104 research outputs found
Switched-current filter structure for synthesizing arbitrary characteristics based on follow-the-leader feedback configuration
This document is the Accepted Manuscript version of the following article: Wenshan Zhao, Yigang He, and Yichuang Sun, ‘Switched-current filter structure for synthesizing arbitrary characteristics based on follow-the-leader feedback configuration’, Analog Integrated Circuits and Signal Processing, (2015), Vol. 82 (2): 479-486. The version of record is available online at doi: 10.1007/s10470-014-0477-8 © Springer Science+Business Media New York 2015Peer reviewedFinal Accepted Versio
Study on the rare radiative decay in the standard model and multiscale walking technicolor model
Applying the perturbative QCD ( PQCD ) method, we study the decay
in the standard model and multiscale walking
technicolor model. In the SM, we find that the contribution of weak
annihilation is more important than that of the electromagnetic penguin. The
presence of Pseudo-Goldstone-Bosons in the MWTCM leads to a large enhancement
in the rate of , but this model is in conflict with
the branching ratio of ( ) and the CLEO data
on the branching ratio BR ( ). If topcolor is further
introduced, the calculated results in the topcolor assisted MWTCM can be
suppressed and be in agreement with the CLEO data for a certain range of the
parameters.Comment: 16 pages, Latex, no macros, 1 figure(in Latex), hard copy is
available upon request. to appear in Phys. Rev.
Developments in unsteady pipe flow friction modelling
This paper reviews a number of unsteady friction models for transient pipe flow. Two distinct unsteady friction models, the Zielke and the Brunone models, are investigated in detail. The Zielke model, originally developed for transient laminar flow, has been selected to verify its effectiveness for "low Reynolds number" transient turbulent flow. The Brunone model combines local inertia and wall friction unsteadiness. This model is verified using the Vardy's analytically deduced shear decay coefficient C* to predict the Brunone's friction coefficient k rather than use the traditional trial and error method for estimating k. The two unsteady friction models have been incorporated into the method of characteristics water hammer algorithm. Numerical results from the quasi-steady friction model and the Zielke and the Brunone unsteady friction models are compared with results of laboratory measurements for water hammer cases with laminar and low Reynolds number turbulent flows. Conclusions about the range of validity for the three friction models are drawn. In addition, the convergence and stability of these models are addressed.Anton Bergant, Angus Ross Simpson, John Vìtkovsk
Taxonomy of prokaryotic viruses : 2017 update from the ICTV Bacterial and Archaeal Viruses Subcommittee
Peer reviewe
Diabetes Alters Intracellular Calcium Transients in Cardiac Endothelial Cells
Diabetic cardiomyopathy (DCM) is a diabetic complication, which results in myocardial dysfunction independent of other etiological factors. Abnormal intracellular calcium ([Ca2+]i) homeostasis has been implicated in DCM and may precede clinical manifestation. Studies in cardiomyocytes have shown that diabetes results in impaired [Ca2+]i homeostasis due to altered sarcoplasmic reticulum Ca2+ ATPase (SERCA) and sodium-calcium exchanger (NCX) activity. Importantly, altered calcium homeostasis may also be involved in diabetes-associated endothelial dysfunction, including impaired endothelium-dependent relaxation and a diminished capacity to generate nitric oxide (NO), elevated cell adhesion molecules, and decreased angiogenic growth factors. However, the effect of diabetes on Ca2+ regulatory mechanisms in cardiac endothelial cells (CECs) remains unknown. The objective of this study was to determine the effect of diabetes on [Ca2+]i homeostasis in CECs in the rat model (streptozotocin-induced) of DCM. DCM-associated cardiac fibrosis was confirmed using picrosirius red staining of the myocardium. CECs isolated from the myocardium of diabetic and wild-type rats were loaded with Fura-2, and UTP-evoked [Ca2+]i transients were compared under various combinations of SERCA, sarcoplasmic reticulum Ca2+ ATPase (PMCA) and NCX inhibitors. Diabetes resulted in significant alterations in SERCA and NCX activities in CECs during [Ca2+]i sequestration and efflux, respectively, while no difference in PMCA activity between diabetic and wild-type cells was observed. These results improve our understanding of how diabetes affects calcium regulation in CECs, and may contribute to the development of new therapies for DCM treatment
Poisson-Like Shape and Shoulder Structure of Hadron Multiplicity Distributions in e+ e- Annihilation at Z0 Energy
Loss of microRNA-128 promotes cardiomyocyte proliferation and heart regeneration
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