33 research outputs found

    Unstable particles in matter at a finite temperature: the rho and omega mesons

    Full text link
    Unstable particles (such as the vector mesons) have an important role to play in low mass dilepton production resulting from heavy ion collisions and this has been a subject of several investigations. Yet subtleties, such as the implications of the generalization of the Breit-Wigner formula for nonzero temperature and density, e.g. the question of collisional broadening, the role of Bose enhancement, etc., the possibility of the kinematic opening (or closing) of decay channels due to environmental effects, the problem of double counting through resonant and direct contributions, are often given insufficient emphasis. The present study attempts to point out these features using the rho and omega mesons as illustrative examples. The difference between the two versions of the Vector Meson Dominance Model in the present context is also presented. Effects of non-zero temperature and density, through vector meson masses and decay widths, on dilepton spectra are studied, for concreteness within the framework of a Walecka-type model, though most of the basic issues highlighted apply to other scenarios as well.Comment: text and figures modifie

    Constraints on possible phase transitions above the nuclear saturation density

    Get PDF
    We compare different models for hadronic and quark phases of cold baryon-rich matter in an attempt to find a deconfinement phase transition between them. For the hadronic phase we consider Walecka-type mean-field models which describe well the nuclear saturation properties. We also use the variational chain model which takes into account correlation effects. For the quark phase we consider the MIT bag model, the Nambu-Jona-Lasinio and the massive quasiparticle models. By comparing pressure as a function of baryon chemical potential we find that crossings of hadronic and quark branches are possible only in some exceptional cases while for most realistic parameter sets these branches do not cross at all. Moreover, the chiral phase transition, often discussed within the framework of QCD motivated models, lies in the region where the quark phases are unstable with respect to the hadronic phase. We discuss possible physical consequences of these findings.Comment: 28 pages, 18 PostScript figures, submitted to Phys. Rev.

    On the reheating stage after inflation

    Full text link
    We point out that inflaton decay products acquire plasma masses during the reheating phase following inflation. The plasma masses may render inflaton decay kinematicaly forbidden, causing the temperature to remain frozen for a period at a plateau value. We show that the final reheating temperature may be uniquely determined by the inflaton mass, and may not depend on its coupling. Our findings have important implications for the thermal production of dangerous relics during reheating (e.g., gravitinos), for extracting bounds on particle physics models of inflation from Cosmic Microwave Background anisotropy data, for the production of massive dark matter candidates during reheating, and for models of baryogenesis or leptogensis where massive particles are produced during reheating.Comment: 8 pages, 2 figures. Submitted for publication in Phys. Rev.

    Effective field theory

    Full text link
    I give a brief review of effective field theory, disscussing the contribution of Feza G\"ursey in particular and focusing on the literature I am most familiar with.Comment: 17 pages, no figs, macros appended, plain te

    Transport Coefficients and Analytic Continuation in Dual 1+1 Dimensional Models at Finite Temperature

    Get PDF
    The conductivity of a finite temperature 1+1 dimensional fermion gas described by the massive Thirring model is shown to be related to the retarded propagator of the dual boson sine-Gordon model. Duality provides a natural resummation which resolves infra-red problems, and the boson propagator can be related to the fermion gas at non-zero temperature and chemical potential or density. In addition, at high temperatures, we can apply a dimensional reduction technique to find resummed closed expressions for the boson self-energy and relate them to the fermion conductivity. Particular attention is paid to the discussion of analytic continuation. The resummation implicit in duality provides a powerful alternative to the standard diagrammatic evaluation of transport coefficients at finite temperature.Comment: 41 pages, 6 figure

    Practicing Imperfect Forgiveness

    Get PDF
    Forgiveness is typically regarded as a good thing - even a virtue - but acts of forgiveness can vary widely in value, depending on their context and motivation. Faced with this variation, philosophers have tended to reinforce everyday concepts of forgiveness with strict sets of conditions, creating ideals or paradigms of forgiveness. These are meant to distinguish good or praiseworthy instances of forgiveness from problematic instances and, in particular, to protect the self-respect of would-be forgivers. But paradigmatic forgiveness is problematic for a number of reasons, including its inattention to forgiveness as a gendered trait. We can account for the values and the risks associated with forgiving far better if we treat it as a moral practice and not an ideal

    Whole-genome sequencing reveals host factors underlying critical COVID-19

    Get PDF
    Critical COVID-19 is caused by immune-mediated inflammatory lung injury. Host genetic variation influences the development of illness requiring critical care1 or hospitalization2,3,4 after infection with SARS-CoV-2. The GenOMICC (Genetics of Mortality in Critical Care) study enables the comparison of genomes from individuals who are critically ill with those of population controls to find underlying disease mechanisms. Here we use whole-genome sequencing in 7,491 critically ill individuals compared with 48,400 controls to discover and replicate 23 independent variants that significantly predispose to critical COVID-19. We identify 16 new independent associations, including variants within genes that are involved in interferon signalling (IL10RB and PLSCR1), leucocyte differentiation (BCL11A) and blood-type antigen secretor status (FUT2). Using transcriptome-wide association and colocalization to infer the effect of gene expression on disease severity, we find evidence that implicates multiple genes—including reduced expression of a membrane flippase (ATP11A), and increased expression of a mucin (MUC1)—in critical disease. Mendelian randomization provides evidence in support of causal roles for myeloid cell adhesion molecules (SELE, ICAM5 and CD209) and the coagulation factor F8, all of which are potentially druggable targets. Our results are broadly consistent with a multi-component model of COVID-19 pathophysiology, in which at least two distinct mechanisms can predispose to life-threatening disease: failure to control viral replication; or an enhanced tendency towards pulmonary inflammation and intravascular coagulation. We show that comparison between cases of critical illness and population controls is highly efficient for the detection of therapeutically relevant mechanisms of disease
    corecore