Ethnic Groups: All or Multiple

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Family & Kin Relationships: All or Multiple

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Voluntary Associations: All or Multiple

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The 3-D shaping of NGC 6741: a massive, fast-evolving Planetary Nebula at the recombination--reionization edge

We infer the gas kinematics, diagnostics and ionic radial profiles, distance and central star parameters, nebular photo- ionization model, spatial structure and evolutionary phase of the PN NGC 6741 by means of long-slit high-resolution spectra at nine position angles. NGC 6741 (distance ~2.0 kpc, age ~1400 yr, ionized mass Mion ~0.06 Mo) is a dense (electron density up to 12,000 cm^(-3)), high-excitation, almost- prolate ellipsoid, surrounded by a sharp low-excitation skin (the ionization front), and embedded into a spherical (radius ~ 0.080 pc), almost-neutral, high-density (n(HI) ~7 x 10^3 atoms cm^(-3)) halo containing a large fraction of the nebular mass (Mhalo>0.20 Mo). The kinematics, physical conditions and ionic structure indicate that NGC 6741 is in a deep recombination phase, started about 200 years ago, and caused by the quick luminosity drop of the massive (M*=0.66-0.68 Mo), hot (logT* ~ 5.23) and faint (log L*/Lo ~ 2.75) post--AGB star, which has exhausted the hydrogen-shell nuclear burning and is moving along the white dwarf cooling sequence. The general expansion law of the ionized gas in NGC 6741, Vexp (km s^(-1)=13 x R", fails in the innermost, highest-excitation layers, which move slower than expected. The observed deceleration is ascribable to the luminosity drop of the central star, and appears in striking contrast to recent reports inferring that acceleration is a common property of the Planetary Nebulae innermost layers. Some general implications on the shaping mechanisms of Planetary Nebulae are discussed.Comment: 27 pages, 18 figures, accepted for publication in A&A, movies of the reconstructed nebula are available at http://web.pd.astro.it/sabbadin

The 3--D ionization structure of NGC 6818: a Planetary Nebula threatened by recombination

Long-slit NTT+EMMI echellograms of NGC 6818 (the Little Gem) at nine equally spaced position angles, reduced according to the 3-D methodology introduced by Sabbadin et al. (2000a,b), allowed us to derive: the expansion law, the diagnostics and ionic radial profiles, the distance and the central star parameters, the nebular photo-ionization model, the 3-D reconstruction in He II, [O III] and [N II], the multicolor projection and a series of movies. The Little Gem results to be a young (3500 years), optically thin (quasi-thin in some directions) double shell (Mion~0.13 Msun) at a distance of 1.7 kpc, seen almost equatorial on: a tenuous and patchy spherical envelope (r~0.090 pc) encircles a dense and inhomogeneous tri-axial ellipsoid (a/2~0.077 pc, a/b~1.25, b/c~1.15) characterized by a hole along the major axis and a pair of equatorial, thick moustaches. NGC 6818 is at the start of the recombination phase following the luminosity decline of the 0.625 Msun central star, which has recently exhausted the hydrogen shell nuclear burning and is rapidly moving toward the white dwarf domain (log T*~5.22 K; log(L*/Lsun)~3.1). The nebula is destined to become thicker and thicker, with an increasing fraction of neutral, dusty gas in the outermost layers. Only over some hundreds of years the plasma rarefaction due to the expansion will prevail against the slower and slower stellar decline, leading to a gradual re-growing of the ionization front. The exciting star of NGC 6818 (mV~17.06) is a visual binary: a faint, red companion (mV~17.73) appears at 0.09 arcsec in PA=190deg, corresponding to a separation >=150 AU and to an orbital period >=1500 yearsComment: 23 pages, 18 figures, A&A accepted. 12 movies of NGC 6818 are available at http://web.pd.astro.it/sabbadin The paper may also be retrivied at http://web.pd.astro.it/supern/preprints.htm

Common Genetic Variants Contribute to Risk of Transposition of the Great Arteries.

RATIONALE: Dextro-transposition of the great arteries (D-TGA) is a severe congenital heart defect which affects approximately 1 in 4,000 live births. While there are several reports of D-TGA patients with rare variants in individual genes, the majority of D-TGA cases remain genetically elusive. Familial recurrence patterns and the observation that most cases with D-TGA are sporadic suggest a polygenic inheritance for the disorder, yet this remains unexplored. OBJECTIVE: We sought to study the role of common single nucleotide polymorphisms (SNPs) in risk for D-TGA. METHODS AND RESULTS: We conducted a genome-wide association study in an international set of 1,237 patients with D-TGA and identified a genome-wide significant susceptibility locus on chromosome 3p14.3, which was subsequently replicated in an independent case-control set (rs56219800, meta-analysis P=8.6x10 CONCLUSIONS: This work provides support for a polygenic architecture in D-TGA and identifies a susceptibility locus on chromosome 3p14.3 nea

Recreational Associations: All or Multiple

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Basketball, Boxing, Football

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Professional Sports All or Multiple

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Trade Associations

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