1,039 research outputs found
A hypercyclic finite rank perturbation of a unitary operator
A unitary operator and a rank operator acting on a Hilbert space
\H are constructed such that is hypercyclic. This answers affirmatively
a question of Salas whether a finite rank perturbation of a hyponormal operator
can be supercyclic.Comment: published in Mathematische Annale
Subspace hypercyclicity
A bounded linear operator T on Hilbert space is subspace-hypercyclic for a
subspace M if there exists a vector whose orbit under T intersects the subspace
in a relatively dense set. We construct examples to show that
subspace-hypercyclicity is interesting, including a nontrivial
subspace-hypercyclic operator that is not hypercyclic. There is a Kitai-like
criterion that implies subspace-hypercyclicity and although the spectrum of a
subspace-hypercyclic operator must intersect the unit circle, not every
component of the spectrum will do so. We show that, like hypercyclicity,
subspace-hypercyclicity is a strictly infinite-dimensional phenomenon.
Additionally, compact or hyponormal operators can never be
subspace-hypercyclic.Comment: 15 page
Conformal dimension and random groups
We give a lower and an upper bound for the conformal dimension of the
boundaries of certain small cancellation groups. We apply these bounds to the
few relator and density models for random groups. This gives generic bounds of
the following form, where is the relator length, going to infinity.
(a) 1 + 1/C < \Cdim(\bdry G) < C l / \log(l), for the few relator model,
and
(b) 1 + l / (C\log(l)) < \Cdim(\bdry G) < C l, for the density model, at
densities .
In particular, for the density model at densities , as the relator
length goes to infinity, the random groups will pass through infinitely
many different quasi-isometry classes.Comment: 32 pages, 4 figures. v2: Final version. Main result improved to
density < 1/16. Many minor improvements. To appear in GAF
Variability of serum markers of erythropoiesis during 6 days of racing in highly trained cyclists
The athlete biological passport for the fight against doping is currently based on longitudinal monitoring for abnormal changes in cellular blood parameters. Serum parameters related to altered erythropoiesis could be considered for inclusion in the passport. The aim of this study was to quantify the changes in such parameters in athletes during a period of intense exercise.
12 highly trained cyclists tapered for 3 days before 6 days of simulated intense stage racing. Morning and afternoon blood samples were taken on most days and analysed for total protein, albumin, soluble transferrin receptor and ferritin concentrations. Plasma volume was determined via total haemoglobin mass measured by carbon-monoxide rebreathing. Percent changes in means from baseline and percent standard errors of measurement (analytical error plus intra-athlete variation) on each measurement occasion were estimated with mixed linear modelling of log-transformed measures.
Means of all variables changed substantially in the days following the onset of racing, ranging from −13% (haemoglobin concentration) to +27% (ferritin). After the second day, errors of measurement were generally twice those at baseline.
Plasma variables were affected by heavy exercise, either because of changes in plasma volume (total protein, albumin, haemoglobin), acute phase/inflammatory reactions (ferritin) or both (soluble transferrin receptor). These effects need to be taken into consideration when integrating a plasma parameter into the biological passport model for athletes
decays
Effective chiral theory of mesons is applied to study the four decay modes of
. Theoretical values of the branching ratios are in
agreement with the data. The theory predicts that the resonance plays a
dominant role in these decays. There is no new parameter in this study.Comment: 12 pages and one figur
Monoclonal antibodies against human astrocytomas and their reactivity pattern
The establishment of hybridomas after fusion of X63-Ag8.653 mouse myeloma cells and splenocytes from mice hyperimmunized against human astrocytomas is presented. The animals were primed with 5 × 106 chemically modified uncultured or cultured glioma cells. Six weeks after the last immunization step an intrasplenal booster injection was administrated and 3 days later the spleen cells were prepared for fusion experiments. According to the specificity analysis of the generated antibodies 7 hybridoma products (MUC 7-22, MUC 8-22, MUC 10-22, MUC 11-22, MUC 14-22, MUC 15-22 and MUC 2-63) react with gliomas, neuroblastomas and melanomas as well as with embryonic and fetal cells but do not recognize non-neurogenic tumors. The selected monoclonal antibodies (McAbs) of IgG1 and IgG2a isotypes are not extensively characterized but these antibodies have been demonstrated to be reactive with a