63 research outputs found
The acute effects of a lunch containing capsaicin on energy and substrate utilisation, hormones, and satiety
- Author
- A Debreceni
- AJ Stunkard
- AJ Stunkard
- Astrid J. Smeets
- B Ahren
- BM Myers
- CA Maggi
- CA Maggi
- CA Maggi
- DA Schoeller
- E Brouwer
- ET Rolls
- F Kawabata
- FW Leung
- H Fukuda
- HE Raybould
- JA Harris
- KR Westerterp
- M Yoshioka
- M Yoshioka
- Margriet S. Westerterp-Plantenga
- MP Lejeune
- MS Westerterp-Plantenga
- PF Schoffelen
- RY Chen
- S Giuliani
- S Manzini
- T Kawada
- T Kawada
- TF Solanke
- TP Eldershaw
- U Holzer-Petsche
- WD Marken Lichtenbelt van
- X Fu-Cheng
- Publication venue
- D. Steinkopff-Verlag
- Publication date
- 01/01/2009
- Field of study
BACKGROUND: Addition of capsaicin to the diet has been shown to increase satiety and thermogenesis. The effects of capsaicin on ghrelin, peptide YY (PYY) and glucagon-like peptide 1 (GLP-1), in relation to changes in hunger and satiety are unknown. AIM: To test the acute effects of a lunch containing capsaicin on gut derived hormones (GLP-1, ghrelin, and PYY), energy expenditure (EE), substrate oxidation and satiety at lunch in the postprandial state. METHODS: Thirty subjects (age: 31 +/- 14 years, BMI: 23.8 +/- 2.8 kg/m(2)) were studied twice in a crossover design. After 30 min resting on a bed, resting metabolic rate was measured by a ventilated hood system. Subsequently lunch (35% of daily energy intake) was served. The two lunch conditions were: (1) lunch without capsaicin and (2) lunch with capsaicin (CAPS). The macronutrient composition (energy percentage) of the lunches was 60% carbohydrates, 10% protein and 30% fat. During 3 h after the lunch diet-induced thermogenesis was measured. Furthermore, anchored 100 mm visual analogue scales on the appetite profile were collected (t = 0, 30, 60, 120, 150, 180 and 240) and blood samples were taken for analysis of GLP-1, PYY, and ghrelin concentrations (t = 0, 45, 60, 120, and 180). RESULTS: Satiety and EE were not different after CAPS lunch as compared to the control lunch. Fifteen minutes after lunch CAPS lunch increased GLP-1 (p < 0.05) and tended to decrease ghrelin (p = 0.07) as compared to the control lunch. PYY responses were not different between the CAPS lunch and the control lunch. CONCLUSIONS: An acute lunch containing capsaicin had no effect on satiety, EE, and PYY, but increased GLP-1 and tended to decrease ghrelin
Role of Abl Kinase and the Wave2 Signaling Complex in HIV-1 Entry at a Post-Hemifusion Step
- Author
- Alexandra Trkola
- B Debreceni
- B Harmon
- BA Connolly
- Brooke Harmon
- C Beer
- C Pak
- CA Derdeyn
- D Sun
- D Yu
- EW Krueger
- G del Real
- G Melikyan
- G Melikyan
- GB Melikyan
- GJK Praefcke
- H Johannsdottir
- HM Kantarjian
- JM Lambert
- JV Melo
- K Burridge
- K Miyauchi
- L Chernomordik
- Lee Ratner
- M Cavrois
- M Viard
- M Viard
- Nancy Campbell
- NL Zandy
- NL Zandy
- R Blumenthal
- R Konig
- RP Subramanian
- S Backert
- S Etienne-Manneville
- S Gallo
- S Gallo
- S Iyengar
- S Jimenez-Baranda
- S Pontow
- S Pontow
- S Roche
- S Roumiantsev
- S Swingler
- SA Gallo
- SE Pontow
- SH Soderling
- T Zavorotinskaya
- TS Stantchev
- WC Greene
- X Wei
- Y Gao
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2010
- Field of study
Entry of human immunodeficiency virus type 1 (HIV-1) commences with binding of the envelope glycoprotein (Env) to the receptor CD4, and one of two coreceptors, CXCR4 or CCR5. Env-mediated signaling through coreceptor results in Gαq-mediated Rac activation and actin cytoskeleton rearrangements necessary for fusion. Guanine nucleotide exchange factors (GEFs) activate Rac and regulate its downstream protein effectors. In this study we show that Env-induced Rac activation is mediated by the Rac GEF Tiam-1, which associates with the adaptor protein IRSp53 to link Rac to the Wave2 complex. Rac and the tyrosine kinase Abl then activate the Wave2 complex and promote Arp2/3-dependent actin polymerization. Env-mediated cell-cell fusion, virus-cell fusion and HIV-1 infection are dependent on Tiam-1, Abl, IRSp53, Wave2, and Arp3 as shown by attenuation of fusion and infection in cells expressing siRNA targeted to these signaling components. HIV-1 Env-dependent cell-cell fusion, virus-cell fusion and infection were also inhibited by Abl kinase inhibitors, imatinib, nilotinib, and dasatinib. Treatment of cells with Abl kinase inhibitors did not affect cell viability or surface expression of CD4 and CCR5. Similar results with inhibitors and siRNAs were obtained when Env-dependent cell-cell fusion, virus-cell fusion or infection was measured, and when cell lines or primary cells were the target. Using membrane curving agents and fluorescence microscopy, we showed that inhibition of Abl kinase activity arrests fusion at the hemifusion (lipid mixing) step, suggesting a role for Abl-mediated actin remodeling in pore formation and expansion. These results suggest a potential utility of Abl kinase inhibitors to treat HIV-1 infected patients
Haemodiafiltration elicits less platelet activation compared to haemodialysis
- Author
- A Czifra
- AD Michelson
- B Canaud
- B Nagy Jr
- CL Meuwese
- E Arellano-Rodrigo
- ER Bladel Van
- Gergely Becs
- H Schmid
- Harjit Pal Bhattoa
- Ildikó Beke Debreceni
- J Cermak
- J Kappelmayer
- J Kappelmayer
- J Kappelmayer
- JA Milburn
- JT Daugirdas
- János Kappelmayer
- József Balla
- K Nagata
- K Pawlak
- M Gritters-van den Oever
- M Gritters-van den Oever
- M Kaplar
- M Schoorl
- M Schoorl
- M Schoorl
- MP Gawaz
- NM Al-Saady
- P Susantitaphong
- PG Kerr
- R Nasr
- Renáta Hudák
- S Itoh
- S Kobayashi
- T Gremmel
