6 research outputs found
Roflumilast in moderate-to-severe chronic obstructive pulmonary disease treated with longacting bronchodilators: two randomised clinical trials
- Author
- A. Altés
- A. Altés
- A. Bettendorf
- A. Bettendorf
- A. C. Roldaan
- A. C. Roldaan
- A. Chopra
- A. Chopra
- A. Delobbe
- A. Delobbe
- A. Delobbe
- A. Dhar
- A. Dhar
- A. F. Kuipers
- A. F. Kuipers
- A. Flemale
- A. Flemale
- A. Graham
- A. Graham
- A. H. Bruning
- A. H. Bruning
- A. J. Schreurs
- A. J. Schreurs
- A. Jackson
- A. Jackson
- A. Kroker
- A. Kroker
- A. Ludwig Sengpiel
- A. Ludwig Sengpiel
- A. M. Moretti
- A. M. Moretti
- A. Mansur
- A. Mansur
- A. Marin
- A. Marin
- A. Mclvor
- A. Mclvor
- A. Nel
- A. Nel
- A. Smithers
- A. Smithers
- A. Zanini
- A. Zanini
- Aguilaniu
- Aguilaniu
- B. B. Von Der Heydt
- B. B. Von Der Heydt
- B. Balint
- B. Balint
- B. Craig
- B. Craig
- B. Glekin
- B. Glekin
- B. Gross
- B. Gross
- B. Mcdonald
- B. Mcdonald
- B. Paradis
- B. Paradis
- B. Pigearias
- B. Pigearias
- B. Terol
- B. Terol
- Brande Van Den
- Brande Van Den
- C. Almonacid
- C. Almonacid
- C. Brotons
- C. Brotons
- C. Bucca
- C. Bucca
- C. Esteban
- C. Esteban
- C. Hutter
- C. Hutter
- C. Laskowitz
- C. Laskowitz
- C. Le Merre
- C. Le Merre
- C. Mckinnon
- C. Mckinnon
- C. Murio
- C. Murio
- C. Pellicer
- C. Pellicer
- C. S. De Graaff
- C. S. De Graaff
- C. Sevette
- C. Sevette
- C. Shum
- C. Shum
- C. Terzano
- C. Verkindre
- C. Verkindre
- Caldwell
- Caldwell
- D. Cheung
- D. Cheung
- D. E. Saracho Jo
- D. E. Saracho Jo
- D. L. Kirsten
- D. L. Kirsten
- D. Marciniuk
- D. Marciniuk
- D. Mcnally
- D. Mcnally
- D. Muller
- D. Muller
- D. R. De Munck
- D. R. De Munck
- D. Rozen
- D. Rozen
- Daniela S. Bundschuh
- E. Bateman
- E. Bateman
- E. Bauchnect
- E. Bauchnect
- E. Chiner
- E. Chiner
- E. Clini
- E. Clini
- E. M. Irusen
- E. M. Irusen
- E. Perez
- E. Perez
- E. Valyon
- E. Valyon
- F. Barbe
- F. Barbe
- F. Bonnaud
- F. Bonnaud
- F. Lemoigne
- F. Lemoigne
- F. Maltais
- F. Maltais
- F. Mazza
- F. Mazza
- F. Sanchez Toril
- F. Sanchez Toril
- F. Vrancken
- F. Vrancken
- Fernando J. Martinez
- Foden
- Foden
- Frognier
- Frognier
- G. Chapman
- G. Chapman
- G. Di Maria
- G. Di Maria
- G. Duschek
- G. Duschek
- G. E. Burns
- G. E. Burns
- G. Garelli
- G. Garelli
- G. Holub
- G. Holub
- G. Juhasz
- G. Juhasz
- G. Moder
- G. Moder
- G. Philteos
- G. Philteos
- G. R. Boyer
- G. R. Boyer
- G. Ras
- G. Ras
- G. Tellier
- G. Tellier
- G. Toma
- G. Toma
- G. Vereecken
- G. Vereecken
- G. W. Canonica
- G. W. Canonica
- G. W. Canonica
- H. A. Trauth
- H. A. Trauth
- H. Artner
- H. E. Damsté
- H. E. Damsté
- H. Grygier
- H. Grygier
- H. H. Weber
- H. H. Weber
- H. Jullian
- H. Jullian
- H. Kafe
- H. Kafe
- H. Kinch
- H. Kinch
- H. Michael
- H. Querfurt
- H. Querfurt
- H. Schiesbühl
- H. Schiesbühl
- H. Steffen
- H. Steffen
- H. V. Hernandez Hector