panel of glioma cell lines with varying patterns of antigen distribution. Using the McAbs described above and a series of cryosections of glioma biopsies and paraffin sections of the same material as well as glioma cultures established from these, variable antigenic profiles among glioma cell populations could be demonstrated. From these results it is evident that there is not only a distinct degree of antigenic heterogeneity among and within brain tumors, but also that the pattern of antigenic expression can change continuously. Some of the glioma associated antigens recognized by the selected antibodies persist after fixation with methanol/acetone and Karnovsky's fixative and probably are oncoembryonic/oncofetal antigen(s). The data suggest that the use of McAbs recognizing tumor associated oncofetal antigens in immunohistochemistry facilitates objective typing of intracranial malignancies and precise analysis of fine needle brain/tumor biopsies in a sensitive and reproducible manner
Southern Ocean control of silicon stable isotope distribution in the deep Atlantic Ocean
Author Posting. © American Geophysical Union, 2012. This article is posted here by permission of American Geophysical Union for personal use, not for redistribution. The definitive version was published in Global Geochemical Cycles 26 (2012): GB2035, doi:10.1029/2011GB004141.The fractionation of silicon (Si) stable isotopes by biological activity in the surface ocean makes the stable isotope composition of silicon (δ30Si) dissolved in seawater a sensitive tracer of the oceanic biogeochemical Si cycle. We present a high-precision dataset that characterizes the δ30Si distribution in the deep Atlantic Ocean from Denmark Strait to Drake Passage, documenting strong meridional and smaller, but resolvable, vertical δ30Si gradients. We show that these gradients are related to the two sources of deep and bottom waters in the Atlantic Ocean: waters of North Atlantic and Nordic origin carry a high δ30Si signature of ≥+1.7‰ into the deep Atlantic, while Antarctic Bottom Water transports Si with a low δ30Si value of around +1.2‰. The deep Atlantic δ30Si distribution is thus governed by the quasi-conservative mixing of Si from these two isotopically distinct sources. This disparity in Si isotope composition between the North Atlantic and Southern Ocean is in marked contrast to the homogeneity of the stable nitrogen isotope composition of deep ocean nitrate (δ15N-NO3). We infer that the meridional δ30Si gradient derives from the transport of the high δ30Si signature of Southern Ocean intermediate/mode waters into the North Atlantic by the upper return path of the meridional overturning circulation (MOC). The basin-scale deep Atlantic δ30Si gradient thus owes its existence to the interaction of the physical circulation with biological nutrient uptake at high southern latitudes, which fractionates Si isotopes between the abyssal and intermediate/mode waters formed in the Southern Ocean.This work was supported by Swiss National Science Foundation
grants 200021-116473 and 200020-130361.2012-12-1
Quantifying loopy network architectures
Biology presents many examples of planar distribution and structural networks
having dense sets of closed loops. An archetype of this form of network
organization is the vasculature of dicotyledonous leaves, which showcases a
hierarchically-nested architecture containing closed loops at many different
levels. Although a number of methods have been proposed to measure aspects of
the structure of such networks, a robust metric to quantify their hierarchical
organization is still lacking. We present an algorithmic framework, the
hierarchical loop decomposition, that allows mapping loopy networks to binary
trees, preserving in the connectivity of the trees the architecture of the
original graph. We apply this framework to investigate computer generated
graphs, such as artificial models and optimal distribution networks, as well as
natural graphs extracted from digitized images of dicotyledonous leaves and
vasculature of rat cerebral neocortex. We calculate various metrics based on
the Asymmetry, the cumulative size distribution and the Strahler bifurcation
ratios of the corresponding trees and discuss the relationship of these
quantities to the architectural organization of the original graphs. This
algorithmic framework decouples the geometric information (exact location of
edges and nodes) from the metric topology (connectivity and edge weight) and it
ultimately allows us to perform a quantitative statistical comparison between
predictions of theoretical models and naturally occurring loopy graphs.Comment: 17 pages, 8 figures. During preparation of this manuscript the
authors became aware of the work of Mileyko at al., concurrently submitted
for publicatio
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