- Zsolt Fejes
- É Csongrádi
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Endocytic and Recycling Endosomes Modulate Cell Shape Changes and Tissue Behaviour during Morphogenesis in Drosophila
- Author
- A Jacinto
- A Jacinto
- A Jacinto
- A Jacinto
- A Martinez Arias
- A Pacquelet
- A Pelissier
- A Wada
- A-K Classen
- AK Satoh
- Alfonso Martinez Arias
- Ana Margarida Mateus
- Andreas Bergmann
- AR Skop
- B Debreceni
- B Riggs
- BG Fernández
- BH Reed
- BM Gumbiner
- C Aguado-Velasco
- CR Magie
- DD Shaye
- DP Kiehart
- DT Fox
- E Marois
- E Perret
- EE Grevengoed
- F Jankovics
- F Ulrich
- FR Maxfield
- G Dollar
- G Jékely
- GB Blanchard
- H Shimizu
- J Compagnon
- J Langevin
- JF Roeth
- JH Lipschutz
- JM Halbleib
- JW Bloor
- JY Lee
- K Sugihara
- KP Harris
- L Lanzetti
- LH Frank
- LH Hartwell
- M Fanto
- M Ren
- M Schober
- M Spaargaren
- M Takahashi
- M Zerial
- MC Jones
- MG Giansanti
- MJ Murray
- MS Bretscher
- MS Bretscher
- MS Hutson
- N Gorfinkiel
- N Gorfinkiel
- Nicole Gorfinkiel
- O Ullrich
- S Eaton
- S Hakeda-Suzuki
- S Kumari
- S Sasikumar
- S Urbé
- Sabine Schamberg
- SD Conner
- T Lecuit
- T Wucherpfennig
- TJC Harris
- TL Le
- U Tepass
- V Derrien
- YT Chen
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2010
- Field of study
During development tissue deformations are essential for the generation of organs and to provide the final form of an organism. These deformations rely on the coordination of individual cell behaviours which have their origin in the modulation of subcellular activities. Here we explore the role endocytosis and recycling on tissue deformations that occur during dorsal closure of the Drosophila embryo. During this process the AS contracts and the epidermis elongates in a coordinated fashion, leading to the closure of a discontinuity in the dorsal epidermis of the Drosophila embryo. We used dominant negative forms of Rab5 and Rab11 to monitor the impact on tissue morphogenesis of altering endocytosis and recycling at the level of single cells. We found different requirements for endocytosis (Rab5) and recycling (Rab11) in dorsal closure, furthermore we found that the two processes are differentially used in the two tissues. Endocytosis is required in the AS to remove membrane during apical constriction, but is not essential in the epidermis. Recycling is required in the AS at early stages and in the epidermis for cell elongation, suggesting a role in membrane addition during these processes. We propose that the modulation of the balance between endocytosis and recycling can regulate cellular morphology and tissue deformations during morphogenesis
Whole-genome sequencing reveals host factors underlying critical COVID-19
- Author
- Abd Elghafar M. S.
- Abdel-Aziz M.
- Abdelrazik M.
- Abdollahi H.
- Abdullah T.
- Abecasis G. R.
- Abedalthagafi M.
- Abel L.
- Abernathy C.
- Abraheem A.
- Abul-Husn N. S.
- Acquilini D.
- Adams C.
- Adams E. L.
- Adams K.
- Adamsara A.
- Adanini O.
- Adeleye O.
- Adra D.
- Afilalo J.
- Afilalo M.
- Afolabi D.
- Afrasiabi Z.
- Agasou A.
- Agrawal S.
- Aguero D.
- Ahmad N.
- Ahmadi S.
- Ahmed A.
- Ahmed C.
- Akeroyd L.
- Akhtar M. N.
- Akinkugbe O.
- Aksentijevich A.
- Al-Afghani H.
- Al-Awdah L.
- Alaamery M.
- Alahmadey Z. Z.
- Alaverdian D.
- Alavere H.
- Albader A.
- Albaiceta G. M.
- Albakri J. K.
- AlBardis H.
- Albeladi M.
- Albesher N.
- Albrich W.
- AlDhawi N.
- Aldridge J.
- Aleagha A. E.
- Alexander P.
- Alfonso J.
- Alghamdi B.
- Alghamdi J.
- Ali A.
- Ali A.
- Ali I. A. M.
- Ali S.
- Aliannejad R.
- Aljawini N.
- AlJohani S.
- Alkwai S.
- Allan A.
- Allan E.
- Allan J.
- Alldis Z.
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- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2022
- Field of study
Critical COVID-19 is caused by immune-mediated inflammatory lung injury. Host genetic variation influences the development of illness requiring critical care1 or hospitalization2–4 after infection with SARS-CoV-2. The GenOMICC (Genetics of Mortality in Critical Care) study enables the comparison of genomes from individuals who are critically ill with those of population controls to find underlying disease mechanisms. Here we use whole-genome sequencing in 7,491 critically ill individuals compared with 48,400 controls to discover and replicate 23 independent variants that significantly predispose to critical COVID-19. We identify 16 new independent associations, including variants within genes that are involved in interferon signalling (IL10RB and PLSCR1), leucocyte differentiation (BCL11A) and blood-type antigen secretor status (FUT2). Using transcriptome-wide association and colocalization to infer the effect of gene expression on disease severity, we find evidence that implicates multiple genes—including reduced expression of a membrane flippase (ATP11A), and increased expression of a mucin (MUC1)—in critical disease. Mendelian randomization provides evidence in support of causal roles for myeloid cell adhesion molecules (SELE, ICAM5 and CD209) and the coagulation factor F8, all of which are potentially druggable targets. Our results are broadly consistent with a multi-component model of COVID-19 pathophysiology, in which at least two distinct mechanisms can predispose to life-threatening disease: failure to control viral replication; or an enhanced tendency towards pulmonary inflammation and intravascular coagulation. We show that comparison between cases of critical illness and population controls is highly efficient for the detection of therapeutically relevant mechanisms of disease
Genetic mechanisms of critical illness in COVID-19.
