11 research outputs found
Biochemical mechanisms implemented by human acute myeloid leukemia cells to suppress host immune surveillance
Get PDFAcute myeloid leukaemia (AML) is a blood/bone marrow cancer originating from myeloid cell precusors capable of self-renewing. AML cells implement biochemical mechanisms which allow them not only to survive, but also to successfully escape immune surveillance. ln this work, we discuss crucial molecular mechanisms used by human AML cells in order to evade immune attack
The dual roles of ginsenosides in improving the anti-tumor efficiency of cyclophosphamide in mammary carcinoma mice
No full textHyperresponsiveness to interferon gamma exposure as a response mechanism to anti-PD-1 therapy in microsatellite instability colorectal cancer
No full textPreparation of soluble dietary fibers from Gracilaria lemaneiformis and its antitumor activity in vivo
No full textMyeloid maturation potentiates STAT3-mediated atypical IFN-Îł signaling and upregulation of PD-1 ligands in AML and MDS
No full textCytotoxicity study of the interleukin-12-expressing recombinant Newcastle disease virus strain, rAF-IL12, towards CT26 colon cancer cells in vitro and in vivo
No full textIL-27 mediates HLA class I up-regulation, which can be inhibited by the IL-6 pathway, in HLA-deficient Small Cell Lung Cancer cells
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