS-CoV-2 receptor gene expression in upper and lower airways
- Author
- Adcock IM
- Aliee H
- Brandsma CA
- Cambridge Lung Cancer Early Detection Programme
- De Biase MS
- Faiz A
- Grimbaldeston M
- Guo Y
- Hiemstra PS
- Hu S
- INER-Ciencias Mexican Lung Program
- Kermani NZ
- Kersten ETG
- Khuder B
- Koppelman GH
- Malmström K
- Massip F
- Mäkelä MJ
- Nawijn MC
- Neighbors M
- NHLBI LungMAP Consortium
- Ponder BA
- Qi C
- Reyfman PA
- Rintoul RC
- Schwarz RF
- Sun K
- Ten Hacken NHT
- Tew GW
- Theis FJ
- Timens W
- U-BIOPRED study group
- Van den Berge M
- Van Nijnatten J
- Vermeulen RCH
- Vonk JM
- Xu CJ
- Zhang X
- Publication venue
- 'Cold Spring Harbor Laboratory'
- Publication date
- 02/09/2020
- Field of study
The recent outbreak of the severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2), which causes coronavirus disease 2019 (COVID-19), has led to a worldwide pandemic. One week after initial symptoms develop, a subset of patients progresses to severe disease, with high mortality and limited treatment options. To design novel interventions aimed at preventing spread of the virus and reducing progression to severe disease, detailed knowledge of the cell types and regulating factors driving cellular entry is urgently needed. Here we assess the expression patterns in genes required for COVID-19 entry into cells and replication, and their regulation by genetic, epigenetic and environmental factors, throughout the respiratory tract using samples collected from the upper (nasal) and lower airways (bronchi). Matched samples from the upper and lower airways show a clear increased expression of these genes in the nose compared to the bronchi and parenchyma. Cellular deconvolution indicates a clear association of these genes with the proportion of secretory epithelial cells. Smoking status was found to increase the majority of COVID-19 related genes including ACE2 and TMPRSS2 but only in the lower airways, which was associated with a significant increase in the predicted proportion of goblet cells in bronchial samples of current smokers. Both acute and second hand smoke were found to increase ACE2 expression in the bronchus. Inhaled corticosteroids decrease ACE2 expression in the lower airways. No significant effect of genetics on ACE2 expression was observed, but a strong association of DNA- methylation with ACE2 and TMPRSS2- mRNA expression was identified in the bronchus
Combination of searches for heavy spin-1 resonances using 139 fb−1 of proton-proton collision data at s = 13 TeV with the ATLAS detector
- Author
- Aad G
- Aakvaag E
- Abbott B
- Abeling K
- Abicht NJ
- Abidi SH
- Aboelela M
- Aboulhorma A
- Abramowicz H
- Abreu H
- Abulaiti Y
- Acharya BS
- Ackermann A
- Adam Bourdarios C
- Adamczyk L
- Addepalli SV
- Addison MJ
- Adelman J
- Adiguzel A
- Adye T
- Affolder AA
- Afik Y
- Agaras MN
- Agarwala J
- Aggarwal A
- Agheorghiesei C
- Ahmad A
- Ahmadov F
- Ahmed WS
- Ahuja S
- Ai X
- Aielli G
- Aikot A
- Ait Tamlihat M
- Aitbenchikh B
- Aizenberg I
- Akbiyik M
- Akimov AV
- Akiyama D
- Akolkar NN
- Aktas S
- Al Khoury K
- Alberghi GL
- Albert J
- Albicocco P
- Albouy GL
- Alderweireldt S
- Alegria ZL
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexopoulos T
- Alfonsi F
- Algren M
- Alhroob M
- Ali B
- Ali HMJ
- Ali S
- Alibocus SW
- Aliev M
- Alimonti G
- Alkakhi W
- Allaire C
- Allbrooke BMM
- Allen JF
- Allendes Flores CA
- Allport PP
- Aloisio A
- Alonso F
- Alpigiani C
- Alvarez Estevez M
- Alvarez Fernandez A
- Alves Cardoso M
- Alviggi MG
- Aly M
- Amaral Coutinho Y
- Ambler A
- Amelung C
- Amerl M
- Ames CG
- Amidei D
- Amirie KJ
- Amor Dos Santos SP
- Amos KR
- An S
- Ananiev V
- Anastopoulos C
- Andeen T
- Anders JK
- Andrean SY
- Andreazza A
- Angelidakis S
- Angerami A
- Anisenkov AV
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- Antel C
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- Zhemchugov A
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- Zimine NI
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- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- Springer Nature
- Publication date
- 19/04/2024
- Field of study
A combination of searches for new heavy spin-1 resonances decaying into different pairings of W, Z, or Higgs bosons, as well as directly into leptons or quarks, is presented. The data sample used corresponds to 139 fb−1 of proton-proton collisions at
