27 research outputs found

    Comparison and flowering valuation of New Guinea Impatiens cultivars from Sonic and Super Sonic groups

    No full text
    In the years 2002-2003 the flowering of four New Guinea Impatiens cultivars from Sonic and Super Sonic groups were compared. They were the following cultivars: 'Sonic Pink', 'Sonic Sweet Cherry', 'Super Sonic Cherry Cream' and 'Super Sonic Hot Pink'. The experiments were carried out from the middle of April till the middle of October. Neutralised sphagnum peat with slow release fertiliser Osmocote Plus 5/6 was used as medium. It was found that the cultivar 'Sonic Pink' began blooming 1-4 weeks earlier than the other cultivars. The cultivars 'Sonic Sweet Cherry' and 'Super Sonic Cherry Cream' had significantly more abundant flowering (105.3-113.3 flowers per plant) than the cultivars 'Sonic Pink' and 'Super Sonic Hot Pink' (72.0-92.8 flowers per plant). All the cultivars had big flowers (6.3-7.8 cm in diameter). The most similar flowers were found in 'Sonic Sweet Cherry' and the least similar in 'Super Sonic Hot Pink'.W latach 2002-2003 oceniano kwitnienie czterech odmian niecierpka nowo- gwinejskiego z grup Sonic i Super Sonic. Były to: 'Sonic Pink', 'Sonic Sweet Cherry',' Super Sonic Cherry Cream' i 'Super Sonic Hot Pink'. Doświadczenia prowadzono od połowy kwietnia do połowy października. Jako podłoże stosowano odkwaszony torf wysoki z dodatkiem nawozu o działaniu spowolnionym Osmocote Plus 5/6. Stwierdzono, że odmiana 'Sonic Pink' zaczęła kwitnąć o 1 4 tygodnie wcześniej niż pozostałe odmiany. Odmiany z grupy Sonic najwięcej kwiatów miały w lipcu, a odmiany z grupy Super Sonic w sierpniu. Odmiany 'Sonic Sweet Cherry' i ' Super Sonic Cherry Cream kwitły istotnie obficiej (105,3-113,3 kwiatów na roślinie) niż odmiany 'Sonic Pink i 'Super Sonic Hot Pink' (72,0-92,8 kwiatów na roślinie). Wszystkie odmiany miały bardzo duże kwiaty (o średnicy 6,3 7,8 cm). Najbardziej wyrównane kwiaty miała odmiana 'Sonic Sweet Cherry', a najmniej 'Super Sonic Hot Pink'

    Wpływ podłoża i nawożenia na cechy morfologiczne i walory odmian niecierpka nowogwinejskiego z grupy Sonic i z grupy Super Sonic. Część I. Wzrost i pokrój roślin

    No full text
    Dwanaście odmian niecierpka nowogwinejskiego z grupy Sonic i dwanaście odmian niecierpka nowogwinejskiego (NGI) z grupy Super Sonic, hodowli niemieckiej firmy Fischer, uprawiano w pięciu podłożach, którymi były: torf wysoki, odkwaszony kredą i dolomitem do pH 6,2, z dodatkiem nawozu o działaniu spowolnionym Osmocote Plus 5 - 6 w dawce 5 g·dm⁻³; odkwaszony torf wysoki, z dodatkiem Osmocote Plus 5 - 6 w dawce 2,5 g·dm⁻³; trzy gotowe podłoża ogrodnicze (TG, TM D 400, TM B 400), produkcji niemieckiej firmy Stender. Wzrost i pokrój roślin zmieniał się w trakcie wegetacji przede wszystkim w zależności od zasobności podłoża w składniki pokarmowe. Spośród gotowych podłoży najlepsze wyniki uzyskano w podłożu TG. Najpóźniej, bo dopiero w czwartym-piątym miesiącu uprawy, u odmian NGI uprawianych w odkwaszonym torfie wysokim tylko z dodatkiem Osmocote Plus 5 - 6 w dawce 5 g·dm⁻³, bez dokarmiania w trakcie uprawy, zaobserwowano zmiany w pokroju i ulistnieniu świadczące o niedoborze składników pokarmowych.Twelve cultivars of NGI belonging to the Sonic group and twelve cultivars belonging to the Super Sonic group from the German breeding firm Fisher, were cultivated in five media: sphagnum peat, neutralised with chalk and dolomite to pH 6.2, supplemented with a slow - released fertilizer Osmocote Plus 5 - 6 at the dose of 5 g·dm⁻³; neutralised sphagnum peat with Osmocote Plus 5 - 6 at the dose of 2.5 g·dm⁻³; three media (CTG, TM D 400, TM B 400) produced by the German firm Stender. The growth and conformation of plants in the course of vegetation depend on the nutrient resources of the medium. The best results were obtained in plants growing on TG. Changes in plant conformation and foliage, indicating nutrient deficiency, were observed very late, in the fourth-fifth month of cultivation, in some NGI cultivars growing in sphagnum peat with 5 g·dm⁻³ of Osmocote Plus 5 - 6 without the additional application during the cultivation

    Activation of the cation channel TRPM3 in perivascular nerves induces vasodilation of resistance arteries

