Primary Idiopathic Frosted Branch Angiitis

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The effect of selective rhythmic movements on, hand- foot coordination in girl children with developmental coordination disorder

زمینه و هدف: اختلال هماهنگی رشدی وقتی رخ می دهد که در رشد مهارت های حرکتی تاخیر بیافتد یا در انجام حرکات هماهنگ مشکلاتی بروز کند که نتیجه آن عدم توانایی انجام وظایف روزمره است. این مطالعه با هدف بررسی اثر یک دوره حرکت های منتخب ریتمیک بر هماهنگی دست و پای کودکان دارای اختلال هماهنگی رشدی انجام شد. روش بررسی: در این تحقیق نیمه تجریی ابتدا توسط غربالگری از طریق پرسشنامه رشدی ویلسون و سیاهه مشاهده حرکتی آموزگاران از بین 475 دانش آموز دختر دبستانی (6 تا 11 ساله) شهرکرد تعداد 30 کودک دارای ناهنجاری هماهنگی رشدی انتخاب شدند. از این گروه پیش‌ـ آزمون توسط مقیاس رشد حرکتی لینکن‌ـ اوزرتسکی (آیتم‌های مربوط به هماهنگی دست و پا) اخذ شد و سپس با تقسیم تصادفی، در دو گروه تجربی و کنترل قرار گرفتند. گروه تجربی در یک دوره برنامه‌ی منتخب حرکات ریتمیک به مدت 8 هفته (سه جلسه در هفته، هر جلسه 45 دقیقه) شرکت کردند، در انتها مجدداً آیتم‌های مربوط به هماهنگی دست – پا مقیاس رشد حرکتی لینکن‌ـ اوزرتسکی از هر دو گروه اخذ شد. داده ها با استفاده از آزمون آماری تی مستقل تجزیه و تحلیل شد. یافته‌ها: نتایج نشان داد که در پیش آزمون میانگین هماهنگی دست و پای (گروه تجربی و کنترل) تفاوت معنادار وجود ندارد، اما در پس آزمون میانگین هماهنگی دست و پا در گروه تجربی بیشتر از گروه کنترل بود (01/0

The effects of Anethum on plasma lipid and lipoprotein in normal and diabetic rats fed high fat diets

زمینه و هدف: هیپرلیپدمی از ریسک فاکتورهای اصلی در پیشرفت بیماری های قلبی عروقی می باشد. آنتوم داروی گیاهی است که به عنوان پایین آورنده چربی خون در ایران استفاده می شود. در این تحقیق اثرات هیپولیپیدمیک عصاره ترکیبی آنتوم (Anethum) در رت های سالم و دیابتی تغذیه شده با جیره غذایی پرچرب مورد بررسی قرار گرفت. روش بررسی: در این مطالعه تجربی تعداد 88 سر موش با تزریق استرپتوزوسین (STZ)دیابتی شدند. دو روز پس از دیابتی نمودن حیوانات، رت های سالم و دیابتی تحت تیمار با جیره غذایی پرچرب قرار گرفتند. یک هفته بعد از شروع جیره غذایی خاص، حیوانات به مدت سه هفته تحت تیمار دارویی با عصاره ترکیبی آنتوم (Anethum) قرار گرفتند. پس از این مدت خون گیری انجام شد و سطح گلوکز، تری گلیسرید تام، کلسترول تام و لیپوپروتئین های با چگالی بالا به روش آنزیمی اندازه گیری شد. مقدار لیپوپروتئین های با چگالی پایین با فرمول فریدولد محاسبه گردید. داده ها به کمک آزمون های آماری آنالیز واریانس یک راهه و توکی تجزیه و تحلیل شد. یافته ها: عصاره ترکیبی آنتوم در رت های سالم تغذیه شده با جیره غذایی پرچرب به صورت معنی داری سطـح تری گلیسرید تام و لیپوپروتئین های با چگالی پایین را کاهش (01/0

