285 research outputs found
In-hospital versus postdischarge adverse events following carotid endarterectomy
- Author
- Publication venue
- Society for Vascular Surgery. Published by Elsevier Inc.
- Publication date
- 30/06/2013
- Field of study
ObjectiveMost studies based on state and nationwide registries evaluating perioperative outcome after carotid endarterectomy (CEA) rely on hospital discharge data only. Therefore, the true 30-day complication risk after carotid revascularization may be underestimated.MethodsWe used the National Surgical Quality Improvement Program database 2005-2010 to assess the in-hospital and postdischarge rate of any stroke, death, cardiac event (new Q-wave myocardial infarction or cardiac arrest), and combinedĀ stroke/death and combined adverse outcome (S/D/CE) at 30 days following CEA. Multivariable analyses were used to identify predictors for in-hospital and postdischarge events separately, and in particular, those that predict postdischarge events distinctly.ResultsA total of 35,916 patients who underwent CEA during 2005-2010 were identified in the National Surgical Quality Improvement Program database; 59% were male, median age was 72 years, and 44% had a previous neurologic event. Thirty-day stroke rate was 1.6% (nĀ = 591), death rate was 0.8% (nĀ = 272), cardiac event rate was 1.0% (nĀ = 350), stroke or death rate was 2.2% (nĀ = 794), and combined S/D/CE rate was 2.9% (nĀ = 1043); 33% of strokes, 53% of deaths, 32% of cardiac events, 40% of combined stroke/death, and 38% of combined S/D/CE took place after hospital discharge. Patients with a prior stroke or transient ischemic attack had similar proportions of postdischarge events compared with patients without prior symptoms. Independent predictors for postdischarge events, but not for in-hospitalĀ events were female sex (stroke [odds ratio (OR), 1.6; 95% confidence interval (CI), 1.2-2.1] and stroke/death [OR, 1.4; 95% CI, 1.1-1.7]), renal failure (stroke [OR, 3.0; 95% CI, 1.4-6.2]) and chronic obstructive pulmonary disease (death [OR, 2.5; 95% CI, 1.6-3.7], stroke/death [OR, 1.8; 95% CI, 1.4-2.4], and S/D/CE [OR 1.8, 95% CI 1.4-2.3]).ConclusionsWith 38% of perioperative adverse events after CEA happening posthospitalization, regardless of symptoms status, we need to be alert to the ongoing risks after discharge particularly in women, patients with renal failure, or chronic obstructive pulmonary disease. This emphasizes the need for reporting and comparing 30-day adverse event rates when evaluating outcomes for CEA, or comparing carotid stenting to CEA
VLDL Hydrolysis by Hepatic Lipase Regulates PPARĪ“ Transcriptional Responses
- Author
- A Chawla
- A Saghatelian
- AC Li
- Alan Saghatelian
- Ben C. B. Ko
- BM Forman
- C Mineo
- CH Lee
- CH Lee
- D Applebaum-Bowden
- Daniel J. Rader
- Eric Oligino
- GE Homanics
- H Jansen
- HK Chiu
- JC Lo
- JD Brown
- JG Duncan
- Jonathan D. Brown
- Jorge Plutzky
- JR Mead
- K Qian
- K Tachibana
- KA Dugi
- L Freeman
- LM Sanderson
- M Duong
- M Teran-Garcia
- MA Ruby
- MG McCoy
- MV Chakravarthy
- N Marx
- N Marx
- NS Tan
- O Ziouzenkova
- P Wiesner
- R Tagore
- R van Haperen
- RA Hegele
- RM Evans
- S Braschi
- S Kersten
- S Kersten
- S Santamarina-Fojo
- S Santamarina-Fojo
- SA Kliewer
- T Kanda
- W Ahmed
- X Yang
- Z Nong
- Publication venue
- Public Library of Science
- Publication date
- 05/07/2011
- Field of study
