112 research outputs found
Erythropoietin Amplifies Stroke-Induced Oligodendrogenesis in the Rat
- Author
- A Nishiyama
- A Polito
- AN Verity
- B Menn
- C Lappe-Siefke
- CK Petito
- D Dewar
- D Sargin
- G Paxinos
- HS Keirstead
- J Chen
- J Lee
- J Zhang
- JA Paterson
- JE Gotts
- Jing Zhang
- JK Ness
- JM Gensert
- JW Zhao
- K Imamoto
- K Mandai
- K Mizuno
- K Tanaka
- L Li
- L Pantoni
- L Vitellaro-Zuccarello
- L Wang
- L Wang
- Lei Wang
- Li Zhang
- M Iwai
- M Iwai
- M Komitova
- M Santra
- M Sugawa
- M Wiznerowicz
- MA van der Kooij
- Manoranjan Santra
- MB Mathews
- Mei Lu
- Mel B. Feany
- Michael Chopp
- N Scolding
- NS Roy
- O Gonzalez-Perez
- PJ Horner
- PS Yam
- Q Jiang
- R Gregersen
- R Reynolds
- RL Zhang
- RL Zhang
- Rui Lan Zhang
- S Belachew
- SE Pfeiffer
- SM LeVine
- SW Levison
- TD Hernandez
- WM Carroll
- WT Norton
- Y Taniguchi
- Yier Toh
- Ying Wang
- Zheng Gang Zhang
- Publication venue
- Public Library of Science
- Publication date
- 01/06/2010
- Field of study
Erythropoietin (EPO), a hematopoietic cytokine, enhances neurogenesis and angiogenesis during stroke recovery. In the present study, we examined the effect of EPO on oligodendrogenesis in a rat model of embolic focal cerebral ischemia.Recombinant human EPO (rhEPO) at a dose of 5,000 U/kg (n = 18) or saline (n = 18) was intraperitoneally administered daily for 7 days starting 24 h after stroke onset. Treatment with rhEPO augmented actively proliferating oligodendrocyte progenitor cells (OPCs) measured by NG2 immunoreactive cells within the peri-infarct white matter and the subventricular zone (SVZ), but did not protect against loss of myelinating oligodendrocytes measured by cyclic nucleotide phosphodiesterase (CNPase) positive cells 7 days after stroke. However, 28 and 42 days after stroke, treatment with rhEPO significantly increased myelinating oligodendrocytes and myelinated axons within the peri-infarct white matter. Using lentivirus to label subventricular zone (SVZ) neural progenitor cells, we found that in addition to the OPCs generated in the peri-infarct white matter, SVZ neural progenitor cells contributed to rhEPO-increased OPCs in the peri-infarct area. Using bromodeoxyuridine (BrdU) for birth-dating cells, we demonstrated that myelinating oligodendrocytes observed 28 days after stroke were derived from OPCs. Furthermore, rhEPO significantly improved neurological outcome 6 weeks after stroke. In vitro, rhEPO increased differentiation of adult SVZ neural progenitor cells into oligodendrocytes and enhanced immature oligodendrocyte cell proliferation.Our in vivo and in vitro data indicate that EPO amplifies stroke-induced oligodendrogenesis that could facilitate axonal re-myelination and lead to functional recovery after stroke
Effects and acceptability of implementing improved cookstoves and heaters to reduce household air pollution: a FRESH AIR study
- Author
- J.F.M. van boven
- L.T. van
- Akylbekov A.
- An P.L.
- Anastasaki M.
- Barton A.
- Bertsias A.
- Binh P.D.U.
- Brakema E.A.
- Burges D.
- Buteme S.
- Cartwright L.
- Chartier R.
- Chatzea V.E.
- Chavannes N.H.
- Cragg L.
- Dang T.N.
- Emilov B.
- Ferarrio I.
- FRESH AIR
- Gemert F. van
- Hedrick B.
- Hong L.H.T.C.
- Hopkinson N.
- Jones R.
- Jong C. de
- Kampen S. van
- Katagira W.
- Kirenga B.
- Kjaergaard J.
- Kleij R.M.J.J. van der
- Kocks J.
- Lan L.T.T.
- Linh T.T.D.
- Lionis C.
- Loan K.X.
- Mademilov M.
- McEwen A.
- Musinguzi P.
- Nantanda R.
- Ndeezi G.
- Papadakis S.
- Pinnock H.
- Pooler J.
- Poot C.
- Postma M.J.
- Poulsen A.
- Powell P.
- Quynh N.N.
- Reventlow S.
- Sifaki-Pistolla D.
- Singh S.
- Sooronbaev T.
- Sousa J.C. de
- Stout J.
- Stubbe-Ostergaard M.
- Tabyshova A.