Autism Spectrum Disorders in Iran

How to Cite this Article: Mohammadi MR, Salmanian M, Akhondzadeh Sh. Autism Spectrum Disorders in Iran. Iranian Journal of Child Neurology2011;5(4):1-9.ObjectiveAutistic disorder, Asperger syndrome, and PDD-Not Otherwise Specified are subsets of autism spectrum disorders (ASDs), which are characterized by impairments in social communication and stereotyped behavior. This article reviews the prevalence, etiology, diagnosis, and treatment of ASDs in Iran.Materials & MethodsWe searched PubMed, ISI Web of Science, and 4 Iranian databases (IranPsych,IranMedex, Irandoc and Scientific Information Database (SID) to find Iranian studies on  ASDs. The results of 39 investigations, comprising original, review and editorial articles; proceedings; and available dissertations were categorized by prevalence, etiology, diagnosis, and treatment.ConclusionSeveral preliminary investigations have been done to evaluate the prevalence of ASDs, and risk factors and effective variables have been studied with regard to etiology. The diagnostic evaluation of ASDs, especially based on EEG, and several pharmacological and behavioral interventions for ASD have been implemented in Iran. Mental health, stress levels, and personality characteristics were examined in the parents of children with ASDs, which were focused on mothers.ReferencesFirst MB, Frances A, Pincus HA. DSM-IV-TR: Handbook of differential diagnosis. United States of America:American Psychiatric Publishing; 2002.Parker S, Zuckerman B, Augustyn M. Developmental and behavioral pediatrics, 2 th ed. United States of America:Lippincott Williams & Wilkins; 2005.Howlin P. Autism and Asperger syndrome, 2 th ed. United States of America: Routledge; 2005.Mohammadi MR, Akhondzadeh S. Autism Spectrum Disorders: Etiology and Pharmacotherapy. Curr Drug ther2007; 2: 97-103.Newschaffer CJ, Croen LA, Daniels J, Giarelli E, GretherJK, Levy SE, et al. The epidemiology of autism spectrumdisorders. Annu Rev Public Health 2007; 28: 235-258.Zaroff CM, Uhm SY. Prevalence of autism spectrum disorders and influence of country of measurement and ethnicity. Soc Psychiatry Psychiatr Epidemiol 2011;published online.Baron-Cohen S, Scott FJ, Allison C, Williams J, Bolton P, Matthews FE, et al. Prevalence of autism-spectrum conditions: UK school-based population study. Br J Psychiatry 2009;194:500-509.Brugha TS, McManus S, Bankart J, Scott F, Purdon S, SmithJ, et al. Epidemiology of autism spectrum disorders inadults in the community in England. Arch Gen Psychiatry 2011; 68: 459-465.Prevalence of autism spectrum disorders - Autism and Developmental Disabilities Monitoring Network, United States, 2006. MMWR Surveill Summ 2009; 58: 1-20.Fombonne E. Epidemiology of pervasive developmental disorders. Pediatr Res 2009; 65: 591-598.Ehlers S, Gillberg C. The epidemiology of Asperger syndrome. A total population study. J Child Psychol Psychiatry 1993; 34: 1327-1350.Kadesjo B, Gillberg C, Hagberg B. Brief report: autismand Asperger syndrome in seven-year-old children: a totalpopulation study. J Autism Dev Disord 1999; 29: 327-331.Williams JG, Higgins JP, Brayne CE. Systematic reviewof prevalence studies of autism spectrum disorders. ArchDis Child 2006; 91: 8-15.Bernier R, Mao A, Yen J. Psychopathology, families, andculture: autism. Child Adolesc Psychiatr Clin N Am 2010;19: 855-867.Shamsi-pour M, Yonesian M, Mansouri A. Epidemiologyof autism: recent challenges in prevalence of autism andits risk factors. Journal of health and knowledge 2010; 5:133.Ghanizadeh A. A preliminary study on screening prevalence of pervasive developmental disorder in school children in Iran. J Autism Dev Disord 2008; 38:759-763.Nejatisafa AA, Kazemi MR, Alaghebandrad J. Autisticfeatures in adult population: evidence for continuity ofautistic symptoms with normality. Advanced in cognitivescience 2003; 5: 34-39.Khoushabi K, Pouretemad HR. Prevalence of pervasive developmental disorders according