537 research outputs found

    Lyman α emitters gone missing: Evidence for late reionization?

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    We combine high resolution hydrodynamical simulations with an intermediate resolution, dark matter only simulation and an analytical model for the growth of ionized regions to estimate the large scale distribution and redshift evolution of the visibility of Lyman-alpha emission in 6<=z<=8 galaxies. The inhomogeneous distribution of neutral hydrogen during the reionization process results in significant fluctuations in the Lyman-alpha transmissivity on large scales. The transmissivity depends not only on the ionized fraction of the intergalactic medium by volume and the amplitude of the local ionizing background, but is also rather sensitive to the evolution of the relative velocity shift of the Lyman-alpha emission line due to resonant scattering. We reproduce a decline in the space density of Lyman-alpha emitting galaxies as rapid as observed with a rather rapidly evolving neutral fraction between z=6-8, and a typical Lyman-alpha line velocity offset of 100 km/s redward of systemic at z=6 which decreases toward higher redshift. The new (02/2015) Planck results indicate such a recent end to reionization is no longer disfavoured by constraints from the cosmic microwave background

    Large 21 cm signals from AGN-dominated reionization

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    We present predictions for the spatial distribution of 21-cm brightness temperature fluctuations from high-dynamic-range simulations for active galactic nucleus (AGN)-dominated reionization histories that have been tested against available Lyα\alpha and cosmic microwave background (CMB) data. We model AGNs by extrapolating the observed MbhM_\text{bh} − σ relation to high redshifts and assign them ionizing emissivities consistent with recent UV luminosity function measurements. We assess the observability of the predicted spatial 21-cm fluctuations in the late stages of reionization in the limit in which the hydrogen 21-cm spin temperature is significantly larger than the CMB temperature. Our AGN-dominated reionization histories increase the variance of the 21-cm emission by a factor of up to 10 compared to similar reionization histories dominated by faint galaxies, to values close to 100 mK2^2 at scales accessible to experiments (kk \lesssim 1 cMpc1^{−1} hh). This is lower than the sensitivity reached by ongoing experiments only by a factor of about 2 or less. When reionization is dominated by AGNs, the 21-cm power spectrum is enhanced on all scales due to the enhanced bias of the clustering of the more massive haloes and the peak in the large scale 21-cm power is strongly enhanced and moved to larger scales due to bigger characteristic bubble sizes. AGN-dominated reionization should be easily detectable by Low Frequency Array (and later Hydrogen Epoch of Reionization Array and Phase 1 of the Square Kilometre Array) at their design sensitivity, assuming successful foreground subtraction and instrument calibration. Conversely, these could become the first non-trivial reionization scenarios to be ruled out by 21-cm experiments, thereby constraining the contribution of AGNs to reionization.Support by ERC Advanced grant 320596 ‘The Emergence of Structure During the Epoch of Reionization’ is gratefully acknowledged. EP gratefully acknowledges support by the Kavli Foundation. We acknowledge PRACE for awarding us access to the Curie supercomputer, based in France at the Tres Grand Centre de Calcul ´ (TGCC). This work used the DiRAC Data Centric system at Durham University, operated by the Institute for Computational Cosmology on behalf of the STFC DiRAC HPC Facility (www.dirac.ac.uk). This equipment was funded by BIS National E-infrastructure capital grant ST/K00042X/1, STFC capital grants ST/H008519/1 and ST/K00087X/1, STFC DiRAC Operations grant ST/K003267/1 and Durham University. DiRAC is part of the National E-Infrastructure. This research was supported by the Munich Institute for Astro- and Particle Physics (MIAPP) of the DFG cluster of excellence ‘Origin and Structure of the Universe’

    Lyman-α emitters gone missing: The different evolution of the bright and faint populations

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    We model the transmission of the Lyman-alpha line through the circum- and intergalactic media around dark matter haloes expected to host Lyman-alpha emitters (LAEs) at z > 5.7, using the high-dynamic-range Sherwood simulations. We find very different CGM environments around more massive haloes (~10^11 M_sun) compared to less massive haloes (~10^9 M_sun) at these redshifts, which can contribute to a different evolution of the Lyman-alpha transmission from LAEs within these haloes. Additionally we confirm that part of the differential evolution could result from bright LAEs being more likely to reside in larger ionized regions. We conclude that a combination of the CGM environment and the IGM ionization structure is likely to be responsible for the differential evolution of the bright and faint ends of the LAE luminosity function at z > 6. More generally, we confirm the suggestion that the self-shielded neutral gas in the outskirts of the host halo can strongly attenuate the Lyman-alpha emission from high redshift galaxies. We find that this has a stronger effect on the more massive haloes hosting brighter LAEs. The faint-end of the LAE luminosity function is thus a more reliable probe of the average ionization state of the IGM. Comparing our model for LAEs with a range of observational data we find that the favoured reionization histories are our previously advocated `Late' and `Very Late' reionization histories, in which reionization finishes rather rapidly at around z ~ 6