- Tsiligianni I.
- Tuan T.D.
- Tumwine J.
- Vinh N.N.
- Walusimbi S.
- Warren L.
- Williams S.
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2019
- Field of study
The objective was to evaluate the effectiveness and acceptability of locally tailored implementation of improved cookstoves/heaters in low- and middle-income countries. This interventional implementation study among 649 adults and children living in rural communities in Uganda, Vietnam and Kyrgyzstan, was performed after situational analyses and awareness programmes. Outcomes included household air pollution (PM2.5 and CO), self-reported respiratory symptoms (with CCQ and MRC-breathlessness scale), chest infections, school absence and intervention acceptability. Measurements were conducted at baseline, 2 and 6-12 months after implementing improved cookstoves/heaters. Mean PM2.5 values decrease by 31% (to 95.1 µg/m3) in Uganda (95%CI 71.5-126.6), by 32% (to 31.1 µg/m3) in Vietnam (95%CI 24.5-39.5) and by 65% (to 32.4 µg/m3) in Kyrgyzstan (95%CI 25.7-40.8), but all remain above the WHO guidelines. CO-levels remain below the WHO guidelines. After intervention, symptoms and infections diminish significantly in Uganda and Kyrgyzstan, and to a smaller extent in Vietnam. Quantitative assessment indicates high acceptance of the new cookstoves/heaters. In conclusion, locally tailored implementation of improved cookstoves/heaters is acceptable and has considerable effects on respiratory symptoms and indoor pollution, yet mean PM2.5 levels remain above WHO recommendations.European Union’s Horizon 2020 programme under grant agreement no. 680997, TRIAL ID NTR5759, http://www.trialregister.nl/trialreg/admin/rctsearch.asp?Term=23332. The devices, measuring the personal HAP, were funded by Netherlands Enterprise Agency (RVO
Shortened Telomere Length Is Associated with Increased Risk of Cancer: A Meta-Analysis
- Author
- A Benetos
- A Smogorzewska
- Alison M. Dunning
- AM Joshua
- AM Valdes
- Amanda Ewart Toland
- BM Unryn
- CB Harley
- CM Raynaud
- EH Blackburn
- EH Blackburn
- ES Epel
- Göran Roos
- H der-Sarkissian
- H. Dean Hosgood
- HD Hosgood 3rd
- HJ Sommerfeld
- Hongxia Ma
- HR Kim
- HX An
- I De Vivo
- IK Moon
- IM Lee
- J Barwell
- J Han
- J Lau
- J Prescott
- J Shen
- J Shen
- J Xing
- JA Van Ziffle
- JP Higgins
- JS Jang
- K Broberg
- K Nordfjall
- KA Pooley
- Karen A. Pooley
- KK Wong
- L Fern
- L Hou
- L Mirabello
- L Mirabello
- L Shao
- LA Panossian
- M Baudis
- M Egger
- M Kammori
- M McGrath
- MA Blasco
- MA Blasco
- Min Shen
- MM Gramatges
- MT Hemann
- N Mantel
- NF Mathon
- PE Slagboom
- Q Lan
- Qingyi Wei
- R Capper
- R DerSimonian
- RA Risques
- RC Allsopp
- RY Zee
- S Brouilette
- SA Stewart
- SE Artandi
- SE Artandi
- Sheng Wei
- SM Gadalla
- SU Wiemann
- T Idei
- T von Zglinicki
- T von Zglinicki
- T von Zglinicki
- U Friedrich
- U Svenson
- Ulrika Svenson
- VG Gorgoulis
- X Liu
- X Wu
- Y Kinouchi
- YL Zheng
- Zhensheng Liu
- Ziyuan Zhou
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2011
- Field of study
BACKGROUND: Telomeres play a key role in the maintenance of chromosome integrity and stability, and telomere shortening is involved in initiation and progression of malignancies. A series of epidemiological studies have examined the association between shortened telomeres and risk of cancers, but the findings remain conflicting. METHODS: A dataset composed of 11,255 cases and 13,101 controls from 21 publications was included in a meta-analysis to evaluate the association between overall cancer risk or cancer-specific risk and the relative telomere length. Heterogeneity among studies and their publication bias were further assessed by the χ(2)-based Q statistic test and Egger's test, respectively. RESULTS: The results showed that shorter telomeres were significantly associated with cancer risk (OR = 1.35, 95% CI = 1.14-1.60), compared with longer telomeres. In the stratified analysis by tumor type, the association remained significant in subgroups of bladder cancer (OR = 1.84, 95% CI = 1.38-2.44), lung cancer (OR = 2.39, 95% CI = 1.18-4.88), smoking-related cancers (OR = 2.25, 95% CI = 1.83-2.78), cancers in the digestive system (OR = 1.69, 95% CI = 1.53-1.87) and the urogenital system (OR = 1.73, 