Optical and near infrared photometry of Butcher-Oemler clusters

Rich clusters of galaxies at moderate redshifts (z approx. .3) have a larger proportion of optically blue galaxies than their low redshift counterparts. Spectroscopic examination of the blue galaxies by various authors has shown that the blue galaxies are generally Seyferts, show evidence for recent star formation, or are foreground objects. Unfortunately, spectroscopy is too time consuming to be used on large samples. Thus, we have looked for a way to separate Seyferts, starbursts, ellipticals and nonmembers using photometry alone. Five moderate redshift clusters, Abell numbers 777, 963, 1758, 1961 and 2218, have been observed in the V, R and K bands. We model the spectral energy distributions of various kinds of galaxies found in clusters and derive observed colors. We have modeled the spectral energy distributions (SED) of several kinds of galaxies and compute their colors as a function of redshift. We expect to see ellipticals, spirals, starbursts, post-starburst and Seyfert galaxies. The SED of elliptical and Sbc galaxies was observed by Rieke and Rieke. The SEDs for the starburst galaxies was created by adding a reddened 10(exp 8) year old burst to a spiral galaxy SED. The post-starburst (E+A) galaxy SEDs are composed of a slightly reddened 10(exp 9) year old burst and elliptical galaxy SED. SEDs for the Seyferts were created by adding a v(exp -1.1) power law, and a hot dust thermal spectrum to the Sbc. From the SEDs the colors of galaxies at various redshifts with assorted filters were computed. Lilly & Gunn (1985) have optical and infrared photometry for a sample of galaxies in CL0024+1654 observed spectroscopically by Dressler, Gunn and Schneider (1985). We have used this data to choose the most appropriate SEDs for our starburst and post-starburst models. The most likely explanation for the optically blue colors in most cluster galaxies is star formation. Very few galaxies lie in the Seyfert locus. Abel 1758 has more Seyfert candidates than the other clusters, we observed. It seems possible to roughly sort types of galaxies in clusters by color alone. The cluster population seems to vary considerably between clusters, but our K selected sample has few Seyferts in any cluster

Urbanization, climate and species traits shape mammal communities from local to continental scales

Human-driven environmental changes shape ecological communities from local to global scales. Within cities, landscape-scale patterns and processes and species characteristics generally drive local-scale wildlife diversity. However, cities differ in their structure, species pools, geographies and histories, calling into question the extent to which these drivers of wildlife diversity are predictive at continental scales. In partnership with the Urban Wildlife Information Network, we used occurrence data from 725 sites located across 20 North American cities and a multi-city, multi-species occupancy modelling approach to evaluate the effects of ecoregional characteristics and mammal species traits on the urbanization-diversity relationship. Among 37 native terrestrial mammal species, regional environmental characteristics and species traits influenced within-city effects of urbanization on species occupancy and community composition. Species occupancy and diversity were most negatively related to urbanization in the warmer, less vegetated cities. Additionally, larger-bodied species were most negatively impacted by urbanization across North America. Our results suggest that shifting climate conditions could worsen the effects of urbanization on native wildlife communities, such that conservation strategies should seek to mitigate the combined effects of a warming and urbanizing world

Initial invasive or conservative strategy for stable coronary disease

BACKGROUND Among patients with stable coronary disease and moderate or severe ischemia, whether clinical outcomes are better in those who receive an invasive intervention plus medical therapy than in those who receive medical therapy alone is uncertain. METHODS We randomly assigned 5179 patients with moderate or severe ischemia to an initial invasive strategy (angiography and revascularization when feasible) and medical therapy or to an initial conservative strategy of medical therapy alone and angiography if medical therapy failed. The primary outcome was a composite of death from cardiovascular causes, myocardial infarction, or hospitalization for unstable angina, heart failure, or resuscitated cardiac arrest. A key secondary outcome was death from cardiovascular causes or myocardial infarction. RESULTS Over a median of 3.2 years, 318 primary outcome events occurred in the invasive-strategy group and 352 occurred in the conservative-strategy group. At 6 months, the cumulative event rate was 5.3% in the invasive-strategy group and 3.4% in the conservative-strategy group (difference, 1.9 percentage points; 95% confidence interval [CI], 0.8 to 3.0); at 5 years, the cumulative event rate was 16.4% and 18.2%, respectively (difference, 121.8 percentage points; 95% CI, 124.7 to 1.0). Results were similar with respect to the key secondary outcome. The incidence of the primary outcome was sensitive to the definition of myocardial infarction; a secondary analysis yielded more procedural myocardial infarctions of uncertain clinical importance. There were 145 deaths in the invasive-strategy group and 144 deaths in the conservative-strategy group (hazard ratio, 1.05; 95% CI, 0.83 to 1.32). CONCLUSIONS Among patients with stable coronary disease and moderate or severe ischemia, we did not find evidence that an initial invasive strategy, as compared with an initial conservative strategy, reduced the risk of ischemic cardiovascular events or death from any cause over a median of 3.2 years. The trial findings were sensitive to the definition of myocardial infarction that was used

Health-status outcomes with invasive or conservative care in coronary disease

BACKGROUND In the ISCHEMIA trial, an invasive strategy with angiographic assessment and revascularization did not reduce clinical events among patients with stable ischemic heart disease and moderate or severe ischemia. A secondary objective of the trial was to assess angina-related health status among these patients. METHODS We assessed angina-related symptoms, function, and quality of life with the Seattle Angina Questionnaire (SAQ) at randomization, at months 1.5, 3, and 6, and every 6 months thereafter in participants who had been randomly assigned to an invasive treatment strategy (2295 participants) or a conservative strategy (2322). Mixed-effects cumulative probability models within a Bayesian framework were used to estimate differences between the treatment groups. The primary outcome of this health-status analysis was the SAQ summary score (scores range from 0 to 100, with higher scores indicating better health status). All analyses were performed in the overall population and according to baseline angina frequency. RESULTS At baseline, 35% of patients reported having no angina in the previous month. SAQ summary scores increased in both treatment groups, with increases at 3, 12, and 36 months that were 4.1 points (95% credible interval, 3.2 to 5.0), 4.2 points (95% credible interval, 3.3 to 5.1), and 2.9 points (95% credible interval, 2.2 to 3.7) higher with the invasive strategy than with the conservative strategy. Differences were larger among participants who had more frequent angina at baseline (8.5 vs. 0.1 points at 3 months and 5.3 vs. 1.2 points at 36 months among participants with daily or weekly angina as compared with no angina). CONCLUSIONS In the overall trial population with moderate or severe ischemia, which included 35% of participants without angina at baseline, patients randomly assigned to the invasive strategy had greater improvement in angina-related health status than those assigned to the conservative strategy. The modest mean differences favoring the invasive strategy in the overall group reflected minimal differences among asymptomatic patients and larger differences among patients who had had angina at baseline