APLIKASI TRAFFIC MONITORING SERVER MENGGUNAKAN SMS

Penelitian ini bertujuan membuat aplikasi traffic monitoring yang dapat membantu network administrator dalam memonitor server kapan dan dimana saja, yaitu dengan menggunakan SMS. Hal – hal yang akan dimonitor adalah lalu lintas data (traffic) dan koneksi jaringan server. Metode yang dilakukanadalahstudilapangan,danstudipustaka,danperancangan. Hasil yang dicapaiberupaaplikasi yang dapat mengirim dan menerima SMS ke / dari network administrator, melakukan cek koneksi ke server, dan memberikan respon kepada network administrator dalamwaktu yang relatifcepatketika koneksi ke server mengalami masalah. Selain itu, aplikasi ini dapat memantau detail paket data yang beredar pada server dan menampilkan bandwidth berdasarkan protokol. Detail paket data tersebut juga dapat dikirimkan melalui SMS. Kesimpulan yang didapat yaitu aplikasi traffic monitoring server sangat berguna karena dapat membantu network administrator untuk memonitor server dimana dan kapan saja. Disarankan untuk menambahkan fasilitas remote pada aplikasi ini

Healthy eaters beat unhealthy eaters in prototype evaluation among men, but abstinence may pose a risk for social standing

Peer reviewe

Robust estimation of bacterial cell count from optical density

Optical density (OD) is widely used to estimate the density of cells in liquid culture, but cannot be compared between instruments without a standardized calibration protocol and is challenging to relate to actual cell count. We address this with an interlaboratory study comparing three simple, low-cost, and highly accessible OD calibration protocols across 244 laboratories, applied to eight strains of constitutive GFP-expressing E. coli. Based on our results, we recommend calibrating OD to estimated cell count using serial dilution of silica microspheres, which produces highly precise calibration (95.5% of residuals <1.2-fold), is easily assessed for quality control, also assesses instrument effective linear range, and can be combined with fluorescence calibration to obtain units of Molecules of Equivalent Fluorescein (MEFL) per cell, allowing direct comparison and data fusion with flow cytometry measurements: in our study, fluorescence per cell measurements showed only a 1.07-fold mean difference between plate reader and flow cytometry data

ANALISIS DAN PERANCANGAN APLIKASI TRAFFIC MONITORING SERVER MENGGUNAKAN SMS PADA PT. SINAR BARU GLASINDO

ANALISIS DAN PERANCANGAN APLIKASI TRAFFIC MONITORING SERVER MENGGUNAKAN SMS PADA PT. SINAR BARU GLASIND

Pertumbuhan PT. Visionet Internasional Dengan Penerapan Strategi Virtualisasi

Pertumbuhan PT. Visionet Internasional Dengan Penerapan Strategi Virtualisasi - Virtualisasi, Virtual Office, langkah Strategis, BSC, pertumbuhan

Integrierte Versorgung - jetzt!

Schaeffer D, Bahrs O, Borchers U, et al. Integrierte Versorgung - jetzt!. In: Hildebrandt H, Stuppardt R, eds. Zukunft Gesundheit – regional, vernetzt, patientenorientiert. Gesundheitswesen in der Praxis. 1st ed. Heidelberg: Medhochzwei; 2021: 3-97

Expanding SPTAN1 monoallelic variant associated disorders:From epileptic encephalopathy to pure spastic paraplegia and ataxia

On behalf of Queen Square Genomics On behalf of Genomics England Research ConsortiumInternational audiencePurpose: Nonerythrocytic αII-spectrin (SPTAN1) variants have been previously associated with intellectual disability and epilepsy. We conducted this study to delineate the phenotypic spectrum of SPTAN1 variants.Methods: We carried out SPTAN1 gene enrichment analysis in the rare disease component of the 100,000 Genomes Project and screened 100,000 Genomes Project, DECIPHER database, and GeneMatcher to identify individuals with SPTAN1 variants. Functional studies were performed on fibroblasts from 2 patients.Results: Statistically significant enrichment of rare (minor allele frequency < 1 × 10-5) probably damaging SPTAN1 variants was identified in families with hereditary ataxia (HA) or hereditary spastic paraplegia (HSP) (12/1142 cases vs 52/23,847 controls, p = 2.8 × 10-5). We identified 31 individuals carrying SPTAN1 heterozygous variants or deletions. A total of 10 patients presented with pure or complex HSP/HA. The remaining 21 patients had developmental delay and seizures. Irregular αII-spectrin aggregation was noted in fibroblasts derived from 2 patients with p.(Arg19Trp) and p.(Glu2207del) variants.Conclusion: We found that SPTAN1 is a genetic cause of neurodevelopmental disorder, which we classified into 3 distinct subgroups. The first comprises developmental epileptic encephalopathy. The second group exhibits milder phenotypes of developmental delay with or without seizures. The final group accounts for patients with pure or complex HSP/HA

Expanding SPTAN1 monoallelic variant associated disorders: From epileptic encephalopathy to pure spastic paraplegia and ataxia

On behalf of Queen Square Genomics On behalf of Genomics England Research ConsortiumInternational audiencePurpose: Nonerythrocytic αII-spectrin (SPTAN1) variants have been previously associated with intellectual disability and epilepsy. We conducted this study to delineate the phenotypic spectrum of SPTAN1 variants.Methods: We carried out SPTAN1 gene enrichment analysis in the rare disease component of the 100,000 Genomes Project and screened 100,000 Genomes Project, DECIPHER database, and GeneMatcher to identify individuals with SPTAN1 variants. Functional studies were performed on fibroblasts from 2 patients.Results: Statistically significant enrichment of rare (minor allele frequency < 1 × 10-5) probably damaging SPTAN1 variants was identified in families with hereditary ataxia (HA) or hereditary spastic paraplegia (HSP) (12/1142 cases vs 52/23,847 controls, p = 2.8 × 10-5). We identified 31 individuals carrying SPTAN1 heterozygous variants or deletions. A total of 10 patients presented with pure or complex HSP/HA. The remaining 21 patients had developmental delay and seizures. Irregular αII-spectrin aggregation was noted in fibroblasts derived from 2 patients with p.(Arg19Trp) and p.(Glu2207del) variants.Conclusion: We found that SPTAN1 is a genetic cause of neurodevelopmental disorder, which we classified into 3 distinct subgroups. The first comprises developmental epileptic encephalopathy. The second group exhibits milder phenotypes of developmental delay with or without seizures. The final group accounts for patients with pure or complex HSP/HA