95% CI = 1.12-2.67). Furthermore, the results also indicated that the association between the relative telomere length and overall cancer risk was statistically significant in studies of Caucasian subjects, Asian subjects, retrospective designs, hospital-based controls and smaller sample sizes. Funnel plot and Egger's test suggested that there was no publication bias in the current meta-analysis (P = 0.532). CONCLUSIONS: The results of this meta-analysis suggest that the presence of shortened telomeres may be a marker for susceptibility to human cancer, but single larger, well-design prospective studies are warranted to confirm these findings
A922 Sequential measurement of 1 hour creatinine clearance (1-CRCL) in critically ill patients at risk of acute kidney injury (AKI)
- Author
- Abbott TE
- Abdallah RI
- Abdel-Aal IR
- Abdelmonem SA
- Abdo WF
- Abellan AN
- Abellán AN
- Abellán AN
- Abellán AN
- Abrams ST
- Ackerley C
- Acuña M
- Adda M
- Adhikari NK
- Adriano G
- Adrie C
- Affatato R
- Affatato R
- Afonso-Rivero D
- Agarossi A
- Agarwal LK
- Ageeva T
- Agrawal R
- Aguayo-DeHoyos E
- Aguilar AL
- Aguilar-Alonso E
- Aguilar-Alonso E
- Aguilar-Alonso E
- Aguirregabyria M
- Ahmadnia E
- Ahn C
- Ahn JY
- Ahn JY
- Ahn MY
- Ahn MY
- Aitken LM
- Akalaev R
- Akbarzadeh A
- Akcan-Arikan A
- Akhlagh SH
- Akhtar N
- AKI Research Group
- Akiduki N
- Akin S
- Akizuki N
- Akkoc T
- Ala-Kokko T
- Alanio A
- Albaiceta GM
- Albaladejo P
- Alberto F
- Albuisson E
- Alcala MA
- Alcázar L
- Aldabo-Pallas T
- Aldana NN
- Aldecoa C
- Alegría L
- Alejandro R
- Alejo GC
- Alejos RM
- Alexandrova E
- Algarte R
- Algarte R
- Algieri I
- Algora-Weber A
- Alhamdi Y
- Aliaga FA
- Aliberti S
- Alkan A
- Almudena PM
- Almudevar PM
- Almudevar PM
- Almudévar PM
- Altıntas ND
- Alvarez J
- Alves SC
- Alzola LM
- Amargianitakis V
- Amaro R
- Amato MB
- Ambler M
- Amerongen MP
- Amininejad T
- Amor LL
- Amorim V
- Anand RK
- Andersen LW
- Andersson S
- Andoh K
- Andrade AH
- Andrade AH
- Andrade AH
- Andrade G
- Andreis DT
- Andreis DT
- Annane D
- Antonelli M
- Antoniadou A
- Antonio C
- Antonucci E
- Antón DG
- Anzueto A
- Appiani F
- Aquevedo A
- Aragon C
- Araujo NJ
- Araujo VF
- ARIAM registry of adult cardiac surgery
- ARIAM-ANDALUCIA
- Arias CC
- Arias N
- Arias-Verdu MD
- Arias-Verdu MD
- Arias-Verdu MD
- Armaganidis A
- Arora MK
- Arora N
- Arrigo M
- Arslantas MK
- Artiguenave M
- Aslanian P
- Aspide R
- Asyralyyeva G
- Ataman S
- Ataman S
- Atchade E
- Atchade E
- Atchasiri S
- Athapattu P
- Atkins G
- Aublanc M
- Aublanc M
- Augustin P
- Augustin P
- Auquier P
- Avilés-Jurado FX
- Avramidis V
- Ayala LY
- Aydin M
- Azevedo JC
- Azevedo JR
- Aznar GP
- Babalis D
- Babini G
- Bachli EB
- Bader A
- Bader A
- Baena J
- Baidya DK
- Baikova VN
- Bakker J
- Balcázar LC
- Balcázar LC
- Baldassarri R
- Baldwin CE
- Balik M
- Ball I
- Ballin A
- Balsera B
- Banderas-Bravo ME
- Banderas-Bravo ME
- Bandert A
- Bangstad IL
- Baptista N
- Baquero JD
- Barbadillo S
- Barberet G
- Barberet G
- Barberet G
- Barbier J
- Barcenilla F
- Barea-Mendoza JA
- Barinas OD
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- Barral M
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- Bassi GL
- Bates D
- Battistini M
- Bauer M
- Bauer PR
- Baumstarck K
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- Beane A
- Beduneau G
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- Beeharry SD
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- Bernal FF
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- Berthelsen RE
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- Bettex D
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- CAPCRI Study
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- Chernyshuk S
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- Chiurazzi C
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- Cholley B
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- Chouvarda I
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- Chrétien JM
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- Cordeiro CP
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- Dimitroulakis K
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- Dimopoulou IK
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- Dos Santos HP
