A Survey of Signal Processing Problems and Tools in Holographic Three-Dimensional Television

Cataloged from PDF version of article.Diffraction and holography are fertile areas for application of signal theory and processing. Recent work on 3DTV displays has posed particularly challenging signal processing problems. Various procedures to compute Rayleigh-Sommerfeld, Fresnel and Fraunhofer diffraction exist in the literature. Diffraction between parallel planes and tilted planes can be efficiently computed. Discretization and quantization of diffraction fields yield interesting theoretical and practical results, and allow efficient schemes compared to commonly used Nyquist sampling. The literature on computer-generated holography provides a good resource for holographic 3DTV related issues. Fast algorithms to compute Fourier, Walsh-Hadamard, fractional Fourier, linear canonical, Fresnel, and wavelet transforms, as well as optimization-based techniques such as best orthogonal basis, matching pursuit, basis pursuit etc., are especially relevant signal processing techniques for wave propagation, diffraction, holography, and related problems. Atomic decompositions, multiresolution techniques, Gabor functions, and Wigner distributions are among the signal processing techniques which have or may be applied to problems in optics. Research aimed at solving such problems at the intersection of wave optics and signal processing promises not only to facilitate the development of 3DTV systems, but also to contribute to fundamental advances in optics and signal processing theory. © 2007 IEEE

Finding an Effective Metric Used for Bijective S-Box Generation by Genetic Algorithms

In cryptography, S-box is a basic component of symmetric key algorithms which performs nonlinear substitution. S-boxes need to be highly nonlinear, so that the cipher can resist linear cryptanalysis. The main criteria for cryptographically strong (n × n) S-box are: • High non linearity; • High algebraic degree; • Balanced structure; • Good auto correlation properties. Our task was to give some suggestions for finding an effective metric used for generation bijective optimal S-Box. Because of the given problem’s complexity, our group considered different approaches and we gave a few suggestions for problem solving

Visible reconstruction by a circular holographic display from digital holograms recorded under infrared illumination

Cataloged from PDF version of article.A circular holographic display that consists of phase-only spatial light modulators is used to reconstruct images in visible light from digital holograms recorded under infrared (10.6 μm) illumination. The reconstruction yields a holographic digital video display of a three-dimensional ghostlike image of an object floating in space where observers can move and rotate around it. © 2012 Optical Society of Americ

Trends in development of dynamic holographic displays

Creation of a dynamic 3-D display based on holography, in which a 3-D scene is encoded in terms of optical diffraction, transformed into the fringe patterns of the hologram that is further converted into a signal for a spatial light modulator (SLM) and displayed in real time, is an extremely challenging enterprise. There are various approaches targeted to solve associated problems

Transcriptome response and spatial pattern of gene expression in the primate subventricular zone neurogenic niche after cerebral ischemia

The main stem cell niche for neurogenesis in the adult mammalian brain is the subventricular zone (SVZ) that extends along the cerebral lateral ventricles. We aimed at characterizing the initial molecular responses of the macaque monkey SVZ to transient, global cerebral ischemia. We microdissected tissue lining the anterior horn of the lateral ventricle (SVZa) from 7 day post-ischemic and sham-operated monkeys. Transcriptomics shows that in ischemic SVZa, 541 genes were upregulated and 488 genes were down-regulated. The transcription data encompassing the upregulated genes revealed a profile typical for quiescent stem cells and astrocytes. In the primate brain the SVZ is morphologically subdivided in distinct and separate ependymal and subependymal regions. The subependymal contains predominantly neural stem cells (NSC) and differentiated progenitors. To determine in which SVZa region ischemia had evoked transcriptional upregulation, sections through control and ischemic SVZa were analyzed by high-throughput in situ hybridization for a total of 150 upregulated genes shown in the www.monkey-niche.org image database. The majority of the differentially expressed genes mapped to the subependymal layers on the striatal or callosal aspect of the SVZa. Moreover, a substantial number of upregulated genes was expressed in the ependymal layer, implicating a contribution of the ependyma to stem cell biology. The transcriptome analysis yielded several novel gene markers for primate SVZa including the apelin receptor that is strongly expressed in the primate SVZa niche upon ischemic insult

Ablation of BAF170 in developing and postnatal dentate gyrus affects neural stem cell proliferation, differentiation, and learning

