30 research outputs found
SMAD3 rs17228212 Gene Polymorphism Is Associated with Reduced Risk to Cerebrovascular Accidents and Subclinical Atherosclerosis in Anti-CCP Negative Spanish Rheumatoid Arthritis Patients
- Author
- A Corrales
- A Willemze
- AG Semb
- Alejandro Balsa
- Alfonso Corrales
- Amr H Sawalha
- Benjamín Fernández-Gutiérrez
- C Gonzalez-Juanatey
- C Gonzalez-Juanatey
- C Shimizu
- Carlos González-Juanatey
- Carmen Gómez-Vaquero
- CS Fox
- CW Lees
- D Aletaha
- DH O'Leary
- DL Scott
- Dora Pascual-Salcedo
- E Naredo
- FC Arnett
- Fernanda Genre
- FJ López-Longo
- Francisco J. López-Longo
- G Kerekes
- GK Hansson
- Isidoro González-Álvaro
- Javier Llorca
- Javier Martín
- Javier Rueda-Gotor
- José A. Miranda-Filloy
- KA Brown
- KK Ho
- L Rodríguez-Rodríguez
- Luis Rodríguez-Rodríguez
- M García-Bermúdez
- MA Gonzalez-Gay
- MA Gonzalez-Gay
- Mercedes García-Bermúdez
- Miguel A. González-Gay
- NA Daha
- NJ Samani
- Patricia Carreira
- PH Dessein
- R López-Mejías
- Raquel López-Mejías
- Ricardo Blanco
- S Viatte
- S Viatte
- Santos Castañeda
- SH Juo
- TW Huizinga
- X Cheng
- Y Tone
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 01/01/2013
- Field of study
Rheumatoid arthritis (RA) is a complex polygenic inflammatory disease associated with accelerated atherosclerosis and increased risk of cardiovascular (CV) disease. Previous genome-wide association studies have described SMAD3 rs17228212 polymorphism as an important signal associated with CV events. The aim of the present study was to evaluate for the first time the relationship between this gene polymorphism and the susceptibility to CV manifestations and its potential association with the presence of subclinical atherosclerosis assessed by the evaluation of carotid intima-media thickness (cIMT) in patients with RA
Influence of elevated-CRP level-related polymorphisms in non-rheumatic Caucasians on the risk of subclinical atherosclerosis and cardiovascular disease in rheumatoid arthritis
- Author
- Alegre Sancho JJ.
- Balsa Criado Alejandro
- Blanco Alonso Ricardo
- Carreira P.
- Castañeda S.
- Corrales A.
- Fernández Gutiérrez B.
- Ferraz Amaro I.
- Genre Fernanda
- González Juanatey Carlos
- González Álvaro Isidoro
- González-Gay Mantecón Miguel Ángel
- Gómez Vaquero C.
- Llorca Díaz Francisco Javier
- López Longo FJ.
- López Mejías R.
- Magro C.
- Martín J.
- Mijares V.
- Miranda Filloy JA.
- Mínguez Sánchez MD.
- Pascual Salcedo D.
- Pina Murcia Trinitario
- Ramírez Huaranga Marco A.
- Raya E.
- Remuzgo Martínez S.
- Robustillo Villarino M.
- Rodríguez Rodríguez Luis
- Tejera Segura B.
- Ubilla B.
- Vicente E.