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- E YY
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- Egginton JS
- Egi M
- El Adawy AS
- El Hamid MA
- El Hossainy R
- El Sayed E
- El Sayed F
- El Sayed I
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- Elbers PW
- Elgendy M
- Eliet J
- Emmanuel J
- Emmerich M
- Encina B
- Endacott R
- Endacott R
- Engelberts D
- ENVIN-HELICS Study Group
- Erb J
- Erdogan M
- Eroles AA
- Escalada SH
- Escalada SH
- Escoval A
- Escribá A
- Escórcio S
- Eslami S
- Espinasse F
- Espinel E
- Estagnasie P
- Esteban A
- Estella A
- Estenssoro E
- Evelein-Brugman S
- Facchin F
- Faivre V
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- Fanzaghi P
- Farias P
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- Feliot E
- Ferez MA
- Fernandes MJ
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- Fernandez NG
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- Fernandez-Zamora MD
- Fernández IF
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- Fernández-Ortega JF
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- Ferré A
- Ferrón FR
- Ferrón FR
- Fiks T
- Fiks T
- Fileković S
- Filho CA
- Filho CA
- Filho CA
- Filipe E
- FINNRESUSCI Study Group
- Fischer G
- Fleischmann E
- Fless K
- Floccard B
- Flowers E
- Follin A
- Follin A
- Fong KY
- Fontaiña LP
- Fontana V
- Fontenot T
- Forel JM
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- Frantzeskaki F
- Frantzeskaki F
- Frat JP
- Frat JP
- Frat JP
- Freile MT
- Frenzel T
- Frigeni B
- Frithiof R
- Fritz C
- FROG ICU Investigators
- Fromentin M
- Frontera PR
- Fuentes M
- Fumagalli F
- Fumagalli F
- Fumis RR
- Fushimi K
- Gabán-Díez Á
- Gadgil S
- Gaide-Chevronnay L
- Gainnier M
- Gaite FB
- Gaizauskas E
- Gajic O
- Galache JA
- Gallego-Corpa AI
- Gameiro J
- Gancedo CH
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- Gando S
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- Gando S
- Gando S
- Gandola A
- Gandolfi S
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- Garcia IP
- Garcia IP
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- Garcia PK
- Garcia-Alvarez JM
- Garcia-Alvarez JM
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- García MF
- García-Martínez MV
- García-Olivares P
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- Gavrychenko D
- Gayat E
- Geantot MA
- Gemmell L
- Gemmell LK
- Genc D
- Genève C
- Georgopoulos D
- Geri G
- GETGAG Working Group
- GETGAG/SEMICYUC
- Ghabina S
- Ghazal S
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- Ghosh S
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- Giannopoulos A
- Giesinger RE
- Gigorro RG
- Gil EM
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- González AS
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- Gonçalves B
- Goodwin S
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- Gordon M
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- Gouveia J
- Gracia RM
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- Grossestreuer A
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- Jardim JJ
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- JSEPTIC (Japanese Society of Education for Physicians and Trainees in Intensive Care) Clinical Trial Group
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- Runge I
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- Rutten AM
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- Sabetian G
- Sabeļņikovs O
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- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2016
- Field of study
Meeting abstrac
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)
- Author
- A. -G. Wu
- A. C. Ma
- A. K. Au
- Abdel-Aziz A. K.
- Abdelfatah S.
- Abdellatif M.
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- Abel S.
- Abeliovich H.
- Abildgaard M. H.
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- Acevedo-Arozena A.
- Adamopoulos I. E.
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- Adornetto A.
- Aflaki E.
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- Publication venue
- 'Informa UK Limited'
- Publication date
- 01/01/2021
- Field of study
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1.
