10 research outputs found
The ESMO programme of certification and training for medical oncology
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/1998
- Field of study
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The ESMO-programme of certification and training for medical oncology
- Publication venue
- Publication date
- 01/01/1998
- Field of study
PRODUCTION OF MONOCLONAL ANTIBODIES AGAINST RT2 ANTIGENS OF THE RAT AND THE DEFINITION OF A NEW LOCUS, RT9, LINKED TO RT2
- Publication venue
- 'Wiley'
- Publication date
- Field of study
The ESMO programme of certification and training for medical oncology
- Author
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
The Pittsburgh insulin-dependent diabetes mellitus (IDDM) study. HLA antigens and haplotypes as risk factors for the development of IDDM in IDDM patients and their siblings
- Author
- A.L. Drash
- B.S. Rabin
- Barbosa
- Barbosa
- Barbosa
- Barbosa
- Bengsch
- Bengtsson
- Boucher
- Breslow
- Cavender
- Cavender
- Colle
- Cudworth
- Cudworth
- Cudworth
- Cutler
- D.E. Cavender
- D.J. Becker
- D.K. Wagener
- Darlow
- Degnbol
- Durruty
- Falk
- Fishbein
- Gamble
- Gorsuch
- Gottlieb
- Gray
- Harris
- Hodge
- Kiesel
- L.H. Kuller
- LaPorte
- Li
- Li
- Ludwig
- M.S. Eberhardt
- MacDonald
- Maclaren
- Miettinen
- Miller
- Morrow
- N.I.A.I.D.: National Instutues of Health
- Orchard
- Paik
- Paik
- Platz
- R.E. LaPorte
- Rodney
- Rossini
- Rubenstein
- Sachs
- Simpson
- Spielman
- Suarez
- Suica-Foca
- Svejgaard
- T.J. Orchard
- Thomsen
- van de Putte
- Wagener
- Walker
- West
- Woolf
- Publication venue
- 'Elsevier BV'
- Publication date
- Field of study
Age and sex variations in glucose tolerance and insulin responses: Parallels with cardiovascular risk
- Author
- A.L. Drash
- Alexander
- Barrett-Connor
- Burkeholder
- Calvert
- Conn
- D.J. Becker
- D.K. Wagener
- du V Florey
- Epstein
- Garcia
- Gordon
- Heller
- Herbert
- Heyden
- Josefsberg
- Keen
- Keen
- Keen
- L.H. Kuller
- LaPorte
- Mirsky
- Nie
- Nikkila
- Nikkila
- Nikkila
- Orchard
- Oskander
- Page
- Palumbo
- Pell
- Persson
- Phillips
- Pyorala
- R.E. LaPorte
- Reaven
- Rosenbloom
- Schmilt
- Stamler
- Stout
- Stout
- T.J. Orchard
- Vital and Health Statistics
- Welborn
- West
- Publication venue
- 'Elsevier BV'
- Publication date
- Field of study
Role of glucocorticoids in coat depigmentation in animals selected for behavior
- Author
- A. Barbazanges
- A. Kapoor
- A. Slominski
- A.A. Tinnikov
- A.A. Zimmermann
- A.J. Drake
- A.L. Markel
- A.V. Badyaev
- B.F. Vanyushin
- C. Guerrero-Bosagna
- D. O’Regan
- D.I. Lugo
- D.K. Belyaev
- D.M. Sloboda
- E. Donet
- E. Vsevolodov
- F. Dean
- G.S. Barsh
- H. Uno
- H.-Sh. Chang
- I. N. Oskina
- I. Wendt-Wagener
- I. Z. Plyusnina
- I.N. Oskina
- J.R. Seckl
- L. A. Prasolova
- L. N. Trut
- L.A. Prasolova
- L.L. Woods
- L.N. Osadchuk
- L.N. Trut
- L.N. Trut
- L.N. Trut
- M. Darnaudery
- M. Fameli
- M. Yamamoto
- M.K. Skinner
- N.N. Dygalo
- R. Klepac
- R. Klepac
- R.G. Gulevich
- R.M. Sapolsky
- S.G. Matthews
- T. Poltyrev
- Publication venue
- 'Allerton Press'
- Publication date
- Field of study
Genetic causes involved in Leishmania Chagasi infection innortheastern: Brazil
- Author
- Abel L.