to gender in a sampleof Iranian children referred to treatment and rehabilitation centers. Journal of Hamedan University of Medical Sciences 2006;13: 58-60.Jannati M. Epidemiology of autism in exceptional studentsin Mashhd. MA thesis in psychology of exceptionalchildren field. Islamic Azad University of Birjand; 2001.Mansouri M, Chalabianlou GR, Malekirad AA, MosadedAA. The comparison of factors affecting the theory of mind development in autistic and normal children. Arak Medical University Journal 2011; 13: 115-125.Salmanian M. Visual memory of shapes and face in children with ASDs as compared to normal children. MS thesis in cognitive psychology. Institute for cognitive science studies; 2010.22. Ghanizadeh A, Mohammadi MR, Sadeghiyeh T, Alavi Shooshtari A, Akhondzadeh S. Symptoms of Childrenwith Autism Spectrum Disorder, a Clinical Sample. Iran J Psychiatry 2009; 4: 165-169. Sasanfar R, Haddad SA, Tolouei A, Ghadami M, Yu D, Santangelo SL. Parental age increases the risk for autismin an Iranian population sample. Mol Autism 2010; 1:2.Abolfazli R, Mirbagheri SA, Zabihi AA, Abouzari M. Autism and Celiac Disease: Failure to Validate the Hypothesis of a Possible Link. Iranian Red Crescent Medical Journal 2009; 11: 442-444.Hamidi Nahrani M, Sedaei M, Fatahi J, Sarough Farahani S, Faghihzadeh S. Auditory brain stem responses in autistic children in comparison with normal children. Audiology 2008; 16: 16-22.Sheikhani A, Behnam H, Mohammadi MR, Noroozian M. Evaluation of Quantitative Electroencephalography in Children with Autistic Disorders in Various Conditions Based on Spectrogram. Iran J Psychiatry 2007; 3: 4-10.Niedermeyer E, Lopes da Silva F. Electroencephalography:Basic principles, clinical applications, and related fields,5th ed. Philadelphia: Lippincott Williams & Wilkins;2005.Sheikhani A, Behnam H, Mohammadi MR, Noroozian M, Mohammadi M. Detection of Abnormalities for Diagnosing of Children with Autism Disorders Using of Quantitative Electroencephalography Analysis. J MedSyst 2010.Noroozian M, Sheikhani A, Behnam H, Mohammadi MR. Abnormalities of Quantitative Electroencephalography inChildren with Asperger Disorder Using Spectrogram and Coherence Values. Iran J Psychiatry 2008; 3: 64-70.Ahmadlou M, Adeli H, Adeli A. Fractality and a wavelet-chaos-neural network methodology for EEG based diagnosis of autistic spectrum disorder. J Clin Neurophysiol 2010; 27: 328-333.Bahari Gharagoz A, Hassanpour A, Amiri SH. Social interactions and repetitive behavior of autistic and trainable mentally retarded children. Developmental Psychology 2010; 7: 39-47.Campbell M, Adams P, Perry R, Spencer EK, Overall JE.Tardive and withdrawal dyskinesia in autistic children: aprospective study. Psychopharmacol Bull 1988; 24: 251-255.Anderson LT, Campbell M, Adams P, Small AM, Perry R, Shell J. The effects of haloperidol on discrimination learning and behavioral symptoms in autistic children. J Autism Dev Disord 1989; 19: 227-239.Shattock P, Kennedy A, Rowell F, et al. Role of neuropeptides in autism and their relationships with classical neurotransmitters. Brain Dysfunct 1990; 3: 328-345.Campbell M, Schopler E, Cueva, J, Hallin A. Treatmentof autistic disorder. J Am Acad Child Adolesc Psychiatry1996; 35: 134-143.Tsia LY. Psychopharmacology in autism. Psychosom Med 1999; 61: 651-665.McCracken JT, McGough J, Shah B, et al. Risperidone in children with autism and serious behavioral problems. N Engl J Med 2002; 347: 314 -321.Posey DJ, McDougle CJ. The pharmacotherapy of target symptoms associated with autistic disorder and other pervasive development disorders. Harv Rev Psychiatry 2000; 4: 45-63.Levy SE, Hyman SL. Novel treatments for autistic spectrum disorders. Ment Retard Dev Disabil Res Rev2005; 11: 131-142.Akhondzadeh S, Fallah J, Mohammadi MR, Imani R, Mohammadi M, Salehi B, et al. Double-blind placebo controlled trial of pentoxifylline added to risperidone:effects on aberrant behavior in children with autism. Prog