165 research outputs found
Magnetic structure of Cd-doped CeCoIn5
The heavy fermion superconductor CeCoIn5 is believed to be close to a
magnetic instability, but no static magnetic order has been found. Cadmium
doping on the In-site shifts the balance between superconductivity and
antiferromagnetism to the latter with an extended concentration range where
both types of order coexist at low temperatures. We investigated the magnetic
structure of nominally 10% Cd-doped CeCoIn5, being antiferromagnetically
ordered below T_N=3 K and superconducting below T_c=1.3 K, by elastic neutron
scattering. Magnetic intensity was observed only at the ordering wave vector
Q_AF = (1/2,1/2,1/2) commensurate with the crystal lattice. Upon entering the
superconducting state the magnetic intensity seems to change only little. The
commensurate magnetic ordering in CeCo(In1-xCdx)5 is in contrast to the
incommensurate antiferromagnetic ordering observed in the closely related
compound CeRhIn5. Our results give new insights in the interplay between
superconductivity and magnetism in the family of CeTIn5 (T=Co, Rh, and Ir)
based compounds.Comment: 4 pages, 4 figure
Effect of magnetic order on the superfluid response of single-crystal ErNiBC: A penetration depth study
We report measurements of the in-plane magnetic penetration depth (T) in single crystals of ErNiBC down to 0.1 K using
a tunnel-diode based, self-inductive technique at 21 MHz. We observe four
features: (1) a slight dip in (T) at the Nel
temperature = 6.0 K, (2) a peak at = 2.3 K, where a weak
ferromagnetic component sets in, (3) another maximum at 0.45 K, and (4) a final
broad drop down to 0.1 K. Converting to superfluid density , we see
that the antiferromagnetic order at 6 K only slightly depresses
superconductivity. We seek to explain some of the above features in the context
of antiferromagnetic superconductors, where competition between the
antiferromagnetic molecular field and spin fluctuation scattering determines
increased or decreased pairbreaking. Superfluid density data show only a slight
decrease in pair density in the vicinity of the 2.3 K feature, thus supporting
other evidences against bulk ferromagnetism in this temperature range.Comment: 15 pages, 5 figure
Evidence for the Coexistence of Anisotropic Superconducting Gap and Nonlocal Effects in the Non-magnetic Superconductor LuNi2B2C
A study of the dependence of the heat capacity Cp(alpha) on field angle in
LuNi2B2C reveals an anomalous disorder effect. For pure samples, Cp(alpha)
exhibits a fourfold variation as the field H < Hc2 is rotated in the [001]
plane, with minima along (alpha = 0). A slightly disordered sample,
however, develops anomalous secondary minima along for H > 1 T, leading
to an 8-fold pattern. The anomalous pattern is discussed in terms of coexisting
superconducting gap anisotropy and non-local effects.Comment: 5 pages, 4 figure
Anomalous Paramagnetic Effects in the Mixed State of LuNi2B2C
Anomalous paramagnetic effects in dc magnetization were observed in the mixed
state of LuNi2B2C, unlike any reported previously. It appears as a kink-like
feature for H > 30 kOe and becomes more prominent with increasing field. A
specific heat jump at the corresponding temperature suggests that the anomaly
is due to a true bulk transition. A magnetic flux transition from a square to
an hexagonal lattice is consistent with the anomaly.Comment: 5 pages, 4 figure
TESS and CHEOPS discover two warm sub-Neptunes transiting the bright K-dwarf HD 15906
We report the discovery of two warm sub-Neptunes transiting the bright (G = 9.5 mag) K-dwarf HD 15906 (TOI 461, TIC 4646810). This star was observed by the Transiting Exoplanet Survey Satellite (TESS) in sectors 4 and 31, revealing two small transiting planets. The inner planet, HD 15906 b, was detected with an unambiguous period but the outer planet, HD 15906 c, showed only two transits separated by ∼ 734 d, leading to 36 possible values of its period. We performed follow-up observations with the CHaracterising ExOPlanet Satellite (CHEOPS) to confirm the true period of HD 15906 c and improve the radius precision of the two planets. From TESS, CHEOPS, and additional ground-based photometry, we find that HD 15906 b has a radius of 2.24 ± 0.08 R⊕ and a period of 10.924709 ± 0.000032 d, whilst HD 15906 c has a radius of 2.93+0.07−0.06 R⊕ and a period of 21.583298+0.000052−0.000055 d. Assuming zero bond albedo and full day-night heat redistribution, the inner and outer planet have equilibrium temperatures of 668 ± 13 K and 532 ± 10 K, respectively. The HD 15906 system has become one of only six multiplanet systems with two warm (≲ 700 K) sub-Neptune sized planets transiting a bright star (G ≤ 10 mag). It is an excellent target for detailed characterization studies to constrain the composition of sub-Neptune planets and test theories of planet formation and evolution
A Novel Dielectric Anomaly in Cuprates and Nickelates: Signature of an Electronic Glassy State
The low-frequency dielectric response of hole-doped insulators
La_{2}Cu_{1-x}Li_{x}O_{4} and La_{2-x}Sr_{x}NiO_{4} shows a large dielectric
constant \epsilon ^{'} at high temperature and a step-like drop by a factor of
100 at a material-dependent low temperature T_{f}. T_{f} increases with
frequency and the dielectric response shows universal scaling in a Cole-Cole
plot, suggesting that a charge glass state is realized both in the cuprates and
in the nickelates.Comment: 5 pages, 4 figure
CERKL regulates autophagy via the NAD-dependent deacetylase SIRT1
<p>Macroautophagy/autophagy is an important intracellular mechanism for the maintenance of cellular homeostasis. Here we show that the <i>CERKL</i> (ceramide kinase like) gene, a retinal degeneration (RD) pathogenic gene, plays a critical role in regulating autophagy by stabilizing SIRT1. <i>In vitro</i> and <i>in vivo</i>, suppressing CERKL results in impaired autophagy. SIRT1 is one of the main regulators of acetylation/deacetylation in autophagy. In CERKL-depleted retinas and cells, SIRT1 is downregulated. ATG5 and ATG7, 2 essential components of autophagy, show a higher degree of acetylation in CERKL-depleted cells. Overexpression of SIRT1 rescues autophagy in CERKL-depleted cells, whereas CERKL loses its function of regulating autophagy in SIRT1-depleted cells, and overexpression of CERKL upregulates SIRT1. Finally, we show that CERKL directly interacts with SIRT1, and may regulate its phosphorylation at Ser27 to stabilize SIRT1. These results show that CERKL is an important regulator of autophagy and it plays this role by stabilizing the deacetylase SIRT1.</p
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