30 research outputs found

    Tropical air-sea interaction in general circulation models

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    An intercomparison is undertaken of the tropical behavior of 17 coupled ocean-atmosphere models in which at least one component may be termed a general circulation model (GCM). The aim is to provide a taxonomy—a description and rough classification—of behavior across the ensemble of models, focusing on interannual variability. The temporal behavior of the sea surface temperature (SST) field along the equator is presented for each model, SST being chosen as the primary variable for intercomparison due to its crucial role in mediating the coupling and because it is a sensitive indicator of climate drift. A wide variety of possible types of behavior are noted among the models. Models with substantial interannual tropical variability may be roughly classified into cases with propagating SST anomalies and cases in which the SST anomalies develop in place. A number of the models also exhibit significant drift with respect to SST climatology. However, there is not a clear relationship between climate drift and the presence or absence of interannual oscillations. In several cases, the mode of climate drift within the tropical Pacific appears to involve coupled feedback mechanisms similar to those responsible for El Niño variability. Implications for coupled-model development and for climate prediction on seasonal to interannual time scales are discussed. Overall, the results indicate considerable sensitivity of the tropical coupled ocean-atmosphere system and suggest that the simulation of the warm-pool/cold-tongue configuration in the equatorial Pacific represents a challenging test for climate model parameterizations

    On the Tropical Atlantic SST warm bias in the Kiel Climate Model

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    Most of the current coupled general circulation models show a strong warm bias in the eastern Tropical Atlantic. In this paper, various sensitivity experiments with the Kiel Climate Model (KCM) are described. A largely reduced warm bias and an improved seasonal cycle in the eastern Tropical Atlantic are simulated in one particular version of KCM. By comparing the stable and well-tested standard version with the sensitivity experiments and the modified version, mechanisms contributing to the reduction of the eastern Atlantic warm bias are identified and compared to what has been proposed in literature. The error in the spring and early summer zonal winds associated with erroneous zonal precipitation seems to be the key mechanism, and large-scale coupled ocean-atmosphere feedbacks play an important role in reducing the warm bias. Improved winds in boreal spring cause the summer cooling in the eastern Tropical Atlantic (ETA) via shoaling of the thermocline and increased upwelling, and hence reduced sea surface temperature (SST). Reduced SSTs in the summer suppress convection and favor the development of low-level cloud cover in the ETA region. Subsurface ocean structure is shown to be improved, and potentially influences the development of the bias. The strong warm bias along the southeastern coastline is related to underestimation of low-level cloud cover and the associated overestimation of surface shortwave radiation in the same region. Therefore, in addition to the primarily wind forced response at the equator both changes in surface shortwave radiation and outgoing longwave radiation contribute significantly to reduction of the warm bias from summer to fall

    Impact of the El Nino/Southern Oscillation on visceral leishmaniasis, Brazil

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    We used time-series analysis and linear regression to investigate the relationship between the annual Nino-3 index from 1980 to 1998 and the annual incidence of visceral leishmaniasis (VL) in the State of Bahia, Brazil, during 1985- 1999. An increase in VL incidence was observed in the post-El Nino years 1989 (+38.7%) and 1995 (+33.5%). The regression model demonstrates that the previous year's mean Nino-3 index and the temporal trend account for approximately 50% of the variance in the annual incidence of VL in Bahia. The model shows a robust agreement with the real data, as only the influence of El Nino on the cycle of VL was analyzed. The results suggest that this relationship could be used to predict high-risk years for VL and thus help reduce health impact in susceptible regions in Brazil

    STOIC: a study of coupled model climatology and variability in tropical ocean regions

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    We describe the behaviour of 23 dynamical ocean-atmosphere models, in the context of comparison with observations in a common framework. Fields of tropical sea surface temperature (SST), surface wind stress and upper ocean vertically averaged temperature (VAT) are assessed with regard to annual mean, seasonal cycle, and interannual variability characteristics. Of the participating models, 21 are coupled GCMs, of which 13 use no form of flux adjustment in the tropics. The models vary widely in design, components and purpose: nevertheless several common features are apparent. In most models without flux adjustment, the annual mean equatorial SST in the central Pacific is too cool and the Atlantic zonal SST gradient has the wrong sign. Annual mean wind stress is often too weak in the central Pacific and in the Atlantic, but too strong in the west Pacific. Few models have an upper ocean VAT seasonal cycle like that observed in the equatorial Pacific. Interannual variability is commonly too weak in the models: in particular, wind stress variability is low in the equatorial Pacific. Most models have difficulty in reproducing the observed Pacific 'horseshoe' pattern of negative SST correlations with interannual Niño3 SST anomalies, or the observed Indian-Pacific lag correlations. The results for the fields examined indicate that several substantial model improvements are needed, particularly with regard to surface wind stress

    A multimodel comparison of centennial Atlantic meridional overturning circulation variability

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    A mechanism contributing to centennial variability of the Atlantic Meridional Overturning Circulation (AMOC) is tested with multi-millennial control simulations of several coupled general circulation models (CGCMs). These are a substantially extended integration of the 3rd Hadley Centre Coupled Climate Model (HadCM3), the Kiel Climate Model (KCM), and the Max Plank Institute Earth System Model (MPI-ESM). Significant AMOC variability on time scales of around 100 years is simulated in these models. The centennial mechanism links changes in the strength of the AMOC with oceanic salinities and surface temperatures, and atmospheric phenomena such as the Intertropical Convergence Zone (ITCZ). 2 of the 3 models reproduce all aspects of the mechanism, with the third (MPI-ESM) reproducing most of them. A comparison with a high resolution paleo-proxy for Sea Surface Temperatures (SSTs) north of Iceland over the last 4,000 years, also linked to the ITCZ, suggests that elements of this mechanism may also be detectable in the real world

    Enu Mutagenesis Identifies a Novel Platelet Phenotype in a Loss-Of-Function Jak2 Allele

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    <div><p>Utilizing ENU mutagenesis, we identified a mutant mouse with elevated platelets. Genetic mapping localized the mutation to an interval on chromosome 19 that encodes the Jak2 tyrosine kinase. We identified a A3056T mutation resulting in a premature stop codon within exon 19 of Jak2 (<i>Jak2</i><sup><i>K915X</i></sup>), resulting in a protein truncation and functionally inactive enzyme. This novel platelet phenotype was also observed in mice bearing a hemizygous targeted disruption of the Jak2 locus (<i>Jak2</i><sup><i>+/-</i></sup>). Timed pregnancy experiments revealed that <i>Jak2</i><sup><i>K915X/K915X</i></sup> and <i>Jak2</i><sup><i>-/-</i></sup> displayed embryonic lethality; however, <i>Jak2</i><sup><i>K915X/K915X</i></sup> embryos were viable an additional two days compared to <i>Jak2</i><sup><i>-/-</i></sup> embryos. Our data suggest that perturbing JAK2 activation may have unexpected consequences in elevation of platelet number and correspondingly, important implications for treatment of hematological disorders with constitutive Jak2 activity.</p> </div
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