- H. V. Hernandez Hector
- Harper
- Harper Ochoa
- I. Abdullah
- I. Abdullah
- I. Albert
- I. Albert
- I. S. Farmer
- I. S. Farmer
- I. Vinkler
- I. Vinkler
- J. A. Van Noord
- J. A. Van Noord
- J. Angrill
- J. Angrill
- J. B. Martinot
- J. B. Martinot
- J. Belda
- J. Belda
- J. Bourbeau
- J. Bourbeau
- J. Carter
- J. Carter
- J. Dupouy
- J. Dupouy
- J. Echave
- J. Echave
- J. Grillenberger
- J. Grillenberger
- J. J. Soler
- J. J. Soler
- J. J. Viljoen
- J. J. Viljoen
- J. Joubert
- J. Joubert
- J. Kidney
- J. Kidney
- J. L. Heredia
- J. L. Heredia
- J. L. Izquierdo
- J. M. Antonana
- J. M. Antonana
- J. Messner
- J. Messner
- J. Patrick
- J. Patrick
- J. Road
- J. Road
- J. Roig
- J. Roig
- J. V. Greses
- J. V. Greses
- J. V. Vaquer
- J. V. Vaquer
- J. Wurtz
- J. Wurtz
- J. Y. Jasnot
- J. Y. Jasnot
- Jose Luis Izquierdo Alonso
- K. H. Franz
- K. H. Franz
- K. Holgate
- K. Holgate
- K. Killian
- K. Killian
- K. Korlipara
- K. Korlipara
- K. R. Chapman
- K. R. Chapman
- K. Rajkay
- K. Rajkay
- Kf M. Rabe
- L. A. De Llano
- L. A. De Llano
- L. Bernabeu
- L. Bernabeu
- L. Blecher
- L. Blecher
- L. Croonenborghs
- L. Croonenborghs
- L. De Teresa
- L. De Teresa
- L. Fabbri
- L. Fouquert
- L. Fouquert
- L. Homik
- L. Homik
- L. P. Krige
- L. P. Krige
- L. Ramos Pde
- L. Savary
- L. Savary
- L. Volgmann
- L. Volgmann
- Leonardo M. Fabbri
- M. Adler
- M. Adler
- M. Blagden
- M. Blagden
- M. Blagden
- M. C. Bride
- M. C. Bride
- M. Czompó
- M. Czompó
- M. Decramer
- M. Decramer
- M. Gagnon
- M. Gagnon
- M. Hoefer
- M. Hoefer
- M. Labrecque
- M. Labrecque
- M. Luengo
- M. Luengo
- M. P. Barbaro
- M. Prins
- M. Prins
- M. Rolke
- M. Rolke
- M. Schiavina
- M. Schiavina
- M. Study Groups Abdulla
- M. Sweilem
- M. Sweilem
- M. Timar
- M. Timar
- M. Vigh
- M. Vigh
- M. Zabaleta
- M. Zabaleta
- M. Zeiner
- M. Zeiner
- Manja Brose
- Mdel M. Garcia
- Mdel M. Garcia
- N. Savani
- N. Savani
- Ochoa
- P. A. Houle
- P. A. Houle
- P. Coulet
- P. Coulet
- P. Fletcher
- P. Fletcher
- P. Greillier
- P. Greillier
- P. Hernandez
- P. Hernandez
- P. Hyvernat
- P. Hyvernat
- P. Kelly
- P. Kelly
- P. L. Wielders
- P. L. Wielders
- P. Larivee
- P. Larivee
- P. M. Mengeot
- P. M. Mengeot
- P. Martin
- P. Martin
- P. Mooney
- P. Mooney
- P. Paggiaro
- P. Paggiaro
- P. T. Monserrat
- P. T. Monserrat
- P. Willoughby
- P. Willoughby
- Pde L. Ramos
- Peter M. A. Calverley
- R. Abdulla
- R. Blanquer
- R. Blanquer
- R. Luton
- R. Luton
- R. Peche
- R. Peche
- R. Popovic
- R. Popovic
- R. Sharma
- R. Sharma
- R. Voves
- R. Voves
- R. W. Dal Negro
- R. W. Dal Negro
- S. Amaducci
- S. Amaducci
- S. Henein
- S. Henein
- S. M. Ali
- S. M. Ali
- S. Nardini
- S. Nardini
- S. Visser
- S. Visser
- T. A. Bantje
- T. A. Bantje
- T. Dapper
- T. Dapper
- T. Ginko
- T. Ginko
- T. Gooding
- T. Gooding
- T. Perez
- T. Perez
- TERZANO Claudio
- U. Gehling
- U. Gehling