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2016
- Field of study
Association between elevated C-reactive protein (CRP) serum levels and subclinical atherosclerosis and cardiovascular (CV) events was described in rheumatoid arthritis (RA). CRP, HNF1A, LEPR, GCKR, NLRP3, IL1F10, PPP1R3B, ASCL1, HNF4A and SALL1 exert an influence on elevated CRP serum levels in non-rheumatic Caucasians. Consequently, we evaluated the potential role of these genes in the development of CV events and subclinical atherosclerosis in RA patients. Three tag CRP polymorphisms and HNF1A, LEPR, GCKR, NLRP3, IL1F10, PPP1R3B, ASCL1, HNF4A and SALL1 were genotyped in 2,313 Spanish patients by TaqMan. Subclinical atherosclerosis was determined in 1,298 of them by carotid ultrasonography (by assessment of carotid intima-media thickness-cIMT-and presence/absence of carotid plaques). CRP serum levels at diagnosis and at the time of carotid ultrasonography were measured in 1,662 and 1,193 patients, respectively, by immunoturbidimetry. Interestingly, a relationship between CRP and CRP serum levels at diagnosis and at the time of the carotid ultrasonography was disclosed. However, no statistically significant differences were found when CRP, HNF1A, LEPR, GCKR, NLRP3, IL1F10, PPP1R3B, ASCL1, HNF4A and SALL1 were evaluated according to the presence/absence of CV events, carotid plaques and cIMT after adjustment. Our results do not confirm an association between these genes and CV disease in RA
Iron Behaving Badly: Inappropriate Iron Chelation as a Major Contributor to the Aetiology of Vascular and Other Progressive Inflammatory and Degenerative Diseases
- Author
- A Abbott
- A Abou-Raya
- A Aljandali
- A Ambesi-Impiombato
- A Aydin
- A Balcerczyk
- A Besarab
- A Brzezinski
- A Campbell
- A Carrillo-Vico
- A Cavalli
- A Chattopadhyay
- A Chattopadhyay
- A Cherubini
- A Dey
- A Doms
- A Donovan
- A Donovan
- A Dávalos
- A Gaeta
- A Garny
- A Gnjec
- A Gomes
- A Gopalakrishnan
- A Hald
- A Henney
- A Hirayama
- A Hoffmann
- A Huber
- A Hugman
- A Iannetti
- A Ide-Ektessabi
- A Jacobs
- A Jeyabalan
- A Kahvejian
- A Karrar
- A Kasztler
- A Kibel
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- A Murakami
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- A Persson
- A Pietrangelo
- A Piga
- A Pirat
- A Post
- A Pradhan
- A Protti
- A Rattner
- A Ravati
- A Rehman
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- A Roetto
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- A Saltelli
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- A Sassano
- A Scalbert
- A Scalbert
- A Scalbert
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- A Smahi
- A Sola
- A Stintzi
- A Szewczyk
- A Taguchi
- A Tedgui
- A Terman
- A Terman
- A Terman
- A Undas
- A Virdis
- A Wagner
- A Wagner
- A Wagner
- A Wagner
- A Wojas-Pelc
- A Wong
- A Xu
- A Yoritaka
- A Yoshimura
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- Ad hoc committee on systemic lupus erythematosus response criteria for fatigue
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- TM Hagen
- TM Hansen
- TM Rooyakkers
- TO Scholl
- TO Scholl
- TP Dalton
- TP Obrenovitch
- TP Tuomainen
- TP Tuomainen
- TR Coleman
- TR Golden
- TR Quinlan
- TS Lee
- TT Lao
- TT Lao
- TT Lao
- TW Jung
- TW Rademacher
- TY Tsai
- U Alon
- U Bartels
- U Buescher
- U Cha'on
- U Eckstein-Ludwig
- U Heinz
- U Laufs
- U Schönbeck
- UN Das
- V Adam-Vizi
- V Adam-Vizi
- V Atanasiu
- V Braun
- V Braun
- V Calabrese
- V Campuzano
- V Campuzano
- V Curcin
- V Emilsson
- V Fellman
- V Gabutti
- V Gordeuk
- V Hower
- V Hvidberg
- V Irazusta
- V Irazusta
- V Kaushal
- V Lahera
- V Prinz
- V Shulaev
- V Siffrin
- V Srinivasan
- VA Kostyuk
- VB O'Donnell
- VD Longo
- VE O'Collins
- VE Steele
- VH Haase
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- VL Kinnula
- VM Campese
- VM Victor
- VM Victor
- VN Anisimov
- VR Gordeuk
- VR Gordeuk
- VR Taqueti
- W Aerbajinai
- W Breuer
- W Craelius
- W Davis Jr
- W Dichtl
- W Dröge
- W Dröge
- W Hu
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- WR Markesbery
- WS Jeong
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- X Chen
- X He
- X Huang
- X Huang
- X Huang
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- X Zhang
- X Zhu
- X Zhu
- XD Huang
- XJ Li
- XM Yuan
- XM Yuan
- XP Huang
- Y Almog
- Y Avramovich-Tirosh
- Y Bao
- Y Bdolah
- Y Chen
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- Y Deugnier
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- Y Wang
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- Y Wu
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- Y Zhang
- Y Zhang
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- Y Zheng
- YE Henrotin
- YF Chu
- YH Lee
- YH Suh
- YH Wei
- YJ Surh
- YJ Surh
- YJ Surh
- YK Wu
- YM Kim
- YS Keum
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- YZ Fang
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- Z Cai
- Z Cheng
- Z Pei
- Z Serdar
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- Z Tong
- Z Yu
- Z Zhang
- ZD Liu
- ZD Liu
- ZE Karanjawala
- ZE Suntres
- ZI Cabantchik
- ZZ Chong
- ZZ Chong
- Publication venue
- Publication date
- 09/08/2008
- Field of study
The production of peroxide and superoxide is an inevitable consequence of
aerobic metabolism, and while these particular "reactive oxygen species" (ROSs)
can exhibit a number of biological effects, they are not of themselves
excessively reactive and thus they are not especially damaging at physiological
concentrations. However, their reactions with poorly liganded iron species can
lead to the catalytic production of the very reactive and dangerous hydroxyl
radical, which is exceptionally damaging, and a major cause of chronic
inflammation. We review the considerable and wide-ranging evidence for the
involvement of this combination of (su)peroxide and poorly liganded iron in a
large number of physiological and indeed pathological processes and
inflammatory disorders, especially those involving the progressive degradation
of cellular and organismal performance. These diseases share a great many
similarities and thus might be considered to have a common cause (i.e.
