Topological mass generation to antisymmetric tensor matter field

We propose a mechanism to give mass to tensor matter field which preserve the U(1) symmetry. We introduce a complex vector field that couples with the tensor in a topological term. We also analyze the influence of the kinetic terms of the complex vector in our mechanism.Comment: 5 pages, to appear in Europhysics Letter

Fruit development of three guava cultivars (Psidium guajava L.).

The knowledge of guava fruiting cycle is of great interest not only due to its biological aspects but also as an important datum for the application of cultural practices. This paper was carried out in the submedio Sao Francisco region on four-year old plants, from the germplasm bank of EMBRAPA-CPATSA. The objective was to study the fruit growth pattern of the guava cultivars Patillo, Paluma and Red Selection of Florida. Fruit length and diameter were monitored on twenty-day intervals, resulting on double sigmoidal curves, and characterizing three growth periods, for the cultivars. The results showed that, within the experimental period, the cultivars Red Selection of FLorida and Paluma had about the same diameter and length growth rate, with a mean value of 0.036 cm/day and 0.042 cm/day, respectively. However ,the biggest fruit size was observed for the cv. Red Selection of Florida. The cv. Patillo had the lowest fruit growth rate, with an increment of 0.027 cm/day in diameter and 0.033 cm/day in lenght

Generating mass and topological terms to the antisymmetric tensor matter field by Higgs mechanism

The interaction between the complex antisymmetric tensor matter field and a scalar field is constructed. We analyze the Higgs mechanism and show the generation of mass and topological terms by spontaneous symmetry breaking.Comment: Accepted for publication in Phys. Lett.

Genome of the Avirulent Human-Infective Trypanosome—Trypanosoma rangeli

Background: Trypanosoma rangeli is a hemoflagellate protozoan parasite infecting humans and other wild and domestic mammals across Central and South America. It does not cause human disease, but it can be mistaken for the etiologic agent of Chagas disease, Trypanosoma cruzi. We have sequenced the T. rangeli genome to provide new tools for elucidating the distinct and intriguing biology of this species and the key pathways related to interaction with its arthropod and mammalian hosts.  Methodology/Principal Findings: The T. rangeli haploid genome is ,24 Mb in length, and is the smallest and least repetitive trypanosomatid genome sequenced thus far. This parasite genome has shorter subtelomeric sequences compared to those of T. cruzi and T. brucei; displays intraspecific karyotype variability and lacks minichromosomes. Of the predicted 7,613 protein coding sequences, functional annotations could be determined for 2,415, while 5,043 are hypothetical proteins, some with evidence of protein expression. 7,101 genes (93%) are shared with other trypanosomatids that infect humans. An ortholog of the dcl2 gene involved in the T. brucei RNAi pathway was found in T. rangeli, but the RNAi machinery is non-functional since the other genes in this pathway are pseudogenized. T. rangeli is highly susceptible to oxidative stress, a phenotype that may be explained by a smaller number of anti-oxidant defense enzymes and heatshock proteins.  Conclusions/Significance: Phylogenetic comparison of nuclear and mitochondrial genes indicates that T. rangeli and T. cruzi are equidistant from T. brucei. In addition to revealing new aspects of trypanosome co-evolution within the vertebrate and invertebrate hosts, comparative genomic analysis with pathogenic trypanosomatids provides valuable new information that can be further explored with the aim of developing better diagnostic tools and/or therapeutic targets

Erratum to: The study of cardiovascular risk in adolescents – ERICA: rationale, design and sample characteristics of a national survey examining cardiovascular risk factor profile in Brazilian adolescents

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