Observations of H-alpha, iron, and oxygen lines in B, Be, and shell stars

We have carried out a spectroscopic survey of several B, Be, and shell stars in optical and near-infrared regions. Line profiles of the H-alpha line and of selected Fe II and O I lines are presented.Comment: 23 pages, 45 figures; accepted in Astronomy & Astrophysic

Spectroscopic analysis of the B/Be visual binary HR 1847

We studied both components of a slightly overlooked visual binary HR 1847 spectroscopically to determine its basic physical and orbital parameters. Basic stellar parameters were determined by comparing synthetic spectra to the observed echelle spectra, which cover both the optical and near-IR regions. New observations of this system used the Ond\v{r}ejov and Rozhen 2-m telescopes and their coud\'e spectrographs. Radial velocities from individual spectra were measured and then analysed with the code {\FOTEL} to determine orbital parameters. The spectroscopic orbit of HR 1847A is presented for the first time. It is a single-lined spectroscopic binary with a B-type primary, a period of 719.79 days, and a highly eccentric orbit with e=0.7. We confirmed that HR 1847B is a Be star. Its H\alpha emission significantly decreased from 2003 to 2008. Both components have a spectral type B7-8 and luminosity class IV-V.Comment: Astronomy and Astrophysics, accepte

Finite Generation of Canonical Ring by Analytic Method

In the 80th birthday conference for Professor LU Qikeng in June 2006 I gave a talk on the analytic approach to the finite generation of the canonical ring for a compact complex algebraic manifold of general type. This article is my contribution to the proceedings of that conference from my talk. In this article I give an overview of the analytic proof and focus on explaining how the analytic method handles the problem of infinite number of interminable blow-ups in the intuitive approach to prove the finite generation of the canonical ring. The proceedings of the LU Qikeng conference will appear as Issue No. 4 of Volume 51 of Science in China Series A: Mathematics (www.springer.com/math/applications/journal/11425)

Sur les exposants de Lyapounov des applications meromorphes

Let f be a dominating meromorphic self-map of a compact Kahler manifold. We give an inequality for the Lyapounov exponents of some ergodic measures of f using the metric entropy and the dynamical degrees of f. We deduce the hyperbolicity of some measures.Comment: 27 pages, paper in french, final version: to appear in Inventiones Mat

The High-Acceptance Dielectron Spectrometer HADES

HADES is a versatile magnetic spectrometer aimed at studying dielectron production in pion, proton and heavy-ion induced collisions. Its main features include a ring imaging gas Cherenkov detector for electron-hadron discrimination, a tracking system consisting of a set of 6 superconducting coils producing a toroidal field and drift chambers and a multiplicity and electron trigger array for additional electron-hadron discrimination and event characterization. A two-stage trigger system enhances events containing electrons. The physics program is focused on the investigation of hadron properties in nuclei and in the hot and dense hadronic matter. The detector system is characterized by an 85% azimuthal coverage over a polar angle interval from 18 to 85 degree, a single electron efficiency of 50% and a vector meson mass resolution of 2.5%. Identification of pions, kaons and protons is achieved combining time-of-flight and energy loss measurements over a large momentum range. This paper describes the main features and the performance of the detector system

Immobilization of Ionic Liquids, Types of Materials, and Applications

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Spontaneous DNA damage to the nuclear genome promotes senescence,redox imbalance and aging

Accumulation of senescent cells over time contributes to aging and age-related diseases. However, what drives senescence in vivo is not clear. Here we used a genetic approach to determine if spontaneous nuclear DNA damage is sufficient to initiate senescence in mammals. Ercc1-/Δ mice with reduced expression of ERCC1-XPF endonuclease have impaired capacity to repair the nuclear genome. Ercc1-/Δ mice accumulated spontaneous, oxidative DNA damage more rapidly than wild-type (WT) mice. As a consequence, senescent cells accumulated more rapidly in Ercc1-/Δ mice compared to repair-competent animals. However, the levels of DNA damage and senescent cells in Ercc1-/Δ mice never exceeded that observed in old WT mice. Surprisingly, levels of reactive oxygen species (ROS) were increased in tissues of Ercc1-/Δ mice to an extent identical to naturally-aged WT mice. Increased enzymatic production of ROS and decreased antioxidants contributed to the elevation in oxidative stress in both Ercc1-/Δ and aged WT mice. Chronic treatment of Ercc1-/Δ mice with the mitochondrial-targeted radical scavenger XJB-5–131 attenuated oxidative DNA damage, senescence and age-related pathology. Our findings indicate that nuclear genotoxic stress arises, at least in part, due to mitochondrial-derived ROS, and this spontaneous DNA damage is sufficient to drive increased levels of ROS, cellular senescence, and the consequent age-related physiological decline

Spontaneous DNA damage to the nuclear genome promotes senescence, T redox imbalance and aging

Accumulation of senescent cells over time contributes to aging and age-related diseases. However, what drives senescence in vivo is not clear. Here we used a genetic approach to determine if spontaneous nuclear DNA damage is sufficient to initiate senescence in mammals. Ercc1-/Δ mice with reduced expression of ERCC1-XPF endonuclease have impaired capacity to repair the nuclear genome. Ercc1-/Δ mice accumulated spontaneous, oxidative DNA damage more rapidly than wild-type (WT) mice. As a consequence, senescent cells accumulated more rapidly in Ercc1-/Δ mice compared to repair-competent animals. However, the levels of DNA damage and senescent cells in Ercc1-/Δ mice never exceeded that observed in old WT mice. Surprisingly, levels of reactive oxygen species (ROS) were increased in tissues of Ercc1-/Δ mice to an extent identical to naturally-aged WT mice. Increased enzymatic production of ROS and decreased antioxidants contributed to the elevation in oxidative stress in both Ercc1-/Δ and aged WT mice. Chronic treatment of Ercc1-/Δ mice with the mitochondrial-targeted radical scavenger XJB-5–131 attenuated oxidative DNA damage, senescence and age-related pathology. Our findings indicate that nuclear genotoxic stress arises, at least in part, due to mitochondrial-derived ROS, and this spontaneous DNA damage is sufficient to drive increased levels of ROS, cellular senescence, and the consequent age-related physiological decline