- Author
- A A Roger Thompson
- A Abraheem
- A Agasou
- A Ahmed
- A Ali
- A Allan
- A Altabaibeh
- A Alvaro
- A Aspinwall
- A Ayers
- A Bamford
- A Barron
- A Bashyal
- A Bellini
- A Bociek
- A Botello
- A Bowes
- A Brady
- A Brayne
- A Brown
- A Brown
- A Butler
- A Campbell
- A Carter
- A Collins
- A Cowley
- A Cowton
- A Cowton
- A Cox
- A Crew
- A Dance
- A Daniel
- A Daniels
- A Dela Rosa
- A Drummond
- A Durie
- A E Heron
- A Easthope
- A Easthope
- A Evans
- A Fofano
- A Gales
- A Ganesan
- A Gardner
- A Garg
- A Gherman
- A Gordon
- A Gratrix
- A Gulati
- A Gupta
- A Haigh
- A Haldeos
- A Harrison
- A Harvey
- A Hayes
- A Higham
- A Higham
- A Hilldrith
- A Holden
- A Hormis
- A Hutcheon
- A Javaid
- A Joseph
- A Kaliappan
- A Katary
- A Kay
- A Kayani
- A Kent
- A Kirkby
- A Knight
- A Kubisz-Pudelko
- A Kuravi
- A Lewis
- A Loveridge
- A Lyle
- A Mayer
- A McAlpine
- A McCarthy
- A McGregor
- A McGregor
- A Meikle
- A Mitchell
- A Mitra
- A Morris
- A Morrison
- A Naranjo
- A Nicholson
- A Nicholson
- A Nilsson
- A Noakes
- A Patel
- A Pickard
- A Poole
- A Price
- A Puxty
- A Quinn
- A Quinn
- A Raithatha
- A Rattray
- A Reddy
- A Reed
- A Reyes
- A Rose
- A Rose
- A Rostron
- A Roy
- A Roynon-Reed
- A S Raj
- A Salberg
- A Sanderson
- A Serrano-Ruiz
- A Solesbury
- A Sukha
- A Swain
- A Tariq
- A Thomas
- A Thomas
- A Todd
- A Tomas
- A Tridente
- A Tucci
- A Turnbull
- A Uriel
- A Ustianowski
- A Vochin
- A Vuylsteke
- A Waite
- A Walden
- A Whileman
- A Wilkinson
- A Williams
- A Williams
- A Wilson
- A Zak
- A Zaki
- Achille Iolascon
- Adam Auton
- Adam Brown
- Agnese Verzuri
- Agostino Ognibene
- Agostino Riva
- Ailsa Golightly
- Alan Maclean
- Alessandra
- Alessandra Stella
- Alessandra Vergori
- Alessia Giorli
- Alexander J Mentzer
- Alice Donati
- Alison M Meynert
- Alistair Nichol
- Ana da Silva Filipe
- Andrea Antinori
- Andrea Cossarizza
- Andrea Ganna
- Andrea Tommasi
- Andrew Rambaut
- Andrew Stenhouse
- Andy Law
- Anjali J Shastri
- Anna Canaccini
- Anna Maria Pinto
- Annarita Giliberti
- Annemarie B Docherty
- Antonella D'Arminio Monforte
- Antonia Ying Wai Ho
- Antonio Di Biagio
- Antony Symons
- Arianna Emiliozzi
- Arianna Gabrieli
- Audrey Coutts
- B Anwar
- B Attwood
- B Baines
- B Blackledge
- B Borislavova
- B Chandler
- B Charles
- B Creagh-Brown
- B David
- B Deacon
- B Deacon
- B Digby
- B Faulkner
- B Fuller
- B Gumbrill
- B Gurung
- B Hadebe
- B Hairsine
- B Hopkins
- B Johnston
- B Lenagh
- B Masunda
- B Ogg
- B Patel
- B Player
- B Purewal
- B Scholefield
- B Shelley
- B Sloan
- B Thomas
- B Wadams
- B Welsh
- B Wilkinson
- B Winter-Goodwin
- B Yates
- Baillie J Kenneth
- Barbara Rossetti
- Beale Rupert
- Beatrice Alex
- Benjamin Bach
- Bretherick Andrew D
- Bruno Frediani
- C Adams
- C Ahmed
- C Almaden-Boyle
- C Ashbrook-Raby
- C Basikolo
- C Battle
- C Beazley
- C Beith
- C Borra
- C Brandwood
- C Brantwood
- C Burnett
- C Castro Delgado
- C Clark
- C Clulow
- C Collins
- C Cruz
- C Davis
- C Demetriou
- C Dennis
- C Dexter
- C Downes
- C Dunmore
- C Eastgate
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- C Finch
- C Gibson
- C Gorman
- C Hays
- C Hewitt
- C Holl
- C Howcroft
- C Humphrey
- C Jackson
- C Jardine
- C Jennings
- C Jones
- C Kaye
- C Lynch
- C Lyons
- C McCulloch
- C McParland
- C McParland
- C Murphy
- C Parmar
- C Pawley
- C Phillips
- C Phillips
- C Pothecary
- C Pothecary
- C Prendergast
- C Price
- C Rishton
- C Sell
- C Shovelton
- C Sicat
- C Stuart
- C Summers
- C Swan
- C Thompson
- C Thorpe
- C Tibke
- C Tierney
- C Vigurs
- C Wells
- C Whitton
- C Whyte
- C Wrey Brown
- Cameron J Fairfield
- Carmelo Piscopo
- Carmen Marciano
- Caroline Abernathy
- Carrol Gamble
- Caterina Lo Rizzo
- Catherine A Shaw
- Catherine H Weldon
- Caulfield Mark
- Ceilia Boz
- Cesira Nencioni
- Chelsea Ye
- Chiara Fallerini
- Chiara Gabbi
- Chiara Spertilli
- Chloe Donohue
- Chris Ponting
- Christoper A Green
- Cinthia E Jannes
- Clare Jackson
- Clohisey Sara
- Cristina Mussini
- D Antcliffe
- D Bakthavatsalam
- D Banach
- D Baptista
- D Bell
- D Branney
- D Brealey
- D Brealey
- D Butcher
- D Butcher
- D Childs
- D Clarke
- D Collier
- D Collier
- D Cristiano
- D Dawson
- D Donaldson
- D Duffin
- D Fottrell-Gould
- D Golden
- D Griffin
- D Hamilton
- D Hawcutt
- D Hope
- D Horner
- D Kallon
- D Kaye
- D Kelly
- D Loader
- D Lomas
- D Martin
- D McGlynn
- D McLaughlanv
- D Melia
- D Menzies
- D Mullan
- D Pogson
- D Potla
- D Potoczna
- D Purohit
- D Raynard
- D Salutous
- D Salutous
- D Scaletta
- D Shaw
- D Skinner
- D Smyth
- D Strachan
- D Tetla
- D Thornton
- D Trodd
- D Vedage
- D Walker
- D Warren
- D Williams
- D Wood
- D Wright
- Daniela Francisci
- Daniella Coker
- Danilo Tacconi
- David A van Heel
- David Bennet
- David L Robertson
- David Stuart
- Dawn Law
- Debby Bogaert
- Derek Murphy
- Domenico A Coviello
- E Abaleke
- E Allan
- E Anastasescu
- E Andrews
- E Apetri
- E B Jude
- E Beech
- E Beranova
- E Bevan
- E Black
- E Brinkworth
- E Collins
- E Combes
- E Davies
- E Dobson
- E Dooks
- E Fisher
- E Gendall
- E Goff
- E Goodwin
- E Gordon
- E Heeney
- E Horsley
- E Hughes
- E Johnson
- E Knights
- E Lee
- E London