PPARs (Ī±,Ī³,Ī“) are a family of ligand-activated transcription factors that regulate energy balance, including lipid metabolism. Despite these critical functions, the integration between specific pathways of lipid metabolism and distinct PPAR responses remains obscure. Previous work has revealed that lipolytic pathways can activate PPARs. Whether hepatic lipase (HL), an enzyme that regulates VLDL and HDL catabolism, participates in PPAR responses is unknown.Using PPAR ligand binding domain transactivation assays, we found that HL interacted with triglyceride-rich VLDL (>HDLā«LDL, IDL) to activate PPARĪ“ preferentially over PPARĪ± or PPARĪ³, an effect dependent on HL catalytic activity. In cell free ligand displacement assays, VLDL hydrolysis by HL activated PPARĪ“ in a VLDL-concentration dependent manner. Extended further, VLDL stimulation of HL-expressing HUVECs and FAO hepatoma cells increased mRNA expression of canonical PPARĪ“ target genes, including adipocyte differentiation related protein (ADRP), angiopoietin like protein 4 and pyruvate dehydrogenase kinase-4. HL/VLDL regulated ADRP through a PPRE in the promoter region of this gene. In vivo, adenoviral-mediated hepatic HL expression in C57BL/6 mice increased hepatic ADRP mRNA levels by 30%. In ob/ob mice, a model with higher triglycerides than C57BL/6 mice, HL overexpression increased ADRP expression by 70%, demonstrating the importance of triglyceride substrate for HL-mediated PPARĪ“ activation. Global metabolite profiling identified HL/VLDL released fatty acids including oleic acid and palmitoleic acid that were capable of recapitulating PPARĪ“ activation and ADRP gene regulation in vitro.These data define a novel pathway involving HL hydrolysis of VLDL that activates PPARĪ“ through generation of specific monounsaturated fatty acids. These data also demonstrate how integrating cell biology with metabolomic approaches provides insight into specific lipid mediators and pathways of lipid metabolism that regulate transcription
A Systematic Analysis of Eluted Fraction of Plasma Post Immunoaffinity Depletion: Implications in Biomarker Discovery
- Author
- A Cabre
- AI Nesvizhskii
- AK Yadav
- AL Krasnoselsky
- Amit Kumar Yadav
- Ashish Kumar Singh
- B Muthusamy
- BL Nicholas
- C Tu
- CF Taylor
- D Kessner
- Debasis Dash
- Dhirendra Kumar
- DJ States
- E Bellei
- Gourav Bhardwaj
- GR Mishra
- GS Omenn
- J Hernandez-Toro
- JK Eng
- K Bjorhall
- KR Coombes
- L Huang
- L Kall
- LA Echan
- LA Liotta
- LG Costa
- LY Geer
- M Zhou
- MPW Smith
- N Gupta
- Niyaz Ahmed
- NL Anderson
- NL Anderson
- NL Anderson
- P Juhasz
- P Shannon
- PJ Kersey
- Q Zhang
- R Craig
- R Stempfer
- RK Dagda
- RL Gundry
- RL Gundry
- Ruby Priyadarshini
- RX Sun
- S Peri
- S Peri
- Shadab Ahmad
- Shantanu Sengupta
- T Liu
- Trayambak Basak
- TS Keshava Prasad
- TS Prasad
- V Faca
- V Faca
- W Derer
- WY Lo
- Y Gong
- Publication venue
- Public Library of Science
- Publication date
- 07/09/2011
- Field of study
Plasma is the most easily accessible source for biomarker discovery in clinical proteomics. However, identifying potential biomarkers from plasma is a challenge given the large dynamic range of proteins. The potential biomarkers in plasma are generally present at very low abundance levels and hence identification of these low abundance proteins necessitates the depletion of highly abundant proteins. Sample pre-fractionation using immuno-depletion of high abundance proteins using multi-affinity removal system (MARS) has been a popular method to deplete multiple high abundance proteins. However, depletion of these abundant proteins can result in concomitant removal of low abundant proteins. Although there are some reports suggesting the removal of non-targeted proteins, the predominant view is that number of such proteins is small. In this study, we identified proteins that are removed along with the targeted high abundant proteins. Three plasma samples were depleted using each of the three MARS (Hu-6, Hu-14 and Proteoprep 20) cartridges. The affinity bound fractions were subjected to gelC-MS using an LTQ-Orbitrap instrument. Using four database search algorithms including MassWiz (developed in house), we selected the peptides identified at <1% FDR. Peptides identified by at least two algorithms were selected for protein identification. After this rigorous bioinformatics analysis, we identified 101 proteins with high confidence. Thus, we believe that for biomarker discovery and proper quantitation of proteins, it might be better to study both bound and depleted fractions from any MARS depleted plasma sample
