Integrating media content analysis, reception analysis, and media effects studies

Every day, the world of media is at our fingertips, whether it is watching movies, listening to the radio, or browsing online media. On average, people spend over 8 h per day consuming messages from the mass media, amounting to a total lifetime dose of more than 20 years in which conceptual content stimulates our brains. Effects from this flood of information range from short-term attention bursts (e.g., by breaking news features or viral ‘memes’) to life-long memories (e.g., of one’s favorite childhood movie), and from micro-level impacts on an individual’s memory, attitudes, and behaviors to macro-level effects on nations or generations. The modern study of media’s influence on society dates back to the 1940s. This body of mass communication scholarship has largely asked, “what is media’s effect on the individual?” Around the time of the cognitive revolution, media psychologists began to ask, “what cognitive processes are involved in media processing?” More recently, neuroimaging researchers started using real-life media as stimuli to examine perception and cognition under more natural conditions. Such research asks: “what can media tell us about brain function?” With some exceptions, these bodies of scholarship often talk past each other. An integration offers new insights into the neurocognitive mechanisms through which media affect single individuals and entire audiences. However, this endeavor faces the same challenges as all interdisciplinary approaches: Researchers with different backgrounds have different levels of expertise, goals, and foci. For instance, neuroimaging researchers label media stimuli as “naturalistic” although they are in many ways rather artificial. Similarly, media experts are typically unfamiliar with the brain. Neither media creators nor neuroscientifically oriented researchers approach media effects from a social scientific perspective, which is the domain of yet another species. In this article, we provide an overview of approaches and traditions to studying media, and we review the emerging literature that aims to connect these streams. We introduce an organizing scheme that connects the causal paths from media content → brain responses → media effects and discuss network control theory as a promising framework to integrate media content, reception, and effects analyses

Adicción a los Videojuegos: ¿Qué podemos aprender desde la perspectiva de la neurociencia de la comunicación?

In recent years, video game addiction has received considerable empirical attention. Unfortunately, this research is stymied by inconsistencies in both conceptual and operational definitions of video game addiction. Moreover, the use of several video game addiction scales makes it difficult to estimate the prevalence and potential effects of video game addiction. While game genre is often treated as a predictor of video game addiction, existing measures often downplay the structural and social characteristics of video games that may contribute to behavioral outcomes such as increased playing time and addiction. In an effort to provide the clarity necessary to overcome these issues, we review research on video game addiction with a focus on the largely ignored unique characteristics of video games that are crucial for a more complete conceptualization of video game addiction. With this review in mind, we offer a conceptual framework for the integration of video game addiction within the broader context of behavioral addictions. Finally, we consider the neurological foundation of addiction and suggest opportunities for media neuroscientists to increase understanding and prediction of video game addiction and explore how game content features interact with reward systems in the brain.En los últimos años, la adicción a los videojuegos ha recibido una atención empírica considerable. Desafortunadamente, la investigación en este campo se encuentra obstaculizada por inconsistencias en definiciones conceptuales y operacionales de la adicción a los videojuegos. Por otro lado, el uso de varias escalas de adicción a los videojuegos dificulta la estimación de la prevalencia y los efectos potenciales de la adicción a los videojuegos. Mientras el género del juego es considerado frecuentemente como un predictor de la adicción a los videojuegos, las medidas existentes restan importancia a las características sociales y estructurales de los videojuegos que pueden contribuir a resultados conductuales tales como un incremento en el tiempo de juego y adicción. En un esfuerzo por proveer de claridad necesaria para subsanar estos problemas, se realiza una revisión sobre investigaciones relacionadas a la adicción a los videojuegos con un enfoque en las características de los videojuegos extensamente ignoradas y que son cruciales para una conceptualización más completa acerca de la adicción a los videojuegos. Con esta revisión, ofrecemos un marco conceptual para la integración de la adicción a los videojuegos dentro un contexto más amplio como el de las adicciones conductuales. Finalmente, consideramos la base neurológica de la adicción y sugerimos oportunidades para los neurocientíficos de la comunicación con el objetivo de incrementar la comprensión y predicción de la adicción a los videojuegos y explorar cómo los elementos de un juego interactúan con los sistemas de recompensa en el cerebro

Intrinsic Reward Motivates Large-Scale Shifts Between Cognitive Control and Default Mode Networks During Task Performance

Cognitive control is an important framework for understanding the neuropsychological processes that underlie and enable the successful completion of everyday tasks. Only recently has research in this area investigated motivational contributions to control allocation. An important gap in our understanding is the way in which intrinsic rewards associated with a task motivate the sustained allocation of cognitive control. In three behavioral and one functional magnetic resonance imaging studies, we use a naturalistic and open-sourced simulator to show that changes in the balance between task difficulty and an individual’s ability to perform the task result in different levels of intrinsic reward, which motivates dynamic shifts between networked brain states. Specifically, high levels of intrinsic reward associated with a balance between task difficulty and individual ability are associated with increased connectivity between cognitive control and reward networks. By comparison, a mismatch between task difficulty and individual ability is associated with lower levels of intrinsic reward and corresponds to increased activity within the default mode network. Insular activation suggests that motivational salience, as defined by the level of intrinsic reward, drives shifts between networked brain states associated with task engagement or disengagement. These results implicate reward processing as a critical component of cognitive control

