Stripped elliptical galaxies as probes of ICM physics : III. Deep Chandra observation of NGC 4552 - Measuring the viscosity of the intracluster medium

We present results from a deep (200 ks) Chandra observation of the early-type galaxy NGC 4552 (M89) which is falling into the Virgo cluster. Previous shallower X-ray observations of this galaxy showed a remnant gas core, a tail to the South of the galaxy, and twin `horns' attached to the northern edge of the gas core [machacek05a]. In our deeper data, we detect a diffuse, low surface brightness extension to the previously known tail, and measure the temperature structure within the tail. We combine the deep Chandra data with archival XMM-Newton observations to put a strong upper limit on the diffuse emission of the tail out to a large distance (10×the radius of the remnant core) from the galaxy center. In our two previous papers [roediger15a,roediger15b], we presented the results of hydrodynamical simulations of ram pressure stripping specifically for M89 falling into the Virgo cluster and investigated the effect of ICM viscosity. In this paper, we compare our deep data with our specifically tailored simulations and conclude that the observed morphology of the stripped tail in NGC 4552 is most similar to the inviscid models. We conclude that, to the extent the transport processes can be simply modeled as a hydrodynamic viscosity, the ICM viscosity is negligible. More generally, any micro-scale description of the transport processes in the high-β plasma of the cluster ICM must be consistent with the efficient mixing observed in the stripped tail on macroscopic scales

Close-up view of an ongoing merger between the NGC 4839 group and the Coma cluster - a post-merger scenario

We study a merger of the NGC 4839 group with the Coma cluster using X-ray observations from the XMM–Newton and Chandra telescopes. X-ray data show two prominent features: (i) a long (∼600 kpc in projection) and bent tail of cool gas trailing (towards south-west) the optical centre of NGC 4839, and (ii) a ‘sheath’ region of enhanced X-ray surface brightness enveloping the group, which is due to hotter gas. While at first glance the X-ray images suggest that we are witnessing the first infall of NGC 4839 into the Coma cluster core, we argue that a post-merger scenario provides a better explanation of the observed features and illustrate this with a series of numerical simulations. In this scenario, the tail is formed when the group, initially moving to the south-west, reverses its radial velocity after crossing the apocenter, the ram pressure ceases and the ram pressure-displaced gas falls back towards the centre of the group and overshoots it. Shortly after the apocenter passage, the optical galaxy, dark matter, and gaseous core move in a north-east direction, while the displaced gas continues moving to the south-west. The ‘sheath’ is explained as being due to interaction of the re-infalling group with its own tail of stripped gas mixed with the Coma gas. In this scenario, the shock, driven by the group before reaching the apocenter, has already detached from the group and would be located close to the famous relic to the south-west of the Coma cluster

A Chandra Study of the NGC 7618/UGC 12491 Major Group Merger at Apogee: Multiple Cold Fronts, Boxy Wings, Filaments, and Arc-shaped Slingshot Tails

Analyses of major group mergers are key to understanding the evolution of large-scale structure in the Universe and the microphysical properties of the hot gas in these systems. We present imaging and spectral analyses of deep Chandra observations of hot gas structures formed in the major merger of the NGC 7618 and UGC 12491 galaxy groups and compare the observed hot gas morphology, temperatures, and abundances with recent simulations. The morphology of the observed multiple cold front edges and boxy wings are consistent with those expected to be formed by Kelvin–Helmholtz instabilities and gas sloshing in inviscid gas. The arc-shaped slingshot tail morphologies seen in each galaxy suggest that the dominant galaxies are near their orbital apogee after having experienced at least one core passage at a large impact parameter

The role of cell-cell adhesion in wound healing

We present a stochastic model which describes fronts of cells invading a wound. In the model cells can move, proliferate, and experience cell-cell adhesion. We find several qualitatively different regimes of front motion and analyze the transitions between them. Above a critical value of adhesion and for small proliferation large isolated clusters are formed ahead of the front. This is mapped onto the well-known ferromagnetic phase transition in the Ising model. For large adhesion, and larger proliferation the clusters become connected (at some fixed time). For adhesion below the critical value the results are similar to our previous work which neglected adhesion. The results are compared with experiments, and possible directions of future work are proposed.Comment: to appear in Journal of Statistical Physic