Characterization of Epstein-Barr Virus miRNAome in Nasopharyngeal Carcinoma by Deep Sequencing
- Author
- A Grundhoff
- AK Lo
- AT Bankier
- C Chen
- Cheng-Yuan Liu
- D Iwakiri
- D Nachmani
- Dong-Yan Jin
- E Marti
- EJ Nam
- EY Choy
- F Kuchenbauer
- GE Crooks
- Gian-Hung Chen
- H Liang
- H Seitz
- HC Chen
- Hua-Chien Chen
- J Lu
- JG Reid
- JG Ruby
- JH Schulte
- JL Mott
- JL Umbach
- JM Middeldorp
- JS Burgos
- JY Zhu
- K Cosmopoulos
- Kai-Ping Chang
- KJ Riley
- L He
- M Fabbri
- MJ de Hoon
- N Walz
- P Landgraf
- PA Gregory
- RD Morin
- RH Edwards
- RL Skalsky
- RW Lung
- S Barth
- S Chatterjee
- S Griffiths-Jones
- S Pfeffer
- S Sengupta
- S Swaminathan
- Shu-Jen Chen
- SM Park
- T Katoh
- T Xia
- T Yoshizaki
- U Burk
- WM Merritt
- X Cai
- Y Hayashita
- Y Karube
- Y Kawahara
- Y Kawahara
- Y Ladeiro
- Y Pekarsky
- Yi-Hsuan Chen
- Yu-Sun Chang
- Z Ghosh
- ZL Pratt
- Publication venue
- Public Library of Science
- Publication date
- 20/09/2010
- Field of study
Virus-encoded microRNAs (miRNAs) have been shown to regulate a variety of biological processes involved in viral infection and viral-associated pathogenesis. Epstein-Barr virus (EBV) is a herpesvirus implicated in nasopharyngeal carcinoma (NPC) and other human malignancies. EBV-encoded miRNAs were among the first group of viral miRNAs identified. To understand the roles of EBV miRNAs in the pathogenesis of NPC, we utilized deep sequencing technology to characterize the EBV miRNA transcriptome in clinical NPC tissues. We obtained more than 110,000 sequence reads in NPC samples and identified 44 EBV BART miRNAs, including four new mature miRNAs derived from previously identified BART miRNA precursor hairpins. Further analysis revealed extensive sequence variations (isomiRs) of EBV miRNAs, including terminal isomiRs at both the 5ā² and 3ā² ends and nucleotide variants. Analysis of EBV genomic sequences indicated that the majority of EBV miRNA nucleotide variants resulted from post-transcriptional modifications. Read counts of individual EBV miRNA in NPC tissue spanned from a few reads to approximately 18,000 reads, confirming the wide expression range of EBV miRNAs. Several EBV miRNAs were expressed at levels similar to highly abundant human miRNAs. Sequence analysis revealed that most of the highly abundant EBV miRNAs share their seed sequences (nucleotides 2ā7) with human miRNAs, suggesting that seed sequence content may be an important factor underlying the differential accumulation of BART miRNAs. Interestingly, many of these human miRNAs have been found to be dysregulated in human malignancies, including NPC. These observations not only provide a potential linkage between EBV miRNAs and human malignancy but also suggest a highly coordinated mechanism through which EBV miRNAs may mimic or compete with human miRNAs to affect cellular functions
Linear and Branched Glyco-Lipopeptide Vaccines Follow Distinct Cross-Presentation Pathways and Generate Different Magnitudes of Antitumor Immunity
- Author
- A Hosmalin
- A Margalit
- A Roth
- AA Chentoufi
- AA Chentoufi
- AA Chentoufi
- AB Nesburn
- AL Ackerman
- AL Sorensen
- AL Sorensen
- Alda Shi
- AM Ercolini
- Anthony B. Nesburn
- AR Hearn
- B Monzavi-Karbassi
- C Bourgeois
- C Durier
- C Hotta
- C Mesa
- C Shepherd
- C Vazquez-Martin
- CC Peeters
- CE Ruby
- D Dorfel
- DC Jackson
- Derya Unutmaz
- DM Lubaroff
- E Belnoue
- E Davila
- E Lazoura
- E Maraskovsky
- G Curigliano
- G Curigliano
- G Dasgupta
- G Deliyannis
- G Hoeffel
- G Moron
- Gargi Dasgupta
- GF Springer
- GT Belz
- H Gahery
