Best Practices for Library Exhibitions

Best Practices for Library Exhibitions provides a comprehensive framework and recommended practices for developing and managing exhibitions in art libraries and similar types of information environments. It offers real-world insights complementing library and information science program curricula enriching students’ learning about exhibition development, which is a growing expectation for librarians. The document is organized by three main areas: Creation, Operation and Logistics, and Management, reflecting the actual cycle of exhibitions. Specific best practices include Curation and Policies, Digital Exhibits, Diversity/Equity/Inclusion/Accessibility, Loaning, Conservation Care, Facilities, Engagement, Marketing and Outreach, Documentation, Evaluation, and Financial Management. The initiative for this project was endeavored by the Exhibitions Special Interest Group with contributions from library professionals representing a variety of institutions and specialties

Nontuberculous Mycobacteria–associated Lung Disease in Hospitalized Persons, United States, 1998–2005

This bacterium is an underappreciated cause of lung disease and infection rates appear to be increasing

Global Genotype-Phenotype Correlations in Pseudomonas aeruginosa

Once the genome sequence of an organism is obtained, attention turns from identifying genes to understanding their function, their organization and control of metabolic pathways and networks that determine its physiology. Recent technical advances in acquiring genome-wide data have led to substantial progress in identifying gene functions. However, we still do not know the function of a large number of genes and, even when a gene product has been assigned to a functional class, we cannot normally predict its contribution to the phenotypic behaviour of the cell or organism - the phenome. In this study, we assessed bacterial growth parameters of 4030 non-redundant PA14 transposon mutants in the pathogenic bacterium Pseudomonas aeruginosa. The genome-wide simultaneous analysis of 119 distinct growth-related phenotypes uncovered a comprehensive phenome and provided evidence that most genotypes are not phenotypically isolated but rather define specific complex phenotypic clusters of genotypes. Since phenotypic overlap was demonstrated to reflect the relatedness of genotypes on a global scale, knowledge of an organism's phenome might significantly contribute to the advancement of functional genomics

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Phone booths at the National Archives - College Park, Maryland.https://digitalcommons.risd.edu/library_stuffwemake_annuallibrarystaffartexhibit_30/1005/thumbnail.jp

Collecting Comics at Fleet Library | a panel discussion

Collecting Comics at Fleet Library | a panel discussion with Bill Adler, music journalist and donor of the Adler Archive of Underground Comix, Tim Finn 00 FAV, owner of Hub Comics (Somerville, MA) and library donor, and Claudia Covert, Special Collections Librarian, Fleet Library. Moderated by Susan Doyle, RISD Illustration (originally planned as Jaleen Grove, RISD Illustration). Introduced by Eric Telfort, RISD Illustration. Tuesday, October 25, 2022, 6:30 pm. Fleet Library, First Floor Living Room , Roger Mandle Building (15 Westminster). In celebration of one of the great and growing strengths of the collections at Fleet Library, this freewheeling conversation about collecting comics, cartoons, and graphic novels from private, commercial, and academic perspectives took place on October 25, 2022. Also considered were the art, ephemera, and sometimes challenging issues related to the fields of comics and comix. The event was co-sponsored by Fleet Library, the Illustration Department, and RISD Institutional Advancement. View Comix from the Adler Archive of Underground Comix.https://digitalcommons.risd.edu/library_events/1000/thumbnail.jp

Karen Idoine, Claudia Covert, ,Kevin McNulty, Diane Blair, Ted Weller, Retirement Party for Karen Idoine, 2019

https://digitalcommons.risd.edu/experimentalandfoundationstudies_history_facultyandstaffportraits/1180/thumbnail.jp

\u3cem\u3eRE:Making: A Documentation of Work by Angela Lorenz\u3c/em\u3e Book Launch

Please join us Thursday, October 12, 2023 at 6:30 PM EDT at the Fleet Library as we celebrate the publication of RE:Making, a volume of 70 essays on historical observation, collections, transformation, and material applications in the work of book artist Angela Lorenz (Brown ‘87) from 1982 to 2022. Lorenz will be in conversation with Special Collections Librarian Claudia Covert, Editor Judith Tolnick Champa and contributors to the volume’s essays, including Massimo Riva, Jennifer Liese, and Margot McIlwain Nishimura. Description from the publisher, the jenny press, New Haven, CT: With essays from over 70 scholars, collectors, librarians, curators, and journalists on Lorenz\u27s work from 1989 to 2022. Includes a reflection from Angela on her juvenilia from 1982 to 1988. Edited by Judith Tolnick Champa with Jae Jennifer Rossman. Trade Edition. Color images of all works plus QR codes to videos. 200 pages. Books will be available for purchase and signing by the author. Free and open to the public.https://digitalcommons.risd.edu/library_events/1003/thumbnail.jp

Key differences in TLR3/poly I:C signaling and cytokine induction by human primary cells: a phenomenon absent from murine cell systems

TLR3 recognizes double-stranded RNA, a product associated with viral infections. Many details of TLR3-induced mechanisms have emerged from gene-targeted mice or inhibition studies in transformed cell lines. However, the pathways activated in human immune cells or cells from disease tissue are less well understood. We have investigated TLR3-induced mechanisms of human primary cells of the innate immune system, including dendritic cells (DCs), macrophages (MØs), endothelial cells (ECs), and synovial fibroblasts isolated from rheumatoid arthritis joint tissue (RA-SFs). Here, we report that while these cells all express TLR3, they differ substantially in their response to TLR3 stimulation. The key antiviral response chemokine IP-10 was produced by all cell types, while DCs and MØs failed to produce the proinflammatory cytokines TNFalpha and IL-6. Unexpectedly, TNFalpha was found secreted by TLR3-stimulated RA-SF. Furthermore, TLR3 stimulation did not activate NFkappaB, MAPKs, or IRF-3 in DCs and MØs, but was able to do so in ECs and RA-SF. These findings were specific for human cells, thereby revealing a complexity not previously expected. This is the first report of such cell type- and species-specific response for any TLR stimulation and helps to explain important difficulties in correlating murine models of inflammatory diseases and human inflammation