A Novel Algorithm for Effective Vibration Control of Portal Frames

Severe vibrations such as earthquakes threaten to demolish or cause damage to built structures during their lifetime. Mitigation of such damage can be done by using control devices such as actuators. In this paper, an algorithm is proposed to analyze the nonlinear behavior of a portal frame supported by an actuator. The results were compared with those for a frame without actuator. The algorithm was developed in accordance with the Timoshenko beam element theory. ANSYS verified the results for the cases of a frame supported by a damper element and a frame without actuator. The results support the efficiency of the algorithm in reducing frame vibration and top-node displacement

The quantum dynamic capacity formula of a quantum channel

The dynamic capacity theorem characterizes the reliable communication rates of a quantum channel when combined with the noiseless resources of classical communication, quantum communication, and entanglement. In prior work, we proved the converse part of this theorem by making contact with many previous results in the quantum Shannon theory literature. In this work, we prove the theorem with an "ab initio" approach, using only the most basic tools in the quantum information theorist's toolkit: the Alicki-Fannes' inequality, the chain rule for quantum mutual information, elementary properties of quantum entropy, and the quantum data processing inequality. The result is a simplified proof of the theorem that should be more accessible to those unfamiliar with the quantum Shannon theory literature. We also demonstrate that the "quantum dynamic capacity formula" characterizes the Pareto optimal trade-off surface for the full dynamic capacity region. Additivity of this formula simplifies the computation of the trade-off surface, and we prove that its additivity holds for the quantum Hadamard channels and the quantum erasure channel. We then determine exact expressions for and plot the dynamic capacity region of the quantum dephasing channel, an example from the Hadamard class, and the quantum erasure channel.Comment: 24 pages, 3 figures; v2 has improved structure and minor corrections; v3 has correction regarding the optimizatio

B -> Xs l_i^+ l_j^+ Decays with R-parity Violation

We derive the upper bounds on certain products of R-parity- and lepton-flavor-violating couplings from B \ra X_s {l_i}^+ {l_j}^- decays. These modes of B-meson decays can constrain the product combinations of the couplings with one or more heavy generation indices which are comparable with or stronger than the present bounds. From the studies of the invariant dilepton mass spectrum and the forward backward asymmetry of the emitted leptons we note the possibility of detecting R-parity-violating signals even when the total decay rate due to R-parity violating couplings is comparable with that in the standard model and discriminating two types of R-parity-violating signals. The general expectation of the enhancement of the forward backward asymmetry of the emitted leptons in the minimal supersymmetric standard model with R-parity may be corrupted by R-parity violation.Comment: 10 pages, Revtex, 1 table and 2 figure

Acupuncture Point Localization Varies Among Acupuncturists

Background: Studies assessing the point-specific effect of acupuncture or the characteristics of acupuncture points (APs) tend to yield inconclusive results. In order to identify a possible confounding factor, we aimed to examine the variability in AP localization by means of a survey. Material and Methods: Attendees of the 14th ICMART (International Council of Medical Acupuncture and Related Techniques) congress as well as DAGfA (German Medical Society of Acupuncture) lecturers and students were asked to locate and mark the APs LI 10 and TH 5 on a research assistant's arm. Identified points were transferred into a coordinate system, and the respective bivariate distribution function was calculated. Additionally, participants filled out a questionnaire about their acupuncture education and experience, the acupuncture style and point localization techniques used most frequently, and their estimation of the size of an AP. Results: The areas of the ellipses, theoretically containing 95% of AP localizations, varied between 44.49 and 5.18 cm(2). The largest distance between 2 identified points was 8.45 cm for LI 10 and 5.3 cm for TH 5. Apart from being trained at the same school, no other factor could be identified that determined the variability in AP localization. Conclusion: Our results indicate that congruity of AP localization among experienced acupuncturists might be low. Although there are some limitations to our results, this possible bias should be taken into account when conducting acupuncture trials and interpreting results of previous acupuncture studies