Neuropsychopharmacol Biol Psychiatry 2010; 34: 32-36.Akhondzadeh S, Tajdar H, Mohammadi MR, Mohammadi M, Nouroozinejad GH, Shabstari OL, et al. A double-blindplacebo controlled trial of piracetam added to risperidonein patients with autistic disorder. Child Psychiatry Hum Dev 2008; 39: 237-245.Rezaei V, Mohammadi MR, Ghanizadeh A, Sahraian A,Tabrizi M, Rezazadeh SA, et al. Double-blind, placebo controlled trial of risperidone plus topiramate in children with autistic disorder. Prog Neuropsychopharmacol Biol Psychiatry 2010; 34: 1269-1272.Akhondzadeh S, Erfani S, Mohammadi MR, Tehrani-Doost M, Amini H, Gudarzi SS, et al. Cyproheptadine in the treatment of autistic disorder: a double-blind placebo controlled trial. J Clin Pharm Ther 2004; 29: 145-150.Mohammadi MR, Asadabadi M, Akhondzadeh S. A double-blind placebo controlled trial of celecoxib added to risperidone in children with autistic disorder. Unpublished paper.Ghanizadeh A. Targeting of glycine site on NMDA receptor as a possible new strategy for autism treatment.Neurochem Res 2011; 36: 922-923.Ghanizadeh A. Targeting neurotensin as a potential novel approach for the treatment of autism. J Neuro inflammation 2010; 7: 58.Bahmanzadegan Jahromi M, Yarmohammadian A,Mousavi H. Efficacy of social skills training in autistic behaviors and social development in children with autism disorder through social stories. New findings inpsychology 2009; 3: 79-93.Dalvand H, Dehghan L, Feizy A, Hosseini SA. The effect of home based Lovaas approach on social interaction,Speech and language, Play and behavior skills, and intensity of autism in young children with Autism. Modern Rehabilitation 2009; 3: 3.Golabi P, Alipour A, Zandi B. the effect of intervention by ABA method on children with autism. Research on exceptional children 2005; 5: 33-54.Arman S, Hakiman S, Golabi P. Three therapeutic methods for autistic children: a clinical trial. Journal of Isfahan Medical School 2005; 23: 44-48.Hatamzadeh A, Pouretemad H, Hassanabadi H. The effectiveness of parent-child interaction therapy for children with high functioning autism. World Conferenceon Psychology, Counselling and Guidance 2010; 5: 994-997.52. Chimeh N, Pouretemad HR, Khoramabadi R. Need assessment of mothers with autistic children. Journal of family research 2007; 3: 697-707.Ghanizadeh A, Alishahi MJ, Ashkani H. Helping families for caring children with autistic spectrum disorders. Arch Iran Med 2009; 12: 478-482.Khoushabi K, Farzad Fard SZ, Kakasoltani B, Pouretemad HR, Nikkhah HR. Coping strategies and stress in mothers with autistic children in comparison with mothers with normal children. Journal of family research 2010; 6: 87-97.Ghobari Bonab B, Estiri Z. A comparative study on personality characteristics and attachment style in mothers of children with and without autism. Research on exceptional children 2006; 6: 787-804.Rajabi Damavandi G, Poushineh K, Ghobari Bonab B. the relationship between personality characteristics and coping strategies in parents of children with ASDs. Research on exceptional children 2009; 9: 133-144.Samadi SA. Parents of children with autism spectrum disorder and children with intellectual disabilities and their stress and general health. Int J Integr Care2009; 9.Khoramabadi R, Pouretemad HR, Tahmasian K,Chimeh N. A comparative study of parental stress in mothers of autistic and non autistic children. Journal of family research 2009; 5: 387-399.59. Poretemad HR, Khoushabi K, Afshari R, Moradi S. Coping strategies and mental health in autistic children mothers. Journal of family research 2006;2: 285-292.Mohammadi A, Pouretemad HR, Khosravi G. Aninitial examination of the effect of guided imagery via music on reduction of stress, depression, and anxiety of mothers with autistic children. Journal of family research 2005; 1: 289-303.Arman S, Zareei N, Farshid nejad AA. Efficacy of group counseling on mothers with autistic children. Research in Behavioral Sciences 2004; 2: 48-52