- Author
- Abdel-Aziz AK
- Abdelfatah S
- Abdellatif M
- Abdoli A
- Abel S
- Abeliovich H
- Abildgaard MH
- Abudu YP
- Acevedo-Arozena A
- Adamopoulos IE
- Adeli K
- Adolph TE
- Adornetto A
- Aflaki E
- Agam G
- Agarwal A
- Aggarwal BB
- Agnello M
- Agostinis P
- Agrewala JN
- Agrotis A
- Aguilar PV
- Ahmad ST
- Ahmed ZM
- Ahumada-Castro U
- Aits S
- Aizawa S
- Akkoc Y
- Akoumianaki T
- Akpinar HA
- Al-Abd AM
- Al-Akra L
- Al-Gharaibeh A
- Alaoui-Jamali MA
- Alberti S
- Alcocer-Gómez E
- Alessandri C
- Ali M
- Alim Al-Bari MA
- Aliwaini S
- Alizadeh J
- Almacellas E
- Almasan A
- Alonso A
- Alonso GD
- Altan-Bonnet N
- Altieri DC
- Alves da Costa C
- Alves S
- Alzaharna MM
- Amadio M
- Amantini C
- Amaral C
- Ambrosio S
- Amer AO
- Ammanathan V
- An Z
- Andersen SU
- Andrabi SA
- Andrade-Silva M
- Andres AM
- Angelini S
- Ann D
- Anozie UC
- Ansari MY
- Antas P
- Antebi A
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- Araújo WL
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- Arikkath J
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- Armstrong-James D
- Arnauné-Pelloquin L
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- Arsov I
- Artero R
- Arévalo M-A
- Asaro DML
- Aschner M
- Ashrafizadeh M
- Ashur-Fabian O
- Atanasov AG
- Au AK
- Auberger P
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- Autelli R
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- Aveleira CA
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- Zimmermann CM
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- Zorzano A
- Zou W
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- Zwerschke W
- Álvarez ÉMC
- Ávalos Y
- Önal G
- Üstün S
- Čolić M
- Đokić J
- Žerovnik E
- Publication venue
- 'Informa UK Limited'
- Publication date
- 01/01/2021
- Field of study
In 2008, we published the first set of guidelines for standardizing research in autophagy. Since then, this topic has received increasing attention, and many scientists have entered the field. Our knowledge base and relevant new technologies have also been expanding. Thus, it is important to formulate on a regular basis updated guidelines for monitoring autophagy in different organisms. Despite numerous reviews, there continues to be confusion regarding acceptable methods to evaluate autophagy, especially in multicellular eukaryotes. Here, we present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes. These guidelines are not meant to be a dogmatic set of rules, because the appropriateness of any assay largely depends on the question being asked and the system being used. Moreover, no individual assay is perfect for every situation, calling for the use of multiple techniques to properly monitor autophagy in each experimental setting. Finally, several core components of the autophagy machinery have been implicated in distinct autophagic processes (canonical and noncanonical autophagy), implying that genetic approaches to block autophagy should rely on targeting two or more autophagy-related genes that ideally participate in distinct steps of the pathway. Along similar lines, because multiple proteins involved in autophagy also regulate other cellular pathways including apoptosis, not all of them can be used as a specific marker for bona fide autophagic responses. Here, we critically discuss current methods of assessing autophagy and the information they can, or cannot, provide. Our ultimate goal is to encourage intellectual and technical innovation in the field
COPD’s early origins in low-and-middle income countries: what are the implications of a false start?
- Author
- J.F.M. van boven
- L.T. van
- Akmatalieva M.
- Akylbekov A.
- An P.L.
- Anastasaki M.
- Barton A.
- Bertsias A.
- Binh P.D.U.
- Brakema E.A.
- Burges D.
- Cartwright L.
- Chatzea V.E.
- Chavannes N.H.
- Cragg L.
- Dang T.N.
- Dautov I.
- Emilov B.
- Ferarrio I.
- FRESH AIR Collaborators
- Gemert F.A. van
- Hedrick B.
- Hong L.H.T.C.
- Hopkinson N.
- Isaeva E.
- Jones R.
- Jong C. de
- Kampen S. van
- Katagira W.
- Kirenga B.
- Kjaergaard J.
- Kleij R.M.J.J. van der
- Kocks J.
- Lan L.T.T.
- Linh T.T.D.
- Lionis C.
- Loan K.X.
- Mademilov M.
- McEwen A.
- Musinguzi P.
- Nantanda R.
- Ndeezi G.
- Papadakis S.
- Pinnock H.
- Pooler J.
- Poot C.
- Postma M.J.
- Poulsen A.
- Powell P.
- Puhan M.
- Quynh N.N.
- Reventlow S.
- Salvi S.
- Sifaki-Pistolla D.
- Singh S.
- Sooronbaev T.
- Sousa J.C. de
- Stout J.
- Stubbe-Ostergaard M.
- Tabyshova A.
- Tsiligianni I.
- Tuan T.D.
- Tumwine J.
- Vinh N.N.
- Walusimbi S.
- Warren L.
- Williams S.