iron-catalysed free radical and especially hydroxyl radical generation). The
studies reviewed include those focused on a series of cardiovascular, metabolic
and neurological diseases, where iron can be found at the sites of plaques and
lesions, as well as studies showing the significance of iron to aging and
longevity. The effective chelation of iron by natural or synthetic ligands is
thus of major physiological (and potentially therapeutic) importance. As
systems properties, we need to recognise that physiological observables have
multiple molecular causes, and studying them in isolation leads to inconsistent
patterns of apparent causality when it is the simultaneous combination of
multiple factors that is responsible. This explains, for instance, the
decidedly mixed effects of antioxidants that have been observed, etc...Comment: 159 pages, including 9 Figs and 2184 reference
Biorefinery cascade processing for creating added value on tomato industrial by-products from Tunisia
- Author
- A Demirbas
- A Kazan
- A Sarkar
- A Scoma
- A Zuorro
- AJ Mattam
- Ayachi Zammel
- C Longo
- C Zhang
- Carsten Zetzl
- D Jiang
- E Cepeda
- E Schettini
- E Vági
- EH Papaioannou
- FJ Eller
- G Brunner
- G Rossini
- G Toscano
- HY Li
- I Campo Del
- I Navarro-González
- IF Strati
- IP Wood
- Irina Smirnova
- J Ishii
- J López-Cervantes
- JA Egydio
- JA Gearhart
- JM Prado
- JM Valle Del
- K Gairola
- K Gairola
- L Müller
- Lisa Marie Schmidt
- M Michelin
- M Mohan
- ME Persia
- MK Roh
- Mouna Kehili
- MS Celiktas
- NL Dickinson
- Noureddine Allouche
- P García Herrera
- P Reddy
- R Parthasarathi
- R Vardanega
- S Imman
- S Machmudah
- Sami Sayadi
- T Baysal
- T Sheng
- W Jiang
- W Reynolds
- WHO/FAO/UNU
- Wienke Reynolds
- X Lu
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Anti-citrullinated protein antibodies contribute to platelet activation in rheumatoid arthritis
- Author
- A Falus
- AC Matzdorff
- AH Mil van der Helm-van
- AM El-Barbary
- AT Nurden
- AY Gasparyan
- AY Gasparyan
- BT Wipke
- D Projahn
- DG Scott
- DL Scott
- E Boilard
- E Boilard
- E.W. Nivine Levarht
- EA Knijff-Dutmer
- F Wang
- FA Gaalen van
- FC Arnett
- FJ López-Longo
- G Cambridge
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- G Lippi
- G Tomasson
- GE Pamuk
- H Demirin
- H Kubagawa
- H Maradit-Kremers
- I Ertenli
- I Matsumoto
- J Beinsberger
- JT Giles
- JT Gustafsson
- JW Semple
- K Nishimura
- Kim L.L. Habets
- KL Habets
- Leendert A. Trouw
- M King
- M Schmitt-Sody
- M Schmitt-Sody
- M Vergeer
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- N Cloutier
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- O FitzGerald
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- Suzanne J.A. Korporaal
- T Cheng
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- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Search for triboson W±W±W∓ production in pp collisions at √s = 8 TeV with the ATLAS detector
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- Zhemchugov A
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- Zobernig G
- Zoccoli A
- Zwalinski L
- Åkesson TPA
- Åsman B
- Ženiš T
- Živković L
- Publication venue
- Publication date
- 01/03/2017
- Field of study
This paper reports a search for triboson
W±W±W∓ production in two decay channels
(W±W±W∓ → ±ν±ν∓ν and W±W±W∓ → ±ν±ν j j
with = e, μ) in proton-proton collision data corresponding
to an integrated luminosity of 20.3 fb−1 at a centreof-mass
energy of 8 TeV with the ATLAS detector at the