- H Gahery
- H Norell
- I Bettahi
- I Bettahi
- I Bettahi
- I Vicente-Suarez
- Ilham Bettahi
- JA Hollenbaugh
- JC Manimala
- JC Tseng
- JY Kang
- KC Roy
- KL Rock
- KN Khan
- KN Syrigos
- L BenMohamed
- L BenMohamed
- L BenMohamed
- L BenMohamed
- L BenMohamed
- L BenMohamed
- L BenMohamed
- L Chen
- Lbachir BenMohamed
- LM Weiner
- M Andrieu
- M Larsson
- M Svensson
- M Triantafilou
- MA Tarp
- MKt Lee
- MV Dhodapkar
- N Monu
- N Petrovsky
- NA Pfender
- O Renaudet
- Olivier Renaudet
- P Nava-Parada
- P Schnorrer
- P van der Bruggen
- Pascal Dumy
- PB Watchmaker
- Q Zhu
- R Belizaire
- R Lo-Man
- R Spohn
- S Bay
- S Borsutzky
- S Casillas
- S Grigalevicius
- S Hakomori
- S Ingale
- S Ingale
- S Khan
- S Radhakrishnan
- S Samanta
- S Vichier-Guerre
- S Vichier-Guerre
- SD Xiang
- SF Slovin
- SJ Keding
- T Buskas
- T Fehr
- T Warger
- TA Groothuis
- TR Smith
- V Kudryashov
- VG Moron
- WR Heath
- X Qi
- X Zhang
- X Zhang
- X Zhu
- Publication venue
- Public Library of Science
- Publication date
- 01/06/2010
- Field of study
Glyco-lipopeptides, a form of lipid-tailed glyco-peptide, are currently under intense investigation as B- and T-cell based vaccine immunotherapy for many cancers. However, the cellular and molecular mechanisms of glyco-lipopeptides (GLPs) immunogenicity and the position of the lipid moiety on immunogenicity and protective efficacy of GLPs remain to be determined.We have constructed two structural analogues of HER-2 glyco-lipopeptide (HER-GLP) by synthesizing a chimeric peptide made of one universal CD4(+) epitope (PADRE) and one HER-2 CD8(+) T-cell epitope (HER(420-429)). The C-terminal end of the resulting CD4-CD8 chimeric peptide was coupled to a tumor carbohydrate B-cell epitope, based on a regioselectively addressable functionalized templates (RAFT), made of four alpha-GalNAc molecules. The resulting HER glyco-peptide (HER-GP) was then linked to a palmitic acid moiety, attached either at the N-terminal end (linear HER-GLP-1) or in the middle between the CD4+ and CD8+ T cell epitopes (branched HER-GLP-2). We have investigated the uptake, processing and cross-presentation pathways of the two HER-GLP vaccine constructs, and assessed whether the position of linkage of the lipid moiety would affect the B- and T-cell immunogenicity and protective efficacy. Immunization of mice revealed that the linear HER-GLP-1 induced a stronger and longer lasting HER(420-429)-specific IFN-gamma producing CD8(+) T cell response, while the branched HER-GLP-2 induced a stronger tumor-specific IgG response. The linear HER-GLP-1 was taken up easily by dendritic cells (DCs), induced stronger DCs maturation and produced a potent TLR- 2-dependent T-cell activation. The linear and branched HER-GLP molecules appeared to follow two different cross-presentation pathways. While regression of established tumors was induced by both linear HER-GLP-1 and branched HER-GLP-2, the inhibition of tumor growth was significantly higher in HER-GLP-1 immunized mice (p<0.005).These findings have important implications for the development of effective GLP based immunotherapeutic strategies against cancers
Residual cancer burden after neoadjuvant chemotherapy and long-term survival outcomes in breast cancer: a multicentre pooled analysis of 5161 patients
- Author
- Abraham Jean e
- Adamson Kathi
- Albain Kathy s.
- Asare Adam l.
- Asare Smita m.
- Balassanian Ron
- Bartlett John m s
- Beckwith Heather
- Berry Donald a.
- Berry Scott m.
- Boughey Judy c.
- Boughey Judy c.
- Buxton Meredith b.
- Caldas Carlos
- Cameron David
- Chen Beiyun
- Chen Yunn-Yi
- Chien A. jo
- Chui Stephen y.
- Clark Amy s.
- Clennell Julia l.
- Cole Kimberly
- Datnow Brian
- De croze Diane
- Del monte-MillƔn Maria
- Demichele Angela m.
- Demichele Angela m.
- Duan Xiuzhen
- Dunn Janet
- Earl Helena
- Edge Stephen b
- Edmiston Kirsten k.