Pelajar tunai tanggungjawab pilih perwakilan dalam pilihan raya kampus

Pilihan Raya Kampus (PRK) Universiti Malaysia Pahang (UMP) bagi memilih calon Majlis Perwakilan Pelajar Sesi 2015/2016 yang diadakan pada 7 Oktober 2015 yang lalu berjalan lancar apabila pelajar masingmasing turun padang memilih calon mereka

The Vicious Circle of Post-Soviet Neopatrimonialism in Russia

Published online: 10 Aug 2015, journal issue (vol.32, N5) appeared in 2016Since the collapse of Communism, Russia and some other post-Soviet states have attempted to pursue socio-economic reforms while relying upon the political institutions of neopatrimonialism. This politico-economic order was established to serve the interests of ruling groups and establish the major features of states, political regimes, and market economies. It provided numerous negative incentives for governing the economy and the state due to the unconstrained rent seeking behavior of major actors. Policy reform programs discovered these institutions to be incompatible with the priorities of modernization, and efforts to resolve these contradictions through a number of partial and compromise solutions often worsened the situation vis-à-vis preservation of the status quo. The ruling groups lack incentives for institutional changes, which could undermine their political and economic dominance, and are caught in a vicious circle: reforms often result in minor returns or cause unintended and undesired consequences. What are the possible domestic and international incentives to reject the political institutions of neopatrimonialism in post-Soviet states and replace them with inclusive economic and political ones?Peer reviewe

An Agenda for Open Science in Communication

In the last 10 years, many canonical findings in the social sciences appear unreliable. This so-called “replication crisis” has spurred calls for open science practices, which aim to increase the reproducibility, replicability, and generalizability of findings. Communication research is subject to many of the same challenges that have caused low replicability in other fields. As a result, we propose an agenda for adopting open science practices in Communication, which includes the following seven suggestions: (1) publish materials, data, and code; (2) preregister studies and submit registered reports; (3) conduct replications; (4) collaborate; (5) foster open science skills; (6) implement Transparency and Openness Promotion Guidelines; and (7) incentivize open science practices. Although in our agenda we focus mostly on quantitative research, we also reflect on open science practices relevant to qualitative research. We conclude by discussing potential objections and concerns associated with open science practices

The Gly2019Ser mutation in LRRK2 is not fully penetrant in familial Parkinson's disease: the GenePD study

<p>Abstract</p> <p>Background</p> <p>We report age-dependent penetrance estimates for leucine-rich repeat kinase 2 (<it>LRRK2</it>)-related Parkinson's disease (PD) in a large sample of familial PD. The most frequently seen <it>LRRK2 </it>mutation, Gly2019Ser (G2019S), is associated with approximately 5 to 6% of familial PD cases and 1 to 2% of idiopathic cases, making it the most common known genetic cause of PD. Studies of the penetrance of <it>LRRK2 </it>mutations have produced a wide range of estimates, possibly due to differences in study design and recruitment, including in particular differences between samples of familial PD versus sporadic PD.</p> <p>Methods</p> <p>A sample, including 903 affected and 58 unaffected members from 509 families ascertained for having two or more PD-affected members, 126 randomly ascertained PD patients and 197 controls, was screened for five different <it>LRRK2 </it>mutations. Penetrance was estimated in families of <it>LRRK2 </it>carriers with consideration of the inherent bias towards increased penetrance in a familial sample.</p> <p>Results</p> <p>Thirty-one out of 509 families with multiple cases of PD (6.1%) were found to have 58 <it>LRRK2 </it>mutation carriers (6.4%). Twenty-nine of the 31 families had G2019S mutations while two had R1441C mutations. No mutations were identified among controls or unaffected relatives of PD cases. Nine PD-affected relatives of G2019S carriers did not carry the <it>LRRK2 </it>mutation themselves. At the maximum observed age range of 90 to 94 years, the unbiased estimated penetrance was 67% for G2019S families, compared with a baseline PD risk of 17% seen in the non-<it>LRRK2</it>-related PD families.</p> <p>Conclusion</p> <p>Lifetime penetrance of <it>LRRK2 </it>estimated in the unascertained relatives of multiplex PD families is greater than that reported in studies of sporadically ascertained <it>LRRK2 </it>cases, suggesting that inherited susceptibility factors may modify the penetrance of <it>LRRK2 </it>mutations. In addition, the presence of nine PD phenocopies in the <it>LRRK2 </it>families suggests that these susceptibility factors may also increase the risk of non-<it>LRRK2</it>-related PD. No differences in penetrance were found between men and women, suggesting that the factors that influence penetrance for <it>LRRK2 </it>carriers are independent of the factors which increase PD prevalence in men.</p