Effect of Surface Treatment with Carbon Dioxide (CO2) Laser on Bond Strength between Cement Resin and Zirconia

Introduction: Since it is not possible to form an adequate micromechanical bond between resin cement and zirconia ceramics using common surface treatment techniques, laser pretreatment has been suggested for zirconia ceramic surfaces. The aim of this study was to evaluate the effect of Carbon Dioxide (CO2) Laser treatment on shear bond strength (SBS) of resin cement to zirconia ceramic.Methods: In this in vitro study thirty discs of zirconia with a diameter of 6 mm and a thickness of 2 mm were randomly divided into two groups of 15. In the test group the zirconia disc surfaces were irradiated by CO2 laser with an output power of 3 W and energy density of 265.39j/cm2. Composite resin discs were fabricated by plastic molds, measuring 3 mm in diameter and 2 mm in thickness and were cemented on zirconia disk surfaces with Panavia F2.0 resin cement (Kuraray Co. Ltd, Osaka, Japan). Shear bond strength was measured by a universal testing machine at a crosshead speed of 0.5 mm/min. The fracture type was assessed under a stereomicroscope at ×40. Surface morphologies of two specimens of the test group were evaluated under SEM before and after laser pretreatment. Data was analyzed by paired t-test (p value < 0.05).Results: The mean SBS values of the laser and control groups were 12.12 ± 3.02 and 5.97 ± 1.14 MPa, respectively. Surface treatment with CO2 laser significantly increased SBS between resin cement and zirconia ceramic (p value = 0.001).Conclusion: Under the limitations of this study, surface treatment with CO2 laser increased the SBS between resin cement and the zirconia ceramic

Comparison of the Effects of Local Injection and Oral Intake of Diclofenac and Atorvastatin in Alveolar Bone Density Assessed with CT in Experimental Periodontitis in Rat