- U. Kleinecke Pohl
- U. Kleinecke Pohl
- U. Steinhauser
- U. Steinhauser
- V. Chan
- V. Chan
- V. Hoffstein
- V. Hoffstein
- V. Seiz
- V. Seiz
- W. Denier
- W. Denier
- W. Holler
- W. Holler
- W. Janisty
- W. Janistyn
- W. Macnee
- W. Macnee
- W. Pohl
- W. Pohl
- W. R. Pieters
- W. R. Pieters
- W. Schröder Babo
- W. Schröder Babo
- W. Schurmann
- W. Schurmann
- W. Vincken
- W. Vincken
- W. Vorderstrasse
- W. Vorderstrasse
- W. Yang
- W. Yang
- W. Zachgo
- W. Zachgo
- Z. Gyori
- Z. Gyori
- Publication venue
- 'Elsevier BV'
- Publication date
- 01/01/2009
- Field of study
Background Patients with chronic obstructive pulmonary disease (COPD) have few options for treatment. The efficacy and safety of the phosphodiesterase-4 inhibitor roflumilast have been investigated in studies of patients with moderate-to-severe COPD, but not in those concomitantly treated with longacting inhaled bronchodilators. The effect of roflumilast on lung function in patients with COPD that is moderate to severe who are already being treated with salmeterol or tiotropium was investigated. Methods In two double-blind, multicentre studies done in an outpatient setting, after a 4-week run-in, patients older than 40 years with moderate-to-severe COPD were randomly assigned to oral roflumilast 500 mu g or placebo once a day for 24 weeks, in addition to salmeterol (M2-127 study) or tiotropium (M2-128 study). The primary endpoint was change in prebronchodilator forced expiratory volume in 1s (FEV(1)). Analysis was by intention to treat. The studies are registered with ClinicalTrials.gov, number NCT00313209 for M2-127, and NCT00424268 for M2-128. Findings In the salmeterol plus roflumilast trial, 466 patients were assigned to and treated with roflumilast and 467 with placebo; in the tiotropium plus roflumilast trial, 371 patients were assigned to and treated with roflumilast and 372 with placebo. Compared with placebo, roflumilast consistently improved mean prebronchodilator FEV(1) by 49 mL (p<0.0001) in patients treated with salmeterol, and 80 mL (p<0.0001) in those treated with tiotropium. Similar improvement in postbronchodilator FEV(1) was noted in both groups. Furthermore, roflumilast had beneficial effects on other lung function measurements and on selected patient-reported outcomes in both groups. Nausea, diarrhoea, weight loss, and, to a lesser extent, headache were more frequent in patients in the roflumilast groups. These adverse events were associated with increased patient withdrawal. Interpretation Roflumilast improves lung function in patients with COPD treated with salmeterol or tiotropium, and could become an important treatment for these patients
Albiglutide and cardiovascular outcomes in patients with type 2 diabetes and cardiovascular disease (Harmony Outcomes): a double-blind, randomised placebo-controlled trial
- Author
- Abbas J.
