686 research outputs found
High Order Multistep Methods with Improved Phase-Lag Characteristics for the Integration of the Schr\"odinger Equation
In this work we introduce a new family of twelve-step linear multistep
methods for the integration of the Schr\"odinger equation. The new methods are
constructed by adopting a new methodology which improves the phase lag
characteristics by vanishing both the phase lag function and its first
derivatives at a specific frequency. This results in decreasing the sensitivity
of the integration method on the estimated frequency of the problem. The
efficiency of the new family of methods is proved via error analysis and
numerical applications.Comment: 36 pages, 6 figure
Winnowing Wheat from Chaff: The Chunking GA
In this work, we investigate the ability of a Chunking GA (ChGA) to reduce the size of variable length chromosomes and control bloat. The ChGA consists of a standard genetic algorithm augmented by a communal building block memory system and associated memory chromosomes and operators. A new mxn MaxSum fitness function used for this work is also described. Results show that a ChGA equipped with memory capacity equal to or greater than the minimal size of an optimal solution naturally eliminates unexpressed genes. © Springer-Verlag Berlin Heidelberg 2004
Unparticles-Higgs Interplay
We show that scalar unparticles coupled to the Standard Model Higgs at the
renormalizable level can have a dramatic impact in the breaking of the
electroweak symmetry already at tree level. In particular one can get the
proper electroweak scale without the need of a Higgs mass term in the
Lagrangian. By studying the mixed unparticle-Higgs propagator and spectral
function we also show how unparticles can shift the Higgs mass away from its
Standard Model value, \lambda v^2, and influence other Higgs boson properties.
Conversely, we study in some detail how electroweak symmetry breaking affects
the unparticle sector by breaking its conformal symmetry and generating a mass
gap. We also show that, for Higgs masses above that gap, unparticles can
increase quite significantly the Higgs width.Comment: 14 pages, 7 figures, typos correcte
Low-energy excitations in the three-dimensional random-field Ising model
The random-field Ising model (RFIM), one of the basic models for quenched
disorder, can be studied numerically with the help of efficient ground-state
algorithms. In this study, we extend these algorithm by various methods in
order to analyze low-energy excitations for the three-dimensional RFIM with
Gaussian distributed disorder that appear in the form of clusters of connected
spins. We analyze several properties of these clusters. Our results support the
validity of the droplet-model description for the RFIM.Comment: 10 pages, 9 figure
Results of Prevention of REStenosis with Tranilast and its Outcomes (PRESTO) trial
BACKGROUND: Restenosis after percutaneous coronary intervention (PCI) is a major problem affecting 15% to 30% of patients after stent placement. No oral agent has shown a beneficial effect on restenosis or on associated major adverse cardiovascular events. In limited trials, the oral agent tranilast has been shown to decrease the frequency of angiographic restenosis after PCI. METHODS AND RESULTS: In this double-blind, randomized, placebo-controlled trial of tranilast (300 and 450 mg BID for 1 or 3 months), 11 484 patients were enrolled. Enrollment and drug were initiated within 4 hours after successful PCI of at least 1 vessel. The primary end point was the first occurrence of death, myocardial infarction, or ischemia-driven target vessel revascularization within 9 months and was 15.8% in the placebo group and 15.5% to 16.1% in the tranilast groups (P=0.77 to 0.81). Myocardial infarction was the only component of major adverse cardiovascular events to show some evidence of a reduction with tranilast (450 mg BID for 3 months): 1.1% versus 1.8% with placebo (P=0.061 for intent-to-treat population). The primary reason for not completing treatment was > or =1 hepatic laboratory test abnormality (11.4% versus 0.2% with placebo, P<0.01). In the angiographic substudy composed of 2018 patients, minimal lumen diameter (MLD) was measured by quantitative coronary angiography. At follow-up, MLD was 1.76+/-0.77 mm in the placebo group, which was not different from MLD in the tranilast groups (1.72 to 1.78+/-0.76 to 80 mm, P=0.49 to 0.89). In a subset of these patients (n=1107), intravascular ultrasound was performed at follow-up. Plaque volume was not different between the placebo and tranilast groups (39.3 versus 37.5 to 46.1 mm(3), respectively; P=0.16 to 0.72). CONCLUSIONS: Tranilast does not improve the quantitative measures of restenosis (angiographic and intravascular ultrasound) or its clinical sequelae
Production and Decay of D_1(2420)^0 and D_2^*(2460)^0
We have investigated and final states and
observed the two established charmed mesons, the with mass
MeV/c and width MeV/c and
the with mass MeV/c and width
MeV/c. Properties of these final states, including
their decay angular distributions and spin-parity assignments, have been
studied. We identify these two mesons as the doublet predicted
by HQET. We also obtain constraints on {\footnotesize } as a function of the cosine of the relative phase of the two
amplitudes in the decay.Comment: 15 pages in REVTEX format. hardcopies with figures can be obtained by
sending mail to: [email protected]
An Emerging Natural History in the Development, Mechanisms and Worldwide Prevalence of Major Mental Disorders
Conciliating recent findings from molecular genetics, evolutionary biology, and clinical observations together point to new understandings regarding the mechanism, development and the persistent worldwide prevalence of major mental disorders (MMDs),
which should be considered the result of an evolutionary downside trade off. Temperamental/trait variability, by facilitating choices
for individual and group responses, confers robustness flexibility and resilience crucial to success of our species. Extreme temperamental variants, originating evolutionarily from the asocial aspect of human nature, also constitute the premorbid personality
of the disorders. The latter create vulnerable individuals out of whom some will develop MMDs but at much higher rate to that of the general population. Significantly, similar temperamental “lopsidedness� enables many of these vulnerable individuals, if intelligent, tenacious, and curious, to be creative and contribute to our survival while some may also develop MMDs. All have a common neural-developmental origin and share characteristics in their clinical expression and pharmacological responses also expressed as mixed syndromes or alternating ones over time. Over-pruning of synaptic neurons may be considered the trigger of such occurrences
or conversely, the failure to prevent them in spite of it. The symptoms of the major mental disorders are made up of antithetical substitutes as an expression of a disturbed over-all synchronizing property of brain function for all higher faculties previously unconsidered in their modeling. The concomitant presence of psychosis is a generic common occurrence
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