    Get PDF
    The Transient Receptor Potential Melastatin 3 (TRPM3) is a Ca2+-permeable non-selective cation channel activated by the neurosteroid pregnenolone sulfate (PS). This compound was previously shown to contract mouse aorta by activating TRPM3 in vascular smooth muscle cells (VSMC), and proposed as therapeutic modulator of vascular functions. However, PS effects and the role of TRPM3 in resistance arteries remain unknown. Thus, we aimed at determining the localization and physiological role of TRPM3 in mouse mesenteric arteries. Real-time qPCR experiments, anatomical localization using immunofluorescence microscopy and patch-clamp recordings in isolated VSMC showed that TRPM3 expression in mesenteric arteries is restricted to perivascular nerves. Pressure myography experiments in wild type (WT) mouse arteries showed that PS vasodilates with a concentration-dependence that was best fit by two Hill components (effective concentrations, EC50, of 14 and 100 μM). The low EC50 component was absent in preparations from Trpm3 knockout (KO) mice and in WT arteries in the presence of the CGRP receptor antagonist BIBN 4096. TRPM3-dependent vasodilation was partially inhibited by a cocktail of K+ channel blockers, and not mediated by β-adrenergic signaling. We conclude that, contrary to what was found in aorta, PS dilates mesenteric arteries, partly via an activation of TRPM3 that triggers CGRP release from perivascular nerve endings and a subsequent activation of K+ channels in VSMC. We propose that TRPM3 is implicated in the regulation of the tone of resistance arteries and that its activation by yet unidentified endogenous damage-associated molecules lead to protective vasodilation responses in mesenteric arteries.This work was supported by grants from the Fund for Scientific Research Flanders (FWO, G.0C68.15, Belgium) to KT, the Ministerio de Economía y Competitividad (BFU2013-45867-R and BFU2016-75360-R, Spain) to JRLL and MTPG and Junta de Castilla y León(VA114P17, Spain) to MTPG. Y.A.A. is a Postdoctoral Fellow of the FWO (Belgium).Peer reviewe

    Type-I interferon signaling through ISGF3 complex is required for sustained Rip3 activation and necroptosis in macrophages

    Get PDF
    Myeloid cells play a critical role in perpetuating inflammation during various chronic diseases. Recently the death of macrophages through programmed necrosis (necroptosis) has emerged as an important mechanism in inflammation and pathology. We evaluated the mechanisms that lead to the induction of necrotic cell death in macrophages. Our results indicate that type I IFN (IFN-I) signaling is a predominant mechanism of necroptosis, because macrophages deficient in IFN-α receptor type I (IFNAR1) are highly resistant to necroptosis after stimulation with LPS, polyinosinic-polycytidylic acid, TNF-α, or IFN-β in the presence of caspase inhibitors. IFN-I-induced necroptosis occurred through both mechanisms dependent on and independent of Toll/IL-1 receptor domain-containing adaptor inducing IFN-β (TRIF) and led to persistent phosphorylation of receptor-interacting protein 3 (Rip3) kinase, which resulted in potent necroptosis. Although various IFN-regulatory factors (IRFs) facilitated the induction of necroptosis in response to IFN-β, IRF-9-STAT1- or -STAT2-deficient macrophages were highly resistant to necroptosis. Our results indicate that IFN-β-induced necroptosis of macrophages proceeds through tonic IFN-stimulated gene factor 3 (ISGF3) signaling, which leads to persistent expression of STAT1, STAT2, and IRF9. Induction of IFNAR1/Rip3-dependent necroptosis also resulted in potent inflammatory pathology in vivo. These results reveal how IFN-I mediates acute inflammation through macrophage necroptosis.status: publishe

    Flexible photonics: Transform rigid materials into mechanically flexible and optically functional systems

    No full text
    none19siAs already done in electronics, passive and active photonic devices demand integration on flexible substrates for a broad spectrum of application ranging from optical interconnection to sensors for civil infrastructure and environments, to coherent and uncoherent light sources and functionalized coatings for integration on biological tissue. In this communication we will present some recent results concerning the fabrication of novel flexible optical layers by sol-gel and radio frequency sputtering deposition techniques. The perspective is to give a technological way to transform intrinsically rigid or brittle materials into a highly mechanically flexible and optically functional systems.noneChiasera A.; Szczurek A.; Tran L.T.N.; Startek K.; Saynger O.; Varas S.; Armellini C.; Chiappini A.; Carpentiero A.; Zonta D.; Bursi O.S.; Ramponi R.; Bollani M.; Scotognella F.; MacRelli G.; Krzak J.; Righini G.C.; Ferrari M.; Lukowiak A.Chiasera, A.; Szczurek, A.; Tran, L. T. N.; Startek, K.; Saynger, O.; Varas, S.; Armellini, C.; Chiappini, A.; Carpentiero, A.; Zonta, D.; Bursi, O. S.; Ramponi, R.; Bollani, M.; Scotognella, F.; Macrelli, G.; Krzak, J.; Righini, G. C.; Ferrari, M.; Lukowiak, A
    corecore