- Elias Anthony d.
- Ellis Erin d.
- Esserman Laura l.
- Esserman Laura l.
- Euhus David m.
- Fadare Oluwole
- Fan Fang
- Fan Fang
- Feldman Michael d
- Forero-Torres Andres
- Godwin Andrew k
- Goetz Matthew p
- Gould Rebekah
- Graham Ashley
- Haley Barbara b.
- Hall Peter
- Hamy Anne-Sophie
- Han Hyo s.
- Harada Shuko
- Haugen Patricia
- Hayward Larry
- Helsten Teresa
- Hiller Louise
- Hirst Gillian l.
- Hoskin Tanya
- Hylton Nola m.
- Isaacs Claudine
- Kemmer Kathleen
- Khan Qamar j.
- Khazai Laila
- Kim Mi-Ok
- Klein Molly e.
- Krings Gregor
- Lang Julie e.
- LaƩ Marick
- Lebeau Lauren g.
- Leyland-Jones Brian
- Liu Minetta c.
- Lo Shelly
- Lu Janice
- LĆ³pez-Tarruella Sara
- Mackintosh Lorna
- Magliocco Anthony
- MartĆn Miguel
- Matthews Jeffrey b.
- Melisko Michelle e.
- Mhawech-Fauceglia Paulette
- Moulder Stacy l.
- Murthy Rashmi k.
- Nanda Rita
- Northfelt Donald w.
- Ocal Idris t.
- Olopade Olufunmilayo
- Osdoit Marie
- Pambuccian Stefan
- Paoloni Melissa
- Park John w.
- Parker Barbara a.
- Perlmutter Jane
- Peterson Garry
- Provenzano Elena
- Pusztai Lajos
- Pusztai Lajos
- Rendi Mara
- Reyal Fabien
- Rugo Hope s.
- Sahoo Sunati
- Sammut Stephen-John
- Sams Sharon
- Sanil Ashish
- Sattar Husain
- Schwab Richard b.
- Schwensen Kelsey
- Shad Sonal
- Sharma Priyanka
- Singhrao Ruby
- Sonke Gabe s
- Steeg Katherine
- Steenbruggen Tessa g
- Stringer-Reasor Erica
- Symmans W. fraser
- Symmans W. fraser
- Tawfik Ossama
- Thomas Jeremy s
- Tripathy Debasish
- Troxell Megan l.
- Valero Vicente
- Van 't veer Laura j
- Van der noordaa Marieke
- Van seijen Maartje
- Van't veer Laura j.
- Venters Sara j.
- Vinh Tuyethoa
- Viscusi Rebecca k.
- Wallace Anne m.
- Wei Jane
- Wei Shi
- Wesseling Jelle
- Wilson Amy
- Yau Christina
- Yau Christina
- Yee Douglas
- Zeck Jay c.
- Publication venue
- Publication date
- 10/12/2021
- Field of study
Measurement of the energy asymmetry in t(t)over-barj production at 13 TeV with the ATLAS experiment and interpretation in the SMEFT framework
- Author
- Aad G
- Abbott B
- Abbott DC
- Abeling K
- Abhayasinghe DK
- Abidi SH
- Aboulhorma A
- Abramowicz H
- Abreu H
- Abud A Abed
- Abulaiti Y
- Abusleme Hoffman AC
- Acharya BS
- Achkar B
- Adam L
- Adamczyk L
- Adamek L
- Addepalli S
- Adelman J
- Adiguzel A
- Adorni S
- Adye T
- Affolder AA
- Afik Y
- Agapopoulou C
- Agaras MN
- Agarwala J
- Aggarwal A
- Agheorghiesei C
- Aguilar-Saavedra JA
- Ahmad A
- Ahmadov F
- Ahmed WS
- Ai X
- Aielli G
- Aizenberg I
- Akatsuka S
- Akbiyik M
- Akesson TPA
- Akimov A
- Al Khoury K
- Alberghi GL
- Albert J
- Albicocco P
- Alconada Verzini MJ
- Alderweireldt S
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexopoulos T
- Alfonsi A
- Alfonsi F
- Alhroob M
- Ali B
- Ali S
- Aliev M
- Alimonti G
- Allaire C
- Allbrooke BMM
- Allport PP
- Aloisio A
- Alonso F
- Alpigiani C
- Alvarez Estevez M
- Alves FL Lucio
- Alviggi MG
- Ambler A
- Ambroz L
- Amelung C
- Amidei D
- Amor Dos Santos SP
- Amoroso S
- Amos KR
- Amrouche CS
- Ananiev V
- Anastopoulos C
- Andari N
- Andeen T
- Anders JK
- Andrean SY
- Andreazza A
- Angelidakis S
- Angerami A
- Anisenkov A
- Annovi A
- Antel C
- Anthony MT
- Antipov E
- Antonelli M
- Antrim DJA
- Anulli F
- Aoki M
- Aparisi Pozo JA
- Aparo MA
- Aranzabal N
- Araya S Tapia
- Arcangeletti C
- Arce ATH
- Arena E
- Arguin J-F
- Argyropoulos S
- Arling J-H
- Armbruster AJ
- Armstrong A
- Arnaez O
- Arnold H
- Artoni G
- Asada H
- Asai K
- Asai S
- Asbah NA
- Asimakopoulou EM
- Asquith L