= 13 TeV collected during 2015–2018 with the ATLAS detector at the CERN Large Hadron Collider. Analyses selecting quark pairs (qq, bb,
, and tb) or third-generation leptons (τν and ττ) are included in this kind of combination for the first time. A simplified model predicting a spin-1 heavy vector-boson triplet is used. Cross-section limits are set at the 95% confidence level and are compared with predictions for the benchmark model. These limits are also expressed in terms of constraints on couplings of the heavy vector-boson triplet to quarks, leptons, and the Higgs boson. The complementarity of the various analyses increases the sensitivity to new physics, and the resulting constraints are stronger than those from any individual analysis considered. The data exclude a heavy vector-boson triplet with mass below 5.8 TeV in a weakly coupled scenario, below 4.4 TeV in a strongly coupled scenario, and up to 1.5 TeV in the case of production via vector-boson fusion
Measurement and interpretation of same-sign W boson pair production in association with two jets in pp collisions at s = 13 TeV with the ATLAS detector
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- Sideras Haddad E
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- Soldatov EY
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- Solodkov AA
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- Solovyanov OV
- Solovyev V
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- Sonay A
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- Sopczak A
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- Sánchez J
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- Taffard A
- Tafirout R
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- Talyshev AA
- Tam KC
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- Tanaka A
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- Tao Z
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- Tarek Abouelfadl Mohamed A
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- Trischuk DA
- Trocmé B
- Troncon C
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- Tskhadadze EG
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- Vasquez GA
- Vasyukov A
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- Veatch J
- Vecchio V
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- Veloce LM
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- Vergis C
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- Vermeulen JC
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- Vgenopoulos A
- Viaux Maira N
- Vickey Boeriu OE
- Vickey T
- Viehhauser GHA
- Vigani L
- Villa M
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- Villhauer EM
- Vilucchi E
- Vincter MG
- Virdee GS
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- Visibile A
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- Vladimirov V
- Voevodina E
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- Weingarten J
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- Wiedenmann W
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- Wiglesworth C
- Wilbern DJ
- Wilkens HG
- Williams DM
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- Williams S
- Willocq S
- Wilson BJ
- Windischhofer PJ
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- Winter BT
- Winter JK
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- Wobisch M
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- Worm SD
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- Wu M
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- Xiang J
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- Xu R
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- Xu Z
- Yabsley B
- Yacoob S
- Yamaguchi Y
- Yamashita E
- Yamauchi H
- Yamazaki T
- Yamazaki Y
- Yan J
- Yan S
- Yan Z
- Yang HJ
- Yang HT
- Yang S
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- Yang X
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- Yang Y
- Yang Y
- Yang Z
- Yao W-M
- Yap YC
- Ye H
- Ye H
- Ye J
- Ye S
- Ye X
- Yeh Y
- Yeletskikh I
- Yeo BK
- Yexley MR
- Yin P
- Yorita K
- Younas S
- Young C
- Young CJS
- Yu C
- Yu Y
- Yuan M
- Yuan R
- Yue L