Extended X-ray study of M49: The frontier of the Virgo cluster

The M49 group, residing outside the virial radius of the Virgo cluster, is falling onto the cluster from the south. We report results from deep XMM-Newton mosaic observations of M49. Its hot gas temperature is 0.8 keV at the group center and rises to 1.5 keV beyond the brightest group galaxy (BGG). The group gas extends to radii of ~300 kpc to the north and south. The observations reveal a cold front ~20 kpc north of the BGG center and an X-ray-bright stripped tail 70 kpc long and 10 kpc wide to the southwest of the BGG. We argue that the atmosphere of the infalling group was slowed by its encounter with the Virgo cluster gas, causing the BGG to move forward subsonically relative to the group gas. We measure declining temperature and metallicity gradients along the stripped tail. The tail gas can be traced back to the cooler and enriched gas uplifted from the BGG center by buoyant bubbles, implying that active galactic nucleus outbursts may have intensified the stripping process. We extrapolate to a virial radius of 740 kpc and derive a virial mass of 4.6 × 1013 M ⊙ for the M49 group. Its group atmosphere appears truncated and deficient when compared with isolated galaxy groups of similar temperatures. If M49 is on its first infall to Virgo, the infall region of a cluster could have profound impacts on galaxies and groups that are being accreted onto galaxy clusters. Alternatively, M49 may have already passed through Virgo once

CLEC-2 and Syk in the megakaryocytic/platelet lineage are essential for development

The C-type lectin receptor CLEC-2 signals through a pathway that is critically dependent on the tyrosine kinase Syk. We show that homozygous loss of either protein results in defects in brain vascular and lymphatic development, lung inflation and perinatal lethality. Furthermore, we find that conditional deletion of Syk in the haematopoietic lineage, or conditional deletion of CLEC-2 or Syk in the megakaryocyte/platelet lineage, also causes defects in brain vascular and lymphatic development, although the mice are viable. In contrast, conditional deletion of Syk in other haematopoietic lineages had no effect on viability or brain vasculature and lymphatic development. We show that platelets, but not platelet releasate, modulate the migration and intercellular adhesion of lymphatic endothelial cells through a pathway that is dependent on CLEC-2 and Syk. These studies demonstrate that megakaryocyte/platelet expression of CLEC-2 and Syk is required for normal brain vasculature and lymphatic development and that platelet CLEC-2 and Syk directly modulate lymphatic endothelial cell behaviour in vitro

Aurintricarboxylic acid prevents GLUR2 mRNA down-regulation and delayed neurodegeneration in hippocampal CA1 neurons of gerbil after global ischemia

Aurintricarboxylic acid (ATA), an inhibitor of endonuclease activity and other protein–nucleic acid interactions, blocks apoptosis in several cell types and prevents delayed death of hippocampal pyramidal CA1 neurons induced by transient global ischemia. Global ischemia in rats and gerbils induces down-regulation of GluR2 mRNA and increased α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-induced Ca(2+) influx in CA1 before neurodegeneration. This result and neuroprotection by antagonists of AMPA receptors suggests that formation of AMPA receptors lacking GluR2, and therefore Ca(2+) permeable, leads to excessive Ca(2+) influx in response to endogenous glutamate; the resulting delayed neuronal death in CA1 exhibits many characteristics of apoptosis. In this study, we examined the effects of ATA on expression of mRNAs encoding glutamate receptor subunits in gerbil hippocampus after global ischemia. Administration of ATA by injection into the right cerebral ventricle 1 h before (but not 6 h after) bilateral carotid occlusion prevented the ischemia-induced decrease in GluR2 mRNA expression and the delayed neurodegeneration. These findings suggest that ATA is neuroprotective in ischemia by blocking the transcriptional changes leading to down-regulation of GluR2, rather than by simply blocking endonucleases, which presumably act later after Ca(2+) influx initiates apoptosis. Maintaining formation of Ca(2+) impermeable, GluR2 containing AMPA receptors could prevent delayed death of CA1 neurons after transient global ischemia, and block of GluR2 down-regulation may provide a further strategy for neuroprotection