- Assahsah J
- Assamagan K
- Astalos R
- Astigarraga ME Pozo
- Atkin RJ
- Atkinson M
- Atlay NB
- Atmani H
- Atmasiddha PA
- Augsten K
- Auricchio S
- Austrup VA
- Avner G
- Avolio G
- Ayoub MK
- Azuelos G
- Babal D
- Bachacou H
- Bachas K
- Bachiu A
- Backman F
- Badea A
- Bagnaia P
- Bahrasemani H
- Bailey AJ
- Bailey VR
- Baines JT
- Bakalis C
- Baker OK
- Bakker PJ
- Bakos E
- Balaji S
- Balasubramanian R
- Baldin EM
- Balek P
- Ballabene E
- Balli F
- Baltes LM
- Balunas WK
- Balz J
- Banas E
- Bandieramonte M
- Bandyopadhyay A
- Bansal S
- Barak L
- Barberio EL
- Barberis D
- Barbero M
- Barbour G
- Barends KN
- Barillari T
- Barisits M-S
- Barkeloo J
- Barklow T
- Barnett BM
- Barnett RM
- Baroncelli A
- Barone G
- Barr AJ
- Barranco Navarro L
- Barreiro F
- Barron U
- Barsov S
- Bartels F
- Bartoldus R
- Bartolini G
- Barton AE
- Bartos P
- Basalaev A
- Basan A
- Baselga M
- Bashta I
- Bassalat A
- Basso MJ
- Basson CR
- Bates RL
- Batlamous S
- Batley JR
- Batool B
- Battaglia M
- Bauce M
- Bauer F
- Bauer P
- Bawa HS
- Bayirli A
- Beacham JB
- Beau T
- Beauchemin PH
- Becherer F
- Bechtle P
- Beck HP
- Becker K
- Becot C
- Beddall AJ
- Bednyakov VA
- Bee CP
- Beermann TA
- Begalli M
- Begel M
- Behera A
- Behr JK
- BeiraoDaCruzESilva C
- Beirer JF
- Beisiegel F
- Belfkir M
- Bella G
- Bella L Aperio
- Bellagamba L
- Bellerive A
- Bellos P
- Beloborodov K
- Belotskiy K
- Belyaev NL
- Benchekroun D
- Benhammou Y
- Benjamin DP
- Benoit M
- Bensinger JR
- Bentvelsen S
- Beresford L
- Beretta M
- Berge D
- Berger N
- Bergmann B
- Bergsten LJ
- Beringer J
- Berlendis S
- Berlingen J Montejo
- Bernardi G
- Bernius C
- Bernlochner FU
- Berry T
- Berta P
- Berthold A
- Bertram IA
- Bethke S
- Betti A
- Bevan AJ
- Bhatta S
- Bhattacharya DS
- Bhattarai P
- Bhopatkar VS
- Bi R
- Bi R
- Bianchi RM
- Biebel O
- Bielski R
- Biesuz N
- Biglietti M
- Billoud TR
- Bindi M
- Bingul A
- Bini C
- Biondi S
- Biondini A
- Birch-sykes CJ
- Bird GA
- Birman M
- Bisanz T
- Biswal JP
- Biswas D
- Bitadze A
- Bittrich C
- Bjorke K
- Bloch I
- Blocker C
- Blue A
- Blumenschein U
- Blumenthal J
- Bobbink GJ
- Bobrovnikov VS
- Boehler M
- Boeriu OE Vickey
- Bogavac D
- Bogdanchikov AG
- Bohm C
- Boisvert V
- Bokan P
- Bolanos G Rabanal
- Bold T
- Bomben M
- Bona M
- Boonekamp M
- Booth CD
- Borbely AG
- Borecka-Bielska HM
- Borgna LS
- Borissov G
- Bortoletto D
- Bosca S Rodriguez
- Boscherini D
- Bosman M
- Bostanabad M Ghasemi
- Bouaouda K
- Boudreau J
- Bouhova-Thacker E
- Boumediene D
- Bouquet R
- Bourdarios C Adam
- Boveia A
- Boyd J
- Boye D
- Boyko IR
- Bozson AJ
- Bracinik J
- Brahimi N
- Brandt G
- Brandt O
- Braren F
- Brau B
- Brau JE
- Brendlinger K
- Brener R
- Brenner L
- Brenner R
- Bressler S
- Bret M Cano
- Brickwedde B
- Britton D
- Britzger D
- Brock I
- Brock R
- Brooijmans G
- Brooks WK
- Brost E
- Brueers B
- Bruncko D
- Bruni A
- Bruni G
- Bruschi M
- Bruscino N
- Bryngemark L
- Buanes T
- Buat Q
- Buchholz P
- Buckley AG
- Budagov IA
- Buescher V
- Bugge MK
- Bulekov O
- Bullard BA
- Burdin S
- Burgard CD
- Burger AM
- Burghgrave B
- Burr JTP
- Burton CD
- Burzynski JC
- Busch EL
- Bussey PJ
- Butler JM
- Buttar CM
- Butterworth JM
- Buttinger W
- Buzykaev AR
- Bylund O Bessidskaia
- Cabras G
- Cabrera Urban S
- Caforio D
- Cai H
- Cairo VMM
- Cakir O
- Calace N
- Calafiura P
- Calderini G
- Calfayan P
- Callea G
- Caloba LP
- Calvet D
- Calvet S
- Calvet TP
- Calvetti M
- Camarda S
- Camarero Munoz D
- Camarri P
- Camelia E Alunno