Public and private communication with a quantum channel and a secret key

We consider using a secret key and a noisy quantum channel to generate noiseless public communication and noiseless private communication. The optimal protocol for this setting is the publicly-enhanced private father protocol. This protocol exploits random coding techniques and "piggybacking" of public information along with secret-key-assisted private codes. The publicly-enhanced private father protocol is a generalization of the secret-key-assisted protocol of Hsieh, Luo, and Brun and a generelization of a protocol for simultaneous communication of public and private information suggested by Devetak and Shor.Comment: 15 pages, 2 figure

Solar and Atmospheric Neutrino Oscillations and Lepton Flavor Violation in Supersymmetric Models with Right-handed Neutrinos

Taking the solar and the atmospheric neutrino experiments into account we discuss the lepton flavor violating processes, such as $\tau\to\mu\gamma$ or $\mu\to e\gamma$, in the minimal supersymmetric standard model with right-handed neutrinos (MSSMRN) and the supersymmetric SU(5) GUT with right-handed neutrinos (SU(5)RN). The predicted branching ratio of $\mu\to e\gamma$ in the MSSMRN with the MSW large angle solution is so large that it goes beyond the current experimental bound if the second-generation right-handed Majorana mass $M_{\nu_2}$ is greater than $\sim 10^{13}(\sim 10^{14})$GeV for $\tan\beta=30(3)$. When we take the MSW small angle solution, the $\mu\to e\gamma$ rate is at most about 1/100 of that of the MSW large angle solution. The 'just so' solution implies $10^{-5}$ of that of the MSW large angle solution. Also, in the SU(5)RN the large $\mu\to e\gamma$ rate naturally follows from the MSW large angle solution, and the predicted rate is beyond the current experimental bound if the typical right-handed Majorana mass $M_N$ is larger than $\sim 10^{13}(\sim 10^{14})$GeV for $\tan\beta=30(3)$, similarly to the MSSMRN. We show the multimass insertion formulas and their applications to $\tau\to\mu\gamma$ and $\mu\to e\gamma$.Comment: 55 pages, 18 figures, Latex, the final version to be published in Phys. Rev.

CP Violating B Decays with R-parity Violation

We study CP violating B decays in the minimal supersymmetric standard model with R-parity violation. We estimate how much R-parity violation modifies the SM predictions for CP asymmetries in B decays within the present bounds. The effects of R-parity- and lepton-number-violating couplings on the ratio of the decay amplitude due to R-parity violation to that of the SM can differ by one or two orders of magnitudes depending on the models of the left-handed quark mixing. It is possible to disentangle the R-parity violating effects from those of the SM and R-parity- conserving supersymmetric models within the present bounds comparing different CP violating decay amplitudes. We also study the effects of R-parity- and baryon-number-violating couplings and find that the effects could be large.Comment: 14 pages. Expanded discussions of the bounds from K decays. To appear in Phys. Rev.

OGLE-2015-BLG-1649Lb:A gas giant planet around a low-mass dwarf

We report the discovery of an exoplanet from the analysis of the gravitational microlensing event OGLE-2015-BLG-1649 that challenges the core accretion model of planet formation and appears to support the disk instability model. The planet/host-star mass ratio is q = 7.2 × 10−3 and the projected separation normalized to the angular Einstein radius is s = 0.9. We conducted high-resolution follow-up observations using the Infrared Camera and Spectrograph (IRCS) camera on the Subaru telescope and are able to place an upper limit on the lens flux. From these measurements we are able to exclude all host stars greater than or equal in mass to a G-type dwarf. We conducted a Bayesian analysis with these new flux constraints included as priors resulting in estimates of the masses of the host star and planet. These are M L = 0.34 ± 0.19 M ⊙ and M p =2.5+1.5 -1.4 M jup, respectively. The distance to the system is D L = 4.23 +1.51-1.64 kpc. The projected star–planet separation is a ⊥ = 2.07+0.65-0.77 au. The estimated relative lens-source proper motion, ~7.1 mas yr−1, is fairly high and thus the lens can be better constrained if additional follow-up observations are conducted several years after the event.PostprintPeer reviewe

Sex-differential genetic effect of phosphodiesterase 4D (PDE4D) on carotid atherosclerosis

