Web-based monitoring tool of the Atlantic Ocean observing system (Europe)

Web-based service tool that monitor data flow and key performance indicators of the Atlantic observing system. The focus here is on the monitoring of European contributions, including the EuroGOOS Atlantic ROOSES

Cocaine-mediated induction of microglial activation involves the ER stress-TLR2 axis

Effects of PBN (ROS scavenger) and APO (NADPH oxidase inhibitor) on cocaine-induced ROS formation in BV2 cells. (A) Green fluorescence was observed using a Zeiss Observer Z1 inverted microscope (Carl Zeiss MicroImaging). It revealed that PBN and APO resulted in significant inhibition of cocaine-mediated induction of ROS production. (JPG 7600 kb

Universality-Breaking Effects in Leptonic Z Decays

We analyze the possibility of universality violation in diagonal leptonic decays of the $Z$ boson, in the context of interfamily "see-saw" models. In a minimal extension of the Standard Model with right-handed neutrino fields, we find that universality-breaking effects increase quadratically with the heavy Majorana neutrino mass and may be observed in the running $LEP$ experiments.Comment: MZ-TH/93-04 #, LaTeX, 14 p. (2 Figs

Short-latency influence of medial frontal cortex on primary motor cortex during action selection under conflict

Medial frontal cortex (MFC) is crucial when actions have to be inhibited, reprogrammed, or selected under conflict, but the precise mechanism by which it operates is unclear. Importantly, how and when the MFC influences the primary motor cortex (M1) during action selection is unknown. Using paired-pulse transcranial magnetic stimulation, we investigated functional connectivity between the presupplementary motor area (pre-SMA) part of MFC and M1. We found that functional connectivity increased in a manner dependent on cognitive context: pre-SMA facilitated the motor evoked-potential elicited by M1 stimulation only during action reprogramming, but not when otherwise identical actions were made in the absence of conflict. The effect was anatomically specific to pre-SMA; it was not seen when adjacent brain regions were stimulated. We discuss implications for the anatomical pathways mediating the observed effects