37 research outputs found
Inhibition of the RAGE products increases survival in experimental models of severe sepsis and systemic infection
RsrI: A new sequence-specific endonuclease from rhodopseudomonas spaeroides
Thesis (B.S.) in Liberal Arts and Sciences--University of Illinois at Urbana-Champaign, 1984.Bibliography: leaves 11-12.U of I OnlyTheses restricted to UIUC community onl
Specific cross-reactivity of antibodies raised against two major stress proteins, stress 70 and chaperonin 60, in diverse species
Rapid and reversible relocalization of heat shock factor 1 within seconds to nuclear stress granules
Requirement of Tyr-992 and Tyr-1173 in Phosphorylation of the Epidermal Growth Factor Receptor by Ionizing Radiation and Modulation by SHP2
Delayed administration of the anti-RAGE antibody is protective in cecal ligation and puncture (CLP)
<p><b>Copyright information:</b></p><p>Taken from "Inhibition of the RAGE products increases survival in experimental models of severe sepsis and systemic infection"</p><p>http://ccforum.com/content/11/6/R122</p><p>Critical Care 2007;11(6):R122-R122.</p><p>Published online 6 Nov 2007</p><p>PMCID:PMC2246216.</p><p></p> The Kaplan-Meier survival analysis following CLP in BALB/c mice compares delayed anti-RAGE monoclonal antibody (mAb) treatment given at various time intervals after CLP with serum control. Each group had a significantly greater survival than the control group (< 0.01), except for the 36-hour delayed treatment group (= 0.12). RAGE, receptor for advanced glycation end products
Modulation of receptor for advanced glycation end products (RAGE) protects mice from the effects of cecal ligation and puncture (CLP)
<p><b>Copyright information:</b></p><p>Taken from "Inhibition of the RAGE products increases survival in experimental models of severe sepsis and systemic infection"</p><p>http://ccforum.com/content/11/6/R122</p><p>Critical Care 2007;11(6):R122-R122.</p><p>Published online 6 Nov 2007</p><p>PMCID:PMC2246216.</p><p></p> Kaplan-Meier survival analysis following CLP comparing wild-type RAGE129SvEvBrd mice (= 15), RAGEmice (= 15), RAGEmice (= 23), and anti-RAGE monoclonal antibody-treated (15 mg/kg intravenously 30 to 60 minutes before CLP) wild-type mice (= 15). < 0.001 for each group in comparison with the wild-type CLP control group. Sham surgery-treated wild-type 129SvEvBrd mice (= 5) were used as an additional control group. WT, wild-type