- E Maccacari
- E Massey
- E McKenna
- E Meadows
- E Moncur
- E Morino
- E Mwaura
- E Peasgood
- E Perkins
- E Radford
- E Raith
- E Raith
- E Salciute
- E Smith
- E Smithson
- E Stoddard
- E Stones
- E Thomas
- E Tilney
- E Timlick
- E Treus Gude
- E Virgilio
- E Watson
- E Wilby
- Edoardo Conticini
- Egle Saviciute
- Elena Bargagli
- Elena Desanctis
- Elisa Benetti
- Elisa Frullanti
- Elisabetta Menatti
- Elisabetta Schiaroli
- Ellie Mcmaster
- Emma C Thomson
- Emmanuelle Marques
- Enrico Martinelli
- Esther Duncan
- Esther Merlini
- Ewen M Harrison
- F Anderson
- F Auld
- F Bibi
- F Davies
- F Farquhar
- F Fisher
- F Hurford
- F Kibutu
- F McNeela
- F Moore
- F Nasir
- F Trim
- F Wakinshaw
- F Williams
- Fabio Lena
- Fawkes Angie
- Federico Anedda
- Federico Franchi
- Ferdinando Giannattasio
- Filip Taneski
- Filippo Aucella
- Filippo Biscarini
- Floriana Valentino
- Fourman Max Head
- Francesca Andretta
- Francesca Fava
- Francesca Gatti
- Francesca Mari
- Francesca Montagnani
- Francesco Castelli
- Francesco Raimondi
- Furniss James
- G Andrew
- G Arbane
- G Bell
- G Bercades
- G Croston
- G Debreceni
- G Durrant
- G Hambrook
- G Hobden
- G Houston
- G Hughes
- G Jones
- G Lubimbi
- G Maloney
- G Martir
- G Millen
- G Mills
- G O'Connor
- G Padden
- G Randell
- G Sadera
- G Seddon
- G Sera Howe
- G Sloane
- G Subramanian
- G Tsinaslanidis
- G Turner
- G Watson
- G Wray
- Gabriella Coiro
- Gabriella Doddato
- Gail Carson
- Gardar Sveinbjornsson
- Gary Leeming
- Genni Spargi
- Georgios Pollakis
- Giacomo Zanelli
- Gian Piero Caldarelli
- Giancarlo Bosio
- Giuseppe Fiorentino
- Giuseppe Merla
- Gountouna Elvina
- Graham S Cooke
- Griffiths Fiona
- Grimes Graeme
- H Bancroft
- H Bates
- H Belfield
- H Blackman
- H Blakemore
- H Brooke
- H Button
- H Coles
- H Curgenven
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- H Jeffrey
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- H Sainsbury
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- H T-Michael
- H Tench
- H Tiveran
- H Turner
- H Whittle
- H Willis
- H Wood
- Haley Chris
- Hanning Mal
- Harrison David
- Hayley Hardwick
- Hayward Caroline
- Helen Szoor-McElhinney
- Hinds Charles
- Ho Antonia
- Horby Peter
- I Ali Mohamed Ali
- I Balagosa
- I Birkinshaw
- I Burn
- I Chadbourn
- I D Clement
- I Edmond
- I Frost
- I Hass
- I Hussain
- I Lancoma-Malcolm
- I Leadbitter
- I Nagra
- I Otahal
- I Otahal
- I Turner-Bone
- I Welters
- I White
- I Wynter
- Ilaria Meloni
- Immacolata Andolfo
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- Luca Cantarini
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- Marco Sita
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- Mari E K Niemi
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- Marie Connor
- Mario Capasso
- Mario U Mondelli
- Marta Corridi
- Maslove David
- Massimiliano Fabbiani
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- Massimo Palmarini
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- Matteo Bassetti
- Matteo Della Monica
- Matteo Siano
- Mattia Cordioli
- Maurizio Sanarico
- Maurizio Sanguinetti
- Maurizio Spagnesi
- McAuley Danny
- Meera Chand
- Meynert Alison
- Michelle Girvan
- Millar Jonathan
- Mirella Bruttini
- Miriana D'Alessandro
- Montgomery Hugh
- Moore Shona C
- Moutsianas Loukas
- Murphy Lee
- Murray Wham
- N Ahmad
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- N Michalak-Glinska
- N Motherwell
- N Muchenje
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- Nichol Alistair
- Nicholas Parkinson
- Nicholas Price
- Nicky Day
- Nicola Picchiotti
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- Oosthuyzen Wilna
- Openshaw Peter J M
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- P Williams
- Pairo-Castineira Erola
- Pamela Raggi
- Paola Magro
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- Parkinson Nick
- Pasko Dorota
- Patrick Sulem
- Patrizia Zucchi
- Paul Finernan
- Paul Klenerman
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- Pier Giorgio Scotton
- Pierpaolo Parravicini
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- Ponting Chris P
- Porteous David J
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- R Herdman-Grant
- R Howle
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- R Jha
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- Raffaele Scala
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- Renzo Scaggiante
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- Richmond Anne
- Riinu Pius
- Rita Perna
- Roberta Russo
- Ross Hendry
- Rossella Tita
- Rowan Kathy
- Russell Clark D
- Ruth Armstrong
- Ruth Lyons
- Ryan S Thwaites
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- S Anderson
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- S Campos
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- S Clamp
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- S Lee
- S Leggett
- S MacFadyen
- S Manna
- S Marriott
- S Masuko
- S McIvor
- S McKechnie
- S Melling
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- S Mepham
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- S Monnery
- S Moore
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- S Moultrie