- Abraham S.
- Ackert J.
- Adams P.
- Aggarwala G.
- Aher N.
- Ahlqvist J.
- Ahn C. W.
- Akers T.
- Akhras R.
- Akhter F.
- Aksentyev S.
- Al Kawas F.
- Alexander K.
- Althouse D.
- Altobelli D.
- Alvarisqueta A.
- Ambrosy A.
- Andersen J.
- Andersen U.
- Anderson M.
- Anglade M.
- Arbanil H.
- Arciszewska M.
- Argoud G.
- Ariani M.
- Ashdji R.
- Avogaro A.
- Avramidis I.
- Axthelm C.
- Aye M.
- Babi C.
- Bachus E.
- Baik S. -H.
- Bailey M.
- Bakhtari L.
- Baksi A.
- Balasubramani M.
- Baldovino J.
- Banach M.
- Banerjee S.
- Bartlett A.
- Bartone S.
- Baum H.
- Bays H.
- Bazhan L.
- Beasley R.
- Beaudry Y.
- Beaulieu G.
- Beboso R.
- Bedard J.
- Beijerbacht H. P.
- Belfort de Aguiar R.
- Benjamin S.
- Berger D.
- Berlingieri J.
- Berndtsson Blom K.
- Besada D.
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- Bhargava A.
- Bhosale A.
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- Bijata-Bronisz R.
- Birkeland K.
- Birkenfeld A.
- Blagden M.
- Bloomfield G.
- Bode B.
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- Bonadonna R.
- Bondar I.
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- Bratcher C.
- Breneman J.
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- Broughton R.
- Brown J.
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- Bull G.
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- Publication venue
- 'Elsevier BV'
- Publication date
- 01/01/2018
- Field of study
Background: Glucagon-like peptide 1 receptor agonists differ in chemical structure, duration of action, and in their effects on clinical outcomes. The cardiovascular effects of once-weekly albiglutide in type 2 diabetes are unknown. We aimed to determine the safety and efficacy of albiglutide in preventing cardiovascular death, myocardial infarction, or stroke. Methods: We did a double-blind, randomised, placebo-controlled trial in 610 sites across 28 countries. We randomly assigned patients aged 40 years and older with type 2 diabetes and cardiovascular disease (at a 1:1 ratio) to groups that either received a subcutaneous injection of albiglutide (30–50 mg, based on glycaemic response and tolerability) or of a matched volume of placebo once a week, in addition to their standard care. Investigators used an interactive voice or web response system to obtain treatment assignment, and patients and all study investigators were masked to their treatment allocation. We hypothesised that albiglutide would be non-inferior to placebo for the primary outcome of the first occurrence of cardiovascular death, myocardial infarction, or stroke, which was assessed in the intention-to-treat population. If non-inferiority was confirmed by an upper limit of the 95% CI for a hazard ratio of less than 1·30, closed testing for superiority was prespecified. This study is registered with ClinicalTrials.gov, number NCT02465515. Findings: Patients were screened between July 1, 2015, and Nov 24, 2016. 10 793 patients were screened and 9463 participants were enrolled and randomly assigned to groups: 4731 patients were assigned to receive albiglutide and 4732 patients to receive placebo. On Nov 8, 2017, it was determined that 611 primary endpoints and a median follow-up of at least 1·5 years had accrued, and participants returned for a final visit and discontinuation from study treatment; the last patient visit was on March 12, 2018. These 9463 patients, the intention-to-treat population, were evaluated for a median duration of 1·6 years and were assessed for the primary outcome. The primary composite outcome occurred in 338 (7%) of 4731 patients at an incidence rate of 4·6 events per 100 person-years in the albiglutide group and in 428 (9%) of 4732 patients at an incidence rate of 5·9 events per 100 person-years in the placebo group (hazard ratio 0·78, 95% CI 0·68–0·90), which indicated that albiglutide was superior to placebo (p<0·0001 for non-inferiority; p=0·0006 for superiority). The incidence of acute pancreatitis (ten patients in the albiglutide group and seven patients in the placebo group), pancreatic cancer (six patients in the albiglutide group and five patients in the placebo