Pairing and continuum effects on low-frequency quadrupole vibrations in deformed Mg isotopes close to the neutron drip line

Low-frequency quadrupole vibrational modes in deformed $^{36,38,40}$Mg close to the neutron drip line are studied by means of the quasiparticle-random-phase approximation based on the coordinate-space Hartree-Fock-Bogoliubov formalism. Strongly collective $K^{\pi}=0^{+}$ and $2^{+}$ excitation modes carrying 10-20 Weisskopf units in the intrinsic isoscalar quadrupole transition strengths are obtained at about 3 MeV. There are two reasons for the enhancement of the transition strengths. First, the quasiparticle wave functions generating these modes possess spatially very extended structure. The asymptotic selection rules characterizing the $\beta$ and $\gamma$ vibrations in stable deformed nuclei are thus strongly violated. Second, the dynamic pairing effects act strongly to enhance the collectivity of these modes. It is suggested that the lowest $K^{\pi}=0^{+}$ collective mode is a particularly sensitive indicator of the nature of pairing correlations in deformed nuclei close to the neutron drip line.Comment: 23 pages including 11 figures and 8 tables. Submitted to NP

Synthesis of both enantiomers of akolactone B and (+)-ancepsenolide

The syntheses of (+)- and (−)-akolactone B and (+)-ancepsenolide were accomplished using a Pd-catalyzed carbonylation. As to the absolute configuration of akolactone B, making a comparison of the specific rotation of both enantiomers of synthetic akolactone B and the natural compound suggests that the absolute configuration at the 4-position of akolactone B is (R).ArticleTETRAHEDRON-ASYMMETRY. 25(20-21):1367-1371 (2014)journal articl

Mutations Found in the Asc1 Gene That Confer Susceptibility to the AAL-Toxin in Ancestral Tomatoes from Peru and Mexico

Tomato susceptibility/resistance to stem canker disease caused by Alternaria alternata f. sp. lycopersici and its pathogenic factor AAL-toxin is determined by the presence of the Asc1 gene. Several cultivars of commercial tomato (Solanum lycopersicum var. lycopersicum, SLL) are reported to have a mutation in Asc1, resulting in their susceptibility to AAL-toxin. We evaluated 119 ancestral tomato accessions including S. pimpinellifolium (SP), S. lycopersicum var. cerasiforme (SLC) and S. lycopersicum var. lycopersicum “jitomate criollo” (SLJ) for AAL-toxin susceptibility. Three accessions, SP PER018805, SLC PER018894, and SLJ M5-3, were susceptible to AAL-toxin. SLC PER018894 and SLJ M5-3 had a two-nucleotide deletion (nt 854_855del) in Asc1 identical to that found in SLL cv. Aichi-first. Another mutation (nt 931_932insT) that may confer AAL-toxin susceptibility was identified in SP PER018805. In the phylogenetic tree based on the 18 COSII sequences, a clade (S3) is composed of SP, including the AAL-toxin susceptible PER018805, and SLC. AAL-toxin susceptible SLC PER018894 and SLJ M5-3 were in Clade S2 with SLL cultivars. As SLC is thought to be the ancestor of SLL, and SLJ is an intermediate tomato between SLC and SLL, Asc1s with/without the mutation seem to have been inherited throughout the history of tomato domestication and breeding