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2019
- Field of study
[Excerpt] The Global Initiative for chronic Obstructive Lung disease (GOLD)guideline of 2018 describes COPD as‘the result of a complexinterplay of long-term cumulative exposure to noxious gases andparticles, combined with a variety of host factors includinggenetics, airway hyper-responsiveness and poor lung growthduring childhood’.1Tobacco smoking is traditionally viewed as themain contributing factor to the development of COPD. However,COPD also occurs among non-smokers, especially in low-incomeand middle-income countries (LMICs).2,3Notably, more than 90%of COPD-related deaths occur in LMICs.4For these countries, otherrisk factors, such as ambient, occupational and household airpollution play a significant role in the development of COPD.1,2,5–7Does COPD in these settings have a different pathophysiologicaltrajectory compared to COPD in high-income countries, and if so:what does this imply?In normal lung development, airway branching is completed bythe 17th week of gestation, after which airways increase in volumeuntil young adulthood. Alveoli are present at birth and developfurther during childhood. Lung volume and airflow continue toincrease as the thorax grows, influenced by age, sex, and ethnicity,reaching a peak at young adulthood. Lung function then remainsconstant for about 10 years (the plateau phase), after which itgradually declines.8In the‘classic’COPD patient, the decline inlung function is more rapid than in healthy individuals. However,in a considerable proportion of COPD patients, lung function doesnot decline rapidly, but reaches a lower plateau phase in earlyadulthood instead. For these patients, a completely differentpathophysiological trajectory seems to lead to the diagnosis ofCOPD: the decline in lung function follows a normal pattern, yetthey seem to have a‘false start’by attaining a lower maximumlung function. [...
Ion formation upon electron collisions with valine embedded in helium nanodroplets
- Author
- A. Mauracher
- A. Mauracher
- A. Mauracher
- A. Mauracher
- A.F. Lago
- A.W. Hauser
- Alexander Kaiser
- B. Bellina
- B.C. Ibǎnescu
- C. Leidlmair
- Diethard K. Böhme
- E. Alizadeh
- F. Bierau
- F. Ferreira da Silva
- F. Ferreira da Silva
- F. Ferreira da Silva
- F. Zappa
- H. Dögg Flosadottir
- H. Schöbel
- H.-W. Jochims
- I. Baccarelli
- J. Postler
- J.-C. Poully
- J.P. Toennies
- K. Martini
- L. An der Lan
- L.F. Gomez
- M. Allan
- M. Goulart
- M. Neustetter
- M. Renzler
- M. Rosenblit
- M. Rosenblit
- M.A. Śmiałek
- Martina Harnisch
- Michael Renzler
- N.B. Brauer
- Nikolaus Weinberger
- O. Echt
- P. Papp
- P. Papp
- Paul Scheier
- R. Janečková
- R. Sliter
- S. Denifl
- S. Denifl
- S. Denifl
- S. Denifl
- S. Maclot
- S. Ralser
- S.E. Huber
- S.F. Yang
- Stefan Denifl
- Stefan Ralser
- T. Schlathölter
- U. Henne
- V. Vorsa
- W. Kong
- W. Wei
- X. Zhang
- Y. Hu
- Y. Ren
- Y. Ren
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Perspectives on the use of transcriptomics to advance biofuels
- Author
- Ahn J-M Mitchell RJ, Gu MB
- Alexandre H Ansanay-Galeote V, Dequin S, et al.
- Alsaker KV Papoutsakis ET
- Alsaker KV Spitzer TR, Papoutsakis ET
- Atsumi S Hanai T, Liao JC
- Bandounas L Wierckx NJP, de Winde JH, et al.
- Barbazuk WB Emrich SJ, Chen HD, et al.
- Bayer EA Belaich J-P, Shoham Y, et al.
- Bhaganna P Volkers RJ, Bell AN, et al.
- Chabannes M Ruel K, Yoshinaga A, et al.
- Chen CY Huang YC, Wei CM, et al.
- Chen F Dixon RA
- Cheung F Haas BJ, Goldberg SM, et al.
- Choi SH Gu MB
- Choi SH Gu MB
- Copley SD Rokicki J, Turner P, et al.
- Corbisier P van der Lelie D, Borremans B, et al.
- Cray JA Stevenson A, Ball P, et al.
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- de Lima Alves F Stevenson A, Baxter E, et al.
- Deng Y Fong SS
- Ezeji T Blaschek HP
- Ezeji T Qureshi N, Blaschek HP
- Feinberg L Foden J, Barrett T, et al.
- Fischer CR Klein-Marcuschamer D, Stephanopoulos
- Fitzgerald D Stratford M, Gasson M, et al.
- Garbelotto M Guglielmo F, Mascheretti S, et al.
- Gaskell J Marty A, Mozuch M, et al.
- Grimmler C Janssen H, Krauβe D, et al.
- Guo T He A, Du T, et al.
- Guo W Jia W, Li Y, et al.
- Guo X Cavka A, Jonsson LJ, et al.
- Hallsworth JE
- Hallsworth JE Heim S, Timmis KN
- Hallsworth JE Prior BA, Nomura Y, et al.
- Hooda R Bhardwaj NK, Singh P
- Hori C Ishida T, Igarashi K, et al.