    Sexual Size Dimorphism and Body Condition in the Australasian Gannet

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    Funding: The research was financially supported by the Holsworth Wildlife Research Endowment. Acknowledgments We thank the Victorian Marine Science Consortium, Sea All Dolphin Swim, Parks Victoria, and the Point Danger Management Committee for logistical support. We are grateful for the assistance of the many field volunteers involved in the study.Peer reviewedPublisher PD

    Non-homologous end-joining pathway associated with occurrence of myocardial infarction: gene set analysis of genome-wide association study data

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    &lt;p&gt;Purpose: DNA repair deficiencies have been postulated to play a role in the development and progression of cardiovascular disease (CVD). The hypothesis is that DNA damage accumulating with age may induce cell death, which promotes formation of unstable plaques. Defects in DNA repair mechanisms may therefore increase the risk of CVD events. We examined whether the joints effect of common genetic variants in 5 DNA repair pathways may influence the risk of CVD events.&lt;/p&gt; &lt;p&gt;Methods: The PLINK set-based test was used to examine the association to myocardial infarction (MI) of the DNA repair pathway in GWAS data of 866 subjects of the GENetic DEterminants of Restenosis (GENDER) study and 5,244 subjects of the PROspective Study of Pravastatin in the Elderly at Risk (PROSPER) study. We included the main DNA repair pathways (base excision repair, nucleotide excision repair, mismatch repair, homologous recombination and non-homologous end-joining (NHEJ)) in the analysis.&lt;/p&gt; &lt;p&gt;Results: The NHEJ pathway was associated with the occurrence of MI in both GENDER (P = 0.0083) and PROSPER (P = 0.014). This association was mainly driven by genetic variation in the MRE11A gene (PGENDER = 0.0001 and PPROSPER = 0.002). The homologous recombination pathway was associated with MI in GENDER only (P = 0.011), for the other pathways no associations were observed.&lt;/p&gt; &lt;p&gt;Conclusion: This is the first study analyzing the joint effect of common genetic variation in DNA repair pathways and the risk of CVD events, demonstrating an association between the NHEJ pathway and MI in 2 different cohorts.&lt;/p&gt

    Cancer mortality in ethnic South Asian migrants in England and Wales (1993–2003): patterns in the overall population and in first and subsequent generations

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    BACKGROUND: Cancer mortality has been examined among ethnic South Asian migrants in England and Wales, but not by generation of migration. METHODS: Using South Asian mortality records, identified by a name-recognition algorithm, and census information, age-standardised rates among South Asians, and South Asian vs non-South Asian rate ratios, were calculated. RESULTS AND CONCLUSIONS: All-cancer rates in ethnic South Asians were half of those in non-South Asians in first-generation (all-cancer-standardised mortality ratio (SMR) in males 0.51 and in females 0.56) and subsequent-generation South Asians (SMR in males 0.43 and in females 0.36). The higher mortality in first-generation South Asians for liver (both sexes), oral cavity and gallbladder cancer (females), particularly marked among Bangladeshis, was reduced in subsequent generations

    Production of phi mesons at mid-rapidity in sqrt(s_NN) = 200 GeV Au+Au collisions at RHIC

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    We present the first results of meson production in the K^+K^- decay channel from Au+Au collisions at sqrt(s_NN) = 200 GeV as measured at mid-rapidity by the PHENIX detector at RHIC. Precision resonance centroid and width values are extracted as a function of collision centrality. No significant variation from the PDG accepted values is observed. The transverse mass spectra are fitted with a linear exponential function for which the derived inverse slope parameter is seen to be constant as a function of centrality. These data are also fitted by a hydrodynamic model with the result that the freeze-out temperature and the expansion velocity values are consistent with the values previously derived from fitting single hadron inclusive data. As a function of transverse momentum the collisions scaled peripheral.to.central yield ratio RCP for the is comparable to that of pions rather than that of protons. This result lends support to theoretical models which distinguish between baryons and mesons instead of particle mass for explaining the anomalous proton yield.Comment: 326 authors, 24 pages text, 23 figures, 6 tables, RevTeX 4. To be submitted to Physical Review C as a regular article. Plain text data tables for the points plotted in figures for this and previous PHENIX publications are (or will be) publicly available at http://www.phenix.bnl.gov/papers.htm
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