The construction, development and transformation of the Young Muslim movement's identity

Предмет истраживања дисертације јесте конструкција, развој и трансформација идентитета покрета Младих муслимана, који је, као транснационалан и панисламистички, настао уочи Другог светског рата у Босни и Херцеговини...This doctoral dissertation focuses on the construction, development and transformation of the identity of the Young Muslims, which was founded as a transnational and pan-Islamist movement on the eve of the Second World War in Bosnia and Herzegovina..

Detailed investigation of the superconducting transition of niobium disks exhibiting the paramagnetic Meissner effect

The superconducting transition region in a Nb disk showing the paramagnetic Meissner effect (PME) has been investigated in detail. From the field-cooled magnetization behavior, two well-defined temperatures can be associated with the appearance of the PME: T_1 (< T_c) indicates the characteristic temperature where the paramagnetic moment first appears and a lower temperature T_p (< T_1) defines the temperature where the positive moment no longer increases. During the subsequent warming, the paramagnetic moment begins to decrease at T_p and then vanishes at T_1 with the magnitude of the magnetization change between these two temperatures being nearly the same as that during cooling. This indicates that the nature of the PME is reversible and not associated with flux motion. Furthermore, the appearance of this paramagnetic moment is even observable in fields as large as 0.2 T even though the magnetization does not remain positive to the lowest temperatures. Magnetic hysteresis loops in the temperature range between T_1 and T_p also exhibit a distinct shape that is different from the archetypal shape of a bulk type-II superconductor. These behaviors are discussed in terms of the so-called 'giant vortex state'.Comment: Total 4 printed pages, 4 Figure

Development of a spectroscopic diagnostic tool for electric field measurements in IShTAR (Ion cyclotron Sheath Test ARrangement)

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Assise: Performance and Availability via NVM Colocation in a Distributed File System

The adoption of very low latency persistent memory modules (PMMs) upends the long-established model of disaggregated file system access. Instead, by colocating computation and PMM storage, we can provide applications much higher I/O performance, sub-second application failover, and strong consistency. To demonstrate this, we built the Assise distributed file system, based on a persistent, replicated coherence protocol for managing a set of server-colocated PMMs as a fast, crash-recoverable cache between applications and slower disaggregated storage, such as SSDs. Unlike disaggregated file systems, Assise maximizes locality for all file IO by carrying out IO on colocated PMM whenever possible and minimizes coherence overhead by maintaining consistency at IO operation granularity, rather than at fixed block sizes. We compare Assise to Ceph/Bluestore, NFS, and Octopus on a cluster with Intel Optane DC PMMs and SSDs for common cloud applications and benchmarks, such as LevelDB, Postfix, and FileBench. We find that Assise improves write latency up to 22x, throughput up to 56x, fail-over time up to 103x, and scales up to 6x better than its counterparts, while providing stronger consistency semantics. Assise promises to beat the MinuteSort world record by 1.5x

Long-Term Results and Prognostic Factors of Gastric Cancer Patients with Microscopic Peritoneal Carcinomatosis

BACKGROUND: Clinical significance of microscopic peritoneal carcinomatosis remained unclear. The aim of this study was to evaluate the prognostic value of microscopic peritoneal carcinomatosis in gastric cancer. METHODS: From 1996 to 2007, 4426 patients underwent gastrectomy for gastric cancer at Fudan University Shanghai Cancer Center. The clinical and pathological data were reviewed to identify patients with microscopic peritoneal carcinomatosis (group 1). The clinicopathological features and prognosis were examined. Additionally, 242 stage-matched gastric cancer patients without microscopic peritoneal carcinomatosis (group 2) and 118 with macroscopic peritoneal carcinomatosis (group 3) were selected as control groups. RESULTS: Microscopic peritoneal carcinomatosis was found in 121 patients. There were 85 males and 36 females (2.36:1). There was a higher incidence rate of large size tumor (≥5 cm) (P = 0.045), Borrmann IV (P = 0.000), and serosal invasion (P = 0.000) in gastric cancer with microscopic peritoneal carcinomatosis compared with the control group. The 5-year survival rate of gastric cancer with microscopic peritoneal carcinomatosis was 24%, significantly poorer than that of the stage-matched control group but better than that of patients with macroscopic peritoneal carcinomatosis. The independent prognostic factors identified included pathological stage and operative curability. CONCLUSIONS: The presence of microscopic peritoneal carcinomatosis was associated with worse prognosis for gastric cancer, but curative surgery showed potential to improve prognosis

Mutations in SLC20A2 are a major cause of familial idiopathic basal ganglia calcification

Familial idiopathic basal ganglia calcification (IBGC) or Fahr's disease is a rare neurodegenerative disorder characterized by calcium deposits in the basal ganglia and other brain regions, which is associated with neuropsychiatric and motor symptoms. Familial IBGC is genetically heterogeneous and typically transmitted in an autosomal dominant fashion. We performed a mutational analysis of SLC20A2, the first gene found to cause IBGC, to assess its genetic contribution to familial IBGC. We recruited 218 subjects from 29 IBGC-affected families of varied ancestry and collected medical history, neurological exam, and head CT scans to characterize each patient's disease status. We screened our patient cohort for mutations in SLC20A2. Twelve novel (nonsense, deletions, missense, and splice site) potentially pathogenic variants, one synonymous variant, and one previously reported mutation were identified in 13 families. Variants predicted to be deleterious cosegregated with disease in five families. Three families showed nonsegregation with clinical disease of such variants, but retrospective review of clinical and neuroimaging data strongly suggested previous misclassification. Overall, mutations in SLC20A2 account for as many as 41 % of our familial IBGC cases. Our screen in a large series expands the catalog of SLC20A2 mutations identified to date and demonstrates that mutations in SLC20A2 are a major cause of familial IBGC. Non-perfect segregation patterns of predicted deleterious variants highlight the challenges of phenotypic assessment in this condition with highly variable clinical presentation