48 research outputs found

    In the Name of the Working Class: Narratives of Labour Activism in Contemporary China

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    Since their appearance in the mid-1990s, Chinese labour NGOs have mostly focused on disseminating labour law and guiding labour disputes through official channels. In so doing, they have assisted the Chinese Communist Party in achieving its paramount goal of maintaining social stability. In line with this approach, activists in these organizations have traditionally framed their work in terms of “public interest” or “legality,” both of which resonate with the hegemonic discourses of the Party-state. However, earlier this decade a minority of Chinese labour activists began to employ some new counterhegemonic narratives centred on the experience of the labour movement and the practice of collective bargaining that attempted to recode the proletarian experience outside of its official representation. In this paper we analyze this discursive shift through the voices of the activists involved, and argue that the rise of these new counterhegemonic voices was one of the reasons that led to the Party-state cracking down on labour NGOs

    Party Spirit Made Flesh: the Production of Legitimacy in the Aftermath of the 2008 Sichuan Earthquake

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    This article argues that the Party retains a tradition of seeking to revitalize its legitimacy through demonstrations of benevolence and glory. The post-2008 Sichuan earthquake provided just such an opportunity to mobilize the discourse of “Party spirit” and display the willingness of cadres to suffer and sacrifice themselves on behalf of the people. In addition to being grist for the propaganda mill, these norms and expectations were implemented in concrete policy directives and work pressures. Local cadres, who were also earthquake survivors, started to suffer from exhaustion, insomnia, and depression. After high-profile suicides by several local cadres, the Party adopted a therapeutic discourse in order to address the psychological needs of individual cadres, though these gentler policies seem doomed to be short-lived

    Neurolinguistic aspects of attrition

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    International audienceThe aim of this paper is to provide the reader with a general overview of the field of attrition. Having situated this relatively new research domain with respect to related fields and approaches, a brief summary of the most important research questions and preliminary findings is given. The discussion then focuses on two issues that are of particular interest with respect to neurolinguistics: the role of the subject's age and of the influence of L2. Concerning the former, a summary of research on the critical period hypothesis is given and discussed in the light of findings from attrition research. Another issue concerns the principal mechanisms involved in L1 attrition, i.e. whether attrition occurs because of lack of L1 use or because of its replacement by the competing L2 structures. Research issues of such scope need integrative approaches and greatly benefit from comparisons with related fields such as normal aging, acquisition and aphasia

    The IFN-Îł-Inducible GTPase, Irga6, Protects Mice against Toxoplasma gondii but Not against Plasmodium berghei and Some Other Intracellular Pathogens

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    Clearance of infection with intracellular pathogens in mice involves interferon-regulated GTPases of the IRG protein family. Experiments with mice genetically deficient in members of this family such as Irgm1(LRG-47), Irgm3(IGTP), and Irgd(IRG-47) has revealed a critical role in microbial clearance, especially for Toxoplasma gondii. The in vivo role of another member of this family, Irga6 (IIGP, IIGP1) has been studied in less detail. We investigated the susceptibility of two independently generated mouse strains deficient in Irga6 to in vivo infection with T. gondii, Mycobacterium tuberculosis, Leishmania mexicana, L. major, Listeria monocytogenes, Anaplasma phagocytophilum and Plasmodium berghei. Compared with wild-type mice, mice deficient in Irga6 showed increased susceptibility to oral and intraperitoneal infection with T. gondii but not to infection with the other organisms. Surprisingly, infection of Irga6-deficient mice with the related apicomplexan parasite, P. berghei, did not result in increased replication in the liver stage and no Irga6 (or any other IRG protein) was detected at the parasitophorous vacuole membrane in IFN-Îł-induced wild-type cells infected with P. berghei in vitro. Susceptibility to infection with T. gondii was associated with increased mortality and reduced time to death, increased numbers of inflammatory foci in the brains and elevated parasite loads in brains of infected Irga6-deficient mice. In vitro, Irga6-deficient macrophages and fibroblasts stimulated with IFN-Îł were defective in controlling parasite replication. Taken together, our results implicate Irga6 in the control of infection with T. gondii and further highlight the importance of the IRG system for resistance to this pathogen

    Understanding Factors Associated With Psychomotor Subtypes of Delirium in Older Inpatients With Dementia

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    Poetry after the Future

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    Aesthetics

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    Communist Hibernation

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    Christian Sorace on 'Shaken Authority'

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