Large Hadron Collider. Events with exactly three charged
leptons, or two leptons with the same electric charge in association
with two jets, are selected. The total number of events
observed in data is consistent with the Standard Model (SM)
predictions. The observed 95% confidence level upper limit
on the SM W±W±W∓ production cross section is found to
be 730 fb with an expected limit of 560 fb in the absence of
SM W±W±W∓ production. Limits are also set on WWWW
anomalous quartic gauge couplings
Simultaneous identification of various antinuclear antibodies using an automated multiparameter line immunoassay system
- Author
- Alarcón-Segovia D
- Beer RG
- Bohan A
- Boire G
- Bozic B
- Clark GM
- De Rooij DJ
- Elkon KB
- Frank MB
- Frank MB
- González CM
- Gordon T
- Hines JJ
- Kallenberg CGM
- Kurata N
- López-Longo FJ
- López-Longo FJ
- López-Longo FJ
- López-Longo FJ
- López-Longo FJ
- López-Longo FJ
- López-Longo FJ
- López-Longo FJ.
- Maddison PJ
- Masi AT
- McCauliffe DP
- Meheus L
- Pettersson I
- Provost TT
- Rodríguez-Mahou M
- Rothfield N
- Rutjes SA
- St-Clair EW
- Tan EM
- Tan EM
- Ter Borg EJ
- Van Dam AP
- Van Venrooij WJ
- Whittingham S.
- Williams G
- Zimmermann C
- Publication venue
- 'SAGE Publications'
- Publication date
- Field of study
Systemic lupus erythematosus: clinical expression and anti-Ro/SS—A response in patients with and without lesions of subacute cutaneous lupus erythematosus
- Author
- Publication venue
- 'SAGE Publications'
- Publication date
- Field of study
Detecting Anti-SSA and Anti-SSB Antibodies in Routine Analysis: A Comparison between Double Immunodiffusion and Immunoblotting
- Publication venue
- 'SAGE Publications'
- Publication date
- Field of study
Differential immunoglobulin class-mediated responses to components of the U1 small nuclear ribonucleoprotein particle in systemic lupus erythematosus and mixed connective tissue disease
- Author
- Publication venue
- 'SAGE Publications'
- Publication date
- 01/11/2013
- Field of study
OBJECTIVE: To determine whether patients with Systemic Lupus Erythematosus (SLE) and Mixed Connective Tissue Disease (MCTD) possess differential IgM-and IgG-specific reactivity against peptides from the U1 small nuclear ribonucleoprotein particle (U1 snRNP). METHODS: The IgM- and IgG-mediated responses against 15 peptides from subunits of the U1 snRNP were assessed by indirect ELISAs in sera from patients with SLE and MCTD and healthy individuals (n = 81, 41 and 31, respectively). Additionally, 42 laboratory tests and 40 clinical symptoms were evaluated to uncover potential differences. Binomial logistic regression analyses (BLR) were performed to construct models to support the independent nature of SLE and MCTD. Receiver Operating Characteristic (ROC) curves corroborated the classification power of the models. RESULTS: We analyzed IgM and IgG anti-U1 snRNP titers to classify SLE and MCTD patients. IgG anti-U1 snRNP reactivity segregates SLE and MCTD from non-disease controls with an accuracy of 94.1% while IgM-specific anti-U1 snRNP responses distinguish SLE from MCTD patients with an accuracy of 71.3%. Comparison of the IgG and IgM anti-U1 snRNP approach with clinical tests used for diagnosing SLE and MCTD revealed that our method is the best classification tool of those analyzed (p ≤ 0.0001). CONCLUSIONS: Our IgM anti-U1 snRNP system along with lab tests and symptoms provide additional molecular and clinical evidence to support the hypothesis that SLE and MCTD may be distinct syndromes