Open data from the third observing run of LIGO, Virgo, KAGRA, and GEO

The global network of gravitational-wave observatories now includes five detectors, namely LIGO Hanford, LIGO Livingston, Virgo, KAGRA, and GEO 600. These detectors collected data during their third observing run, O3, composed of three phases: O3a starting in 2019 April and lasting six months, O3b starting in 2019 November and lasting five months, and O3GK starting in 2020 April and lasting two weeks. In this paper we describe these data and various other science products that can be freely accessed through the Gravitational Wave Open Science Center at https://gwosc.org. The main data set, consisting of the gravitational-wave strain time series that contains the astrophysical signals, is released together with supporting data useful for their analysis and documentation, tutorials, as well as analysis software packages

ACTN3 and ACE genotypes in elite Jamaican and US sprinters

Acta 3 and ace genotypes in elite jamaican and us sprinters. Med. Sci. Sports exerc., Vol. 42, No. 1, Pp. 107-112, 2010. The angiotensin-converting enzyme (Ace) And the alpha-actinin-3 (Actn3) Genes are two of the most studied "Performance genes" And both have been associated with sprint/power phenotypes and elite performance. Purpose: To investigate the association between the ace and the actn3 genotypes and sprint athlete status in elite jamaican and us african american sprinters. Methods: The actn3 r577x and the ace i/d and a22982g (Rs4363) Genotype distributions of elite jamaican (J-a: N = 116) And us sprinters (Us-a: N = 114) Were compared with controls from the jamaican (J-c n = 311) And us african american (Us-c; N = 191) Populations. Frequency differences between groups were assessed by exact test. Results: For actn3, the xx genotype was found to be at very low frequency in both athlete and control cohorts (J-c = 2%, j-a = 3%. Us-c = 4%, us-a = 2%). Athletes did not differ front controls in actn3 genotype distribution (J, p = 0.87; Us, p = 0.58). Similarly, neither us nor jamaican athletes differed from controls in genotype at ace i/d (J, p = 0.44; Us, p = 0.37). Jamaican athletes did not differ from controls for a22982g genotype (P = 0.28), Although us sprinters did (P = 0.029), Displaying all excess of heterozygotes relative to controls but no excess of gg homozygotes (Us-c 22%, us-a = 18%). Conclusions: Given that actn3 xx genotype is negatively associated with elite sprint athlete status, the underlying low frequency in these populations eliminates the possibility of replicating this association in jamaican and us african american sprinters. The finding of no excess in ace dd or gg genotypes in elite sprint athletes relative to controls suggests that ace genotype is not a determinant of elite sprint athlete status