- Camerlingo MT
- Cameron D
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- Xu Z
- Yabsley B
- Yacoob S
- Yamaguchi N
- Yamaguchi Y
- Yamatani M
- Yamauchi H
- Yamazaki T
- Yamazaki Y
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- Yao W-M
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- Yexley MR
- Yildiz H Duran
- Yin P
- Yorita K
- Yoshihara K
- Young C
- Young CJS
- Yuan M
- Yuan R
- Yue X
- Zaazoua M
- Zabinski B
- Zacharis G
- Zagazeta LF Gutierrez
- Zaid E
- Zaitsev AM
- Zakareishvili T
- Zakharchuk N
- Zambito S
- Zanzi D
- Zaplatilek O
- Zeissner S
- Zeitnitz C
- Zeng JC
- Zenger DT
- Zenin O
- Zenis T
- Zenz S
- Zerradi S
- Zerwas D
- Zhang B
- Zhang DF
- Zhang G
- Zhang J
- Zhang K
- Zhang L
- Zhang M
- Zhang R
- Zhang S
- Zhang X
- Zhang X
- Zhang Z
- Zhao P
- Zhao T
- Zhao Y
- Zhao Z
- Zhemchugov A
- Zheng Z
- Zhong D
- Zhou B
- Zhou C
- Zhou H
- Zhou N
- Zhou Y
- Zhu C
- Zhu CG
- Zhu H
- Zhu HL
- Zhu J
- Zhu Y
- Zhuang X
- Zhukov K
- Zhulanov V
- Zieminska D
- Zimine N
- Zimmermann S
- Zinsser J
- Ziolkowski M
- Zivkovic L
- Zoccoli A
- Zoch K
- Zorbas TG
- Zormpa O
- Zou W
- Zwalinski L
- Publication venue
- 'Springer Fachmedien Wiesbaden GmbH'
- Publication date
- 28/04/2022
- Field of study
A measurement of the energy asymmetry in jet-associated top-quark pair production is presented using 139fbā1
139
fb
-
1
of data collected by the ATLAS detector at the Large Hadron Collider during pp collisions at sā=13TeV
s
=
13
TeV
. The observable measures the different probability of top and antitop quarks to have the higher energy as a function of the jet scattering angle with respect to the beam axis. The energy asymmetry is measured in the semileptonic ttĖ
t
t
ĀÆ
decay channel, and the hadronically decaying top quark must have transverse momentum above 350GeV
350
GeV
. The results are corrected for detector effects to particle level in three bins of the scattering angle of the associated jet. The measurement agrees with the SM prediction at next-to-leading-order accuracy in quantum chromodynamics in all three bins. In the bin with the largest expected asymmetry, where the jet is emitted perpendicular to the beam, the energy asymmetry is measured to be ā0.043Ā±0.020
-
0.043
Ā±
0.020
, in agreement with the SM prediction of ā0.037Ā±0.003
-
0.037
Ā±
0.003
. Interpreting this result in the framework of the Standard Model effective field theory (SMEFT), it is shown that the energy asymmetry is sensitive to the top-quark chirality in four-quark operators and is therefore a valuable new observable in global SMEFT fits
Measurement of the ttĀÆttĀÆ production cross section in pp collisions at ās=13 TeV with the ATLAS detector
- Author
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- Abhayasinghe DK
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- Ćncel ĆO
- ŽeniŔ T
- ŽivkoviÄ L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 16/11/2021
- Field of study
A measurement of four-top-quark production using proton-proton collision data at a centre-of-mass energy of 13 TeV collected by the ATLAS detector at the Large Hadron Collider corresponding to an integrated luminosity of 139 fbā1 is presented. Events are selected if they contain a single lepton (electron or muon) or an opposite-sign lepton pair, in association with multiple jets. The events are categorised according to the number of jets and how likely these are to contain b-hadrons. A multivariate technique is then used to discriminate between