group), medullary thyroid carcinoma (zero patients in both groups), and other serious adverse events did not differ between the two groups. There were three (<1%) deaths in the placebo group that were assessed by investigators, who were masked to study drug assignment, to be treatment-related and two (<1%) deaths in the albiglutide group. Interpretation: In patients with type 2 diabetes and cardiovascular disease, albiglutide was superior to placebo with respect to major adverse cardiovascular events. Evidence-based glucagon-like peptide 1 receptor agonists should therefore be considered as part of a comprehensive strategy to reduce the risk of cardiovascular events in patients with type 2 diabetes. Funding: GlaxoSmithKline
Albiglutide and cardiovascular outcomes in patients with type 2 diabetes and cardiovascular disease (Harmony Outcomes): a double-blind, randomised placebo-controlled trial
- Author
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- Publication venue
- Publication date
- 01/01/2018
- Field of study
Glucagon-like peptide 1 receptor agonists differ in chemical structure, duration of action, and in their effects on clinical outcomes. The cardiovascular effects of once-weekly albiglutide in type 2 diabetes are unknown. We aimed to determine the safety and efficacy of albiglutide in preventing cardiovascular death, myocardial infarction, or stroke
Edoxaban versus warfarin in patients with atrial fibrillation
- Author
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- Abril SB
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- Ŝpinar J
- Publication venue
- 'Massachusetts Medical Society'
- Publication date
- 01/01/2013
- Field of study
Contains fulltext :
125374.pdf (publisher's version ) (Open Access)BACKGROUND: Edoxaban is a direct oral factor Xa inhibitor with proven antithrombotic effects. The long-term efficacy and safety of edoxaban as compared with warfarin in patients with atrial fibrillation is not known. METHODS: We conducted a randomized, double-blind, double-dummy trial comparing two once-daily regimens of edoxaban with warfarin in 21,105 patients with moderate-to-high-risk atrial fibrillation (median follow-up, 2.8 years). The primary efficacy end point was stroke or systemic embolism. Each edoxaban regimen was tested for noninferiority to warfarin during the treatment period. The principal safety end point was major bleeding. RESULTS: The annualized rate of the primary end point during treatment was 1.50% with warfarin (median time in the therapeutic range, 68.4%), as compared with 1.18% with high-dose edoxaban (hazard ratio, 0.79; 97.5% confidence interval [CI], 0.63 to 0.99; P<0.001 for noninferiority) and 1.61% with low-dose edoxaban (hazard ratio, 1.07; 97.5% CI, 0.87 to 1.31; P=0.005 for noninferiority). In the intention-to-treat analysis, there was a trend favoring high-dose edoxaban versus warfarin (hazard ratio, 0.87; 97.5% CI, 0.73 to 1.04; P=0.08) and an unfavorable trend with low-dose edoxaban versus warfarin (hazard ratio, 1.13; 97.5% CI, 0.96 to 1.34; P=0.10). The annualized rate of major bleeding was 3.43% with warfarin versus 2.75% with high-dose edoxaban (hazard ratio, 0.80; 95% CI, 0.71 to 0.91; P<0.001) and 1.61% with low-dose edoxaban (hazard ratio, 0.47; 95% CI, 0.41 to 0.55; P<0.001). The corresponding annualized rates of death from cardiovascular causes were 3.17% versus 2.74% (hazard ratio, 0.86; 95% CI, 0.77 to 0.97; P=0.01), and 2.71% (hazard ratio, 0.85; 95% CI, 0.76 to 0.96; P=0.008), and the corresponding rates of the key secondary end point (a composite of stroke, systemic embolism, or death from cardiovascular causes) were 4.43% versus 3.85% (hazard ratio, 0.87; 95% CI, 0.78 to 0.96; P=0.005), and 4.23% (hazard ratio, 0.95; 95% CI, 0.86 to 1.05; P=0.32). CONCLUSIONS: Both once-daily regimens of edoxaban were noninferior to warfarin with respect to the prevention of stroke or systemic embolism and were associated with significantly lower rates of bleeding and death from cardiovascular causes. (Funded by Daiichi Sankyo Pharma Development; ENGAGE AF-TIMI 48 ClinicalTrials.gov number, NCT00781391.)