Objectives The first cause of tooth loss in developed countries is periodontitis. Chronic periodontitis is the most common form of periodontitis and it is characterized by loss of periodontal attachment, destruction of alveolar bone and eventual loss of teeth. Atorvastatin is a statin drug used for the treatment of high cholesterol. Statins can stop the inflammatory process by inhibiting the cholesterol pathway. Diclofenac is an NSAID with anti-inflammatory, anti-pyretic and analgesic effects. Its primary mechanism is through the inhibition of prostaglandins synthesis by the inhibition of the cyclooxygenase enzyme (COX). The purpose of this study is to compare the effects of local injected and oral intake of Diclofenac and atorvastatin on alveolar bone density measured in HUs with the use of a CT scan in a periodontitis-induced model in rats.Methods Thirty rats were randomly divided into 6 groups of 5 rats each. Ligatures were placed around the left second maxillary molar to induce periodontitis for 10 days. Administration of 12.5 mg/kg of oral atorvastatin (group 1), 0.25 mg/kg of injectable atorvastatin (group 2), 7.5mg/kg of oral Diclofenac (group 3), 6.25mg/kg of injectable Diclofenac (group 4), the oral solvent without medicine as oral control (groups 5), and the injectable solvent without medicine as injectable control (group 6). In each group, the right side of maxilla was considered as control group (without ligature and drug interaction). At day eleven, the rats were sacrificed and the maxillary bone was separated from the soft tissue and fixed in 4% formalin. The prepared samples were then radiologically evaluated to determine the bone density with CT in fixed exposure conditions.Results There was a statistically significant difference between the alveolar bone density of the oral atorvastatin group and the oral Diclofenac (P = 0.006). There was no statistical significant difference in alveolar bone density between the injectable atorvastatin and the injectable Diclofenac groups (P=0.228).Conclusion Both atorvastatin and Diclofenac have shown better results when assessing bone density in a periodontitis rat model  as compared to controls. Additionally, Diclofenac has been shown to be more effective at both oral and injectable administrations as compared with atorvastatin in the prevention of loss of bone density in a rat model with periodontitis

Subcutaneous Reaction of Rat Tissues to Nanosilver Coated Gutta-Percha

Introduction: Gutta-percha (GP), is a neutral and non-toxic material. The aim of this animal study was to compare the biocompatibility of nanosilver coated GP (NS-GP) with conventional GP in subcutaneous tissues in a rat model. Methods and Materials: Conventional GP and NS-GP were subcutaneously implanted in the backs of 20 male Wistar rats (n=10). A control animal was assigned for each trial period. Ten animals were sacrificed after 7 and 30 days and light microscopic evaluation of tissue reaction to NS-GP (n=20) and conventional GP (n=20) was accomplished. The Mann-Whitney U, Wilcoxon Signed Ranks, Fisher Exact, and McNemar tests were used for statistical analysis of the data. Results: After 7 days, inflammation was moderate and mild for NS-GP and conventional GP, respectively (P<0.001). After 30 days, no inflammation was discernible in conventional GP. However, mild inflammation was reported for NS-GP (P<0.001). Regarding inflammatory cell type, there was a significant difference between two experimental groups at both times (P<0.001). Conclusion: Inflammation decreased over time in both groups. Fibrous connective tissue, a representative of healing and control of inflammatory process, surrounded both test materials. NS-GP was biocompatible and might be a reasonable endodontic obturation material.Keywords: Gutta-Percha; Inflammation; Nanosilver Coated Gutta-Percha; Subcutaneous Connective Tissue

Successful Management of an Aluminum Phosphide Poisoned Patient Following Ventricular Tachycardia

Aluminum Phosphide (ALP) poisoning is regarded as fatal; however, it is commonly used indeveloping countries. In some countries, such as India and Iran, it is still being used to protectgrain against pests at home. As yet, no known antidote is reported to be effective against ALPpoisoning. ALP acts through blockage of cell respiration, alteration of cell permeability, andinduction of oxidative stress. Some factors can worsen the prognosis of its intoxication, whichinclude hyperglycemia on admission, ingestion of fresh tablets, cardiac abnormality, especiallyVentricular Tachycardia (VT), severe metabolic acidosis, cardiogenic shock and other typesof shocks, electrolyte disturbances, end-organ damage, and a need for mechanical ventilation.ALP kills its victims by induction of multi-organ failure.In this study, we controlled the patient’s VT using MgSO4, electric shock, amiodarone, andlidocaine. We also encountered the cardiogenic shock using norepinephrine, High-Dose Insulin(HDI), antioxidants, sodium bicarbonate, and normal saline. Moreover, myocardial ischemia,recurrent VTs, and adverse cardiac effects were protected via tight control of magnesium andother electrolytes

The Efficacy of Cupping Therapy Added to Electroacupuncture and Exercise Therapy on Knee Osteoarthritis