<p>Abstract</p> <p>Background</p> <p>The phosphodiesterase 4D (PDE4D) gene was reported as a susceptibility gene to stroke. The genetic effect might be attributed to its role in modulating the atherogenic process in the carotid arteries. Using carotid intima-media thickness (IMT) and plaque index as phenotypes, the present study sought to determine the influence of this gene on subclinical atherosclerosis.</p> <p>Methods</p> <p>Carotid ultrasonography was performed on 1013 stroke-free subjects who participated in the health screening programs (age 52.6 ± 12.2; 47.6% men). Genotype distribution was compared among the high-risk (plaque index ≥ 4), low-risk (index = 1-3), and reference (index = 0) groups. We analyzed continuous IMT data and further dichotomized IMT data using mean plus one standard deviation as the cutoff level. Because the plaque prevalence and IMT values displayed a notable difference between men and women, we carried out sex-specific analyses in addition to analyzing the overall data. Rs702553 at the PDE4D gene was selected because it conferred a risk for young stroke in our previous report. Previous young stroke data (190 cases and 211 controls) with an additional 532 control subjects without ultrasonic data were shown as a cross-validation for the genetic effect.</p> <p>Results</p> <p>In the overall analyses, the rare homozygote of rs702553 led to an OR of 3.1 (p = 0.034) for a plaque index ≥ 4. When subjects were stratified by sex, the genetic effect was only evident in men but not in women. Comparing male subjects with plaque index ≥ 4 and those with plaque index = 0, the TT genotype was over-represented (27.6% vs. 13.4%, p = 0.008). For dichotomized IMT data in men, the TT genotype had an OR of 2.1 (p = 0.032) for a thicker IMT at the common carotid artery compared with the (AA + AT) genotypes. In women, neither IMT nor plaque index was associated with rs702553. Similarly, SNP rs702553 was only significant in young stroke men (OR = 1.8, p = 0.025) but not in women (p = 0.27).</p> <p>Conclusions</p> <p>The present study demonstrates a sex-differential effect of PDE4D on IMT, plaque index and stroke, which highlights its influence on various aspects of atherogenesis.</p

Role of the CCAAT-Binding Protein NFY in SCA17 Pathogenesis

Spinocerebellar ataxia 17 (SCA17) is caused by expansion of the polyglutamine (polyQ) tract in human TATA-box binding protein (TBP) that is ubiquitously expressed in both central nervous system and peripheral tissues. The spectrum of SCA17 clinical presentation is broad. The precise pathogenic mechanism in SCA17 remains unclear. Previously proteomics study using a cellular model of SCA17 has revealed reduced expression of heat shock 70 kDa protein 5 (HSPA5) and heat shock 70 kDa protein 8 (HSPA8), suggesting that impaired protein folding may contribute to the cell dysfunction of SCA17 (Lee et al., 2009). In lymphoblastoid cells, HSPA5 and HSPA8 expression levels in cells with mutant TBP were also significantly lower than that of the control cells (Chen et al., 2010). As nuclear transcription factor Y (NFY) has been reported to regulate HSPA5 transcription, we focused on if NFY activity and HSPA5 expression in SCA17 cells are altered. Here, we show that TBP interacts with NFY subunit A (NFYA) in HEK-293 cells and NFYA incorporated into mutant TBP aggregates. In both HEK-293 and SH-SY5Y cells expressing TBP/Q61∼79, the level of soluble NFYA was significantly reduced. In vitro binding assay revealed that the interaction between TBP and NFYA is direct. HSPA5 luciferase reporter assay and endogenous HSPA5 expression analysis in NFYA cDNA and siRNA transfection cells further clarified the important role of NFYA in regulating HSPA5 transcription. In SCA17 cells, HSPA5 promoter activity was activated as a compensatory response before aggregate formation. NFYA dysfunction was indicated in SCA17 cells as HSPA5 promoter activity reduced along with TBP aggregate formation. Because essential roles of HSPA5 in protection from neuronal apoptosis have been shown in a mouse model, NFYA could be a target of mutant TBP in SCA17