Cardiovascular Efficacy and Safety of Bococizumab in High-Risk Patients
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- Zubek V
- Publication venue
- 'Massachusetts Medical Society'
- Publication date
- 01/01/2017
- Field of study
Bococizumab is a humanized monoclonal antibody that inhibits proprotein convertase subtilisin- kexin type 9 (PCSK9) and reduces levels of low-density lipoprotein (LDL) cholesterol. We sought to evaluate the efficacy of bococizumab in patients at high cardiovascular risk. METHODS In two parallel, multinational trials with different entry criteria for LDL cholesterol levels, we randomly assigned the 27,438 patients in the combined trials to receive bococizumab (at a dose of 150 mg) subcutaneously every 2 weeks or placebo. The primary end point was nonfatal myocardial infarction, nonfatal stroke, hospitalization for unstable angina requiring urgent revascularization, or cardiovascular death; 93% of the patients were receiving statin therapy at baseline. The trials were stopped early after the sponsor elected to discontinue the development of bococizumab owing in part to the development of high rates of antidrug antibodies, as seen in data from other studies in the program. The median follow-up was 10 months. RESULTS At 14 weeks, patients in the combined trials had a mean change from baseline in LDL cholesterol levels of -56.0% in the bococizumab group and +2.9% in the placebo group, for a between-group difference of -59.0 percentage points (P<0.001) and a median reduction from baseline of 64.2% (P<0.001). In the lower-risk, shorter-duration trial (in which the patients had a baseline LDL cholesterol level of ≥70 mg per deciliter [1.8 mmol per liter] and the median follow-up was 7 months), major cardiovascular events occurred in 173 patients each in the bococizumab group and the placebo group (hazard ratio, 0.99; 95% confidence interval [CI], 0.80 to 1.22; P = 0.94). In the higher-risk, longer-duration trial (in which the patients had a baseline LDL cholesterol level of ≥100 mg per deciliter [2.6 mmol per liter] and the median follow-up was 12 months), major cardiovascular events occurred in 179 and 224 patients, respectively (hazard ratio, 0.79; 95% CI, 0.65 to 0.97; P = 0.02). The hazard ratio for the primary end point in the combined trials was 0.88 (95% CI, 0.76 to 1.02; P = 0.08). Injection-site reactions were more common in the bococizumab group than in the placebo group (10.4% vs. 1.3%, P<0.001). CONCLUSIONS In two randomized trials comparing the PCSK9 inhibitor bococizumab with placebo, bococizumab had no benefit with respect to major adverse cardiovascular events in the trial involving lower-risk patients but did have a significant benefit in the trial involving higher-risk patients
Cardiovascular Efficacy and Safety of Bococizumab in High-Risk Patients
- Author
- Abbott J. Dawn
- Abdullah S.
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- Zubek V.
- Publication venue
- 'Massachusetts Medical Society'
- Publication date
- 01/01/2017
- Field of study
BACKGROUN