Single Proton Knock-Out Reactions from 24,25,26F

The cross sections of the single proton knock-out reactions from 24F, 25F, and 26F on a 12C target were measured at energies of about 50 MeV/nucleon. Ground state populations of 6.6+-.9 mb, 3.8+-0.6 mb for the reactions 12C(24F,23O) and 12C(25F,24O) were extracted, respectively. The data were compared to calculations based on the many-body shell model and the eikonal theory. In the reaction 12C(26F,25O) the particle instability of 25O was confirmed

Nuclear vorticity and the low-energy nuclear response - Towards the neutron drip line

The transition density and current provide valuable insight into the nature of nuclear vibrations. Nuclear vorticity is a quantity related to the transverse transition current. In this work, we study the evolution of the strength distribution, related to density fluctuations, and the vorticity strength distribution, as the neutron drip line is approached. Our results on the isoscalar, natural-parity multipole response of Ni isotopes, obtained by using a self-consistent Skyrme-Hartree-Fock + Continuum RPA model, indicate that, close to the drip line, the low-energy response is dominated by L>1 vortical transitions.Comment: 8 pages, incl. 4 figures; to appear in Phys.Lett.

Soft Dipole Modes in Neutron-rich Ni-isotopes in QRRPA

The soft dipole modes in neutron rich even-even Ni-isotopes are investigated in the quasiparticle relativistic random phase approximation. We study the evolution of strengths distribution, centroid energies of dipole excitation in low-lying and normal GDR regions with the increase of the neutron excess. It is found in the present study that the centroid energies of the soft dipole strengths strongly depend on the thickness of neutron skin along with the neutron rich even-even Ni-isotopes.Comment: 14 pages, 7 figure

Structure of the N=27 isotones derived from the 44^{44}Ar(d,p)45^{45}Ar

Expérience GANIL/SPIRAL, détecteur CATS, détecteur MUST, 7 figures,International audienceThe $^{44}$Ar(d,p)$^{45}$Ar neutron transfer reaction was performed at 10~A.MeV. Measured excitation energies, deduced angular momenta and spectroscopic factors of the states populated in $^{45}$Ar are reported. A satisfactory description of these properties is achieved in the shell model framework using a new $sdpf$ interaction. The model analysis is extended to more exotic even-Z nuclei down to $^{41}_{14}$Si$_{27}$ to study how collectivity impacts the low lying structure of N~=~27 neutron-rich nuclei

Direct evidence of transfer with weakly bound isotopes of He near the Coulomb barrier and implications of fusion

NESTERPartial residue cross sections for fusion and transfer have been measured from the intensities of characteristic gamma-rays for the $^{4,6}$ He + $^{63,65}$Cu systems at energies near the Coulomb barrier (Vb)

Crossing the Dripline to 11N Using Elastic Resonance Scattering

The level structure of the unbound nucleus 11N has been studied by 10C+p elastic resonance scattering in inverse geometry with the LISE3 spectrometer at GANIL, using a 10C beam with an energy of 9.0 MeV/u. An additional measurement was done at the A1200 spectrometer at MSU. The excitation function above the 10C+p threshold has been determined up to 5 MeV. A potential-model analysis revealed three resonance states at energies 1.27 (+0.18-0.05) MeV (Gamma=1.44 +-0.2 MeV), 2.01(+0.15-0.05) MeV, (Gamma=0.84 +-$0.2 MeV) and 3.75(+-0.05) MeV, (Gamma=0.60 +-0.05 MeV) with the spin-parity assignments I(pi) =1/2+, 1/2- and 5/2+, respectively. Hence, 11N is shown to have a ground state parity inversion completely analogous to its mirror partner, 11Be. A narrow resonance in the excitation function at 4.33 (+-0.05) MeV was also observed and assigned spin-parity 3/2-.Comment: 14 pages, 9 figures, twocolumn Accepted for publication in PR

De novo mutations in SMCHD1 cause Bosma arhinia microphthalmia syndrome and abrogate nasal development

Bosma arhinia microphthalmia syndrome (BAMS) is an extremely rare and striking condition characterized by complete absence of the nose with or without ocular defects. We report here that missense mutations in the epigenetic regulator SMCHD1 mapping to the extended ATPase domain of the encoded protein cause BAMS in all 14 cases studied. All mutations were de novo where parental DNA was available. Biochemical tests and in vivo assays in Xenopus laevis embryos suggest that these mutations may behave as gain-of-function alleles. This finding is in contrast to the loss-of-function mutations in SMCHD1 that have been associated with facioscapulohumeral muscular dystrophy (FSHD) type 2. Our results establish SMCHD1 as a key player in nasal development and provide biochemical insight into its enzymatic function that may be exploited for development of therapeutics for FSHD