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- S Mulla
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- S Sousa Arias
- S Spencer
- S Srikaran
- S Stone
- S Swann
- S Tilbey
- S Turki
- S Turney
- S Twiss
- S Wadd
- S Whiteley
- S Williams
- S Wood
- S Yussuf
- Sabino Scolletta
- Samreen Ijaz
- Sandro Mancarella
- Sandro Panese
- Sara Amitrano
- Sara McDonald
- Sarah Law
- Sarah McCafferty
- Saverio Giuseppe Parisi
- Saye Khoo
- Scott Richard
- Semple Malcolm G
- Serafina Valente
- Serena Ludovisi
- Sergio Daga
- Seán Keating
- Shankar-Hari Manu
- Shen Xia
- Shih Barbara
- Shiranee Sriskandan
- Shona C Moore
- Silvia Cappelli
- Simona Marcantonio
- Simone Furini
- Sophie Halpin
- Sophie Venturelli
- Stefania Mantovani
- Stefano Baratti
- Stefano Ceri
- Stefano Rusconi
- Stella Aslibekyan
- Stephanie Roberts
- Stephen R Knight
- Summers Charlotte
- Susanna Croci
- Susanna Guerrini
- T Anderson
- T Arden
- T Baird
- T Behan
- T Bemand
- T Cosier
- T Coventry
- T Duncan
- T Felton
- T Geary
- T Hazleton
- T Joefield
- T Kannan
- T Lawton
- T Marsden
- T Martin
- T McHugh
- T Newman
- T Nortcliffe
- T O Jones
- T Pogreban
- T Rees
- T Samakomva
- T Smith
- T Smith
- T Szakmany
- T Varghes
- T Williams
- T Wood
- Tammy Gilchrist
- Tenesa Albert
- Teresa Filshtein-Sonmez
- Thomas M Drake
- Thushan de Silva
- Tim Walsh
- Tom Fletcher
- Tom Solomon
- Tony Wackett
- Trevor Paterson
- Tullio Trotta
- Turtle Lance
- U Poultney
- V Amin
- V Anumakonda
- V Bastion
- V Cannons
- V Crickmore
- V Gopal
- V Irvine
- V Kasipandian
- V Krishnamurthy
- V Lake
- V Linnett
- V Martinson
- V Nagarajan
- V Page
- V Parris
- V Parris
- V Pristopan
- V Ratnam
- V Rivers
- V Sarathy
- V Thomas
- V Thwaites
- V Tuckey
- V Turner
- V Wagstaff
- V Waugh
- Valentina Anemoli
- Vanessa Sancho-Shimizu
- Victoria Shaw
- Vitart Veronique
- W Harrison
- W Khaliq
- W Khaliq
- W McCormick
- W Woodyatt
- Walker Susan
- Walsh Timothy
- Wang Bo
- Wei Shen Lim
- Wendy S Barclay
- William A Paxton
- William Greenhalf
- Wilson James F
- Wrobel Nicola
- Wu Yang
- X Qiu
- Y Baird
- Y Choudhury
- Y Hussain
- Y Jackson
- Y Thirlwall
- Yang Jian
- Yang Zhijian
- Z Alldis
- Z Belagodu
- Z Bradshaw
- Z Coton
- Z Daly
- Z Farzad
- Z Fernandez
- Z Garland
- Z Maqsood
- Z Omar
- Z Prime
- Z Scott
- Zechner Marie
- Zhai Ranran
- Zheng Chenqing
- Publication venue
- Nature
- Publication date
- 01/01/2020
- Field of study
Host-mediated lung inflammation is present1, and drives mortality2, in the critical illness caused by coronavirus disease 2019 (COVID-19). Host genetic variants associated with critical illness may identify mechanistic targets for therapeutic development3. Here we report the results of the GenOMICC (Genetics Of Mortality In Critical Care) genome-wide association study in 2,244 critically ill patients with COVID-19 from 208 UK intensive care units. We have identified and replicated the following new genome-wide significant associations: on chromosome 12q24.13 (rs10735079, P = 1.65 × 10-8) in a gene cluster that encodes antiviral restriction enzyme activators (OAS1, OAS2 and OAS3); on chromosome 19p13.2 (rs74956615, P = 2.3 × 10-8) near the gene that encodes tyrosine kinase 2 (TYK2); on chromosome 19p13.3 (rs2109069, P = 3.98 ×  10-12) within the gene that encodes dipeptidyl peptidase 9 (DPP9); and on chromosome 21q22.1 (rs2236757, P = 4.99 × 10-8) in the interferon receptor gene IFNAR2. We identified potential targets for repurposing of licensed medications: using Mendelian randomization, we found evidence that low expression of IFNAR2, or high expression of TYK2, are associated with life-threatening disease; and transcriptome-wide association in lung tissue revealed that high expression of the monocyte-macrophage chemotactic receptor CCR2 is associated with severe COVID-19. Our results identify robust genetic signals relating to key host antiviral defence mechanisms and mediators of inflammatory organ damage in COVID-19. Both mechanisms may be amenable to targeted treatment with existing drugs. However, large-scale randomized clinical trials will be essential before any change to clinical practice
Effect of high- and low- frequency transcutaneous electrical nerve stimulation (TENS) on angiogenesis and wound contraction in acute excisional wounds in rat skin
- Author
- Abram SE
- Agha R
- Ansell DM
- Atalay C
- Bae SH
- Baker LL
- Baker LL
- Buonocore M
- Chen C
- Cosmo P
- Debreceni L
- Finsen L
- Forst T
- Gürgen SG
- Hochman B
- Johnson MI
- Kaada B
- Karba R
- Khalil Z
- Khalil Z
- Kloth LC
- Kutlu AK
- Li J
- Liebano RE
- Liebano RE
- Liebano RE
- Liebano RE
- Machado AFP
- Medrado ARAP
- Nnoaham KE
- Ogrin R
- Ogrin R
- Papazoglou ES
- Sebastian A
- Starkey C
- Valacchi G
- Wikström OS
- Yarboro DD
- Publication venue
- 'FapUNIFESP (SciELO)'
- Publication date
- Field of study
Epigenetic associations in relation to cardiovascular prevention and therapeutics
- Author
- A Aartsma-Rus
- A Baccarelli
- A Bedalov
- A Crescenti
- A Granger
- A Ramirez Bosca
- A Schober
- A Vogt
- AD Johnson
- AH Lichtenstein
- AJ Innes
- AK Chomistek
- B Buijsse
- B Debreceni
- B Debreceni
- B Heltweg