- Abel L.
- Abel L.
- Akaike H.
- Barton C.H.
- Blackwell J.M.
- Blackwell J.M.
- Blackwell J.M.
- Blackwell J.M.
- Blackwell J.M.
- Blackwell J.M.
- Blackwell J.M.
- Bradley D.J.
- Bradley D.J.
- Brown I.N.
- Cabello P.H.
- Demenais F.
- Eliane Azevêdo
- Elston R.C.
- Feitosa M.F.
- Feitosa M.F.
- Goto Y.
- Govoni G.
- Gros P.
- Haile R.W.C.
- Henrique Krieger
- Lalouel J.M.
- Lalouel J.M.
- Levee G.
- Lima A.M.M.D.
- Malo D.
- Mary Furlan Feitosa
- Mock B.
- Morgan K.
- Morton N.E.
- Morton N.E.
- Newport M.
- Plant J.
- Plant J.
- Reed S.G.
- Roberts M.
- Roberts M.
- Sachdeva G.
- Schurr E.
- Schurr E.
- Schurr E.
- Schurr E.
- Serjeantson S.W.
- Shaw M.A.
- Shaw M.A.
- Shields E.D.
- Skamene E.
- Skamene E.
- Skamene E.
- Skamene E.
- Smith D.G.
- Vidal S.M.
- Wagener D.K.
- Ângela Maria Lima
- Publication venue
- 'FapUNIFESP (SciELO)'
- Publication date
- Field of study
References
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- Publication venue
- 'Informa UK Limited'
- Publication date
- Field of study
Pan-cancer analysis of whole genomes
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- Aaltonen L.A.
- Abascal F.
- Abeshouse A.
- Aburatani H.
- Adams D.J.
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- Wouters B.G.
- Wright A.J.
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- Yachida S.
- Yakneen S.
- Yamaguchi R.
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- Yellapantula V.
- Yellapantula V.D.
- Yoon C.J.
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- Yousif F.
- Yu J.
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- Yung C.
- Yung C.K.
- Zaikova O.
- Zamora J.
- Zapatka M.
- Zenklusen J.C.
- Zenz T.
- Zeps N.
- Zhang C.Z.
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Cancer is driven by genetic change, and the advent of massively parallel sequencing has enabled systematic documentation of this variation at the whole-genome scale. Here we report the integrative analysis of 2,658 whole-cancer genomes and their matching normal tissues across 38 tumour types from the Pan-Cancer Analysis of Whole Genomes (PCAWG) Consortium of the International Cancer Genome Consortium (ICGC) and The Cancer Genome Atlas (TCGA). We describe the generation of the PCAWG resource, facilitated by international data sharing using compute clouds. On average, cancer genomes contained 4-5 driver mutations when combining coding and non-coding genomic elements; however, in around 5% of cases no drivers were identified, suggesting that cancer driver discovery is not yet complete. Chromothripsis, in which many clustered structural variants arise in a single catastrophic event, is frequently an early event in tumour evolution; in acral melanoma, for example, these events precede most somatic point mutations and affect several cancer-associated genes simultaneously. Cancers with abnormal telomere maintenance often originate from tissues with low replicative activity and show several mechanisms of preventing telomere attrition to critical levels. Common and rare germline variants affect patterns of somatic mutation, including point mutations, structural variants and somatic retrotransposition. A collection of papers from the PCAWG Consortium describes non-coding mutations that drive cancer beyond those in the TERT promoter; identifies new signatures of mutational processes that cause base substitutions, small insertions and deletions and structural variation; analyses timings and patterns of tumour evolution; describes the diverse transcriptional consequences of somatic mutation on splicing, expression levels, fusion genes and promoter activity; and evaluates a range of more-specialized features of cancer genomes