- Zaazoua M
- Zabinski B
- Zaid E
- Zakareishvili T
- Zakharchuk N
- Zambito S
- Zamora Saa JA
- Zang J
- Zanzi D
- Zaplatilek O
- Zeitnitz C
- Zeng H
- Zeng JC
- Zenger DT
- Zenin O
- Zenz S
- Zerradi S
- Zerwas D
- Zhai M
- Zhang B
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- Zhang J
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- Zhang K
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- Zhang P
- Zhang R
- Zhang S
- Zhang T
- Zhang X
- Zhang X
- Zhang Y
- Zhang Y
- Zhang Z
- Zhang Z
- Zhao H
- Zhao P
- Zhao T
- Zhao Y
- Zhao Z
- Zhemchugov A
- Zheng J
- Zheng K
- Zheng X
- Zheng Z
- Zhong D
- Zhou B
- Zhou H
- Zhou N
- Zhou Y
- Zhu CG
- Zhu J
- Zhu Y
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- Zhulanov V
- Zimine NI
- Zinsser J
- Ziolkowski M
- Zoccoli A
- Zoch K
- Zorbas TG
- Zormpa O
- Zou W
- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- Springer Nature
- Publication date
- 01/04/2024
- Field of study
This paper presents the measurement of fducial and diferential cross sections for both the inclusive and electroweak production of a same-sign W-boson pair in association with two jets (W±W±jj) using 139 fb−1 of proton-proton collision data recorded at a centre-of-mass energy of √s = 13 TeV by the ATLAS detector at the Large Hadron Collider. The analysis is performed by selecting two same-charge leptons, electron or muon, and at least two jets with large invariant mass and a large rapidity diference. The measured fducial cross sections for electroweak and inclusive W±W±jj production are 2.92 ± 0.22 (stat.) ± 0.19 (syst.)fb and 3.38±0.22 (stat.)±0.19 (syst.)fb, respectively, in agreement with Standard Model predictions. The measurements are used to constrain anomalous quartic gauge couplings by extracting 95% confdence level intervals on dimension-8 operators. A search for doubly charged Higgs bosons H±± that are produced in vector-boson fusion processes and decay into a same-sign W boson pair is performed. The largest deviation from the Standard Model occurs for an H±± mass near 450 GeV, with a global signifcance of 2.5 standard deviations
Horizontal gene transfer in an acid mine drainage microbial community
- Author
- A Krogh
- AF Andersson
- B Wittmann-Liebold
- C Brochier
- C Pál
- C Pál
- CD Nadell
- CP Andam
- CS Riesenfeld
- D Biase De
- DF Birt
- EV Koonin
- F Branco dos Santos
- F Cui
- Fumeng Wang
- G Schönknecht
- GQ Hu
- GW Tyson
- H Grosjean
- H Noguchi
- H Ochman
- H Shimodaira
- H Shimodaira
- Huaiqiu Zhu
- I Bertini
- IL Jung
- J Filée
- J Handelsman
- J Tamames
- J Wiedenbeck
- JD Thompson
- JG Lawrence
- JG Lawrence
- Jiangtao Guo
- Jinxian Yao
- JM Logsdon Jr
- JP Gogarten
- JR Cole
- JR Zaneveld
- JW Foster
- K Lagesen
- K Tamura
- LM Iyer
- M Gandhi
- M Wu
- MN Price
- MP Castanie-Cornet
- MR Barer
- MS Poptsova
- N Goldenfeld
- N Iwabe
- O Zhaxybayeva
- PD Cotter
- Qi Wang
- RS Mueller
- RS Mueller
- SA Lincoln
- SF Altschul
- SR Waterman
- TJ Treangen
- TW Schoenfeld
- V Kunin
- VJ Denef
- W Carpentier
- Xiaoqi Wang
- Z Yang
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Accuracy versus precision in boosted top tagging with the ATLAS detector
- Author
- Aad G
- Aakvaag E
- Abbott B
- Abdelhameed S
- Abeling K
- Abicht NJ
- Abidi SH
- Aboelela M
- Aboulhorma A
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- Abreu H
- Abulaiti Y
- Acharya BS
- Ackermann A
- Adamczyk L
- Addepalli SV
- Addison MJ
- Adelman J
- Adiguzel A
- Adye T
- Affolder AA
- Afik Y
- Agaras MN
- Agarwala J
- Aggarwal A
- Agheorghiesei C
- Agullo PM
- Ahmadov F
- Ahmed WS
- Ahuja S
- Ai X
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- Akimov AV
- Akiyama D
- Akolkar NN
- Aktas S
- Al Khoury K
- Alberghi GL
- Albert J
- Albicocco P
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- Aleksandrov IN
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- Honan BP
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- Kalaitzidou I
- Kalderon CW
- Kang NJ
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- Karyukhin AN
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- Klein U
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- Klimentov A
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- Koch SF
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- Kodyš P