- B Sarria
- BC Bernardo
- BD Strahl
- BF Baker
- C Abi Khalil
- C Gebhard
- C Heiss
- C Heiss
- C Napoli
- C Napoli
- C Pantos
- C Schiano
- C Schleithoff
- CA Klattenhoff
- CC Matouk
- CD Mathers
- CE Burd
- CM Grozinger
- CM Ramirez
- CR Harper
- CW Lee
- D Lloyd-Jones
- D Mozaffarian
- D Rein
- D Wang
- D Won
- DA Bell
- DE Handy
- DM Patrick
- DS Wald
- E Galkina
- E Lonn
- E Rooij van
- E Rooij van
- E Zhang
- EE Morrisey
- EJ Hennessy
- EN Frankel
- ER Miller 3rd
- F Cacciapuoti
- F Tabet
- F Visioli
- FH Nazki
- G Albertoni
- G Arunachalam
- G Bora-Tatar
- G Rimbach
- GH Kim
- H Kuhl
- H McNulty
- H Okamoto
- H Ota
- H Ryu
- H Schroeter
- H Suzuki
- H Taniura
- HL Li
- HL Liu
- HM Stauss
- HS Soifer
- HT Yang
- HY Seok
- I Gouni-Berthold
- I Janszky
- I Mattagajasingh
- I Rahman
- J Balzer
- J Chiu
- J Kim
- J Mursu
- J Zhang
- JA Broderick
- JA Vita
- JD Spence
- JE Fish
- JE Fish
- JE Fish
- JG Herman
- JH Chan
- JH Choi
- JJ Ho
- JJ Ho
- JL Medina-Franco
- JL Rinn
- JN Wilcox
- JT Willerson
- JY Ooi
- K Balasubramanyam
- K Ried
- KB Soni
- KH Bonaa
- KJ Murphy
- KJ Rayner
- KJ Rayner
- KK Meja
- KT Howitz
- L Cui
- L Folkersen
- L Hajra
- L Hooper
- L Hooper
- L Jia
- L Rossig
- L Zhang
- LJ Ignarro
- LM Holdt
- LM Holdt
- LM Holdt
- LP Lim
- LP McQuillan
- M Ciaccio
- M Ehrlich
- M Sun
- M Vinod
- MD Prado-Uribe
- MF Corsten
- MH Pan
- MM Martin
- MP Turunen
- MS Cunnington
- MS Ebert
- MU Kaikkonen
- MV Latronico
- N Engel
- N Halfon
- N Khan
- N Khan
- N Osakabe
- N Sato
- N Singh
- N Tsuda
- ND Fisher
- O Jarinova
- P Caruso
- P Grote
- P Libby
- PJ Barnes
- PJ Turgeon
- PM Kris-Etherton
- R Barres
- R Corti
- R Feil
- R Menghini
- R Papait
- R Visone
- R Wang
- R Ziesche
- RC Friedman
- RC Kaplan
- RL Montgomery
- RR Holt
- S Baba
- S Baba
- S Bekkering
- S Chen
- S Dimmeler
- S Fichtlscherer
- S Friso
- S Kar
- S Rajan
- S Reuter
- S Shishodia
- S Singh
- S Udali
- SA Hannou
- SJ Duthie
- SM Krishna
- SP Guay
- SP Romaine
- T Morimoto
- T Thum
- T Wallerath
- TA Harris
- TG Hullinger
- TPP Vanhaecke
- VG Athyros
- W Wongcharoen
- W Yu
- X Sun
- XL Liu
- Y Ben-Shlomo
- Y Chan
- Y Chen
- Y D’Alessandra
- Y Huang
- Y Liu
- Y Takahashi
- Y Zhao
- Y Zhao
- Z Faridi
- Z Liu
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Whole-genome sequencing reveals host factors underlying critical COVID-19
- Author
- Abd Elghafar M.S.
- Abdel-Aziz M.
- Abdelrazik M.
- Abdollahi H.
- Abdullah T.
- Abecasis G.R.
- Abecasis G.R.
- Abedalthagafi M.
- Abel L.
- Abernathy C.
- Abraheem A.
- Abul-Husn N.S.
- Acquilini D.
- Adams C.
- Adams E.L.
- Adams K.
- Adamsara A.
- Adanini O.
- Adeleye O.
- Adra D.
- Afilalo J.
- Afilalo M.
- Afolabi D.
- Afrasiabi Z.
- Agasou A.
- Agrawal S.
- Agüero D.
- Ahmad N.
- Ahmadi S.
- Ahmed A.
- Ahmed C.
- Akeroyd L.
- Akhtar M.N.
- Akinkugbe O.
- Aksentijevich A.
- Al Harthi F.
- Al Mutairi A.
- Al-Afghani H.
- Al-Awdah L.
- Alaamery M.
- Alahmadey Z.Z.
- Alaverdian D.
- Alavere H.
- Albader A.
- Albaiceta G.M.
- Albakri J.K.
- AlBardis H.
- Albeladi M.
- Albesher N.
- Albrich W.
- AlDhawi N.
- Aldridge J.
- Aleagha A.E.
- Alexander P.
- Alfonso J.
- Alghamdi B.
- Alghamdi J.
- Ali A.
- Ali A.
- Ali I.A.M.
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- Vulesevic B.
- Vuylsteke A.
- Wackett T.
- Wadams B.
- Waddington N.
- Wagstaff V.
- Wain L.V.
- Waite A.A.C.
- Wakefield P.
- Wakinshaw F.
- Waldron J.
- Walker D.
- Walker R.
- Walker S.
- Walker S.
- Walker S.
- Wall A.
- Walsh T.
- Walsh T.
- Walters R.K.
- Walton O.
- Wang B.
- Wang J.
- Wang X.
- Wanying Z.
- Ward G.
- Ward L.
- Ward M.
- Warden S.
- Warmerdam R.
- Warren D.
- Wassall H.
- Wasson C.
- Watkins C.
- Watson E.
- Watson G.
- Watson J.
- Watters M.
- Waugh V.
- Weaver J.
- Webster A.
- Webster D.
- Webster K.
- Wei S.
- Weiss S.T.
- Weldon C.H.
- Wells C.
- Welters I.D.
- Weston H.
- Whileman A.
- Whitbread J.
- White D.
- White I.
- White K.
- White M.
- White N.
- White N.
- Whiteley S.
- Whiteside J.
- Whittle H.
- Whitton C.
- Whyte C.
- Wicks S.J.
- Wilby E.
- Wilcox L.
- Wilcox R.
- Wild H.
- Wild L.
- Wild L.
- Wilding L.
- Wiles M.
- Wilkins J.
- Wilkinson A.
- Wilkinson B.
- Willer C.
- Williams A.
- Williams A.
- Williams A.
- Williams A.T.
- Williams D.
- Williams E.
- Williams F.
- Williams G.
- Williams H.
- Williams K.
- Williams M.
- Williams P.
- Williams S.
- Williams T.
- Willis C.
- Willis H.
- Wills M.
- Willson J.
- Wilson A.
- Wilson D.J.
- Wilson J.
- Wilson J.F.
- Wilson J.F.
- Winnard S.
- Winter-Goodwin B.
- Wolf-Roberts R.
- Wolford B.
- Wong J.
- Wood D.
- Wood H.-L.
- Wood S.
- Wood T.
- Woodford C.
- Woodruff M.
- Woods L.
- Woodyatt W.
- Woolley A.E.
- Worner R.
- Worsley L.
- Wray G.
- Wren L.
- Wrey Brown C.
- Wright D.
- Wright J.
- Wright M.
- Wrobel N.
- Wu Y.
- Wulandari R.
- Wyllie D.H.
- Wynter I.
- Xavier K.
- Xue X.
- Yadav A.
- Yang J.
- Yassen A.M.
- Yates B.
- Ye C.
- Yun N.
- Zainy T.
- Zak A.
- Zaki A.
- Zamikula J.
- Zanella I.
- Zborowski A.C.
- Zeberg H.
- Zechner M.
- Zechner M.
- Zguro K.
- Zhang M.
- Zhao J.H.
- Zheng C.
- Zhou W.
- Zitter L.
- Zlatopolsky M.
- Zongo O.
- Zucchi P.
- Zyndorf M.
- Zöllner S.
- Ã…svold B.O.