Background: Electroacupuncture and exercise therapy have been used to treat knee osteoarthritis, but evidence for adding cupping to this treatment is lacking. Therefore, this study aimed to investigate the effect of cupping and acupuncture combined with exercise on knee osteoarthritis. Materials and Methods: This randomized control trial was done on 56 patients with knee osteoarthritis. We had two groups: a control and an intervention group. Both groups received electroacupuncture and exercise therapy programs. The intervention group received cupping after electroacupuncture plus exercise therapy. The Western Ontario and McMaster Universities Index (WOMAC) questionnaire and Visual Analogue Scale (VAS) measured patient outcomes before and after treatment. Results: All patients' VAS and WOMAC scores decreased in these two groups after treatment. The difference between VAS and WOMAC scores and pain and knee function was significant compared to the intervention group with the control group (p<0.05). The difference in knee stiffness was not significant comparing the intervention group with the control group (p>0.05). Conclusion: Adding cupping therapy following electroacupuncture and exercise therapy significantly decreased pain and improved function

Estimativa da composição elementar de solos do Azerbaijão oeste, Irã, utilizando-se modelos espectrais de infravermelho

[Abstract] Characterizing the elemental composition provides useful information about the weathering degree of soils. In Miandoab County, Northern Iran, this characterization was missing, and thus the objectives of this work were to evaluate the weathering degrees for the most typical soils in the area from their elemental compositions, and to estimate this elemental composition using Fourier transform infrared spectroscopy and Random Forest models. Five soil profiles, including Aridisols and Inceptisols, were selected as the most representative of the area. Major elemental oxides were determined in each genetic horizon by X-ray fluorescence, showing that these soils were at early developmental stages. Only Al2O3 and CaO were accurately estimated, with R2 values of 0.8, and out-of-bag mean square errors of 0.2 and 1.1, respectively. The other oxides were not predicted satisfactorily, probably due to small differences in their elemental compositions. Random Forest provided the important spectral bands related to the content of each element. For Al2O3, these bands were between 500 and 650 cm-1, which represent out-of-plane OH bending vibrations and Al-O gibbsite and alumino-silicate vibrations. For CaO, the most important bands are related to carbonate content. A combination of Fourier transform infrared spectra and Random Forest models can be used as a rapid and low-cost technique to estimate the elemental composition of arid and semi-arid soils of Northern Iran.[Resumo] A caracterização da composição elementar fornece informações úteis para caracterizar o grau de alteração dos solos. Em Miandoab, norte do Irã, esta caracterização não existe. Os objetivos deste trabalho foram avaliar o grau de intemperismo dos solos típicos da região usando a sua composição elementar e estimar esta composição usando espectroscopia infravermelha com transformada de Fourier (FTIR) e modelos Random Forest (RF). Foram selecionados cinco perfis de solo, incluindo Aridisolos e Inceptisolos, como os mais representativos da área. Os principais óxidos elementares foram determinados por fluorescência de raios-X em cada horizonte genético, mostrando que estes solos estavam em um estágio de baixo grau de desenvolvimento. Apenas o Al2O3 e o CaO foram estimados com precisão, com valores de R2 de 0,8 e erro quadrático médio nos dados utilizados para validação de 0,2 e 1,1, respectivamente, enquanto os outros óxidos não foram preditos satisfatoriamente, provavelmente devido às pequenas diferenças na sua composição. O modelo Random Forest forneceu importantes bandas espectrais relacionadas com o conteúdo de cada elemento. Para o Al2O3, estes atingiram a região 500 a 650 cm-1, o que foi atribuído a vibrações de flexão de OH e vibrações de Al-O de gibbsita e alumino-silicatos. Para o CaO, as bandas mais importantes estavam relacionadas ao teor de carbonatos. Os resultados indicam que uma combinação de espectros infravermelha de transformada de Fourier e modelos Random Forest pode ser usada como uma técnica rápida e de baixo custo para estimar a composição elementar de solos do norte do Irã