219 research outputs found

    A comparison of terrestrial laser scanning and structure-from-motion photogrammetry as methods for digital outcrop acquisition

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    Terrestrial laser scanning (TLS) has been used extensively in Earth Science for acquisition of digital outcrop data over the past decade. Structure-from-­motion (SfM) photogrammetry has recently emerged as an alternative and competing technology. The real-world performance of these technologies for ground-based digital outcrop acquisition is assessed using outcrops from North East England and the United Arab Emirates. Both TLS and SfM are via­ble methods, although no single technology is universally best suited to all situations. There are a range of practical considerations and operating conditions where each method has clear advantages. In comparison to TLS, SfM benefits from being lighter, more compact, cheaper, more easily replaced and repaired, with lower power requirements. TLS in comparison to SfM provides intrinsically validated data and more robust data acquisition in a wide range of operating conditions. Data post-processing is also swifter. The SfM data sets were found to contain systematic inaccuracies when compared to their TLS counterparts. These inaccuracies are related to the triangulation approach of the SfM, which is distinct from the time-of-flight principle employed by TLS. An elaborate approach is required for SfM to produce comparable results to TLS under most circumstances

    "Level Curvature" Distribution for Diffusive Aharonov-Bohm Systems: analytical results

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    We calculate analytically the distributions of "level curvatures" (LC) (the second derivatives of eigenvalues with respect to a magnetic flux) for a particle moving in a white-noise random potential. We find that the Zakrzewski-Delande conjecture is still valid even if the lowest weak localization corrections are taken into account. The ratio of mean level curvature modulus to mean dissipative conductance is proved to be universal and equal to 2π2\pi in agreement with available numerical data.Comment: 12 pages. Submitted to Phys.Rev.

    Relating the Lorentzian and exponential: Fermi's approximation,the Fourier transform and causality

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    The Fourier transform is often used to connect the Lorentzian energy distribution for resonance scattering to the exponential time dependence for decaying states. However, to apply the Fourier transform, one has to bend the rules of standard quantum mechanics; the Lorentzian energy distribution must be extended to the full real axis <E<-\infty<E<\infty instead of being bounded from below 0E<0\leq E <\infty (``Fermi's approximation''). Then the Fourier transform of the extended Lorentzian becomes the exponential, but only for times t0t\geq 0, a time asymmetry which is in conflict with the unitary group time evolution of standard quantum mechanics. Extending the Fourier transform from distributions to generalized vectors, we are led to Gamow kets, which possess a Lorentzian energy distribution with <E<-\infty<E<\infty and have exponential time evolution for tt0=0t\geq t_0 =0 only. This leads to probability predictions that do not violate causality.Comment: 23 pages, no figures, accepted by Phys. Rev.

    Microscopic theories of neutrino-^{12}C reactions

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    In view of the recent experiments on neutrino oscillations performed by the LSND and KARMEN collaborations as well as of future experiments, we present new theoretical results of the flux averaged 12C(νe,e)12N^{12}C(\nu_e,e^-)^{12}N and 12C(νμ,μ)12N^{12}C(\nu_{\mu},{\mu}^-)^{12}N cross sections. The approaches used are charge-exchange RPA, charge-exchange RPA among quasi-particles (QRPA) and the Shell Model. With a large-scale shell model calculation the exclusive cross sections are in nice agreement with the experimental values for both reactions. The inclusive cross section for νμ\nu_{\mu} coming from the decay-in-flight of π+\pi^+ is 15.2×1040cm215.2 \times 10^{-40} cm^2 to be compared to the experimental value of 12.4±0.3±1.8×1040cm212.4 \pm 0.3 \pm 1.8 \times 10^{-40} cm^2, while the one due to νe\nu_{e} coming from the decay-at-rest of μ+\mu^+ is 16.4×1042cm216.4 \times 10^{-42} cm^2 which agrees within experimental error bars with the measured values. The shell model prediction for the decay-in-flight neutrino cross section is reduced compared to the RPA one. This is mainly due to the different kind of correlations taken into account in the calculation of the spin modes and partially due to the shell-model configuration basis which is not large enough, as we show using arguments based on sum-rules.Comment: 17 pages, latex, 5 figure

    A tetragonal-to-monoclinic phase transition in a ferroelectric perovskite: the structure of PbZr(0.52)Ti(0.48)O3