- Publication venue
- Springer Science and Business Media LLC
- Publication date
- 07/07/2022
- Field of study
Critical COVID-19 is caused by immune-mediated inflammatory lung injury. Host genetic variation influences the development of illness requiring critical care1 or hospitalization2,3,4 after infection with SARS-CoV-2. The GenOMICC (Genetics of Mortality in Critical Care) study enables the comparison of genomes from individuals who are critically ill with those of population controls to find underlying disease mechanisms. Here we use whole-genome sequencing in 7,491 critically ill individuals compared with 48,400 controls to discover and replicate 23 independent variants that significantly predispose to critical COVID-19. We identify 16 new independent associations, including variants within genes that are involved in interferon signalling (IL10RB and PLSCR1), leucocyte differentiation (BCL11A) and blood-type antigen secretor status (FUT2). Using transcriptome-wide association and colocalization to infer the effect of gene expression on disease severity, we find evidence that implicates multiple genes—including reduced expression of a membrane flippase (ATP11A), and increased expression of a mucin (MUC1)—in critical disease. Mendelian randomization provides evidence in support of causal roles for myeloid cell adhesion molecules (SELE, ICAM5 and CD209) and the coagulation factor F8, all of which are potentially druggable targets. Our results are broadly consistent with a multi-component model of COVID-19 pathophysiology, in which at least two distinct mechanisms can predispose to life-threatening disease: failure to control viral replication; or an enhanced tendency towards pulmonary inflammation and intravascular coagulation. We show that comparison between cases of critical illness and population controls is highly efficient for the detection of therapeutically relevant mechanisms of disease
Cardiopoietic cell therapy for advanced ischemic heart failure: results at 39 weeks of the prospective, randomized, double blind, sham-controlled CHART-1 clinical trial
- Author
- A. Aminian
- A. Ancion
- A. Anesini
- A. Asif
- A. Bogdanovic
- A. Bogdanovic
- A. Bogdanovic
- A. Casado
- A. Csepregi
- A. Cziraki
- A. Darago
- A. Djokovic
- A. Djordjevic Dikic
- A. Dominguez
- A. Fernandez
- A. Frankiewicz
- A. Frimerman
- A. Gambaro
- A. Gelei
- A. Goldberg
- A. Hernandez
- A. Ka\u2dbdzielski
- A. Korda
- A. Kosztin
- A. Kovacs
- A. Mazurek
- A. Mijovic
- A. Mu~noz
- A. Nagy
- A. Nemetz
- A. Ochala
- A. Radinov
- A. Ristic Andjelkov
- A. Rossi
- A. Ruck
- A. Shotan
- A. Shturman
- A. Vazan
- A. Yordanov
- Alexandre Bertrand
- Alexandrovska University Hospital
- Andreka . M. Atanasova Hungary: Gottsegen Gyo\ua8 rgy Orszagos Kardiologiai Inte\ub4zet: P.
- Andreka Peter
- AOUI Verona \u2013 Borgo Trento Hospital: C. Vassanelli
- Arandjelovic Aleksandra
- Atar Shaul
- AZ Glorieux: A. Heyse
- B. El Nakadi
- B. Kittka
- B. Magyari
- B. Muk
- B. Obrenovic Kircanski
- B. Polgar
- Bartunek Jozef
- Barzilai Medical Center: A. Katz
- Bayes Genis Antoni
- Behfar Atta
- Beleslin Branko
- Bieganski Hospital: J. Kasprzak
- C. Bergamini
- C. Biagioni
- C. Botev
- C. Camporini
- C. Crljenica
- C. Doyen
- C. Jenei
- C. Labata
- C. Lombardi
- C. Monti
- C. Pascual
- C. Plymen
- C. Silva
- C. Sylve\ub4n
- Cardiology Clinic: A. Arandjelovic
- Cardiology Clinic: B. Beleslin
- Cardiology Clinic: P. Seferovic
- Cardiology Department: S. Radovanovic
- CHU Mont Godinne UCL: A. Guedes
- City Hospital Clinic Cardiology Center: I. Petrov
- Clinic of Emergency Internal Medicine
- Clinical Center of Kragujevac: N. Jagic
- Clinical Centre of Serbia
- Clinical Centre of Serbia
- Clinical Hospital Center Bezanijska Kosa
- Cotter Gad
- D. Ljubinka Spain: Hospital Universitario Germans Trias I. Pujol: A. Bayes Genis
- D. S. Blondheim
- D. Boljevic
- D. Ciecwierz
- D. Cotra
- D. Dalla Mura
- D. Fiutowska
- D. Haval Qawoq
- D. Markovic
- D. Mcloman
- D. Nenov
- D. Nikolic
- D. Orlic
- D. Russo
- D. Saleem
- D. Simeunovic
- D. Su\ua8rder
- D. Trifunovic
- Davison Beth A
- Debreceni Egyetem Orvos e\ub4s Ege\ub4szse\ub4gtudomanyi Centrum Altalanos Orvostudomanyi Kar Kardiologia Inte\ub4zet: I. Edes
- Dolatabadi Dariouch
- Domaine Universitaire du Sart Tilman: V. Legrand
- E. Baudoux
- E. Chiari
- E. De Vuyst
- E. Dimitrov
- E. Drasar
- E. Ferrer
- E. Gara
- E. Gaszewska Zurek
- E. Gunyeli
- E. Hadzhiev
- E. Pasotti
- E. Zatarain
- Edes Istvan
- El Nakadi Badih
- F. Carrasco
- F. Jouhra
- F. Van Durme
- Fernandez Aviles Francisco
- Filippatos Gerasimos S
- G. Dolci United Kingdom: Kings College Hospital: J. Hill
- G. Amin Youssef
- G. Dimitrov
- G. Foldes
- G. Fontos
- G. Kerecsen
- G. Loncar
- G. Loughlin
- G. Misic
- G. Pesarini Poland: Jagiellonian University Department of Cardiac
- G. Ponti
- G. Raczak
- G. Szombath
- G. Tzvetkova
- G. Uzonyi
- G. Vajda
- Gelev Valeri L
- Gersh Bernard J
- Gornosla\u2dbskie Centrum Medyczne Sla\u2dbskie j. Akademii Medycznej: W. Wojakowski
- Gruchala Marcin
- Gu\ue9d\ue8s Antoine
- H. Alizadeh
- H. Stoyanov
- Hajjar Roger J
- Halabi Majdi
- Henry Timothy D
- Hernandez Garcia Jose Maria
- Heyndrickx Guy R
- Heyse Alex
- Hillel Yaffe Medical Center: M. Shochat
- Ho\u2c6pital Civil de Charleroi: D. Dolatabadi
- Horvath Ivan G
- Hospital Clinico San Carlos: P. Jimenez Quevedo
- Hospital Clinico Universitario Virgen de la Victoria: J. M. Hernandez Garcia
- Hospital General Universitario Gregorio Mara~non: F. Fernandez Aviles
- I. Amine
- I. Blankoff
- I. Bonadei
- I. Garvanski
- I. Marciniak
- I. Milinkovic
- I. Nedeljkovic
- I. Orlov
- I. Petrova
- I. Rosenfeld
- I. Stopczynska
- I. Tasheva Dimitrova
- I. Webb
- I. Zheleva Kichukova
- J. J. Gomez deDiego
- J. M. Garcia Pinilla
- J. Anguita
- J. Bartunek
- J. Bellwon
- J. Bermejo
- J. Byrne
- J. Ciosek
- J. Fabian
- J. Fo\ua8rstedt Switzerland: CardioCentro Ticino
- J. Jafari
- J. Lalmand
- J. Podolec
- J. Ruiz
- J. Saric
- J. Skopal
- J. Stepanovic
- J. Tresnjak
- J. Verschuere
- J. Wdowczyk
- J. Wroblewska
- Jagic Nikola
- Jimenez Quevedo Pilar
- K. Aehab
- K. Karamfiloff
- K. Kichukov
- K. Leblanc
- K. Mosakowska
- K. Zmudka
- Kasprzak Jaroslaw D
- Katz Amos
- Kiss . M.