The BAF chromatin remodeling complex plays an essential role in brain development. However its function in postnatal neurogenesis in hippocampus is still unknown. Here, we show that in postnatal dentate gyrus (DG), the BAF170 subunit of the complex is expressed in radial glial-like (RGL) progenitors and in cell types involved in subsequent steps of adult neurogenesis including mature astrocytes. Conditional deletion of BAF170 during cortical late neurogenesis as well as during adult brain neurogenesis depletes the pool of RGL cells in DG, and promotes terminal astrocyte differentiation. These derangements are accompanied by distinct behavioral deficits, as reflected by an impaired accuracy of place responding in the Morris water maze test, during both hidden platform as well as reversal learning. Inducible deletion of BAF170 in DG during adult brain neurogenesis resulted in mild spatial learning deficits, having a more pronounced effect on spatial learning during the reversal test. These findings demonstrate involvement of BAF170-dependent chromatin remodeling in hippocampal neurogenesis and cognition and suggest a specific role of adult neurogenesis in DG in adaptive behavior

Yet Another Ranking Function for Automatic Multiword Term Extraction

International audienceTerm extraction is an essential task in domain knowledge acquisition. We propose two new measures to extract multiword terms from a domain-specific text. The first measure is both linguistic and statistical based. The second measure is graph-based, allowing assessment of the importance of a multiword term of a domain. Existing measures often solve some problems related (but not completely) to term extraction, e.g., noise, silence, low frequency, large-corpora, complexity of the multiword term extraction process. Instead, we focus on managing the entire set of problems, e.g., detecting rare terms and overcoming the low frequency issue. We show that the two proposed measures outperform precision results previously reported for automatic multiword extraction by comparing them with the state-of-the-art reference measures

BAF(mSWI/SNF) complex regulates mediolateral cortical patterning in the developing forebrain

Early forebrain patterning entails the correct regional designation of the neuroepithelium, and appropriate specification, generation, and distribution of neural cells during brain development. Specific signaling and transcription factors are known to tightly regulate patterning of the dorsal telencephalon to afford proper structural/functional cortical arealization and morphogenesis. Nevertheless, whether and how changes of the chromatin structure link to the transcriptional program(s) that control cortical patterning remains elusive. Here, we report that the BAF chromatin remodeling complex regulates the spatiotemporal patterning of the mouse dorsal telencephalon. To determine whether and how the BAF complex regulates cortical patterning, we conditionally deleted the BAF complex scaffolding subunits BAF155 and BAF170 in the mouse dorsal telencephalic neuroepithelium. Morphological and cellular changes in the BAF mutant forebrain were examined using immunohistochemistry and in situ hybridization. RNA sequencing, Co-immunoprecipitation, and mass spectrometry were used to investigate the molecular basis of BAF complex involvement in forebrain patterning. We found that conditional ablation of BAF complex in the dorsal telencephalon neuroepithelium caused expansion of the cortical hem and medial cortex beyond their developmental boundaries. Consequently, the hippocampal primordium is not specified, the mediolateral cortical patterning is compromised, and the cortical identity is disturbed in the absence of BAF complex. The BAF complex was found to interact with the cortical hem suppressor LHX2. The BAF complex suppresses cortical hem fate to permit proper forebrain patterning. We provide evidence that BAF complex modulates mediolateral cortical patterning possibly by interacting with the transcription factor LHX2 to drive the LHX2-dependent transcriptional program essential for dorsal telencephalon patterning. Our data suggest a putative mechanistic synergy between BAF chromatin remodeling complex and LHX2 in regulating forebrain patterning and ontogeny

Interaction between Axons and Specific Populations of Surrounding Cells Is Indispensable for Collateral Formation in the Mammillary System

An essential phenomenon during brain development is the extension of long collateral branches by axons. How the local cellular environment contributes to the initial sprouting of these branches in specific points of an axonal shaft remains unclear.The principal mammillary tract (pm) is a landmark axonal bundle connecting ventral diencephalon to brainstem (through the mammillotegmental tract, mtg). Late in development, the axons of the principal mammillary tract sprout collateral branches at a very specific point forming a large bundle whose target is the thalamus. Inspection of this model showed a number of distinct, identified cell populations originated in the dorsal and the ventral diencephalon and migrating during development to arrange themselves into several discrete groups around the branching point. Further analysis of this system in several mouse lines carrying mutant alleles of genes expressed in defined subpopulations (including Pax6, Foxb1, Lrp6 and Gbx2) together with the use of an unambiguous genetic marker of mammillary axons revealed: 1) a specific group of Pax6-expressing cells in close apposition with the prospective branching point is indispensable to elicit axonal branching in this system; and 2) cooperation of transcription factors Foxb1 and Pax6 to differentially regulate navigation and fasciculation of distinct branches of the principal mammillary tract.Our results define for the first time a model system where interaction of the axonal shaft with a specific group of surrounding cells is essential to promote branching. Additionally, we provide insight on the cooperative transcriptional regulation necessary to promote and organize an intricate axonal tree