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    The perovskite-like ferroelectric system PbZr(1-x)Ti(x)O3 (PZT) has a nearly vertical morphotropic phase boundary (MPB) around x=0.45-0.50. Recent synchrotron x-ray powder diffraction measurements by Noheda et al. [Appl. Phys. Lett. 74, 2059 (1999)] have revealed a new monoclinic phase between the previously-established tetragonal and rhombohedral regions. In the present work we describe a Rietveld analysis of the detailed structure of the tetragonal and monoclinic PZT phases on a sample with x= 0.48 for which the lattice parameters are respectively: at= 4.044 A, ct= 4.138 A, at 325 K, and am= 5.721 A, bm= 5.708 A, cm= 4.138 A, beta= 90.496 deg., at 20K. In the tetragonal phase the shifts of the atoms along the polar [001] direction are similar to those in PbTiO3 but the refinement indicates that there are, in addition, local disordered shifts of the Pb atoms of ~0.2 A perpendicular to the polar axis.. The monoclinic structure can be viewed as a condensation along one of the directions of the local displacements present in the tetragonal phase. It equally well corresponds to a freezing-out of the local displacements along one of the directions recently reported by Corker et al.[J. Phys. Condens. Matter 10, 6251 (1998)] for rhombohedral PZT. The monoclinic structure therefore provides a microscopic picture of the MPB region in which one of the "locally" monoclinic phases in the "average" rhombohedral or tetragonal structures freezes out, and thus represents a bridge between these two phases.Comment: REVTeX, 7 figures. Modifications after referee's suggestion: new figure (figure 5), comments in 2nd para. (Sect.III) and in 2nd & 3rd para. (Sect. IV-a), in the abstract: "...of ~0.2 A perpendicular to the polar axis.

    Respiratory plasticity in response to changes in oxygen supply and demand

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    Aerobic organisms maintain O2 homeostasis by responding to changes in O2 supply and demand in both short and long time domains. In this review, we introduce several specific examples of respiratory plasticity induced by chronic changes in O2 supply (environmental hypoxia or hyperoxia) and demand (exercise-induced and temperature-induced changes in aerobic metabolism). These studies reveal that plasticity occurs throughout the respiratory system, including modifications to the gas exchanger, respiratory pigments, respiratory muscles, and the neural control systems responsible for ventilating the gas exchanger. While some of these responses appear appropriate (e.g., increases in lung surface area, blood O2 capacity, and pulmonary ventilation in hypoxia), other responses are potentially harmful (e.g., increased muscle fatigability). Thus, it may be difficult to predict whole-animal performance based on the plasticity of a single system. Moreover, plastic responses may differ quantitatively and qualitatively at different developmental stages. Much of the current research in this field is focused on identifying the cellular and molecular mechanisms underlying respiratory plasticity. These studies suggest that a few key molecules, such as hypoxia inducible factor (HIF) and erythropoietin, may be involved in the expression of diverse forms of plasticity within and across species. Studying the various ways in which animals respond to respiratory challenges will enable a better understanding of the integrative response to chronic changes in O2 supply and deman

    Detecting non-binomial sex allocation when developmental mortality operates

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    Optimal sex allocation theory is one of the most intricately developed areas of evolutionary ecology. Under a range of conditions, particularly under population sub-division, selection favours sex being allocated to offspring non-randomly, generating non-binomial variances of offspring group sex ratios. Detecting non-binomial sex allocation is complicated by stochastic developmental mortality, as offspring sex can often only be identified on maturity with the sex of non-maturing offspring remaining unknown. We show that current approaches for detecting non-binomiality have limited ability to detect non-binomial sex allocation when developmental mortality has occurred. We present a new procedure using an explicit model of sex allocation and mortality and develop a Bayesian model selection approach (available as an R package). We use the double and multiplicative binomial distributions to model over- and under-dispersed sex allocation and show how to calculate Bayes factors for comparing these alternative models to the null hypothesis of binomial sex allocation. The ability to detect non-binomial sex allocation is greatly increased, particularly in cases where mortality is common. The use of Bayesian methods allows for the quantification of the evidence in favour of each hypothesis, and our modelling approach provides an improved descriptive capability over existing approaches. We use a simulation study to situations where current methods fail, and we illustrate the approach in real scenarios using empirically obtained datasets on the sexual composition of groups of gregarious parasitoid wasps demonstrate substantial improvements in power for detecting non-binomial sex allocation in situations where current methods fail, and we illustrate the approach in real scenarios using empirically obtained datasets on the sexual composition of groups of gregarious parasitoid wasps

    iNOS activity is critical for the clearance of Burkholderia mallei from infected RAW 264.7 murine macrophages

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    Burkholderia mallei is a facultative intracellular pathogen that can cause fatal disease in animals and humans. To better understand the role of phagocytic cells in the control of infections caused by this organism, studies were initiated to examine the interactions of B. mallei with RAW 264.7 murine macrophages. Utilizing modified kanamycin-protection assays, B. mallei was shown to survive and replicate in RAW 264.7 cells infected at multiplicities of infection (moi) of ≤ 1. In contrast, the organism was efficiently cleared by the macrophages when infected at an moi of 10. Interestingly, studies demonstrated that the monolayers only produced high levels of TNF-α, IL-6, IL-10, GM-CSF, RANTES and IFN-β when infected at an moi of 10. In addition, nitric oxide assays and inducible nitric oxide synthase (iNOS) immunoblot analyses revealed a strong correlation between iNOS activity and clearance of B. mallei from RAW 264.7 cells. Furthermore, treatment of activated macrophages with the iNOS inhibitor, aminoguanidine, inhibited clearance of B. mallei from infected monolayers. Based upon these results, it appears that moi significantly influence the outcome of interactions between B. mallei and murine macrophages and that iNOS activity is critical for the clearance of B. mallei from activated RAW 264.7 cells
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