- Krampera Mauro
- L. Arobov Italy: A. O. Spedali Civili di Brescia: M. Metra
- L. Perez de Isla Sweden: Karolinska University Hospital: A. Mansson Broberg
- L. Akria
- L. Balogh
- L. Chiumiento
- L. Davin
- L. Drabik
- L. Gabriel
- L. Geller
- L. Inama
- L. Nikitasov
- L. Tekieli
- L. Tomovic
- L. Turchetto
- L. Vladimirova
- L. Zanolla
- Legrand Victor
- M. A. Cicoria
- M. E. Ferna ndez Santos
- M. P. Queipo de Llano
- M. Andre\ub4
- M. Angelova
- M. Banovic
- M. Banovic
- M. Batlle
- M. Biggiogero
- M. Brezins
- M. Chmielecki
- M. Dobric
- M. Dupont
- M. Dzieciuch Rojek
- M. Dzierzak
- M. Gujic
- M. Janssens
- M. Jimenez Navarro
- M. Kilimnik
- M. Kozlowski
- M. Magatelli
- M. Markiewicz
- M. Markiewicz Serbia: Clinical Hospital Center Zvezdara
- M. Mielczarek
- M. Minchev
- M. Moccetti
- M. Mombelli
- M. Nedeljkovic
- M. Olszowska
- M. Plewka
- M. Rossi
- M. Rozmyslowska
- M. Rynkiewicz
- M. Sobczak
- M. Sreckovic
- M. Tarnawska
- M. Tasic
- M. Taszner
- M. Tesic
- M. Wraga
- M. Zielinska
- Merkely Bela
- Metra Marco
- MH Ege\ub4szse\ub4gu\ua8gyi Ko\ua8zpont Kardiologiai Osztaly: N. Nyolczas
- Military Medical Academy: S. Obradovic
- Moccetti Tiziano
- Musialek Piotr
- N. Cvetinovic
- N. Dally
- N. Djenic
- N. Sekularac
- N. Sydo
- N. Vallejo
- N. Zornic
- Nazareth Hospital EMMS: M. Omory N. Kogan Nielsen
- Nyolczas No\ue9mi
- O. Gach
- O. Markovic
- O. Petrovic
- O. Radev
- O. Rodriguez Leor
- O. Sofer
- Obradovic Slobodan
- OLV Ziekenhuis Aalst \u2013 Cardiologie: M. Vanderheyden
- P. Meeus Bulgaria: Tokuda Hospital Sofia: V. Gelev
- P. Blomberg
- P. Djurdjevic
- P. Dubois
- P. Farkas
- P. Gurnet
- P. Lancellotti
- P. Lipiec
- P. Podolec
- P. Polomski
- P. Rubis
- P. Weijner Mik
- Pe\ub4csi Tudomanyegyetem Klinikai Ko\ua8zpont Sz\u131vgyogyaszati Klinika: I. Horvath
- Penicka . M.
- Petrov Ivo
- Plazak . W.
- Povsic Thomas J
- R. A. Gonzalez
- R. Dulgheru
- R. Dworakowski
- R. Faludi
- R. Guerra
- R. Krecki
- R. Melamed
- R. Murzilli
- R. Nadolna
- R. Parapunova
- R. Sanz Ruiz
- R. Tarnovska Kadreva
- R. Winter
- Radovanovic Slavica
- Ribichini Flavio
- Ribichini Flavio Luciano
- S. 0ordevic
- S. Burakowski
- S. Dworowy
- S. Gergely
- S. Hinic
- S. Maspoli
- S. Meisel
- S. Polledri
- S. Sardovski
- S. Seldrum
- S. Stamenkovic
- S. Verstreken
- S. Vukotic
- S. Zivkovic
- Sanz Ruiz Ricardo
- Seferovic Petar M
- Semmelweis Egyetem Varosmajori Sz\u131v e\ub4s Ergyogyaszati Klinika: B. Merkely
- Seron Aymeric
- Sherman Warren
- Shochat Michael K
- sites Belgium: Ziekenhuis Oost Limburg: J. Dens
- Sofia: D. Vassilev
- Stough Wendy Gattis
- Switzerland: T. Moccetti
- T. Borsanyi
- T. Drogenikov
- T. Fulop
- T. Jadczyk
- T. Levinas
- T. Mcdonagh
- T. Plaev
- T. Rechcinski
- Teerlink John R
- Tendera Michal
- Terzic Andre
- U. Sutor
- Uniwersyteckie Centrum Kliniczne: M. Gruchala
- V. Bisenic
- V. Carubelli
- V. D\u2019Orio
- V. Giga
- V. Lazzarini
- V. Locicero
- V. Malenkovic
- V. Miloradovic
- V. Potter
- V. Vuckovic
- V. Vukcevic
- Vanderheyden Marc
- Vascular Diseases at John Paul II Hospital in Krakow: P. Musialek
- W. Cybulski
- W. Mullens
- W. Orczyk
- W. Wanha
- W. Wijns
- Waldman Scott A
- Wijns W. i. l. l. i. a. m. Collaborators Clinical investigators
- Wojakowski Wojciech
- Y. M. Smyth Israel:Western Galilee Hospital: S. Atar
- Y. Barshay
- Y. De Bruyne
- Z. Bari Ireland: HRB Clinical Research Facility: F. Sharif
- Z. Jovic
- Z. Parma
- Z. Stankov
- Z. Walter
- Ziv Medical Center: M. Halabi
- Publication venue
- 'Oxford University Press (OUP)'
- Publication date
- 01/01/2016
- Field of study
Cardiopoietic cells, produced through cardiogenic conditioning of patients' mesenchymal stem cells, have shown preliminary efficacy. The Congestive Heart Failure Cardiopoietic Regenerative Therapy (CHART-1) trial aimed to validate cardiopoiesis-based biotherapy in a larger heart failure cohort
- …