- Hu S Zheng H, Gu Y, et al.
- Jin Y Fang Y, Huang M, et al.
- Kalluri UC Keller M
- Kim HJ Lee S, Kim J, et al.
- Klinke HB Thomsen AB, Ahring BK
- Knoshaug EP Zhang M
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- Kumari R Pramanik K
- Lan EI Liao JC
- Larsson S Nilvebrant N, Jönsson L
- Lee S Lee JH, Mitchell RJ
- Lee S Mitchell RJ
- Lee S Monnappa AK, Mitchell RJ
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- Lütke-Eversloh T Bahl H
- MacDonald J Doering M, Canam T, et al.
- Macdonald J Master ER
- Machado IM Atsumi S
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- Mills TY Sandoval NR, Gill RT
- Mitchell RJ Gu MB
- Mitchell RJ Gu MB
- Mitchell RJ Gu MB
- Monnappa AK Lee JH, Mitchell RJ
- Monnappa AK Lee S, Mitchell RJ
- Nilvebrant NO Reimann A, Larsson S, et al.
- Oudshoorn A van der Wielen LA, Straathof AJ
- Pauly M Keegstra K
- Penuelas J Carnicer J
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- Shi Z Blaschek HP
- Stead D
- Stevenson DM Weimer PJ
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- Tomas CA Beamish J, Papoutsakis ET
- Tomas CA Welker NE, Papoutsakis ET
- Van Acker R Leple JC, Aerts D, et al.
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- Winkler J Kao KC
- Wong DW
- Wong MM Cannon CH, Wickneswari R
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- You KM Rosenfield C-L, Knipple DC
- Zhang Y Ezeji TC
- Zubieta C Krishna SS, Kapoor M, et al.
- Publication venue
- 'American Institute of Mathematical Sciences (AIMS)'
- Publication date
- 01/01/2015
- Field of study
As a field within the energy research sector, bioenergy is continuously expanding. Although much has been achieved and the yields of both ethanol and butanol have been improved, many avenues of research to further increase these yields still remain. This review covers current research related with transcriptomics and the application of this high-throughput analytical tool to engineer both microbes and plants with the penultimate goal being better biofuel production and yields. The initial focus is given to the responses of fermentative microbes during the fermentative production of acids, such as butyric acid, and solvents, including ethanol and butanol. As plants offer the greatest natural renewable source of fermentable sugars within the form of lignocellulose, the second focus area is the transcriptional responses of microbes when exposed to plant hydrolysates and lignin-related compounds. This is of particular importance as the acid/base hydrolysis methods commonly employed to make the plant-based cellulose available for enzymatic hydrolysis to sugars also generates significant amounts of lignin-derivatives that are inhibitory to fermentative bacteria and microbes. The article then transitions to transcriptional analyses of lignin-degrading organisms, such as Phanerochaete chrysosporium, as an alternative to acid/base hydrolysis. The final portion of this article will discuss recent transcriptome analyses of plants and, in particular, the genes involved in lignin production. The rationale behind these studies is to eventually reduce the lignin content present within these plants and, consequently, the amount of inhibitors generated during the acid/base hydrolysis of the lignocelluloses. All four of these topics represent key areas where transcriptomic research is currently being conducted to identify microbial genes and their responses to products and inhibitors as well as those related with lignin degradation/formation.clos
Inhibition of protein ubiquitination by paraquat and 1-methyl-4-phenylpyridinium impairs ubiquitin-dependent protein degradation pathways
- Author
- A Anandhan
- A Chorfa
- A Chorfa
- A Garcia-Garcia
- A Jong de
- A Lau
- A Yamamuro
- AB Manning-Bog
- AB Manning-Bog
- AM Choi
- AM Cuervo
- AN Carvalho
- Annadurai Anandhan
- Annemieke de Jong
- B Caneda-Ferron
- B Fauvet
- Betzabet Quintanilla-Vega
- Bo Yarabe
- BP Monia
- BY Zeng
- C Cook
- C Dixon
- C Klein
- C Liu
- C Marques
- C Oh
- CL Simms
- CM Tanner
- CR Berkers
- CS Cho
- D Ebrahimi-Fakhari