- Koeck DM
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- Koffas T
- Kolay O
- Koletsou I
- Komarek T
- Kong AXY
- Kono T
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- Kontaxakis P
- Konya B
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- Kotwal A
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- Kourkoumeli-Charalampidi A
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- Kourlitis E
- Kovanda O
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- Kramarenko VA
- Kramberger G
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- Kurdysh O
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- Kyriacou NG
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- Ladygin E
- Lafarge A
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- Lahbabi FZ
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- Lanni F
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- Lanza A
- Lançon E
- Laporte JF
- Lari T
- Lassnig M
- Latonova V
- Latour BMD
- Laurier A
- Lawlor SD
- Lawrence Z
- Lazaridou R
- Lazzaroni M
- Le Boulicaut EM
- Le Pottier LT
- Le B
- Leban B
- Lebedev A
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- Lee S
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- Leeuw LL
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- Leinonen W
- Leisos A
- Leite MAL
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- Leitner R
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- Leonidopoulos C
- Leopold A
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- Li X
- Li Z
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- Liberti B
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- Lindon JH
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- Lipniacka A
- Lister A
- Little JD
- Liu B
- Liu BX
- Liu D
- Liu EHL
- Liu JB
- Liu JKK
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- Llácer MM
- Lobodzinska EM
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- Lohwasser K
- Loiacono E
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- Lomas JD
- Long JD
- Longarini I
- Longo R
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- Lopez-canelas NA
- Lory AM
- Losada M
- Loseva O
- Lou X
- Lou X
- Lounis A
- Love PA
- Lu G
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- Lubatti HJ
- Luci C
- Luehring F
- Luengo SIF
- Luise I
- Lukianchuk O
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- Lundberg O
- Luongo NA
- Lutz MS
- Lux AB
- Lynn D
- Lysak R
- Lytken E
- Lyubushkin V
- Lyubushkina T
- Lyukova MM
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- Ma Y
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- Maeda J
- Maeno T
- Maguire H
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- Maio A
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- Zhemchugov A
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- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- IOP Publishing
- Publication date
- 01/08/2024
- Field of study
Abstract
The identification of top quark decays where the top quark has a large momentum transverse to the beam axis, known as top tagging, is a crucial component in many measurements of Standard Model processes and searches for beyond the Standard Model physics at the Large Hadron Collider.
Machine learning techniques have improved the performance of top tagging algorithms, but the size of the systematic uncertainties for all proposed algorithms has not been systematically studied.
This paper presents the performance of several machine learning based top tagging algorithms on a dataset constructed from simulated proton-proton collision events measured with the ATLAS detector at √
s
= 13 TeV.
The systematic uncertainties associated with these algorithms are estimated through an approximate procedure that is not meant to be used in a physics analysis, but is appropriate for the level of precision required for this study.
The most performant algorithms are found to have the largest uncertainties, motivating the development of methods to reduce these uncertainties without compromising performance.
To enable such efforts in the wider scientific community, the datasets used in this paper are made publicly available.</jats:p
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