signal and background events. The measured four-top-quark production cross section is found to be 26+17ā15 fb, with a corresponding observed (expected) significance of 1.9 (1.0) standard deviations over the background-only hypothesis. The result is combined with the previous measurement performed by the ATLAS Collaboration in the multilepton final state. The combined four-top-quark production cross section is measured to be 24+7ā6 fb, with a corresponding observed (expected) signal significance of 4.7 (2.6) standard deviations over the background-only predictions. It is consistent within 2.0 standard deviations with the Standard Model expectation of 12.0 Ā± 2.4 fb
Measurements of W+Wā+ ā„ 1 jet production cross-sections in pp collisions at sā = 13 TeV with the ATLAS detector
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- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/06/2021
- Field of study
Fiducial and differential cross-section measurements of W+Wā production in
association with at least one hadronic jet are presented. These measurements are sensitive
to the properties of electroweak-boson self-interactions and provide a test of perturbative
quantum chromodynamics and the electroweak theory. The analysis is performed using
proton-proton collision data collected at p
s = 13TeV with the ATLAS experiment, corresponding
to an integrated luminosity of 139 fbā1. Events are selected with exactly one oppositely
charged electron-muon pair and at least one hadronic jet with a transverse momentum
of pT > 30 GeV and a pseudorapidity of |ļæ½| < 4.5. After subtracting the background
contributions and correcting for detector effects, the jet-inclusive W+Wā+ ļæ½ 1 jet fiducial
cross-section and W+Wā+ jets differential cross-sections with respect to several kinematic
variables are measured. These measurements include leptonic quantities, such as the lepton
transverse momenta and the transverse mass of the W+Wā system, as well as jet-related
observables such as the leading jet transverse momentum and the jet multiplicity. Limits on
anomalous triple-gauge-boson couplings are obtained in a phase space where interference
between the Standard Model amplitude and the anomalous amplitude is enhanced
Measurements of sensor radiation damage in the ATLAS inner detector using leakage currents
- Author
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- Publication venue
- IOP
- Publication date
- 01/01/2021
- Field of study
Non-ionizing energy loss causes bulk damage to the silicon sensors of the ATLAS pixel and strip detectors. This damage has important implications for data-taking operations, charged-particle track reconstruction, detector simulations, and physics analysis. This paper presents simulations and measurements of the leakage current in the ATLAS pixel detector and semiconductor tracker as a function of location in the detector and time, using data collected in Run 1 (2010ā2012) and Run 2 (2015ā2018) of the Large Hadron Collider. The extracted fluence shows a much stronger |z|-dependence in the innermost layers than is seen in simulation. Furthermore, the overall fluence on the second innermost layer is significantly higher than in simulation, with better agreement in layers at higher radii. These measurements are important for validating the simulation models and can be used in part to justify safety factors for future detector designs and interventions.publishedVersio
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