- D Lan
- D Martins-Branco
- D Petroi
- D Scholz
- D Yao
- DC Rubinsztein
- DJ Klionsky
- DM Sampathu
- DW Miller
- E Gomez-Tortosa
- E Kuusisto
- E Kuusisto
- Erin Bradley
- F Buttgereit
- F Fornai
- F Kamel
- F Liu
- F Meier
- F Shang
- F Shang
- F Shang
- F Sun
- F Sun
- F Yang
- FK Lee
- FM Cunha da
- G Asher
- G Srivastava
- GK Tofaris
- GK Tofaris
- H Jang
- H Rodriguez-Rocha
- H Rodriguez-Rocha
- H Sawada
- HA Lashuel
- Huib Ovaa
- I Ahmed
- Iryna Bohovych
- J Bove
- J Choi
- J Fang
- J Gal
- J Hanna
- J Jahngen-Hodge
- J Klucken
- J Lim
- J Ling
- J Long
- J Navarro-Yepes
- J Trinh
- J Wills
- JB Martins
- JF Linares
- JG Lee
- JH Kordower
- JH Stack
- JL Webb
- JM Gorell
- JR Babu
- JT Lee
- Juliana Navarro-Yepes
- JX Song
- K Okada
- K Prasad
- K Tanaka
- K Tanji
- K Wakabayashi
- K Zatloukal
- KK Chung
- KS McNaught
- KS McNaught
- KS McNaught
- L Wang
- L Yang
- LG Friedman
- LJ Corcoran
- M Caballero
- M Haj-Yahya
- M Kastle
- M Komatsu
- M Mark van der
- M Obin
- M Shamoto-Nagai
- M Tanaka
- M Zhu
- MF Notter
- MG Spillantini
- MJ Clague
- ML Seibenhener
- MW Wooten
- N Chondrogianni
- N Kadoguchi
- N Myeku
- N Myeku
- N Wei
- NF Bence
- NF Bence
- Oleh Khalimonchuk
- P Corish
- P Goel
- PH Weinreb
- Q Ding
- Q Ding
- Q Ding
- Q Xu
- R Betarbet
- R Endo
- R Shringarpure
- RA Dunlop
- RL Levine
- Rodrigo Franco
- RS Sohal
- S Giordano
- S Kageyama
- S Kuzuhara
- S Lei
- S Luo
- S Mitra
- S Odagiri
- S Shaid
- S Waelter
- S Zucchelli
- SA Mulherkar
- SB Janen
- SM Goldman
- SR Subramaniam
- T Bartels
- T Hara
- T Ishii
- T Ravid
- TA Yacoubian
- U Dettmer
- U Nagaoka
- UB Pandey
- V Khurana
- V Rogov
- VI Korolchuk
- VI Korolchuk
- W Seufert
- W Xie
- W Yang
- WM Caudle
- X Li
- X Li
- X Shan
- X Shan
- X Zhang
- XF Wang
- XM Cotto-Rios
- Y Bang
- Y Chang
- Y Huang
- Y Saito
- Y Watanabe
- Y Watanabe
- Y Yang
- YH Seo
- YM Kim
- You Zhou
- ZI Alam
- Publication venue
- DigitalCommons@University of Nebraska - Lincoln
- Publication date
- 01/01/2016
- Field of study
Intracytoplasmic inclusions of protein aggregates in dopaminergic cells (Lewy bodies) are the pathological hallmark of Parkinson’s disease (PD). Ubiquitin (Ub), alpha [α]-synuclein, p62/sequestosome 1 and oxidized proteins are major components of Lewy bodies. However, the mechanisms involved in the impairment of misfolded/oxidized protein degradation pathways in PD are still unclear. PD is linked to mitochondrial dysfunction and environmental pesticide exposure. In this work, we evaluated the effect of the pesticide paraquat (PQ) and the mitochondrial toxin 1-methyl-4-phenylpyridinium (MPP+) on Ub-dependent protein degradation pathways. No increase in the accumulation of Ub-bound proteins or aggregates was observed in dopaminergic cells (SK-N-SH) treated with PQ or MPP+, or in mice chronically exposed to PQ. PQ decreased Ub protein content, but not its mRNA transcription. Protein synthesis inhibition with cycloheximide depleted Ub levels and potentiated PQ–induced cell death. Inhibition of proteasomal activity by PQ was found to be a late event in cell death progression, and had no effect on either the toxicity of MPP+ or PQ, or the accumulation of oxidized sulfenylated, sulfonylated (DJ-1/PARK7 and peroxiredoxins) and carbonylated proteins induced by PQ. PQ- and MPP+-induced Ub protein depletion prompted the dimerization/inactivation of the Ub-binding protein p62 that regulates the clearance of ubiquitinated proteins by autophagic. We confirmed that PQ and MPP+ impaired autophagy flux, and that the blockage of autophagy by the overexpression of a dominant-negative form of the autophagy protein 5 (dnAtg5) stimulated their toxicity, but there was no additional effect upon inhibition of the proteasome. PQ induced an increase in the accumulation of α-synuclein in dopaminergic cells and membrane associated foci in yeast cells. Our results demonstrate that inhibition of protein ubiquitination by PQ and MPP+ is involved in the dysfunction of Ub-dependent protein degradation pathways
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